Ch. 46 Flashcards

(49 cards)

1
Q

GERD stands for

A

gastroesophageal reflux disease

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2
Q

most common upper GI disorder in the US

A

GERD

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3
Q

__ % of adults in North America have reflux disorder

A

18-28%

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4
Q

GERD occurs as a result of

A

backward flow of the stomach contents into esophagus

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5
Q

causes of GERD

A
  • no single causative agent
  • NG tube
  • genetic connection: fam hx
  • lifestyle: alc, caffeine, smoking
  • Barrett’s epithelium
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6
Q

Barret’s esophagus

A
  • With Barretts esophagus, body substitutes Barrett epithelium instead of normal squamous cell
  • New tissue is more resistant to acid, but is premalignant, increased risk for esophageal cancer
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7
Q

normal healing of GERD (type of epithelium)

A

Normal healing of GERD with squamous cell epithelium

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8
Q

GERD prevention

A
  • healthy eating habits
  • limit fried, fatty, spicy foods and caffeine
  • sit upright for one hour after eating
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9
Q

GERD assessment: history (sx)

A
  • heartburn
  • morning hoarseness
  • coughing or wheezing at night
  • when do they experience these symptoms? usually report 1-2 hrs after meal
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10
Q

GERD assessment: s/sx

A
  • may be asymptomatic at first
  • dyspepsia, dysphagia
  • auscultate lungs for crackles (should have clear sounds with GERD, listen for crackles to ensure food is not backing up into the lungs)
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11
Q

GERD assessment: psychosocial

A
  • preference of what patient likes to eat
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12
Q

GERD assessment: diagnostics

A
  • upper endoscopy (UGD) (conscious sedation used, need vs monitor, someone to drive home; scope into esophagus; more invasive but confirmatory dx)
  • ambulatory esophageal pH monitoring: 24 hr reading of the pH levels (how acidic the esophagus is; no sedation)
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13
Q

priority patient problems of GERD

A
  • potential for compromised nutrition status due to dietary selection
  • acute pain due to reflux of gastric contents
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14
Q

GERD interventions: nutrition

A
  • balanced nutrition
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15
Q

GERD interventions: for minimizing pain

A

non-surgical mangement:
- lifestyle changes: no caffeine, alc, smoking, fried/spicy foods
- drug therapy
- endoscopic therapies

surgical management

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16
Q

Acid reflux diet: foods to avoid

A
  • coffee
  • wine
  • fast food
  • soft drinks
  • tomatoes (acidic)
  • dairy
  • spices
  • peppermint
  • citrus (oranges, grapefruit)
  • chocolate
  • garlic
  • onion
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17
Q

Acid reflux diet: foods that are okay to eat

A
  • ginger
  • leafy greens
  • brown rice
  • coconut
  • banana
  • pear
  • apple
  • avocado
  • fennel
  • celery
  • berries
  • melon
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18
Q

GERD care coordination

A
  • make appropriate dietary selections
  • adhere to drug therapy
  • teach signs of esophageal restriction and Barrett esophagus
  • RDN
  • local support groups
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19
Q

GERD meds include (classes)

A
  • proton pump inhibitors
  • H2 receptor blockers
  • antacids
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20
Q

GERD meds: proton pump inhibitors

A

the “-prazole” meds
- omeprazole
- pantoprazole
- lansoprazole

21
Q

GERD meds: H2 receptor blockers

A

end in “-tidine”
- rantidine
- famotidine
- cemetidine

22
Q

GERD meds: antacids

A
  • maalox and mylanta
23
Q

antacids

A
  • increase gastric pH by neutralizing acid
  • aluminum base =
  • magnesium base =
  • take 1-3 hours AFTER meals
  • taken PRN (as needed)
24
Q

histamine-receptor blocks (H2R blockers)

