CDS Oral Surgery Flashcards

(570 cards)

1
Q

Process of acute apical abscess formation

A

Irreversible pulpitis - loss of tooth vitality - apical periodontitis - acute apical abscess

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2
Q

Infection from maxillary molar spreads buccally above the insertion of the buccinator

A

Spreads into buccal space causing buccal swelling

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3
Q

Maxillary molar infection breaks through buccally below the insertion of the buccinator

A

Drains into the mouth creating a draining sinus on the attached mucosa
Quite painless usually but a bad taste and possibly a bubble present

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4
Q

Why is infection from a maxillary molar more likely to spread buccally than palatally?

A

Infection follows the path of least resistance, bone is less dense buccaly

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5
Q

Which tooth in the upper arch is most likely to have infection spread palatally?

A

Lateral incisor - root is quite palatally placed

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6
Q

What happens if infection from maxillary tooth spreads upwards?

A

Into maxillary sinus - can cause sinusitis, rare

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7
Q

Infection from lower tooth spreads lingually and perforates the bone above the insertion of mylohyoid

A

Spreads into the sublingual space, creating a sublingual abscess

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8
Q

Infection from lower tooth spreads lingually and perforates the bone below the insertion of mylohyoid

A

Spreads into the submandibular space

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9
Q

Does sublingual or submandibular infection cause more problems?

A

Submandibular

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10
Q

Infection from lower tooth spreads buccally and perforates the bone above the insertion of buccinator

A

Draining sinus into the mouth

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11
Q

Infection from lower tooth spreads buccally and perforates the bone below the insertion of buccinator

A

Buccal space infection and swelling

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12
Q

Which direction is infection in the posterior lower teeth likely to spread?

A

Lingually - bone is thinner than buccally

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13
Q

Which direction is infection in the lower anterior teeth likely to spread?

A

Labially

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14
Q

What determines whether infection spreads into sublingual or submandibular space from the lower teeth?

A

Which tooth is affected in relation to the mylohyoid line
Premolars more likely to end up in the sublingual space
7 or 8 infections most likely into the submandibular space

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15
Q

Masticatory spaces

A

Pterygomandibular space
Infratemporal space
Deep temporal space
Superficial temporal space
Masseteric space
Infection can easily spread between lots of these spaces as all of them communicate with each other

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16
Q

Result of infection spreading to masticatory spaces on the muscles

A

Severe trismus - the muscles go into spasm

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17
Q

Pterygomandibular space

A

Bound by the mandible, medial and lateral pterygoid muscles

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18
Q

Infratemporal space

A

Infratemporal fossa region

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19
Q

Deep temporal space

A

Deep to temporalis

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20
Q

Superficial temporal space

A

Superficial to temporalis

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21
Q

Masseteric space

A

Between the masseter and the ramus of the mandible

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22
Q

Path of infection spreading to masticatory spaces

A

Sublingual/submandibular spreads backwards into the jaw

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23
Q

Where can infection spread to from masticatory spaces?

A

Lateral pharyngeal space
then
Retropharyngeal space
then
Prevertebral space

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24
Q

Clinical appearance of infection in the lateral pharyngeal space

A

Oral cavity has an area being pushed in around the lateral pharyngeal space, a bulge in the pharynx

