Caries Flashcards

1
Q

Why is caries management important in modern dentistry? (4 reasons)

A
  1. Second most prevalent non-communicable disease in adults worldwide and it affects quality of life
  2. Known links between caries and systemic health - caries is preventable
  3. Aesthetic restorations are part of the rehab of med/high caries-risk pts motivation, value, responsibility)
  4. Manufacturers producing high quality direct, adhesive aesthetic materials/cements
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2
Q

What is ‘MI’ dentistry?

A

Minimum Intervention Oral Care

Holistic and patient -focussed and involves all members of oral health care team. Aims to achieve long-term oral health.

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3
Q

Name the 4 domains of oral care plans

A
  1. Identify
  2. Prevent and control
  3. MI restore
  4. Recall - pt focused
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4
Q

What is involved in the ‘identify’ stage of the oral care plan?

A

Verbal history, oral exam, caries lesion detection, radiographs, aetiological factors for susceptibility

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5
Q

What is the (long) definition of dental caries?

A

Reversible disease process of hard tissues.

Instigated by action of bacteria on fermentable carbs in plaque biofilm at tooth surfaces.

This leads to formation of carious lesion: acid demineralization and ultimate proteolytic destruction of the organic component of dental tissues

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6
Q

Outline what dental caries is

A

Progressive, non communicable disease initiated at the surface in the biofilm that is reversible up to a point

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7
Q

What is a carious lesion?

A

It is where the tooth substance has softened and has been destroyed

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8
Q

What is the caries PROCESS?

A

The histopathological metabolic interactions occurring in the plaque biofilm causing disease

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9
Q

What is a carious LESION?

A

The signs of the disease on dental hard tissues i.e. early lesions/discolouration/opacities/cavities etc

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10
Q

Which bacteria is primarily associated with the caries process?

A

Strep Mutans

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11
Q

What 4 factors need to occur in order for caries to develop?

A

Bacteria (relevant bacteria)

Susceptible tooth surface

Carbohydrates

Time

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12
Q

At what pH is the enamel particularly susceptible to acid attack (critical pH)?

A

5.5

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13
Q

What’s the critical pH of dentine?

A

6.2

it’s more susceptible to acid attack

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14
Q

What 5 factors are required for good visual detection?

A

Sharp eyes + magnification

Good light

Clean, dry tooth surface

Dental explorer (blunt) - e.g. perio probe

Time

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15
Q

What steps are involved in diagnosing caries? (4)

A

Caries history/susceptibility assessment

Signs- detection

Symptoms i.e. pain history

Special investigations: sensibility tests (temp, electrical, percussion etc), radiographs

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16
Q

Name the factors that can increase susceptibility of caries

A

Medical (certain drugs, sucrose-based meds)

Social: stress, lifestyle change

Dietary: prolonged breast feeding, grazing

Host resistance: previous caries experience, lesions on certain tooth surface, soft,light coloured lesions

Salivary: Low secretion and buffering

Microbiology: high numbers S Mutans and Lactobacilli

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17
Q

What does it mean if a pt is low risk?

A

Inactive/controlled:

0-1 active lesion/no history of recent restorations

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18
Q

What does it mean if a pt is medium risk?

A

Active/modifiable:

> 1 active lesions
2 new, progressive or filled in last 2 years

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19
Q

What does it mean if a pt is high risk?

A

Active/unmodifiable or unidentifiable risk factors:

> 1 active lesions
2 new, progressive or filled lesions in last 2 years

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20
Q

How would you control caries risk for low risk patients?

A

OH, fluoride, standard home care

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21
Q

How would you control caries risk for medium risk patients?

A

OH, supplementary fluoride mouthwash or gels.

Dietary modifications

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22
Q

How would you control caries risk for high risk patients?

A

Control at individual pt level

Same as medium risk plus salivary flow stimulation

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23
Q

What causes the enamel to dimineralise and form a lesion?

A

The biofilm releases acids that lower the pH to 5.5 or below. This causes the dissociation of minerals/ions in the enamel (but the surface can still be intact and demineralised) = underlying enamel is porous

24
Q

How are the enamel crystals arranged?

