CCM wiser material Flashcards

(81 cards)

1
Q

Initial evaluation and management of unstable patient

A

1) introduce yourself to the patient, if they respond, ABC intact. 2) If pt unresponsive, check for pulse

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2
Q

what if the patient is unresponsive

A

Check for pulses, if no pulse: Call for help, start CPR, or think pulseless rhythms

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3
Q

What if the patient is unresponsive but they do have a pulse

A

Check BP,

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4
Q

What if the patient is unresponsive but they do have a pulse and its low

A

begin resuscitation

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5
Q

What if the patient is unresponsive but they do have a pulse and its normal

A

Narcan (0.4 mg diluted in 9 ccs; start with 1 mg, 2 mg, 4 mg) given 1 amp D 50 or glucagon 1 mg

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6
Q

What if the patient is unresponsive but they do have a pulse and its normal, and you given narcan and glucose,

A

Secure airway, evaluate for other causes

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7
Q

Unresponsive patient

A

Able to protect airway (secretions)?, breathing (spontaneously, or increased work of breathing), circulation (pulse)

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8
Q

Increased work of breathing

A

Tachypnea, tachycardia, diaphoresis, accessory muscle use

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9
Q

RR decreased, ddx

A

meds (benzos or opiates), brainstem (mid structural lesion or metabolic encephalopathy), neuromuscular, metabolic alkalosis (vomiting, meds, contractile alkalosis)

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10
Q

RR increased? what else are you looking for (indications for BVM)

A

1) increased WOB 2) refractory hypoxia, 3) apnea, 4) inability to protect airway 5) hypoxia with bradycardia

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11
Q

Pt fits indications for BVM, but they are still hypoxic with no chest rise

A

place oral/nasal airway

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12
Q

Pt starting on BVM, then you place ora/nasal airway

A

Direct laryngoscope and intubation (begin prep)

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13
Q

Pt w/ increased RR but no indications for BVM

A

if the tidal volume is low (rapid, shallow breathing), beging BVM, if no, and the patient is hypoxic but responding to supplemental O2, go through ddx

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14
Q

ddx for hypoxic patient that does respond to supplemental O2

A

1) Low FiO2, 2) low barometric pressure 3) shunt (> 40%) 4) deat space 5) diffusion or O2 carrying abn 6) hypoventilation

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15
Q

Minute ventilation equation

A

RR x Tidal volume, normal 8L

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16
Q

PCO2

A

production / RR x (tidal volume - dead space)

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17
Q

What is a shunt and and % is needed for refractory hypoxia

A

perfusion of alveoli that are not ventilated; > 40% leading to refractory hypoxia; does not improve with increasing FiO2, but responds to PEEP

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18
Q

Etiology of shunt

A

Blood (hemorrhage), water (pulmonary edema), pus (PNA), atelectasis, ARDS

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19
Q

What is deadspace

A

Ventilation of alveoli that are not perfused; responds to FIO2, doesnt respond to PEEP

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20
Q

etiology of deadspace

A

PE;

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21
Q

Preparation for rapid sequence induction and intubation

A

1) evaluate airway 2) IV access 3) pre-oxygenate 4) contraindications for Succs 5) last time you ate?

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22
Q

Evaluating airway

A

mallampatti, thyomental distance, thyrohyoid distance

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23
Q

Contraindications to succynylchonile

A

kidney problems, unable to move extremities? myalgias? prior problems with anesthesia? ( burns, eye trauma, denervation, hyperkalemia)

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24
Q

After initial preparation for intubation, what equipment do you need?

A

1) laryngoscope (mac, miller, glydoscope) 2) endotracheal tube (7.5 fem, 8 male) 3) stylet 4) syringe) 5)CO2 detector 6) cardiac, O2, BP monitor,