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25
Where can infection in the retropharyngeal space spread to?
Upwards - base of the skull Downwards - superior mediastinum
26
Where can infection in the prevertebral space spread to?
Upwards - base of the skull Downwards - inferior mediastinum
27
What can infection spread to the mediastinum cause?
Cardiac tamponade
28
What can infection spreading to the base of the skull lead to?
Abscess on the brain
29
Infection in the cavernous sinus
Infection can spread into the cavernous sinus, resulting in a cavernous sinus thrombosis - rare
30
Path of upper vs lower tooth infection to cavernous sinus
Upper - infraorbital space - valveless veins in this region - cavernous sinus Lower - Lateral pharyngeal space - infratemporal space - pterygoid plexus which communicates with the brain - valveless veins - cavernous sinus
31
Infection from upperr anterior spread
Lip Nasiolabial Lower eyelid
32
Infection from upper lateral incisor spread
Palate Face
33
Upper premolars and molars infection spread
Cheek Infra-temporal region Maxillary antrum (v rare) Palate
34
Chronic draining abscess
Infection drains into mout Blister forms, bursts, bad taste, disappears then comes back
35
Clinical sign of infraorbital infection
Loss of nasiolabial fold
36
Lower anterior teeth infection spread
Mental and submental space Tend to stick there but could spread into the sublingual or submandibular space
37
Lower premolars and molars infection spread
Buccal space Submasseteric space Sublingual space Submandibular space Lateral pharyngeal space
38
Clinical sign of submandibular space infection
Can't feel the border of the mandible
39
Surgical management of large abscesses
Sometimes under local, otherwise it requires hospital admission and GA Establishment of drainage - get rid of pus Incise the skin if necessary - if possible do this intra-orally, may need to be extra-orally Remove source of infection - extirpate pulp or extract - ideally immediately but v difficult to anaesthetise pt when they have severe infection
40
Antibiotic therapy for large abscesses
Depends on lots of factors Not offered if you can remove the cause and obtain proper drainage and no need in pts that are not systemically unwell Consider Toxicity Desirability Medical history
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SIRS
Systemic inflammatory response syndrome Raised temp Raised HR Raised resp rate Raised white cell count
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What does SIRS indicate?
Antibiotic therapy and urgent hospital referral
43
Anatomical consideration when incising submandibular abscess
Consider the marginal mandibular branch of the facial nerve, which runs down the border of the mandible and ends at the corner of the mouth. Always go at least 2 fingers with below the inferior border of the mandible. If the branch is damaged it can stop patients smiling on that side of the face.
44
How to drain abscess once incised
Finger into the hole or Hilton technique
45
Hilton technique
To drain an abscess - use scissors or instrument with two ends, insert instrument in closed position and open in the incision, stretching the tissues.
46
Once you have surgically drained an abscess how is it managed?
Drain is sutured in for a few days and covered with a dressing Dressing is repeatedly replaced until it is clean
47
Ludwig's angina
Bilateral cellulitis of the sublingual and submandibular spaces
48
Ludwig's angina features
Raised tongue Difficulty breathing Difficulty swallowing Drooling Diffuse redness and swelling bilaterally in submandibular region SIRS
49
Why does Ludwig's angina require urgent treatment?
Can compromise the airway
50
Is gram positive or gram negative purple?
Gram positive
51
Cocci vs bacili
Cocci - round Bacili - rod shape
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Streptococcus structure
chains
53
Capnophilic bacteria meaning
Need carbon dioxide to survive and grow
54
What type of microorganism does metronidazole work on?
Only strict anaerobes
55
Types of acquired resistance
Mutation Acquisition of new DNA - transformation, transduction or conjugation
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DNA transformation
Uptake of short fragments of naked DNA by naturally transformable bacteria
57
DNA transduction
Transfer of DNA from one bacterium into another via bacteriophages
58
DNA conjugation
Transfer of DNA material via sexual pilus, required cell to cell contact
59
Altered target site antibiotic resistance
The path of entry for the antibiotic into the bacteria cell has changed shape and the antibiotic can no longer get in the attack the inside of the bacteria
60
Pulp hyperaemia
Increased blood supply to the pulp Pulp can recover from minor trauma at this stage, going back to normal or an acute pulpitis could develop Sharp pain lasting for seconds when stimulated, resolving after stimulus Caries approaching pulp but tooth can still be restored without treating pulp Reversible pulpitis
61
Acute pulpitis
Sudden onset inflammation of the pulp Constant severe pain Reacts to thermal stimuli Referral of pain/ poorly localised pain Little response to analgesics Becoming irreversible This can become a chronic pulpitis which will flare up every now and then (goes between acute and chronic)
62
Chronic pulpitis spread out of the pulp chamber becomes
Acute apical periodontitis
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Acute apical periodontitis can develop into ___ when actual infection is present (not just inflammation)
Acute apical abscess
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Abscess
A collection of dead neutrophils and other cells
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Chronic apical infection
Can go back and forth with acute apical abscess, when the abscess subsides but there is ongoing low grade infection around the apex
66
Apical cyst
Can form from long term chronic apical infection Painless but can increase in size over time or become infected - painful
67
Open pulpitis symptoms
Can be less severe as the exposed pulp releases pressure
68
Acute pulpitis diagnosis
History Visual exam Negative TTP usually as PDL is not yet inflamed Pulp testing is ambiguous Radiographs won't show much except from a big cavity Diagnostic LA - numb the pt next to suspected tooth and see if the pain goes away Removal of restorations if necessary
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What can happen once there is an acute pulpitis?
It can to and fro with chronic pulpitis or develop into an acute apical periodontitis
70
Acute apical periodontitis diagnosis
Easy diagnosis TTP Tooth is non vital (unless traumatic) Radiographs - loss of lamina dura, radiolucent shadow (may indicate an old lesion eg. flare up of apical granuloma), delay in changes of apex of tooth, widening of apical periodontal space and possible resorption of the root
71
Cause of traumatic periodontitis
Parafunction - clenching or grinding
72
Diagnosis of traumatic periodontitis
Clinical exam of the occlusion - functional positioning, posturing TTP Normal vitality Radiographs - may show generalised widening or periodontal space
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Treatment for traumatic periodontitis
Occlusal adjustment Therapy for parafunction
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Causes of acute apical abscess
Acute apical periodontitis can develop into this Pericoronitis - inflammation around a crown, usually PE Periodontal abscess - pulp is fine, abscess develops directly in the periodontium Sialadenitits - infection of the glands, usually one of the major salivary glands, resulting in swelling and redness, dryness and pus
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Acute apical abscess organisms
Polymicrobial Anaerobes play an important part Strep anginosus, prevotella intermedia both common
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Antibiotics effective against strep anginosus examples
Penicillin Erythromycin
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When does antimicrobial resistance occur?
When microorganisms such as bacteria viruses fungi and parasites change in ways that render the medications used to cure the infections they cause ineffective
78
How do antibiotics fight infection?
Inhibit bacterial cell wall synthesis, damage cell membranes, disrupt bacterial metabolism and restrict the ability to multiply
79
Broad spectrum antibiotics
Target several classes including good bacteria, making them unsuitable for self limiting conditions
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Where can antibiotic resistant bacteria occur?
In anyone who uses antibiotics, and can live in that person for up to a year
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Altered target site
Mechanism of antibiotic resistance The way that the antibiotic gets into the bacteria cell has changed shape and the antibiotic can no longer get in to attack the inside of the bacteria
82
Enzymatic inactivation
Mechanism of antibiotic resistance Bacteria produces enzymes which destroy antibiotics or prevent binding to target sites and having effect on the bacteria
83
What is the usual antibiotic resistance mechanism of prevotella and fusobacterium?
Beta-lactamase enzymes
84
Carbapenems
New class of beta lactam antiobiotics developed to counteract ESBLs Almost generate a forcefield around the beta lactam molecule (BUT bacteria have developed a carbapenemase enzyme)
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ESBLs
Extended spectrum beta lactamases Enzyme produced by bacteria which reduce the choice of antibiotics that can work
86
CPE
Carbapenemase producing eneterobacterales - almost untreatable
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Enterobacterales
Group of gram negative bacili found in common infections such as E.coli
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Last therapeutic option to treat complex infections caused by multi drug resistant bacteria
Carbapenems
89
Decreased uptake mechanism
Mechanism of antibiotic resistance Thick gelatinous capsule around bacterial cell wall - very difficult to get antibiotics to penetrate the mucopolysaccharide
90
What is the key to treating any dental infection?
Remove the source - extraction or extirpation of the pulp
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Methods of resistance
Mutation Inactivation Efflux - antimicrobials pumped out of cell
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Two routes of infection from oral cavity to periapical region
Through crown - carious cavity or trauma -> pulp -> apical foramen Via periodontal ligament
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Endogenous infection
Infectious agent is derived from our own flora
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Examples of microbial agents involved in acute dental infection
Strep anginosus (gram +ve cocci) Prevotella intermedia (gram -ve bacili)
95
What clinical specimen is best for investigation of microbials in acute periapical infection?
Aspirated pus (has not been contaminated with saliva flora)
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Examples of microbial agents involved often in periodontal abscess
Anaerobic streptococci Prevotella intermedia
97
Treatment for localised infection
Establish and document a diagnosis Remove the source of infection
98
Examples of microbial agents involved in pericoronitis
Predominantly mixed oral anaerobes - e.g. P intermedia, S.anginosis
99
Treatment for pericoronitis
Local measures such as operculectomy, systemic tx only if systemic symptoms, metronidazole if appropriate
100
Microbial cause of dry socket and tx
Localised alveolar osteitis Mixed oral flora Does NOT require antibiotics, local tx such as curettage of socket, rinse, alvogyl
101
Predisposing factors for osteomyelitis of the jaws
Bisphosphonate therapy Impaired vascularity of bone (radiotherapy, Pagets disease) Foreign bodies (implants) Compound fractures Impaired host defences (diabetes)
102
Treatment of osteomyelitis of the mandible
LA, curettage, IV antibiotics
103
Microbiology involved in osteomyelitis of the mandible
