Cardiovascular - Part #2 Flashcards
(80 cards)
Know the types of CAD
Hint: Use flowchart
- Chronic Stable Angina
- Acute Coronary Syndrome
-> Unstable angina
-> NSTEMI
-> STEMI
Infarction
Necrosis or cell death that occurs when severe ischemia is prolonged and decreased perfusion causes irreversible damage to tissue
What is the cause of Coronary Artery Disease?
build up of plaque (usually due to atherosclerosis), which causes ischemia
Compare Chronic Stable Angina from Unstable Angina
Chronic Stable Angina
- Fixed, stable plaque that partially blocks (narrows) coronary artery, causing temporary myocardial ischemica because of imbalance between oxygen supply and demand in the myocardium
-> Release of lactic acid irritates nerve fibers and causes pain in the cardiac nerves
Unstable Angina
- Ruptured or unstable plaque that causes a sudden clot or narrowing, causing formation of a platelet plug (clot) and the thrombus -> reduce O2 supply to myocardium
-> If thrombus enlarges and fully occludes artery -> MI
What are nonmodifiable and modifiable risk factors for Coronary Artery Disease (Chronic Stable Angina and Unstable Angina)
Nonmodifiable:
- Age
-> middle aged men, but once 65+ incidence becomes equal - Gender
-> Women with CAD may have atypical symptoms - Ethnicity (African American)
- Genetics
Modifiable:
- Atherosclerosis
-> Elevated lipids - Smoking (Tobacco Use)
-> Substance Abuse - Sedentary Lifestyle
-> HTN
-> Obesity
-> Diabetes - Chronic, uncontrolled stress
- High Homocysteine level
Risk factors for unstable angina overlap with risk factors for CAD. What are 4 specific risk factors that are specific to unstable angina?
- Thrombus formation
- Plaque rupture
- Worsening of previously stable CAD
- Severe or sudden increase in oxygen demand (ex: emotional or physical stress)
What are the symptoms of Chronic Stable Angina?
How does it compare between men and women?
Intermittent SXS: - Same pattern of onset, duration, and intensity of symptoms
-
Referred cardiac pain
-> Occurs with exertion/activitiy in familiar pattern
-> Goes away with rest (5 - 15 minutes)
-> Pressures/Aches
-> Suffocating sensation - Pulse deficit
- Indigestion (burning)
- Cool, clammy, pale skin
Men:
- L sided chest pain, that radiates down to left arm
Female:
- Epigastric pain
- Neck/jaw/s pain
- Heartburn
What are the symptoms of Unstable Angina?
-
Chest pain at rest (or with exertion), not relieved by nitro
-> Pressure/discomfort 15+ minutes
-> Increase frequency of pain - Anxious/Restless
- Tachycardia
- SOB
What are some assessments that should be done for Chronic Stable Angina
- Check JVD
- Listen to heart and lung sounds
-> Check peripheral pulses
-> Tachycardia, Bradycardia
What Imaging/Radiology should be taken for a a patient with Chronic Stable Angina or Unstable Angina
-
Troponin Imaging assessment
-> No change in troponin values = unstable angina
-> if elevated, NOT chronic stable angina = MI - CTCA - CT Coronary Angiography (assess coronary arter narrowing)
- CXR (look at heart size)
- Thallium Scans (ID areas of ischemia during stress/rest)
- CMR, echocardiography
What tests should be run if patient suspected to have Chronic Stable Angina or Unstable Angina
- 12-lead EKG (dysrhythmias)
- Exercise tolerance (stress test)
What surgery should be run for patient with Chronic Stable Angina or Unstable Angina
-
Cardiac catherization/Angiography
-> Visualize blockages (diagnostic)
-> Open blockages (interventional)
-» PCI
-» Balloon angioplasty
-» Stent
What interventions should be done for a patient with Chronic Stable Angina if they have difficulty breathing?
- High/Semi-fowlers (if difficulty breathing)
- Supplemental O2 (if difficulty breathing)
What medications (general) should be given to a patient with Chronic Stable Angina?
- Administer nitrate (nitroglycerin) (x3 max every 5 minutes) followed by opioid analgesic
-> Opiod: Morphine and Aspirin
What specific medicaitons should be given to a patient with Chronic Stable Angina.
What other suggestoins may you suggest this patient to change?
-
Short-acting nitrates (nitroglycerin; chest pain)
-> Sublingual tablets/spray -
Long acting nitrates (isosorbide, nitro topical; chest pain)
-> 12 hours on, 12 hours off - Antihypertensives (ACEI, CCB, B blocker)
- Antiplatlets
- Lipid lowernig agent
Recommend:
- Lifestyle modifications
A patient has Chronic Stable Angina. They took Nitrostat (short-acting) for chest pain at 0800. By 0805, the patient still feels chest pain. What is the first action the patient should take?
Call 911 for pain that doesnt go away with rest or nitro after the FIRST dose. After calling 911, they may go ahead and repeat a second dose
What medications are given to someone with Unstable Angina.
MONA
- Morphine
- Oxygen
- Nitroglycerin
- Aspirin
With CAD and older adults, _________ pain may not be as obvious or reported. They may have symtoms of unexplained _________, _________, and ________ symptoms
chest
unexplained dyspnea
confusion
GI symptoms
T/F: Dysrhythmia may be a normal age-related change rather than a complication of MI
True
For patients with CAD, nurses should plan to slowly and steadily increase in activity. What should the nurse expect to do when having these patients participate in exercise?
- Gradually increase activity
- Plan longer warm-up and cool-down periods when participating in exercise (bc pulse rates may not return to baseline for 30 minutes)
Review Heart Failure chart in master notes
review if you want, but i would focus on knowing L vs R heart failure
What are the categories of heart failure in regards to severity.
-
Acute Heart failure
-> Caused by another condition (ex: severe anemia) - Chronic Heart failure (Congestive Heart Failure)
What are common causes of systolic left-sided heart failure?
Systolic left-sided heart failure is due to the heart’s inability to pump effectively because of…
- Impaired contraction (MI)
- Increased afterload (HTN)
- Cardiomyopathy
- Mechanical abnormalities (valvular heart disease)
What structural changes occur in the left ventricle during systolic heart failure?
- The left ventricle loses the ability to generate enough ejection pressure to pump blood out through the aorta.
-> it becomes dilated and hypertrophied