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25
Meds needed for rapid sequence intubation
sedation: etomidate (0.3 mg/kg); ketamine 3 mg/kg; versed, propofol Paralytics: Succinylcholine, roc( 1mg per kg) Push dose: neo (20mg/D5W 250ml) give 80 mcg/cc; IVF
26
Initial ventilator settings
Mode (assisst control), tidal volume ( 6cc/kg IBW), RR (target MV/tidal volume), PEEP (5 mmHg, higher for shunt physiology), FIO2 (start 100%, decrease to \<60)
27
MV w PCO2
MV PaCO2 8 40 14 30 20 20
28
Hypotension following intubation
IVF if systolic is low Phenylephedrine if diastolic (20 mg into 250 cc NS) give 2 cc at a time
29
VT/VF arrest witnessed
shock first, charge to 200
30
VT/VF arrest unwitnessed
2 min
31
Arrest at 0 min
Help CPR IV access BVM Get defibrillator
32
Arrest at 1:30 min
Continue CPR Prep meds (epi 1 mg 1:10.000; 300 mg amio) Place pads, begin to charge for defib
33
Arrest at 2 min
Check pulse/rhythm Give meds - (epi 1 mg 1:10.000; 300 mg amio) BVM Shock if can
34
Arrest at 3 min
Continue CPR Prep meds (epi 1 mg 1:10.000; 150 mg amio) meds for intubation Place pads, begin to charge for defib
35
Arrest at 4 min
Check pulse/rhythm epi 1 mg 1:10.000 150 mg amio intubate Shock if can
36
Arrest at 5 min
Continue CPR Prep meds (epi 1 mg 1:10.000; BVM q 5-6 hr Shock
37
Persistent VF/VT
Repeat epinephrine/shock q 5 min Repeat amio/shock 150 mg IV x1 Things to consider: - Check ABG/lytes - Give Na Bicarb 1 amp IV - Give Mg sulfate 2 g IV push
38
PEA ddx
Severe hypotension/obstructive shock 1) Massive PE (tPA, takes 24 hr) 2) Hypovolemia -\> Fluids5 3) Temponade -\> surgical 4) Tension pneumo 5) Hypoxia -\> BVM 6) High K, 7) MI/PE
39
What to do for Hyperkalemia
Give Ca IV, NA bicarb, Insulin IV, D50 IV
40
Shock in terns of equation
CO = HR x SV
41
Shock w/ decreased HR
Asystole vs bradycardia
42
Shock w/ asystole
PEA
43
Shock w/ bradycardia and hypoxic
Emergent BVM - treat hypoxia reverse meds, pacing,
44
Etiology of bradycardia
1) severe hypoxia (BVM), 2) well conditioned ppl, 3) MI RCA supplying sinus node, 4) Meds (CCB, BB, digoxin (digibind), vasovagal) 5) hypothermia
45
How to reverse bradycardia 2/2 BB
epi, glucagon, high dose insulin
46
How to reverse bradycardia 2/2 CCB
Ca, epi, high dose insulin, lipid emulsion
47
Tachycardia wide complex, NBS?
+ p wave, no p wave
48
Tachycardia wide complex, + p wave?
Treat underlying cause: hyperkalemia, L or R BBB, TCA OD, WPW, pacemaker
49
Tachycardia wide complex, - p wave?
stable vs unstable
50
Tachycardia wide complex, - p wave, stable- dx and tx?
V tach, AVNRT w/ abberancy -\> Rate and rhythm (amiodarone) cardioversion.
51
Tachycardia wide complex, - p wave, unstable?
unstable V tach: AMS, CP, hypotensive -\> electric cardioversion
52
Tachycardia narrow complex, unstable?
AMS, CP, hypotensive -\> electric cardioversion
53
Tachycardia narrow complex, stable, irregular, ddx?
MAT Sinus tach w/ PACs or PVCs a fib
54
Tachycardia narrow complex, stable, irregular, new onset?
Rate control/cardioversion (in \< 72 hr) and correct trigger
55
Tachycardia narrow complex, stable, irregular, old onset?
Rate control Anticoagulate and fix underlying trigger
56
Causes of sinus tachycardia (triggers)
Hypoxia Increased WOB Shock Hypoglycemia Anemia Increased DO2 Meds and electrolytes Pain/Anxiety
57
Tachycardia narrow complex thats regular, categories
divided based on P waves
58
Tachycardia narrow complex thats regular, + P waves
A flutter Sinus tachy
59
Tachycardia narrow complex thats regular, no P waves
AVNRT * Reentry pathway * Signal comes down the AV node * Fast channel depolarizes * PAC occurs and goes down slow channel as fast channel is refractory from earlier beat * When PCA gets to the end of slow channel, fast channel is no longer refractory from earlier beat, re-entry rhythm starts.
60
Tachycardia narrow complex thats regular, ? P waves