Anaerobic gram -ve bacilli Anaerobic streptococci S anginosus, S aureus
104
Management of salivary gland infection
Drain the pus Flucloxacillin and metronidazole
105
Microbiology of salivary gland infection
S aureus and mixed anaerobes
106
What needs documented when treating an infection?
Diagnosis Antibiotic choice, dose, route and duration Review date Document deviation from guidance
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Ludwig's angina
Bilateral infection of the submandibular space Most commonly caused by anaerobic gram negative bacilli Strep anginosus and anaerobic streptococci, could be staph aureus
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Most common microbial associated with hospitalisation from dental infection
S milerii and mixed anaerobes
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Sepsis
Life threatening organ dysfunction caused by disregulated host response to infection SIRS + suspected/confirmed infection
110
SIRS
Systemic inflammatory response syndrome Temp <36 or 38+ Heart rate >90/min Resp rate >20/min WCC <4000/μL or >12000/μL
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Sepsis six
Give high flow oxygen Take blood cultures Give IV antibiotics Give a fluid challenge Measure lactate Measure urine output
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S I R for choice of antibiotics
Susceptible at standard dose Susceptible at increased dose Resistant even with increased dose
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What is a breakpoint of an antibiotic?
A chosen concentration (mg/L) of an antibiotic which defines whether a species of bacteria is susceptible or resistant to the antibiotic
114
Resistance
A high likelihood of therapeutic failure even when there is increased exposure
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Stewardship
An organisational or healthcare system wide approach to promoting and monitoring judicious use of antimicrobials to preserve their future effectiveness A coherent set of actions which promote using antimicrobials responsibly
116
2 key ways of tackling AMR
Reducing the need for and unintentional exposure to antimicrobials Optimising the use of antimicrobials
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Responsibilities of stewardship
Vaccination Infection prevention and control Public health interventions - e.g sanitation, oral health Antimicrobial prescribers
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WHO global action plan on antimicrobial resistance 5 strategic objectives
Improve awareness and understanding Strengthen the knowledge through surveillance and research Reduce the incidence of infection Optimise the use of antimicrobial medicines Ensure sustainable investment
119
Recommended first line when antibiotics required for acute dento-alveolar infection
Phenoxymethylpenicillin
120
What is penicillin V most effective against?
Oral streptococci Anaerobes Selected gram negative cocci
121
Amoxicillin compared to Pen V
Possesses the same spectrum plus more active against gram negative cocci and members of the family enterobacteriaceae
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Strep anginosus is invariably sensitive to _____
Amoxicillin and Pen V
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what is the problem with using amoxicillin over Pen V?
Broader spectrum of activity so it has a greater impact on selection of resistance in the host micro flora
124
Recommended dose of first line antibiotics for acute dentoalveolar infection
Phenyoxymethylpenicillin 500mg 6 hourly 5 days Important to review after 24-48 hours
125
Examples of unacceptable reasons for dental antibiotic prescribing
Workload pressures Unsure of diagnosis Treatment had to be delayed
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Three examples of oral health conditions that antibiotics DO NOT WORK FOR
Acute pulpitis Gingivitis Sinusitis
127
Alternative causes of acute apical abscess (not usual route of acute pulpitis to apical periodontitis)
Periodontal abscess - pulp is fine, abscess develops directly in the periodontium Pericoronitis - inflammation around a crown, usually partially erupted Sialadenitis - infection of the glands, usually one of the major salivary glands, resulting in swelling and redness, dryness and pus
128
Cervico-facial actinomycosis
Very chronic pus producing infection which can develop after an extraction
129
5 cardinal signs of inflammation
Heat Swelling Pain Redness Loss of function
130
Symptoms of early stage abscess before the infection has broken through bone and into soft tissues
Almost identical to acute apical periodontitis Severe unremitting pain Acute tenderness in function Acute tenderness to percussion No swelling, redness or heat until the abscess spreads out from within the jaw bone
131
Symptoms of acute apical abscess that has perforate through bone
Pain often remits initially due to release in pressure (unless in the palate) Swelling, redness and heat (in soft tissues) become increasingly apparent As swelling increases pain returns There is initial reduction in tenderness to percussion of the tooth as pus escapes into the soft tissues
132
Factors determining the site of spread of dental infection
Position of the tooth in the arch Root length Muscle attachments
133
Potential spaces in proximity to lesion that dental infection could spread into
Submental space Sublingual space Submandibular space Buccal space Infraorbital space Lateral pharyngeal space Palate
134
Need for antibiotics in case of dental infection is determined by
Severity Absence of adequate drainage Patients medical condition Local factors
135
Local factors denoting need for antibiotics
Toxicity (if pt is systemically unwell) Airway compromisation Dysphagia Trismus Lymphadenitis Location - floor of mouth
136
Systemic factors denoting need for antibiotics in case of dental infection
Immunocompromised pt - acquired causes (HIV), drug induces (steroids/cytostatics), blood disorders (leukaemias), poorly controlled diabetes, extremes of age
137
Reversible pulpitis
A level of inflammation in which returning to a normal state is possible if noxious stimuli removed The pulp is inflamed due to caries or a restoration etc, if the cause is removed, the pulp will recover to normal health Usually mild to moderate tooth pain, no pain without stimulus, subsides in <5secs, no mobility, no pain on percussion
138
Irreversible pulpitis
Higher level of inflammation where the dental pulp has been damaged beyond the point of recovery Sharp throbbing sever pain upon stimulation and pain may be spontaneous or occur without stimulation, pain persists after stimulus is removed, >5secs TX - RCT or extraction
139
Periapical granuloma
Chronic apical periodontitis Mass of chronically inflamed granulation tissue at apex of tooth (plasma cells, lymphocytes and few histocytes with fibroblasts and capillaries) NOT a true granuloma because not granulomatous inflammation (it has epithelioid histiocytes mixed with lymphocytes and giant cells)
140
Etiology of an apical radicular cyst
Caries/trauma/periodontal disease Death of dental pulp Apical bone inflammation Dental granuloma Stimulation of epithelial rests of Malassez (remnants of embryological origin) Epithelial proliferation Periapical cyst formation
141
% of minor salivary gland tumours that are malignant
40-50%
142
Coughing blood suggests
Lung cancer Could be pharynx or larynx
143
When to make an urgent referral for suspected oral cancer?
Persistent unexplained head and neck lumps >3 weeks Unexplained ulceration or swelling/induration of the oral mucosa persisting >3 weeks All unexplained red or mixed red and white patches of the oral mucosa persisting for > 3weeks Persistent (not intermittent) hoarseness lasting for >3 weeks Persistent pain in the throat or pain on swallowing lasting for >3weeks
144
Ludwig's angina
Pus either side of the mylohyoid line
145
Mylohyoid line
Bony ridge on the internal surface of the mandible, running posteriosuperiorly. The site of origin for the mylohyoid muscle
146
Most important issue associated with Ludwig's angina
Airway problems due to the swelling causing the tongue to rise up
147
Upper central infection tends to cause swelling where?
Lip
148
Upper laterals, canines and first premolar infections are likely to cause swelling where?
Eye
149
Upper 2nd premolar and molar infections are likely to spread and swell where?
Sinus, cheek, temple
150
151
152
Where is a lower incisor infection likely to cause infection?
Mental/submental
153
Where are infections of lower canines and premolars likely to cause swelling?
Submandibular
154
Where are lower molar infections likely to cause swelling?
Pharynx/cheek
155
What determines the spread of infection of a dental infection?
The root length of the tooth and its position in the arch in relation to the mylohyoid line
156
Triangle of danger
Named so because of the connection from the facial veins into the cavernous sinus Infection which enters the veins in this area can cause cerebral abscess
157
Cavernous sinus
A vascular space under the pituitary gland, with nerves and blood vessels running through it
158
Types of elevators
Warwick James - right left and straight Cryer's - right and left Coupland's - size 1, 2, 3
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Name and what they are used for
Left to right Upper root forceps - retained roots Upper straight forceps - upper anteriors Upper universals - canines and premolars Upper molar forceps (left and right) Upper third molar bayonet forceps
160
Describe upper molar forceps and why are they shaped this way?
One rounded and one pointed beak The pointed beak is designed to engage the furcation on the buccal side
161
Name and what they are used for
Left to right Lower root forceps - retained roots Lower universal forceps - lower incisors, canines and premolars Lower molar forceps Cowhorn forceps - lower molars, often broken down ones that are not easily gripped
162
Patient positioning for extractions
Upper tooth - supine 45-90 degrees Lower tooth - more upright 0-45 degrees At a comfortable height for the operator
163
Where should the operator be positioned for an extraction of a lower right tooth?
Behind the right shoulder
164
Where should the operator be positioned for extraction of an upper right tooth?
To the front of the patient on their RHS
165
Where should the operator be positioned for extraction of an upper left tooth?
To the front of the patient on their RHS
166
Where should the operator be positioned for extraction of an lower left tooth?
To the front of the patient on their RHS
167
Describe extraction technique with forceps
Make sure soft tissues are clear of the forceps Position forceps as far down the tooth as possible without traumatising the gingivae Place the thumb and forefinger of the non dominant hand on the alveolar bone to support it Apply apical pressure and carry out extraction movements - on multi rooted teeth such as molars this is buccal expansion and figure of 8, on single rooted teeth (and sometimes premolars) use rotation
168
Procedure following extraction
Check apices are intact Check socket is clear If RR or bony sequestrum that is easily removed, remove it Place dampened gauze and get patient to bite, applying pressure Check for haemostasis after 5-10 minutes Give post-op instructions
169
What are luxators and elevators for?
Used to aid extraction of retained roots and teeth Used to aid mobility before using forceps
170
Luxator
Used to tear and sever the periodontal ligament, creating mobility
171
How do elevators work?
By creating space by using - Wedge - Lever or - wheel and axle motions
172
How to tell the difference between a luxator and a coupland's elevator?
Luxators have a rounded edge
173
Name and what are they used for
Warwick James (left, straight, right) Used as elevators to create space Particularly handy when extracting third molars
174
Left and right Used as elevators to create space Particularly useful when used in the furcation area of a molar tooth
175
How to hold elevators?
Bottom of the handle in the palm Curve middle, ring and pinkie fingers around the handle Put index finger on the shank for support and control
