Ch 19 Peri Implant Health & Diseases Flashcards
(41 cards)
Dental implant
-a nonbiological device surgically inserted into the jawbone to replace a missing tooth and/or provide support for a prosthetic denture
Implant body
The “root” of the implant that is surgically placed into the living alveolar bone
Abutment
-titanium post that attaches to the implant body
-protrudes partially or fully through the gingival tissue
-supports the crown or denture
-biocompatible (not rejected) with the body
Peri implant issues
The soft tissue surrounding the dental implant
-similar in many ways to the periodontium of a natural tooth, but there are important differences
Implant to epithelial tissue interface
-the epithelium adapts to the titanium abutment post, creating a biological seal
-the biological seal functions as a barrier between the implant and the oral cavity
-the sulcular epithelium surrounds the implant abutment post surgery
Implant to connective tissue interface
-significantly different than that of a natural tooth
-implant surface lacks cementum
-gingival fibers and periodontal ligament cannot insert into the titanium surface
-periodontal pathogens can destroy bone faster along a dental implant than a natural tooth
Implant to bone interface
-osseointegration is the direct contact of bone with the implant surface
-major requirement for implant success
-osseointegration is successful if no mobility, absence of inflammation of tissues, no discomfort or pain when functioning, no increased bone loss or radiolucency
Tissue surrounding a dental implant
-Junctional epithelium: attaches to the implant surface (biologic seal)
-Connective tissue: run parallel to or encircle the implant
-PDL: no periodontal ligament
-Cementum: no cementum
-Alveolar bone: makes direct contact with the implant surface (osseointegration)
Peri Implant Health
-characterized by an absence of erythema, bleeding upon probing, swelling, and suppuration
-does not appear clinically different from clinically healthy periodontal tissues
-probing depths may be deeper compared to a healthy tooth site (due to orientation of CT tissue fibers
Peri implant disease
Peri implant tissue inflammation
-plaque deposits on implants
-can result in inflammation of soft tissues around the implant
-as disease progresses, partial or total loss of osseointegration occurs
2 forms of peri implant disease
-peri implant mucositis: (also known as peri implant gingivitis) plaque-induced gingivitis in tissues surrounding the implant
-peri implantitis- periodontitis in tissues surrounding osseointegrated implant, resulting in bone loss
Peri implant mucositis
-also called peri implant gingivitis
-plaque biofilm induced inflammation of the soft tissues
-no loss of supporting bones
-reversible if plaque is removed; if not, may progress to peri implantitis
-occurs in 80% of patients and 50% of implant sites
Peri implant mucositis requires close monitoring to notice signs of mucositis. What are signs?
Red tissues
Swelling
bleeding
Increased probing depths may
Peri implantitis
-periodontitis affecting soft and hard tissues surrounding a functioning osseointegrated dental implant
-plaque biofilm-induced inflammation
-progressive loss of alveolar bone
-may progress in a nonlinear and accelerating pattern
-prevalence ranges from 6.61-47%
Peri implantitis diagnosis requires
-Signs of inflammation
-Presence of bleeding and suppuration upon probing
-increased probing depths
-progressive bone loss as seen on radiographs
Peri implantitis begins where?
Begins at the coronal portion of the implant while the apical portion continues to be osseointegrated
-the implant does not become mobile until final stages of diseases
-mobile implants that show signs of loss of osseointegration should be removed
Radiographic signs of implant failure
-vertical destruction of crestal bone around implant
-bottom portion of implant remains osseointegrated
-may be wedge shaped defects along implant
Peri-implant disease etiology
Both forms share common etiology
-polymicrobial bacterial infection
-biomechanical factors
Bacterial infection
-perio disease in both the periodontium in natural teeth and the peri implant tissues of implants progresses in a similar fashion
-both require bacterial plaque biofilm and host inflammatory response
-perimucositis does not always progress to peri implantitis
-rate of destruction is more rapid in peri implant tissues than in a natural dentition
Risk factors for implant failure
-history of previous periodontal disease
-poor plaque biofilm control
-smoking
-residual cement
-biomechanical overload
Biomechanical overload
-collective forces placed on an implant
-influenced by a number of factors: position of the implant fixture, number of implants supporting prosthesis, occlusal force distribution among implants and remaining teeth
-implants lack the protective structure of periodontal ligaments
-implants are in direct contact with the bone
-forces placed in an implant are transmitted directly to the bone
-it is critical to minimize forces placed on an implant
Detection of a failing implant: clinical signs in soft tissue
-peri implant pocket
-bleeding
-suppuration
-possible swelling
-pain usually not present
Detection of a failing implant: changes to bone support
-mobility is best indicator of implant failure
-should not be mobile if healthy
-may indicate presence of loose abutment or rupture of cement seal
-severe mobility associated with pain may indicate a fracture of the implant
Radiographic signs of failure
-vertical destruction of crestal bone around implant
-bottom portion of implant may remain osseointegrated
-radiolucency indicates bone loss adjacent to implant