Ch 10 - Motor Neurons Flashcards

(59 cards)

1
Q

Freedforward refers to

A

the anticipatory use of sensory information to prepare for movement
(like using visual info when driving)

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2
Q

Feedback refers to

A

the use of sensory information during or after movement to make corrections either to ongoing movement or to future movements (propioceptive and visual information)

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3
Q

LMS reside in

A

brainstem and spinal cord

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4
Q

Total resistance to muscle stretch is determiend by

A

active contraction, titin, and weak actin-myosin bonds

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5
Q

Muscle tone is the

A

resistance to stretch in a resting muscle

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6
Q

What is used clinically to assess muscle tone

A

PROM

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7
Q

Normal resting muscle tone is provided by

A

titin and weak actin myosin bonds and low level tonic activation from descending motor input

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8
Q

Loss of sarcomeres is a

A

structural adaptation to the shortened position so that the muscle generates optimal force at the new resting length

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9
Q

If a mm is immobilized in a lengthened position it will

A

add new sarcomeres

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10
Q

Cocontraction refers to

A

simultaneous contraction of antagonist muscles

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11
Q

LMNs are the only neurons that convey signals to

A

extrafusal and intrafusal skeletal muscle fibers

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12
Q

Two types of LMNs

A

alpha and gamma

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13
Q

AMNs are MNs with

A

large cells bodies and large myelinated axons that reside in brainstem and spinal cord

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14
Q

Gamma motor neurons consists of

A

medium sized myelinated axons

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15
Q

Axons of AMNs project to

A

extrafusal skeletal muscle, branching into numerous terminals as they approach mm

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16
Q

Axons of GMNs project to

A

intrafusal fibers in the muscle spindle

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17
Q

H reflexes are

A

monosynaptic reflexes elicited by electrically stimulating a nerve

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18
Q

Purpose of H reflexes

A

quantify the level of alpha motor neuron facilitation of inhibition

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19
Q

Medial UMN tracts (4)

A

Reticulospinal
Medial vestibulospinal tracts
Lateral vestibulospinal tract
Medial corticospinal tract

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20
Q

Reticulospinal tract

A

Facilitates bilateral LMN innervating postural and gross limb movement of mm throughout the body

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21
Q

Medial vestibulospinal tracts

A

receives info about head mvmnt and position from vestibular apparatus

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22
Q

Lateral vestibulospinal tract

A

responds to gravity information from vestibular apparatus

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23
Q

Medial corticospinal tract

A

Has direct connection from cerebral cortex to the spinal cord

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24
Q

Fractionation is

A

the ability to activate individual muscles independently of other mm (mvmnt of hands)

25
Which is the most important pathway controlling goal-directed, voluntary movements
lateral corticospinal tract
26
Origin of corticospinal tracts
primary motor, premotor, and supplementary motor cortex
27
Signs of MN lesions
``` paresis, paralysis muscle atrophy involuntary mm contractions abnormal mm tone abnormal reflexes disturbances of mvmnt efficiency and speed impaired postural control ```
28
Paresis is the
partial loss of voluntary contraction
29
Muscle atrophy
loss of muscle bulk
30
Most severe atrophy is produced by
denervation of skeletal muscle
31
Flaccidity is the
lack of resistance to passive stretch
32
Disorders of LMNs
Trauma, infection, degenerative or vascular disorders, and tumors can LMNs
33
If LMN cell bodies and/or axons are destroyed then the affected muscles can undergo
loss of reflexes atrophy flaccid paralysis fibrillations
34
UMN lesions can produce several changes in mvmnt control including
paresis or paralysis loss of fractionation of mvmnt abnormal reflexes velocity dependent hypertonia
35
Paresis occurs in ___ lesions as a result of ___
UMN | inadequate facillitation of LMNs
36
3 most common abnormal reflexes in those with chronic SCIs are
muscle stretch hyperreflexia clonus clasp-knife response
37
Excessive mm contraction occurs when
spindles are stretched as a result of excessive firing of LMNs
38
Clonus is
involuntary, repeating, and rhythmic muscle contractions
39
Clasp-knife response occurs when
a paretic muscle is slowly and passively stretched and resistance drops at a specific point in the ROM
40
____ __ ____ elicit the clasp knife response
type II afferents
41
velocity dependent hypertonia limits
joint ROM, interferes with function, and may cause deformity
42
myoplasticity is
adaptive changes within a muscle in response to changes in neuromuscular activity level and to prolonged positioning
43
Spasticity is
neuromuscular overactivity secondary to UMN lesion
44
What is the primary cause of stroke spasticity
brainstem UMN overactivity
45
Measuring hypertonia
EMG | Ashworth scale
46
Spastic cerebral palsy
abnormal supraspinal influences, failure of normal neuronal selection, and consequent aberrant muscle development lead to movement dysfunction
47
Complete spinal cord injury
all descending neuronal control is lost below the level of the lesion
48
Incomplete spinal cord injury
function of some ascending and/or descending fibers is preserved within the spinal cord
49
Primary impairment with spastic cerebral palsy
paresis of agonist postural muscles
50
Continuous prolonged immobilization of skeletal mm results in
contracture from loss of sarcomeres
51
purpose of alpha gamma coactivation
maintain mm spindle sensitivity when extrafusal mm fibers contract
52
Phasic stretch reflex AKA
myotatic reflex muscle stretch reflex deep tendon reflex
53
reciprocal inhibition produces inhibition of the alpha motor neuron to what
antagonist muscle
54
What contributes to spinal control of walking
afferent inpur interneuorns reciprocal signals crossing dorsal columb
55
if LMN to mm is destroyed what sign
loss of reflexes and atrophy
56
abnormal cutaneous reflexes that occur after an UMN lesion include what
Babinski | muscle spasm
57
What limits movement in those who had stroke
paresis (dec agonist) cant fractionate abnormal timing of mm activation
58
Clonus is characterized by what
involuntary, repetitive, and rhythmic mm contractions in response to mm stretch
59
Decorticate rigidity occurs with what
lesions of superior midbrain