176
How to use a luxator?
Position the same as when using forceps - Support alveolar bone with non dominant hand - Luxate in the buccal sulcus from mesial to distal Lack of stability can cause soft tissue trauma
177
How to use a Coupland's?
Lever or wedge motion
178
Correct positioning when extracting a tooth allows you to..
Keep your arms close to your body Provides stability and support Allows you to keep your wrists straight enough to deliver adequate force with your arm and shoulder, and not with your fingers/hand, the force can thus be controlled in the face of sudden loss of resistance from a root or fracture of the bone
179
Types of suture technique
Simple interrupted Horizontal mattress Vertical mattress Figure of 8 Continuous
180
What type of suture is this?
Vertical mattress
181
What type of suture is this?
Vertical mattress
182
What type of suture is this?
Horizontal mattress
183
Approximately how far from the wound edge should sutures be placed?
2-5mm
184
Describe a vertical mattress suture
A shallower, more superficial suture closer to the wound edge, within a deeper, further from the wound edge suture
185
Why use resorbable sutures?
In areas where the suture requires to be buried, or is difficult to remove Used for most intra-oral wounds
186
Why use non resorbable sutures?
Sutures retain tensile strength and remain in the tissue until removed Often used in areas where high tensile strength is required for a longer period of time Such as OAC, skin closure, or to hold dressings when exposing canines
187
Example of resorbable mono filament suture
Monocryl
188
Example of resorbable polyfilament suture
Vicryl, Velosorb, Polysorb
189
Example of non resorbable monofilament suture
Nylon Prolene
190
Example of non resorbable poly filament suture
Silk
191
What is the difference between monofilament and polyfilament?
Monofilament - sutures are made from a single strand. These are less likely to facilitate an infection because it is more difficult for bacteria to colonise on a single strand Polyfilament - Sutures are made from several smaller strands twisted together and can be easier to handle. They are often contraindicated in contaminated wounds due to wicking
192
What is wicking/the wick effect?
Studies have shown multifilament sutures can absorb fluids and bacteria thus enabling infection to penetrate the body along the suture tract
193
Advantage of polyfilament sutures
Can be easier to handle
194
Disadvantage of polyfilament sutures
Wicking/ the wick effect
195
Advantage of monofilament sutures
Less likely to facilitate an infection because it is more difficult for bacteria to colonise a single strand
196
Consideration of LA in pregnant women
Citanest contains felypressin which may induce labour
197
Which type of elevator's are you more likely to use wheel and axle motion with?
Cryer's
198
Required INR for warfarin patients for extraction and which guidelines?
SDCEP 1-4
199
How do new oral anti-coagulants work?
by inhibiting the action of factor 10a on the coagulation cascade
200
Bones associated with the TMJ
Temporal Sphenoid Zygomatic Maxilla Mandible
201
Describe the structure of the mandible
2 superior processes - condyloid and coronoid The mandibular notch is between the two Inferior to this is the neck, the ramus and then the angle Moving anteriorly there is the body of the mandible, beginning just lateral to the mental foramina, where the mental nerve exits to provide sensory innervation to the chin and some of the mandibular teeth. In the midline of the body is the mandibular symphysis.
202
Describe the internal aspect of the mandible
The mandibular foramen, where the inferior alveolar nerve enters into the mandibular canal and the submandibular fossa, which the submandibular gland is pressed against are on the internal aspect
203
A - Coronoid process B - Angle C - Ramus D - Neck E - Condyloid process F - Mandibular notch
204
Body of the mandible (left) Mental foramen
205
Where is the mental nerve derived from?
It is a branch of the inferior alveolar nerve which is a branch of the mandibular nerve which is the third division of the trigeminal nerve
206
What does the mental nerve innervate?
It provides sensory innervation to the chin and lip, anterior buccal mucosa and some mandibular teeth
207
Where does the mental nerve exit the skull through?
Mental foramen
208
Through what does the inferior alveolar nerve enter the mandibular canal?
Through the mandibular foramen
209
Where does the condyloid process articulate with to give the TMJ?
Mandibular fossa of the temporal bone
210
What is anterior movement of the TMJ limited by?
Articular eminence
211
Where is the articular eminence located?
On the zygomatic arch, just anterior to TMJ
212
A - zygomatic arch B - articular tubercle/eminence C - mandibular fossa D - Styloid process E - Mastoid process F - External auditory meatus
213
What type of joint is the TMJ?
Synovial
214
What is the TMJ?
A synovial joint at the articulation between the condyloid process of the mandible and the mandibular fossa of the temporal bone, found in the a region known as infratemporal fossa
215
What is the region of the skull that TMJ is found in?
Infratemporal fossa
216
What splits the TMJ into an upper and a lower compartment?
Articular disc
217
A - articular disc B - articular tubercle/eminence C - Articular capsule D - Ramus of mandible E - Mandibular condyle F - Inferior joint cavity G - Superior joint cavity H - Mandibular fossa
218
what is the difference between the upper and lower compartments of the TMJ?
Gliding movements such as protrusion and retraction or side to side are permitted in the upper Rotational movements such as elevation and depression are permitted in the lower
219
What must happen before depression of the mandible will be allowed?
The condylar process must move anteriorly within the upper compartment of the TMJ
220
What will happen if the condylar process moves anteriorly beyond the articular eminence and how is this fixed?
MoM will spasm and TMJ will become dislocated Put downward pressure on the lower molars and guide the head of the mandible back into the mandibular fossa
221
What is the lateral temporomandibular ligament and its role?
Attaches to zygomatic arch and posterior portion of the neck of the mandible Limits posterior movement of the mandible
222
What limits posterior movement of the mandible?
Lateral temporomandibular ligament
223
1 - joint capsul 2 - lateral temporomandibular ligement 3 - sphenomandibular ligament 4 - styloid process 5 - stylomandibular ligament
224
What are the medial ligaments to the TMJ?
Sphenomandibular ligament Stylomanidbular ligament
225
What is the function of the sphenomandibular and stylomandibular ligaments?
Limiting lateral movement of the TMJ
226
Where does the sphenomandibular ligament run from/to?
From the ramus to the sphenoid bone
227
Where does the stylomandibular ligament run from/to?
From the ramus to the styloid process
228
Muscles of mastication
Temporalis Masseter Lateral pterygoid Medial pterygoid
229
Which bone are the pterygoid plates part of?
Sphenoid
230
Where are the pterygoid muscles located in relation to the mandible?
The pterygoid muscles are located medial to the mandible
231
Describe medial pterygoid structure
Has two heads Deep head - attached to medial aspect of the lateral pterygoid plate Superficial - attached to the maxilla and the palatine bones Both heads run posterior inferiorly to reach a point at which they fuse and will then insert onto the ramus of the mandible
232
1 Lateral pterygoid 2 Medial pterygoid
233
Describe lateral pterygoid structure
Superior head attaches to the roof of the infratemporal fossa and the lateral portion of the lateral pterygoid plate Inferior head attaches to lateral portion of lateral pterygoid plate Moving posteriorly both heads will fuse and attach to the condylar process of the mandible
234
What happens when lateral pterygoid muscles act bilaterally?
They protrude the jaw
235
Which action of the lateral pterygoids is paramount to the opening of the mouth?
Acting bilaterally to protrude the jaw
236
What does unilateral contraction of the lateral pterygoids do?
Swings the jaw to the contralateral side
237
What does bilateral action of the medial pterygoid muscles do?
Closes the jaw and assists in protrusion
238
What does unilateral contraction of medial pterygoid muscles do?
Swings the jaw to the contralateral side
239
Where does temporalis arise from and attach to?
From the temporal fossa, attaching to the coronoid process of the mandible
240
Describe the difference between the anterior and posterior fibres of temporalis
The anterior fibres run vertically and will assist in closing the jaw The posterior fibres run horizontally and will retract the mandible
241
Where is masseter located?
On the lateral side of the mandible
242
Structure of masseter
Superficial head - attached to zygomatic bone Deep head - attached to zygomatic arch Inferiorly both heads attach to the ramus and angle of the mandible
243
Action of masseter
Elevates the mandible to close the mouth
244
Innervation of muscles of mastication
All innervated by the mandibular division of the trigeminal nerve
245
When is the TMJ most stable?
When the jaw is closed with teeth in occlusion
246
What is the most common kind of TMJ displacement?
Anterior dislocation - the condyloid process moves anterior to the articular eminence and the MoM spasm, preventing retraction of the mandible
247
What opposes anterior dislocation of the TMJ?
Articular eminence, lateral temporomandibular ligament, and the contraction of medial pterygoid, masseter and temporalis
248
Origin of the masseter
2 origins Zygomatic buttress of the zygomatic bone and medial aspect of zygomatic process of the temporal bone
249
Insertion of the masseter
Lateral surface of the angle of the mandible
250
Origin of temporalis
Temporal fossa
251
Insertion of temporalis
Coronoid process
252
Origin of medial pterygoid
Medial surface of the lateral pterygoid plate
253
Insertion of the medial pterygoid
Medial side of the angle of the mandible
254
Lateral pterygoid origin
2 origins Lateral surface of lateral pterygoid plate and the base of skull/top of infratemporal fossa
255
Insertion of the lateral pterygoid
Pterygoid fovea at the condyle
256
Blood supply to the TMJ
Deep auricular artery (branch of the 1st part of the maxillary artery)
257
Nerve supply to the TMJ and why is this relevant to symptoms?
Auriculotemporal, masseteric, posterior deep temporal nerve Auriculotemporal nerve also supplies parts of EAM and some with TMD get pain in the ear
258
Suprahyoid muscles
Digastric Mylohyoid Geniohyoid Stylohyoid
259
Infrahyoid muscles
Thyrohyoid Sternohyoid Sternothyroid Omohyoid
260
A Digastric (anterior belly) B Geniohyoid C Mylohyoid D Stylohyoid E Digastric (posterior belly)
261
A Thyrohyoid B Sternothyroid C Omohyoid (superior belly) D Sternohyoid E Omohyoid (inferior belly)
262
Which part of the articular disc can not feel pain and why?
The anterior band of the articular disc Does not have sensory innervation
263
What does the articular disc do during jaw movements?
Slides back and forth with the condyle
264
Causes of TMD
Myofascial pain (muscles) Disc displacement (anterior with or without reduction) Degenerative such as osteoarthritis or rheumatoid arthritis Chronic recurrent dislocation Ankylosis (very rare) Hyperplasia (one condyle grows more) Neoplasia (tumours) Infection (incredibly rare)
265
What could be a localised degenerative cause of TMD?
Osteoarthritis
266
What could be a systemic degenerative cause of TMD?
Rheumatoid arthritis
267
What does it mean if anterior disc displacement is 'with reduction'?
With reduction - disc eventually slips back into place Without reduction - stuck in front of the condyle permanently
268
Appearance of condylar hyperplasia
Facial asymmetry Chin points away from the side of the condyle that is growing more
269
Possible neoplastic causes of TMJ (rare)
Osteochondroma - overgrowth of bone and cartilage Osteoma - benign bony tumour Osteosarcoma - malignant bone tumour
270
How to relocate a dislocated TMJ?