A
  • block action of histamine on H2 receptors
  • decreases HCl acid secretion
  • decreases conversion of pepsinogen to pepsin
  • increase ulcer healing
  • taken before meals
25
proton pump inhibitors (PPIs)
- main treatment for GERD - block HCl acid secretions - promote ulcer healing - DO NOT CRUSH OR CHEW (PO) - must be given IV for short-term use (usually given PO) - promote rapid healing, but reoccurrence common with cessation - can interfere with calcium absorption - taken in morning, empty stomach - maintenance med; pretty much on it forever
26
main treatment for GERD
proton pump inhibitors (PPIs)
27
GERD evaluation
- adhere to appropriate diet, medication therapy, and lifestyle modifications - experience minimized or absence of pain
28
hiatal hernia
- Protrusion of the stomach through the esophageal hiatus of the diaphragm into the chest
29
hiatial hernia assessment: sx
many people are asymptomatic; others have sx similar to GERD - heartburn regurgitation - dysphagia sx often increase after meal or when lying supine
30
hiatal hernia assessment: diagnostics
- barium swallow study with fluoroscopy: barium illuminates the hernia - EGD - high resolution manometry with esophageal pressure topography
31
hiatal hernia non-surgical interventions
- drug therapy - nutrition therapy: no spicy, greasy foods - lifestyle changes: caffeine, alc, smoking
32
hiatal hernia surgical interventions
- LNF- laproscopic nissen fundoplication (sm incision in abdomen; monitor s/sx of infection) - may need to lose weight before surgery
33
hiatal hernia care coordination
- nutrition modifications - stool softeners or bulk laxatives (more so post-op) - daily incisional inspection - signs to report to the HCP - avoid sick people (don't want pt to cough bc of abdominal incision)
34
diarrhea is caused by
- pathogens: infections; gastroenteritis - medical conditions: inflammatory GI diseases - food intolerance: lactose, gluten
35
diarrhea sx
- frequent liquid stool - hyperactive BS - abdominal tenderness, distention
36
diarrhea diagnostics
- stool culture (for patients with suspected infectious disease, ie C Diff)
37
diarrhea collaborative care (goals of care)
- stop sx - hydration: worried about hypovolemia - treat underlying cause (infectious: ABT; anti-inflammatories for inflammatory d/o)
38
acute infectious diarrhea: assessment
- s/sx of dehydration - skin turgor - tachycardia - dry mucous membranes - low BP (hypotension) (mostly older population)
39
acute infectious diarrhea: nursing diagnoses
- Deficit fluid volume r/t excessive fluid loss - Decrease CO r/t decreased plasma volume - Impaired oral mucous membranes r/t inadequate oral secretions - Risk for falls r/t orthostatic hypotension - Confusion (acute or chronic) r/t neuro changes (d/t electrolyte changes from being dehydrated)
40
clostridium difficile
aka c. diff - bacteria that causes severe diarrhea - commonly starts 4-9 days after beginning antibiotics (s/e of ABT)
41
c. diff: dx tests
- stool testing (culture)
42
c. diff: treatment
drug therapy - vancomycin PO - metronidazole
43
c. diff: interventions
- private room/contact isolations - contact precautions (gloves & gown) - hand washing with soap and water
44
constipation
- Stool moves slowly through GI tract - Many factors can cause -Muscles used to move bowels are not coordinated can be caused by: - use of opioids - post-op/anesthesia - sedentary lifestyle - diet: not enough fiber/fluids - taking iron supplements
45
constipation: s/sx
- hard stools - straining with BM - sense of rectal fullness or blockage - incomplete evacuation
46
constipation: complications
- hemorrhoids (from straining) - bowel obstruction
47
constipation: diagnostics
- barium enema - sigmoidoscopy/ colonoscopy
48
constipation: interventions
- nutrition and lifestyle therapy - not being sedentary/exercising - increasing fluid intake - increasing fiber intake/ high fiber diet - drug therapy
49
constipation: drug therapy
- bulk-forming: metamucil - stool softener: docusate - stimulants: senna - osmotic solutions: miralax - saline laxatives: milk of magnesia (MOM) - lubricants: fleets (all oral agents; except fleets) - try high fiber diet first, then PO meds, then fleet enema or suppository as the last resorts