A

Arranged into prisms

25
Which particular part of the enamel suffers from acid attack?
The enamel prisms and boundaries
26
How would you describe an early white spot lesion on enamel?
Frosty white appearance, chalky and softened, increased porosity
27
How would you describe an early white spot lesion on enamel?
Frosty white appearance, chalky and softened, increased porosity. The surface becomes roughened and cavitated
28
What is a brown spot lesion
It's where a white spot lesion has arrested. It has been stained from the diet
29
Which part of dentine plays a critical part in matrix and mineralisation production?
Non-collagenous proteins | 10% of dentine
30
What 3 components make up dentine?
Mineral, collagenous matrix, tubules
31
What are the biochemical affects of caries attack on dentine?
Demineralisation due to acid attack Bacterial penetration via tubules and branches Proteolysis- collagen breakdown via host and bacterial enzymes (primarily host enzymes)
32
Why does dentine become various shades of brown when attacked?
Maillard reaction: between proteins and carbs in acidic environment
33
What signs would suggest that the dentine-pulp complex has triggered a defence reaction?
Translucent dentine Reparative (tertiary) dentine
34
Why does dentine become soft when attacked by acid or caries?
Due to demineralisation
35
What is translucent dentine made up of?
Plate-like crystals of whitlockite
36
What structural factor plays a part in the hardness of the tissue?
The way the crystals are arranged in each layer - i.e the crystalline structure
37
Name 3 ways in which dentine-pulp complex defends itself to attack?
Translucent dentine, reparative dentine, inflammatory/serum proteins in pulpal fluid (these are then pushed up into tubules)
38
Outline stage 0 of the modified ICDAS scale
0 - no or slight change in enamel translucency after prolonged drying >5secs.No enamel demineralisation or narrow surface zone of pacity
39
Outline stage 1 of the modified ICDAS scale
Opacity or discolouration hardly visible on a wet surface, but distinctly visible after air drying. Enamel demineraliseation limited to outer 50% of the enamel layer
40
Outline stage 2 of the modified ICDAS scale
Opacity or discolouration distinctly visible without air drying. No clinical cavitation detectable. Demineralisation involving between 50% of the enamel and the outer third of dentine
41
Outline stage 3 of the modified ICDAS scale
Localised enamel breakdown in opaque or discoloured enamel +/- greyish discolouration from underlying dentine. Demineralisation involving the middle third of dentine.
42
Outline stage 4 of the modified ICDAS scale
Cavitation in opaque or discoloured enamel exposing the underlying dentine. Demineralisation involving the inner third of dentine
43
Why can caries affected dentine be remineralized?
Because the collagen in the affected dentine has been damaged NOT denatured
44
What is the difference between a D1 and D2 carious lesion?
D2 is closer to the dentine layer
45
What is the histology of D1 & D2 carious lesions?
Early subsurface demineralisation Early porosity ? Bacterial penetration ? tertiary dentine
46
What are the clinical signs of D1 & D2 carious lesions?
White spot lesion, frosty Chalky, roughened surface Arrested brown spot lesion
47
What are the symptoms of D1 & D2 carious lesions?
Minimal symptoms Slight sensitivity to H/C/S if at EDJ
48
What would the treatment of D1 & D2 carious lesions be?
Monitor OHI, fluoride, diet ? Fissure seal/PRR
49
What is the histology of D3 carious lesions?
Enamel demineralisation Increased porosity Bacterial penetration organic/inorganic dentine/tubular destruction Translucent dentine Tertiary/reparative dentine
50
What are the signs of D3 carious lesions?
Cavitated (open)/non-cavitated (closed) Discolouration/opacities
51
What are the symptoms of D3 carious lesions?
? acute, reversible pulpitis
52
What is the treatment for D3 carious lesions?
Monitor/OHI/Fluoride/diet Minimal cavity prep Sealed restoration
53
What is the histology of D4 carious lesions?
Gross demineralisation Gross bacterial penetration Tubular destruction with pulp exposure
54
What are the signs of D4 carious lesions?
Cavitation Gross discolouration Visible necrotic pulp ?
55
What are the symptoms of D4 carious lesions?
Chronic, irreversible pulpitis Loss of function
56
What is the treatment for D4 carious lesions?
Pulp capping Sealed, layered, complex restoration Pulp extirpation and/or RCT