Hold jaw with thumbs inside the mouth on the buccal surfaces of lower molars and push down and backwards slowly Sometimes muscle relaxants such as benzodiazepines are necessary
271
Possible pathogenesis to myofascial pain TMJ
Inflammation in the MoM or TMJ secondary to parafunctional habits Trauma - either directly to the joint or indirectly such as sustained opening Stress Psychogenic Occlusal abnormalities (no evidence)
272
Social history considerations for a TMD patient
Occupation, stress, home life, sleeping pattern, recent bereavement, relationships, habits, hobbies
273
E/O exam of TMD patient
MoM Joint - clicks/ crepitus Jaw movements Facial asymmetry
274
Intra-oral exam for TMD patient
Interincisal mouth opening - could use Willis bite gauge Signs of parafunction - tongue scalloping, biting cheeks, linea alba, occlusal NCTSL
275
When are you more likely to do special investigations for TMD?
Suspicion of pathology
276
Common clinical features of TMD
Females > males Age 18-30 Intermittent pain of several months or years Muscle/joint/ear pain, particularly on waking Trismus/locking Clicking/popping Headaches
277
Potential differential diagnoses for TMD
Dental pain Sinusitis Ear pathology Salivary gland pathology Referred neck pain Headache Atypical facial pain Trigeminal neuralgia Angina Condylar fracture Temporal arteritis
278
What is temporal arteritis?
Inflammation of the temporal artery which can result in blindness Presents as very severe pain in the temporal region
279
Treatment for TMJ dysfunction
Patient education Counselling Jaw exercises Medication (Electromyographic recording)
280
What medication can be used to treat TMD?
NSAIDs Muscle relaxants Tricyclic antidepressants Botox Steroids
281
What does TMD counselling include?
Soft diet Masticate bilaterally No wide opening No chewing gum Don't incise food Cut food into small pieces Stop parafunctional habits Support mouth on opening such as yawning
282
Physical therapy for TMD
Physiotherapy Massage/heat Acupuncture Relaxation Ultrasound therapy TENS Hypnotherapy
283
Examples of irreversible treatment for TMD
Occlusal adjustment TMJ surgery
284
How might patients with TMJ disc displacement present?
Painful clicking TMJ The click is due to a lack of coordinated movement between the condyle and the articular disc Jaw tightness/locking The mandible may deviate initially to the affect side
285
What can happen if TMJ disc displacement is left untreated?
Osteoarthritis
286
Treatment for TMJ disc displacement
Counselling, limited mouth opening, bite raising appliance (occasionally surgery)
287
What is this?
Jaw screw For trismus Placed between incisors then on twisting, opens the mouth
288
Treatment for trismus
Physio therapy Therabite jaw rehabilitation system Jaw screw
289
Guidelines for TMJ dysfunction
NICE
290
When would you refer a TMD patient to oral med or OMFS?
History of trauma or fracture to TMJ complex Markedly limited mouth opening suggesting disc displacement without reduction Pain or reduced function in people with rheumatic joint disease Worsening symptoms lasting >3 months despite primary care tx Other chronic pain comorbidities Recurrent dislocation Severe pain and dysfunction not responding to conservative management
291
What should you consider before assessing a trauma case?
ATLS - Advanced Trauma Life Support, treat the greatest threat to life first ABCD - Airway, breathing, circulation, disability
292
What is the Glasgow Coma Scale?
Used to assess head injuries Eye opening, verbal response and motor response are all considered and given points in order to give a GCS score
293
Glasgow Coma Scale scores
Mild 13-15 Moderate 9-12 Severe 3-8
294
Glasgow Coma Scale Eye opening scores
Spontaneous - 4 To sounds - 3 To pressure - 2 None - 1
295
Glasgow Coma Scale verbal response scores
Orientated - 5 Confused - 4 Words - 3 Sounds - 2 None - 1
296
Glasgow Coma Scale motor response scores
Obey commands - 5 Localising - 5 Normal flexion - 4 Abnormal flexion - 3 Extension - 2 None - 1
297
Indicators of mandible fracture
Sublingual haematoma 2 point mobility vertically Abnormal sensation contralateral to the side of injury Pain contralateral to the side of injury Numbness that can't be explained by direct injury to the nerve
298
What is needed to diagnose a mandible fracture?
2 regular Xrays or a CT scan (there is no role for half OPGs)
299
How to manage a mandible fracture
Fast the patient Analgesia - can swallow tablets with small amount of water Antibiotics for open fractures - amoxicillin and metronidazole or equivalent Liquid diet Immediate discussion with OMFS team
300
What region would be considered a midface fracture?
Eyebrows to maxillary teeth, including zygoma
301
Important signs of broken bone in the midface
Epistaxis (nose bleed) without a blow to the nose V2 numbness without a direct blow to the nerve Subconjunctival bleed Midface mobility Malocclusion Surgical emphysema around eye Swelling after nose blowing Diplopia Change of appearance Clear liquid CSF running out of nose
302
Diplopia
Double vision
303
What causes a subconjunctival bleed?
Seen in some midface fractures Conjunctiva has been torn by the fracture, and blood turns the white of the eye deep red (not the whole eye)
304
Epistaxis
Nose bleed
305
How to check for midface mobility?
Hold forehead still and pull palate backwards and forwards
306
What is meant by surgical emphysema?
Air in the soft tissues Seen around the eye in some midface fractures
307
What is required to assess a midface fracture?
2 Xrays
308
Le fort fractures
The face breaks in three particular patterns in the midface All fractures extend into the pterygoid columns, if the pterygoids aren't broken then you don't have a Le Fort fracture
309
How to tell which type of Le Fort fracture?
Hold head and pull on teeth If teeth move - Le Fort I If nose moves - Le Fort II If eyes move - Le Fort III
310
Management of zygoma fracture
No indication for routine antibiotics Call OMFS - vast majority will be followed up in 7-10 days No nose blowing Soft diet for their comfort Give warning re retrobulbar bleed, as this can lead to blindness (rare)
311
Retrobulbar bleed
Rare, sight-threatening emergency, that results in accumulation of blood in the retrobulbar space
312
Orbit fracture
Breaking of just the eye socket, deep segments rather than the rim - would be zygoma fracture
313
Management of orbit fracture
Ensure visual acuity and diplopia documented Discuss with OMFS Don't bother CTing No need for routine antibiotics No nose blowing Give warning re retrobulbar bleed
314
Indicators of an orbit fracture
Eyebrow sign - gas leaks and rises to just below eyebrow Eye may be sunken or dropped down
315
When would you give warning about retrobulbar bleed?
Zygoma fracture Orbit fracture
316
Management of maxilla fracture (le fort type)
Fast the patient Antibiotics Discussion with OMFS Liquid diet No nose blowing Most will need assessed on the day
317
Pathognomic features of zygoma fracture
Unilateral epistaxis when the noe has not been injured Eyebrow sign Paraesthesia when trauma was distant to extraosseous infraorbital nerve Buttress tenderness
318
Nasal Orbital Ethmoidal fracture pathognomic features
Retropositioned nose Buttress not tender Epistaxis Often numb Steps at IOR, pyriform, glabella Hyperteloric (eyes drift apart)
319
Naso-maxillary fracture pathognomic features
Tender IOR/pyriform Buttress intact Often numb Unilateral epistaxis without blow to the nose Same as zygoma EXCEPT buttress
320
Where is the parasymphasis of the mandible?
Anterior to mental foramen
321
Most common imaging for midface fractures?
OPG and PA mandible (two angulations of PA mandible for zygoma or orbit)
322
What does SIRS stand for?
Systemic inflammatory response syndrome
323
SIRS Systemic Inflammatory Response Syndrome criteria
Fever 38C + Hypothermia <36C Tachycardia - high pulse >90bpm Tachypnoea - high breathing rate >20 breaths/min Change in blood count (WBC count >1200) Partial pressure CO2 <32mmHg
324
Hot potato syndrome
Infection causes raised floor of mouth, patient talks like they have hot potato in their mouth
325
Where must you never incise an intraoral abscess in GDP?
Floor of mouth
326
Should you suture the wound from draining an abscess?
No - allows more drainage
327
What are rongeurs?
Bone nibblers Used to remove small fragments of bone
328
What excisional soft tissue surgery might be used for before provision of dentures?
Frenoplasty Papillary hyperplasia Flabby ridges Denture induced hyperplasia Maxillary tuberosity reduction Retromolar pad reduction
329
Why might a labial frenoplasty be required?
Oral hygiene issues
330
What risk must be considered during a buccal frenoplasty?
Risk of damaging the mental nerve
331
When might a lingual frenoplasty be necessary?
Tongue tie
332
When might a buccal frenoplasty be necessary?
High buccal frenum in a denture patient This would break the seal and displace the lower denture every time the patient moves
333
What should be done if denture associated papillary hyperplasia does not resolve with removal of the denture?
Excisional surgery
334
Vestibuloplasty
Surgery to deepen the sulcus in order to achieve better denture retention by having more space for extending the flange
335
When might hard tissue excisional surgery be considered before provision of dentures?
Removal of retained teeth/roots/pathology Ridge defect correction (alveoplasty) Mandibular tori Maxillary tori Maxillary tuberosity Exostoses Undercuts Genial tubercle reduction Mylohyoid ridge reduction
336
Why might retained roots or "buried" teeth become a problem in denture provision?
If the ridge resorbs these may end up palpable or visible and interfere with the fitting surface
337
What can be seen here and what is the most likely cause?
Well defined, unilocular, corticated radiolucency Residual cyst - apical radicular cyst in relation to one of the teeth in the region, tooth has been removed but the cyst has remained
338
How would you manage this?
Biopsy to determine what it is CBCT to see relation to the IAN Then decide on final tx plan
339
What surgical technique would be used for this and why?
Alveoplasty to prevent interference with denture retention
340
What is the likely cause of this?
Retained lower anteriors for much longer than the posteriors
341
What is this and how would you manage it/why?
Knife edge ridge This is sharp and can be uncomfortable when wearing a denture or traumatic to soft tissues Smooth this ridge to make it more comfortable but do not take too much away as this could negatively affect denture retention
342
Management of a maxillary torus in denture patients
Surgically remove the bony projection OR design denture around it e.g. horseshoe shape
343
Why might a large maxillary tuberosity occur?
Large bony tuberosity OR lots of excess fibrous tissue surrounding normal bone
344
Which two structures on the mandible can become prominent and might require removal/reduction with very sever bone resorption?
Genial tubercle Mylohyoid ridge
345
Examples of hard tissue augmentation procedures that might be done for denture patients
Autografts Allografts Xenografts Synthetic grafts
346
Autografts
Bone taken from elsewhere in the body for a graft eg hip bone being used to augment the maxillary ridge
347
Allografts
Bone taken from human cadavers for bone grafts Commonly used for alveolar bone for implants
348
Xenografts
Bone from animals, usually cows, can be horses Used to provide framework for bone regeneration
349
Pros/cons of synthetic bone grafts
No risk of disease transmission No cultural/religious/ethical issues Can be very effective but some can have a lower rate of being accepted by the body Customisable to be exact shape/size/porosity that you want
350
When would inferior alveolar nerve relocation be done? And describe the procedure
In severe cases of bone resorption the mental foramen can end up at the surface In some cases the entire IAN can be just covered by soft tissue rather than by bone Denture fitted to these tissues will press on the nerve causing numbness and pain To reposition you would open up to expose the nerve, drill a channel deeper down in the body of the mandible and reposition the nerve further down
351
Why might implants be provided for complete denture patients?
