cardiovascular system Flashcards

(12 cards)

1
Q

What are the fundamental components of the cardiovascular system?

A

Heart, blood vessels, and blood. It includes the systemic and pulmonary circulations.

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2
Q

What regulates blood flow through the heart?

A

Mechanical processes such as the opening and closing of valves and chamber contractions during the cardiac cycle.

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3
Q

How are cardiac action potentials initiated and propagated?

A

Initiated at the SA node and propagated through the atria, AV node, Bundle of His, bundle branches, and Purkinje fibers.

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4
Q

What ionic fluxes occur during the cardiac action potential?

A

Phase 0: Na⁺ influx; Phase 1: K⁺ efflux; Phase 2: Ca²⁺ influx and K⁺ efflux; Phase 3: K⁺ efflux; Phase 4: Resting membrane potential maintained.

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5
Q

How does the ECG relate to the cardiac cycle?

A

P wave = atrial depolarization, QRS = ventricular depolarization, T wave = ventricular repolarization.

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6
Q

How do structural features of arteries enable their function?

A

Thick walls with elastic tissue and smooth muscle allow arteries to withstand and regulate high pressure.

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7
Q

What is the function of veins?

A

Return blood to the heart; thin walls and valves prevent backflow and act as blood reservoirs.

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8
Q

What is the role of capillaries?

A

Enable exchange of gases, nutrients, and waste between blood and tissues through thin walls and large surface area.

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9
Q

How is arterial blood pressure measured?

A

Using a sphygmomanometer and stethoscope to detect Korotkoff sounds during cuff deflation.

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10
Q

How does the baroreceptor reflex control blood pressure?

A

By sensing pressure changes and adjusting heart rate, stroke volume, and vessel diameter via the autonomic nervous system.

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11
Q

How do vasoconstriction and vasodilation control peripheral resistance?

A

Vasoconstriction increases resistance and blood pressure; vasodilation decreases them.

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12
Q

What is the role of the RAAS in blood pressure regulation?

A

Renin from the kidney initiates a cascade converting angiotensinogen to angiotensin II, leading to vasoconstriction and aldosterone-mediated volume retention.

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