ch 19 pulmonary edema Flashcards

(33 cards)

1
Q

what is cardiogenic pulmonary edema classified as

A

restrictive lung disease

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2
Q

what is cardiogenic pulmonary edema also called

A

hydrostatic pulmonary edema

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3
Q

what is it essentially caused by

A

left side heart failure or fluid overload

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4
Q

what is pulmonary edema

A

excessive amount of fluid in the lung tissue or alveoli due to an increase in pulmonary capillary pressure resulting from abnormal left heart function

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5
Q

what is the etiology of cardiogenic pulmonary edema

A

left heart failure (ex. coronary artery disease)
aortic stenosis
mitral valve stenosis
systemic hypertension
fluid overload
decrease in osmotic pressure (pressure in blood to keep blood in inter vascular space - protein - albumin level)

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6
Q

what does the etiology result in

A

backup of fluid from the heart into the pulmonary capillaries which becomes engorged

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7
Q

what happens when pulmonary capillaries become engorged

A

fluid leaks into interstitial space and then into the alveoli

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8
Q

what is the swan ganz catheter

A

it measures the back pressure from the pulmonary veins - done from the carotid vein or subclavian vein and into right atrium, tricuspid, right ventricle

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9
Q

is it possible to measure the pressure from the left heart

A

no

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10
Q

is it possible to measure from the right heart

A

yes

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11
Q

what carries the swan ganz

A

the right ventricle into the pulmonary artery

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12
Q

where does the swan ganz measure pressure and it should be no more than what

A

5-8 and only in front of the catheter is where it is measured

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13
Q

how do they measure the progress of the treatment of the pulmonary edema

A

swan ganz catheter

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14
Q

how can you treat pulmonary edema

A

ionotropic - make left side work harder (digoxin)

lasics - get the excess fluid out of the body like a diuretic

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15
Q

what is atrial fibrillation

A
atopic phoxi (spelling lol) 
350-600 / min
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16
Q

what is atrial fibrillation caused by

A

congestive heart of the left ventrical, academia, alkalemia, electrical imbalances

17
Q

vital signs on admission

A
increased RR 22 
pH 7.56 
co2 28
hco3 20
pao2 51
spo2 70%
respiratory alkalemia - caused by hypoxemia
18
Q

patient inspection

A
distended neck veins
frequent cough - pink frothy sputum
distended abdomen
pitting edema 
blue lips
19
Q

xray

A

faint opacities in lower lobes bilaterally

heart was enlarged (left ventrical)

20
Q

treatment

A

oxygen
intravenous digitalis - dobutamine makes heart beat faster
furosemide - laxis takes liquid out of body to make you pee

21
Q

digitalis

A
  • inotrophic
  • increases myocardial force of contraction
  • increases stroke volume
  • antiarrhythmic used to treat atrail flutter and fibrillation
22
Q

dobutamine

A

increase myocardial contraction and stroke volume without increasing systemic vascular resistance (doesn’t increase blood pressure)
beta one

23
Q

how does furosemide work

A

causes diuresis by inhibiting reabsorption of sodium, also causes loss of chlorine, and potassium

24
Q

what abg can furosemide cause

A

metabolic alkalosis

25
auscultation
inspiratory crackles expiratory wheezes over lower lobes
26
example of shunt in this disease
alveoli filled with fluid
27
venous admixture
good lung mixing with bad lung leaded to lower pao2
28
pulmonary mechanics what happens to lung volumes
all lung volumes get smaller
29
what needs to be resupplied for lung volumes to go back to normal
surfactant
30
forced vital capacity
flow would unaffected but forced vital capacity would be smaller (middle line in picture)
31
lung compliance?
decreased compliance
32
WOB ?
increased work of breath
33
what is the gold standard treatment for cardiogenic pulmonary edema
cpap mask