For implant retained overdentures
352
23 year old pt attends reporting pain from LL for 18 months, pt says pain is from wisdom tooth and asks for it to be removed. What do you want to know?
SOCRATES HPC - past episodes of pericoronitis/antibiotics/swelling history Systemic MH, meds, allergies PDH, anxiety, previous extractions, any problems SH - smoking, drinking, occupation, caring responsibilities
353
How will you carry out a comprehensive assessment of someone reporting signs of pericoronitis?
Extra oral TMJs Lymphnodes Asymmetry MoM Mouth opening Intra oral Soft tissues Dentition Caries Perio OH Working distance L8 erupted? Condition of adjacent 7 Presence of other 8s
354
Assessment findings
LL8 partially erupted- approx 1/2 occlusal surface is visible LL8 appears vertically impacted on clinical examination Operculum appears infllamed
355
Patient reports recurrent episodes of pain from LL quadrant and on examination you find this. What is your next step?
OPT
356
Report
OPT diagnostically acceptable Vertical/slighty distoangular LL8 superficial impaction LL8 crown healthy - no signs of caries LL8 crown wider than roots LL8 has at least 2 roots LL8 apices appear close to ID canal but no signs of intimate relationship LL7 appears sound Adequate bone levels
357
Pericoronitis recurring LL8 What are the treatment options
Clinical review - monitor at regular examination, only require radiographs if change in signs or symptoms Surgical removal of LL8 (No indication for further imaging or coronectomy in this case as no intimate relationship with ID canal, no indication for XLA UL8 as doesn't look to be occluding against operculum)
358
What information do you need to give to ensure pt is making informed decision about surgical removal of LL8?
Discuss option of LA/Conscious sedation/GA (and referral if required) Regarding procedure: Pain, Swelling, Bleeding, Infection, Jaw stiffness, Dry socket Temporary (2-20%) or Permanent (<1%) damage to nerve, possibility of numbness, tingling or painful sensation Areas affected could include side of chin, lip, tongue, gums or cheek Small risk of loss of taste Surgical approach: cut in gum, bone removal which will feel like vibration/water, pressure, stitches (dissolving)
359
Usual age of eruption for third molars
18-24 years
360
When can crown calcification of 8s start to be seen radiographically?
7-9 for uppers 8-10 for lowers (completed by about 18)
361
What age is root calcification of 8s complete?
18-25
362
How common is it that and adult has 1 or more third molars present?
1 in 4
363
Agenesis
Failure of an organ to develop
364
Is third molar agenesis more common in mandible or maxilla?
Maxilla
365
Is third molar agenesis more common in men or women?
Women
366
What age would you expect to see third molars radiographically, after which they almost never develop?
14
367
What is a common reason for older patients to have issues with third molars?
Third molars causing problems with dentures
368
Impacted
Tooth eruption is blocked Failure to erupt into either a full or partial functional position, or at all
369
What is the most common reason for third molars failing to erupt?
Impaction
370
What are M3Ms usually impacted against?
Adjacent tooth, alveolar bone, surrounding mucosal soft tissue or a combination of these factors
371
Eruption of impacted third molars
Impacted third molars can be unerupted, partially erupted or fully erupted
372
Fully erupted
Whole occlusal surface through the mucosa and exposed to the oral cavity
373
Incidence of impacted lower 3rd molars
36-59%
374
Consequences of third molar impaction
Caries (in 8 or 7) Pericoronitis Cyst formation (often results from the failure of the follicle to separate)
375
What does it mean if a tooth appears unerupted clinically, but radiographically there is caries?
This suggests that there is a communication between this tooth and the oral cavity, since the bacteria is able to reach it and cause caries Probe carefully distal to 7 to try to find this communication
376
4 nerves at risk during 3rd molar surgery
Inferior alveolar nerve Lingual nerve Nerve to mylohyoid Long buccal nerve
377
Which two nerves are most likely to be damaged during third molar surgery?
Inferior alveolar nerve Lingual nerve
378
Inferior alveolar nerve
Peripheral sensory nerve formed from the mandibular division of the trigeminal nerve. Supplies all the mandibular teeth (on its side) and mucosa and skin of the lower lip and chin on that side
379
Inferior alveolar nerve and third molar relationship
Position in relation to the mandibular third molar varies greatly Usually need radiograph before third molar surgery to see this Risk to the nerve should be considered
380
Lingual nerve
Branch of the mandibular division of the trigeminal nerve supplying anterior two thirds of the dorsal and ventral mucosa of the tongue Also gives off a branch which supplies lingual gingivae and FoM
381
Lingual nerve position
Lingual nerve varies in position Close relationship to the lingual plate in the mandibular and retromolar area At or above level of the lingual plate in 15-18% Between 0.3-5mm medial to mandible Must be very careful to avoid during third molar surgery
382
Guidelines for 3rd molar surgery
NICE and SIGN basically dictate that because of the related risks, you must be able to justify third molar surgery with pathology - caries, significant infection, perio, cyst RCS FDS - more recent acknowledges that you might be delaying inevitable surgery which could make it more difficult in future, and recommended changing from a solely therapeutic approach to a mixed range of interventions
383
Therapeutic indications for third molar surgery
Infection - caries, pericoronitis, perio, local bone infection, most common Cysts Tumours External resorption of 7 or 8
384
If there is any history of pericoronitis..
Removal of any symptomatic third molar should always be considered
385
1st and 2nd most common reasons for removal of third molars
1st caries 2nd pericoronitis
386
Why is restoring a caries lower 8 usually not done?
Access and moisture control make it very difficult 8s are not really necessary
387
What type of third molar impaction is more prone to causing bone loss distal to the lower 7
Horizontal and mesio angular impaction
388
What is the best way to reduce bone loss distal to a 7, if there is a horizontally or mesio angular impacted 8?
Early extraction of the third molar Late removal, especially after age 30 has not been shown to improve periodontal status of the adjacent 7
389
Most common age range for cyst formation
20s-50s
390
When do cysts normally first become symptomatic?
When they become very large and/or infected
391
Most common type of cyst to be found associated with third molars
Indigenous cyst
392
Indigenous cyst
Arises from the reduced enamel epithelium separation from the crown
393
Are cysts associated with third molars more common in mandible or maxilla?
Mandible
394
Why is prophylactic removal or coronectomy of a disease free lower 8 to prevent cyst formation not routine?
This would prevent development of a cyst but the number of cysts that you're going to prevent by doing this would be fairly small so it is not usually an indication
395
Tumours as indication to removal third molars
If the tumour is close to the lower 8 it might be extracted as part of a cancer, in part of the dissection as may other teeth If the pt has tumour anywhere in the body and will be having radiotherapy, it might be indication to remove lower 8 because of the future ORN risk
396
Why might resorption be indication for third molar removal?
External resorption is the destruction of tissue, the cause of inflammation is often unclear but left untreated it is usually progressive External resorption of the third molar or of the second molar caused by the third molar should always make us consider third molar removal
397
Most common age for external resorption of lower 7 by lower 8
21-30 (relatively rare)
398
Non therapeutic indications for extractions of third molars
Surgical indications High risk of disease Medical indications Accessibility Patient age Auto transplantation General anaesthetic
399
Examples of possible surgical indications for third molar removal
Tooth is within surgical field - orthognathic, fractured mandible, in resection of diseased tissue
400
What are high risk of disease indications for third molar removal?
Mesio angular or horizontally impacted lower 8 High risk of caries for 8 or 7 and of periodontal bone loss
401
Medical indications for removal of third molars
needing signed off dentally fit e.g. Awaiting cardiac surgery Immunosuppressed (or about to become) Before going on bisphosphonates Before starting radiotherapy or chemotherapy
402
When might you be more likely to consider removal of 8s in a perfectly healthy patient?
If they have limited access to the dentist e.g. submariners
403
Why is patient age an indicator in 3rd molar removal?
Complications and recovery time increase with age, so it might make sense to taje the tooth out while the patient is young
404
Where would an 8 usually be moved to in autotransplantation?
Lower 6 position
405
Why might you be more inclined to take out a third molar in someone alongside other dental treatment?
If they were going for GA
406
Pericoronitis
Inflammation around the crown of a partially erupted tooth Tooth is normally partially erupted
407
What causes pericoronitis?
Food and debris gets trapped under the operculum of a partially erupted tooth, resulting in inflammation or infection
408
Describe pericoronitis infection
Usually transient and self-limiting
409
Most common age for pericoronitis
20-40
410
General health factor in pericoronitis
Upper respiratory tract infection can become pericoronitis
411
What type of microbes are likely to be involved in pericoronitis?
Anaerobes
412
Examples of bacteria found in pericoronitis infections
Streptococci Actinomyces Prioponibacterium Beta-lactamase producing prevotella Bacteroides Fusobacterium Staphylococci
413
Pericoronitis signs and symptoms
Pain Swelling Bad taste/halitosis Pus Occlusal trauma to operculum Ulceration of operculum Evidence of cheek biting Limited mouth opening Dysphagia Pyrexia Malaise Regional lymphadenopathy
414
What might be seen in severe pericoronitis?
Extra oral swelling Most commonly starting at the angle of the mandible Can spread anywhere, commonly submandibular areas
415
If pericoronitis infection spreads distobuccally under the masseter, what is this called? and give a characteristic sign
Submasseteric abscess Pt can not open mouth
416
What symptom might arise from spread of pericoronitis infection into the parapharyngeal space?
Dysphagia This might present with drooling
417
Pericoronitis treatment
IF pt is acutely symptomatic you might - Incise pericoronal abscess - Irrigate under operculum with chlorhexidine or saline using blunt syringe (10-20ml) - Extract upper third molar if traumatising operculum - Instruct pt on warm salt water or chlorhexidine MW - Antibiotics if systemically unwell, extra-oral swelling, immunocompromised - If large extra oral swelling, significantly unwell, trismus, dysphagia - refer to OMFS or A&E (generally do not remove the third molar during an acute episode of pericoronitis)
418
Why is operculectomy now not done very often?
Within weeks or months the operculum grows back to where it was so not much is gained
419
Method of pericoronitis management that is no longer in favour
Operculectomy
420
Predisposing factors for pericoronitis
Partial eruption and vertical or distoangular impaction Opposing maxillary 2nd or 3rd molar causing mechanical trauma URTI Stress/fatigue Poor OH Insufficient space between ascending ramus and distal aspect of M2M White race Full dentition
421
Most current guidelines on third molar management
RCS FDS
422
When is it appropriate to take out a third molar with no associated pathology?
If the risk and likelihood of it causing problems at some point in the future is too high
423
History during assessment of patient presenting with third molar
General appearance - asymmetry, difficulty speaking, look unwell C/O and HPC Medical history Dental history Social history
424