Adenosine, avoid in pt/s wiht bronchospasm (asthma, COPD)
61
Rate control
CCB, BB
62
Rhythm control
Amiodarone BB Digoxin
63
Etiology of sinus tachycardia - decreased oxygen delivery
DO2 = 1.34 x Hgb x CO x SaO2 CO = HR x SV DO2 = 1.34 x Hgb x [HR x SV] x SaO2 Examples: severe anemia, hypoxia, dereased SV from changes in preload, afterload and contractility.
64
Etiology of sinus tachycardia - decreased resistance
BP = CO x Resistance HR will rise to provide compensatory increase in CO Examples: Sepsis, anaphylaxis, medications-vasodilator, adrenal insufficiency
65
Etiology of sinus tachycardia - increased WOB
Increased catecholamines as pt struggles to breath
66
meds
Beta agonists, withdrawal - EtOH, BB
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68
Multifocal Atrial Tachycardia
3 or more distinct P wave morphologies Irregular PR interval Common to chronic lung
69
Determining if SVT is stable vs unstable
SOB CP Hypotension * IF UNSTABLE, CARDIOVERSION (sedate, intubate, cardiovert, set on synchronized cerdioverstion start at 100J)
70
If stable SVT, how to dx
* if P waves visible, tben based on EKG * If no p waves, need to use adenosine toslow rhythm down enough to make dx * Adenosine IV - block AV node for 5 seconds
71
Adenosine IV to dx SVT
* Adenosine IV - block AV node for 5 seconds * need pt to be monitored, be prepared for rare complications of asystole * Give adenosine IV * peripheral: 6 mg repeat at 12 mg * central: 3 mg repeat at 6 mg * AVNRT - converts to NSR * A flutter - converts to flutter waves * Sinus tach - converts to 3rd degree AV block
72
How to do rate control of SVT
* Increase level of care to place where you can manage possible hypotension or arrhythmia * Beta Blocker – IV metoprolol or esmolol drip * Calcium Channel Blockers- diltiazem IV or drip * Side Effect of Rate control could be hypotension * Reverse hypotension of Calcium Channel Blockers with IV calcium * Reverse hypotension with Neosynephrine IV
73
How to do rhythm control of SVT
* Amiodarone IV * Loading Dose 150mg IV over 10 minutes * Drip: 1mg/hour x 8 hours then 0.5mg/hour x 16 hours * Start PO after drip completed
74
Hypotension categories
* low pulse pressure (preserved diastolic) * Cardiogenic (HF, ACS) -\> Ionotropes, pressors * Obx (PE, tamponade, tx) -\> reverse * Hypovolemia (hemorrhage, dehydration) * wide pulse pressure (decreased diastolic) * Distributive (sepsis, AI, anaphylaxis, meds) * Neurogenic (usually with bradycardia) * Sepsis tx, IVF, pressors * Adrenal Insuff: steroids * Anaphylaxis: IVF, epi, H1 and 2, steroids * Neurogenic: ionotropic and pressors *
75
**Narrow Pulse Pressure Hypotension** * Example: 120/80 decreases to 100/70 * Change more in systolic blood pressure * Systolic Blood Pressure related to \_\_\_\_\_
* Stroke Volume and Aortic Compliance *
76
Stroke Volume is dependent on 3 main factors
* Preload (loss of intravascular volume) * Afterload (blood not leaving heart) * Contractility (decreased L ventricular function)
77
Hypovolemia- loss of intravascular volume
* Dry Mucous Membrane, Jugular Veins flat * Non-Hemorrhagic - vomiting, third spacing, diuresis * Hemorrhagic- Trauma, acute GI bleed * **Treatment-** Volume Resuscitation- IV fluid 2L wide open
78
Obstruction- prevents return of fluid to the heart
* JVD, Clear lungs * Massive Pulmonary Embolism – vascular blockage * Cardiac Tamponade- pericardial pressure preventing ventricular filling * Tension pneumothorax- intrathoracic pressure preventing blood return * RV Infarct * Treatment- Volume Resuscitation and relieve underlying condition
79
Afterload- Prevents blood from leaving heart
* Severe valvular stenosis * **Treatment- Decrease afterload**
80
Contractlity- Decreased Left ventricular function
* Myocardial Infarction * Arrhythmia * Cardiomyopathy * Infiltrative Disease * Treatment- Inotropic Agents
81