3 components of assessment of pt presenting with third molar
History Clinical examination Radiographic assessment (if indicated)
425
HPC for pericoronitis
How long, how many episodes, how often, severity, requirement for antibiotics SOCRATES
426
Underlying systemic diseases that might interfere with normal healing
Diabetes Chronic renal disease Liver disease Bleeding disorder Immunosuppressed Radio or chemotherapy
427
Medications relevant during third molar treatment planning
Contraceptive - increased risk of dry socket Steroid therapy - increased risk of wound infection and delayed healing Bisphosphonates - MRONJ Anticoagulants and antiplatelets
428
What would you ask if a patient has previous extractions, and you are planning third molar management?
How did they find it? Any sedation/GA Surgical Any delay in healing Any issues post op
429
Extra-oral exam of patient presenting with third molars
TMJ Mouth opening Lymphadenopathy Facial asymmetry MoM
430
Why is it important to examine TMJ when a patient presents with third molars?
TMD can give pre-auricular pain very similar to pericoronitis Good to know if there's a click before surgery so that pt does not think surgery caused this
431
Intra-oral examination of patient presenting with third molar
Soft tissues Dentition M2M Working space Eruption status of M3Ms Condition of dentition Occlusion Oral hygiene Caries Perio
432
When would you do radiographic assessment of a patient presenting with third molars?
Only if surgical intervention is being considered
433
What radiograph(s) would you take to assess third molars?
OPT
434
What can be determined from an OPT to assess third molars?
Presence or absence of disease Anatomy of 3M (crown size, shape, condition, root formation, crown:root) Depth of impaction Orientation of impaction Working distance Follicular width Periodontal status Relationship or proximity of U8s to maxillary sinus, and L8s to IAN canal Any other associated pathology
435
Superficial impaction
When the crown of the 8 is sitting at the same height as the crown of the adjacent seven
436
Deep impaction
When the crown of the 8 is at the same level as the roots of the adjacent 7
437
Moderate impaction
When the crown of the 8 is level with crown and root of the adjacent 7
438
Dental follicle
Tissue that surrounds the crown of a developing tooth As the tooth pushes into oral cavity you normally lose this but if a tooth is unerupted this would appear as radiolucency
439
What does dental follicle becoming bigger than expected indicate?
Suggests pathology such as a cyst Concern at anything over 2.5-3mm
440
Why is it important to discuss the relationship of the M3M with the inferior dental CANAL (not the nerve) when discussing radiographs?
Nerves can not be seen radiographically, you are looking at the canal
441
Signs of close proximity of M3Ms to the inferior dental canal
Interruption of the white lines/lamina dura of the canal Darkening of the root where crossed by the canal Diversion/deflection of the inferior dental canal Deflection of root Narrowing of canal Narrowing of root Dark and bifid root apex Juxta apical area
442
Three radiographic signs associated with significantly increased risk of nerve injury during third molar surgery
Interruption of the white lines of the canal Diversion of the canal Darkening of the root where crossed by the canal
443
Juxta apical area
A well circumscribed radiolucent region lateral to the root of the third molar, usually not right at the apex Usually well defined, can appear corticated Lamina dura of the root still intact and appearance not pathological
444
What do guidelines suggest about further imaging after OPT for third molars
Where conventional imaging has shown a close relationship between the third molar and the inferior dental canal, CBCT may be of benefit
445
What information can CBCT give you on the relationship between M3M and inferior alveolar nerve canal?
Is there bone between the ID canal and apices of the tooth Is the tooth actually compressing the canal
446
Why is CBCT better than CT for third molar assessment?
Limited FoV of CBCT is advantageous in terms of image reconstruction and the radiation dose to the patient
447
% of different angulations of third molars
Vertical 30-38% Mesial around 40% Distal around 6-15% Horizontal 3-15% Transverse or aberrant less common
448
Most difficult third molar angulation to remove
Distal
449
Most common third molar angulation
Mesial
450
What is angulation of third molars measured against?
Curve of Spee
451
Curve of Spee
Curve as you follow the natural cusps of the dentition
452
Angulation of M3Ms
LR8 is mesially impacted LL8 is horizontally impacted
453
Why is it important to measure angulation of lower third molar impactions against the curve of spee?
It is easy to confuse a distal angulation with a vertical angulation Distal is much more difficult to extract
454
Why are distally impacted M3Ms so difficult to extract
it is very unlikely you will get it out intact without distal bone removal, and the roots of distally impacted 8s are often very close to roots of the 7 so it can be difficult to get and application point and you need to be very careful not to damage the 7
455
Angulation of L8s
LR8 is horizontally impacted LL8 is distally impacted
456
Angulation of L8s
LR8 is vertically impacted LL8 is distally impacted
457
Angulation of LR8
LR8 is transversely impacted
458
Position of LL8
Aberrant position
459
Why is it good to assess depth of impaction radiographically?
Gives an indication of the amount of bone removal required
460
Depth of impactions of L8s
LR8 is moderate LL8 is superficial
461
Depth of impaction
Superficial
462
Depth of impaction
Moderate impaction
463
What must be considered if 7 next to an impacted 8 has a large or overhanging restoration?
Risk of restoration fracture during extraction of the 8 - pt must be warned of this if they have - Large restoration - Crown - Overhangs If this happens the tooth would need to be temporised and dealt with later
464
Common treatment options for impacted M3Ms
Referral Clinical review Removal Extraction of the maxillary 3rd molar Coronectomy
465
Clinical review of M3M
Review signs and symptoms associated, can be done at regular review No indication for radiographic assessment unless clinically there are signs/symptoms
466
Coronectomy of M3M
Removing the crown and leaving the roots in situ Only usually considered if close relationship between M3M and IAN canal
467
Less common treatment options for M3Ms
Operculectomy Surgical exposure Pre-surgical orthodontics Autotransplantation
468
Important aspects of decision making in M3M treatment planning
Decision should be made jointly between patient and clinician Patient involvement - communicate findings, risk status, tx options including risks and benefits Good notekeeping Current status of patient and M3M Risk of complication Patient access to treatment
469
How to manage asymptomatic 3rd molars with disease present or high risk of disease development
Clinician should use their expertise to assess the risk eg of caries, perio, etc then consider surgical intervention This might be affected by risk of complications eg proximity to IAN canal in which case the decision might be active surveillance
470
Active surveillance of M3M
Monitoring the tooth with radiographs at regular intervals Sometimes done for asymptomatic M3Ms either with disease present or high risk of disease developing
471
What can you consult for M3M treatment planning?
Summary of the management of patients with Mandibular Third Molars from the RCS FDS guidelines
472
Management of asymptomatic M3M with no disease present/low risk of disease
Clinical review is likely to be the most appropriate management Pt medical history may change this e.g. if tooth is in surgical field
473
Management of symptomatic M3M with high risk of or disease present
Consider cause of the symptoms - caries, perio etc Then consider tx options
474
Management of symptomatic M3Ms with no disease present and low risk of disease
Consider other causes of symptoms - consider TMJ, salivary gland disease It would not be indicated to remove a deeply impacted M3M if it is disease free
475
Which patients may only be able to tolerate 3rd molar removal under GA?
Extremely anxious Contraindications to sedation Other factors complicating the surgery e.g. extensive resection being done at the same time
476
When would you explain risk of jaw fracture to a patient having extraction of M3M?
Edentulous/atrophic mandible Aberrant lower 8 close to lower border Large cystic lesion associated with the tooth
477
How would you explain the procedure of M3M surgical extraction to a patient?
Minor surgical procedure Cut flap Possible drilling Stitches - 2-3 weeks to dissolve If tooth is likely to need sectioned explain this
478
Risks to explain to pt having M3M removal
If 2nd molars have large restorations explain risk of restoration fracture Pain Swelling Bruising Jaw stiffness/limited mouth opening Bleeding Infection Dry socket Numbness or tingling
479
Why is the jaw sometimes stiff with limited opening after M3M extraction?
Mouth has been open for a long time with pressure on the lower jaw
480
Factors making dry socket more common
Extraction of 8s Females Mandible Smokers If pt has had dry socket before Contraceptive pill
481
Nerve anaesthesia feeling
Numbness
482
Nerve paraesthesia feeling
Tingling
483
% of patients who experience temporary IAN damage after M3M extraction
10-20%
484
% of patients who experience permanent IAN damage after M3M extraction
<1%
485
% of patients who experience temporary lingual nerve damage after M3M extraction
0/25-23%
486
% of patients who experience permanent lingual nerve damage after M3M extraction
0.14-2%
487
What is the time frame for nerve recovery?
Most will happen within 9 months but nerves have been shown to have recovery up to 18-24months after surgery After this any recovery very unlikely
488
Chorda tympani
Carries taste sensation from anterior two third of the tongue Carries fibres via lingual nerve Arises from facial nerve
489
Most common sensation from nerve damage during M3M surgery
Numbness - anaesthesia Tingling - paraesthesia
490
Rarer sensations from nerve damage during M3M surgery
Painful uncomfortable sensation - dysaesthesia Reduced sensation - hypoaesthesia Increased sensation - hyperaesthesia
491
When would you not opt for CBCT having seen close relationship of M3M and IAN canal on OPT?
IF the results will not change the txp - Patient wants full surgical removal regardless of the risk - Grossly carious lower 8 not suitable for coronectomy
492
What must be included in a M3M referral letter
SBAR Situation Background (HPC) Assessment Recommendation
493
When is a surgical removal required?
When tooth cannot be removed with forceps alone
494
Basic principles of surgical extraction
Risk assessment Aseptic technique Minimal trauma to hard and soft tissues
495
Surgical removal process
Anaesthesia Access Bone removal as necessary Tooth division as necessary Debridement - ensure all apices are accounted for Suture Achieve haemostasis Post op instructions
496
Access for surgical removal
Access to the tooth is gained by raising a buccal mucoperiosteal flap (+/- raising a lingual flap) Maximum access with minimal trauma Larger flaps heal just as quickly as smaller Use scalpel in one continuous stroke Minimise trauma to papillae
497
Reflection of soft tissues for surgical extraction
Commence reflection at base of relieving incision Undermine/free the papillae before proceeding with reflection distally to avoid tears (Warwick James) Reflect with periosteal elevator firmly on bone in order to avoid dissection occurring superior to periosteum and reduce soft tissue bruising/trauma
498
Instruments used to reflect surgical flap
Mitchel's trimmer Howarth's periosteal elevator Ash periosteal elevator
499
Instruments used to retract surgical flap
Howarth's periosteal elevator Rake retractor Minnesota retractor
500
Why is it important to retract the flap during surgical extractions
Access to the operative field Protect the soft tissues
501
Name left to right
Howarth's periosteal elevator Rake retractor Minnesota retractor
502
What is important for atraumatic retraction of soft tissues during surgical extractions?
Rest firmly on bone Awareness of adjacent structures e.g. mental nerve
503
Why are air driven handpieces not used during surgical extractions?
May cause surgical emphysema
504
Bone removal process for surgical extraction
Electrical straight handpiece with SS or Tungsten carbide saline cooled bur (to avoid bone necrosis) Round bur used to cut buccal gutter and on to distal aspect of impaction, starting distally and coming mesial (reduces risk to lingual nerve and other soft tissues behind M3M) Buccal gutter as narrow and deep as poss Bone removed to allow application of elevators
505
What is done after bone removal during surgical extraction?
Operator must assess the possibility of removing the tooth in one piece with elevators and forceps If this is not possible and adequate bone has been removed the tooth should then be sectioned with drillOPerator Most commonly sections between crown and roots, then sometimes further separation of the roots from each other Operator may prefer to section vertically
506
A 68 year old female with history of a fractured neck of femur has been given 2 drugs to prevent her getting another fracture. Give two drug types and examples that this could be
Anti-resorptives - bisphosphonates - zoledronic acid Vitamin supplements - Vit D
507
What significant oral condition may arise from taking some anti-resorptive drugs?
Medication related osteo necrosis of the jaw
508
Four ways in which MRONJ can be prevented
Patient education OHI Consider high fluoride toothpaste Make pt dentally fit before starting antiresorptives Remove risk factors where poss eg sharp denture flange Smoking cessation advice Non-invasive alternative treatment such as RCT
509
Management options for MRONJ
Monitoring Specific OHI for exposed bone Antiseptic MW Occasionally antibiotics Minimal surgical debridement in select cases Primary closure where possible Remove traumatic causes Consult GMP to check if drug modification or replacement is appropriate Symptomatic relief Topical analgesics Radiographs to establish differential diagnosis Referral to secondary care
510
Top to bottom
Frontal sinus Sphenoid sinus Ethmoidal air cells Maxillary sinus
511
Which of the sinuses are most well formed at birth?
Maxillary and ethmoid Formation occurs withing 3-4th foetal month
512
Functions of paranasal sinuses
Resonance to the voice Reserve chambers for warming inspired air Reduce the weight of the skull
513
Which of the sinuses is usually the largest?
Maxillary
514
Maxillary sinus description
Pyramid shaped cavity within the body of each maxilla Volume approx 15ml in average adult 37mmH 27mmW 35mmAP on average
515
Ostium of the maxillary sinus
Located medially near the roof of the maxillary sinus Drains into middle meatus Approx 4mm diameter Lined with mucosa Can become narrowed or blocked during episodes of inflammation or disease
516
What is generally found on the posterior wall of the maxillary sinus cavity?
The alveolar canals that transport the posterior superior alveolar vessels and nerves to the maxillary posterior teeth
517
Roots of maxillary molars and sometimes premolars may project into ____
The floor of the maxillary sinus The roots may perforate the bone so that only the mucosal lining of the sinus covers them
518
Epithelium of the sinuses
Pseudostratified ciliated columnar epithelium
519
What is the role of the cilia in the maxillary sinus
1. Mobilised trapped particulate matter and foreign material within the sinus 2. Move this material towards the ostia for elimination into the nasal cavity
520
Possible issues with maxillary sinus
OAC - acute OAF - chronic Root (or entire tooth) pushed into the sinus Sinusitis Benign lesion Malignant lesions
521
How is an OAC usually created?
On extraction of upper molar with roots projecting onto the floor of the maxillary sinus, a communication between the sinus and the mouth is created by either breaking the bone, or tearing the lining of the sinus
522
Why is an OAC a problem?
Bacteria from the mouth can now enter into the sinus, which can result in sinusitis and make other functions problematic
523
How do you diagnose an OAC?
Size of tooth Radiographic position of roots in relation to sinus Bone at trifurcation of roots Bubbling of blood Nose holding test Direct vission Good light and suction - echo Blunt probe
524
Which two ways that you would investigate whether an OAC has been created could potentially create an OAC if not careful?
Nose holding test Probing
525
OAF
Oro-antral communication that persists as an opening, and a sinus tract forms and become epithelialized Oro antral fistula
526
When might a pre-op assessment suggest likelihood of and OAC?
Roots of the tooth for XLA appear to by within the sinus or projecting onto lining of the sinus Very thin bone in the area
527
What is this?
OAC
528
What is this?
OAF
529
What is shown here?
OAF radiographic appearance
530
Management of OAC
Tell the patient If small or sinus lining intact - encourage clot, suture margins, possible antibiotic, post op instructions minimising pressure formation within sinuses and the mouth - avoid straws, balloons, singing, smoking, blowing nose If large or lining torn - close with buccal advancement flap, can refer urgently to OS for this
531
OACs <2mm prognosis
Usually heal with normal blood clot formation and routine mucosal healing
532
What type of flap is usually used to close an OAC?
Buccal advancement flap
533
Buccal advancement flap
Three sided flap with a crestal incision and two relieving incisions Flare base to ensure that there is bloody supply to all of the flap
534
Procedure of closing OAC with buccal advancement flap
Lift the flap of mucosa, sometimes need to chip away some buccal bone. This flap will not stretch easily across so you need to incise the periosteum of the flap (fresh blade) on the underside. This will make the flap loose enough to stretch it over the OAC. Be very careful when incising the periosteum not to cut the flap off. One initial suture. Usually resorbable sutures, but sometimes these dissolve too quickly. Then follow with more sutures for complete primary closure.
535
Patients with an OAF may complain of
* Problems with fluid consumption, fluids from nose * Nasal quality to speech and singing * Problems playing brass/wind instruments * Problems smoking or using a straw * Bad taste/odour/halitosis/pus discharge * Pain/sinusitis type symptoms
536
OAF management
The same as the OAC closure except first you must excise the epithelialized sinus tract, then perform buccal advancement flap. Sometimes an antral washout is also required - if chronic sinusitis and sinus is full of infection, this is flushed out and aspirated. This can cause reduction in sulcus depth.
537
Potential flap designs for OAC/ OAF closure
* Buccal advancement flap * Buccal fat pad with buccal advancement flap - if bigger OAF or OAC, two layer closure, very effective * Palatal flap - incredibly painful, leaves exposed bone on the palate * Bone graft/collagen membrane * Rotated tongue flap (historical)
538
Aetiology of fractured maxillary tuberosity
* Single standing molar * Unknown unerupted molar or wisdom tooth * Pathological gemination/concrescence * Extracting in wrong order (you should start posterior and move forward) * Inadequate alveolar support
539
Diagnosis of fractured maxillary tuberosity
* Noise * Movement noted both visually or with supporting fingers * More than one tooth movement * Tear in soft tissue of palate
540
Management of maxillary tuberosity fracture
if noticed early enough Reduce and stabilise * Orthodontic buccal arch wire with composite * Arch bar * Splints (lab made) If bone removed - dissect out and primary closure of wound
541
If maxillary tuberosity fracture is treated by splinting tooth must remember:
* Remove or treat pulp * Ensure it is out of occlusion * Consider antibiotics and antiseptics * Post-op instructions * Remove the tooth surgically 4-8 weeks later
542
Management of a root or tooth in the maxillary sinus
Confirm radiographically by OPT, occlusal or periapical (+/-CBCT) CBCT should be done on the day of retrieval as it can move around Decision on retrieval If in doubt or retrieval difficult - refer
543
How to manage?
You can leave this piece of root because if it has not torn the lining, it will not cause sinusitis or other problems and it will not move from there
544
Removal of root in the maxillary sinus process
Open fenestration with care Suction - efficient and narrow bore Small curettes Irrigation or ribbon gauze Close as for OAC Careful not to tear lining If this doesn't work - Caldwell-Luc approach (buccal window cut in bone) If unretrievable - refer to ENT for endoscopic retrieval
545
What must you remember when examining patients with maxillary discomfort?
- Close relationship of the sinuses and the posterior maxillary teeth - The aetiology of paranasal sinus inflammation and infection - Patients with sinusitis often present to the dentist first
546
Aetiology of Sinusitis
Mostly precipitated by the effects of a viral infection (debate over antibiotics) Inflammation and oedema Obstruction of ostia Trapping of debris within sinus cavity Mucociliary clearance patterns may be altered by allergens, inflammation, anatomic abnormalities Normal function further disrupted by cellular damage to mucosal lining, affecting ciliary function Build up in sinus and bacterial overgrowth
547
2 effects of sinus not being able to evacuate its contents efficiently
Build up of pressure Stagnation in sinuses - opportune situation for bacterial overgrowth of normal flora
548
Signs and symptoms of sinusitis
* Facial pain * Pressure * Congestion * Nasal obstruction * Paranasal drainage * Hyposmia * Fever * Headache * (dental pain) * Halitosis * Fatigue * Cough * Ear pain * Anaesthesia/paraesthesia over cheek
549
Dental causes that must be ruled out with similar symptoms to sinusitis
- PA abscess - Periodontal infection - Deep caries - Recent extraction socket - TMD - Neuralgia or atypical facial pain/chronic midfacial pain
550
Specific indicators of sinusitis, that indicate the symptoms do not have a dental cause
Discomfort on palpation infraorbitally A diffuse pain the maxillary teeth Equal sensitivity from percussion of multiple teeth in the same region Pain that worsens with head or facial movement (jump up and down, bend and stand up)
551
Treatment aims for patients with sinusitis
Treat presenting symptoms Reduce tissue oedema Reverse obstruction of the ostia
552
Sinusitis treatments
Decongestants to reduce mucosal oedema - ephedrine nasal drops 0.5% one drop up each nostril up to three times daily when required (max 7 days as will cause atrophy of sinus and nose lining) Humidified air also helpful (steam/menthol inhalations)
553
Why can't you use decongestants long term?
Will cause atrophy of the lining of the sinus and the nose
554
Antibiotics for sinusitis
Only to be used if symptomatic treatment not effective/symptoms worse AND Signs and symptoms point to bacterial sinusitis Amoxicillin 500mg 3x daily 7 days Doxycycline 100mg 1x daily 7 days (200mg loading dose) SDCEP guidance
555
Fungal infections of sinuses
Very rarely non-resolving sinusitis may be due to a fungal infection This can cause expansion of the bony walls by increased mucous secretion and fungal growth (this can happen with other types of infection too)
556
What types of trauma can cause sinusitis by violating the integrity of the bony cavity and sinus membrane?
Sinus wall fractures Orbital floor fractures RCT Extractions Implants/sinus lifts Deep perio treatment Nasal packing Nasogastric tubes Mechanical (nasal) intubation
557
What is a sinus lift?
Procedure used to reduce volume of the sinus to increase amount of bone available usually for implants
558
Benign sinus lesions
Polyps, papillomas, antral pseudocysts, mucoceles, mucous retention cysts, odontogenic cysts/tumours expanding into the sinus
559
Malignant lesions of the maxillary sinus
Primary tumours Local spread from adjacent sites
560
561
562
563
564
565
566
567
568
569
How much alveolar bone support should you have for a post?
At least half of post length into the root in bone
570