chap 1 principle and concepts Flashcards

1
Q

New pain that is often severe, continuous, and perhaps disabling. Tend to be irritable

A

Acute pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Aggravating, not as intense, has been experienced before

A

Chronic pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Peripheral sensitization of nocireceptors

A

AKA primary hyperalgesia. when tissue has been damaged, substances are released leading to inflammation, resulting localized pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Central sensitization

A

AKA secondary hyperalgesia: more central process involving Spinal cord and brain, manifests itself as widespread hypersensitivity to such physical mental and emotional stressors

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Acute condition (how many days)

A

7-10days

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

subacute conditions

A

10 days to 7 weeks

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Chronic conditions

A

longer than 7 weeks

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

cramping, dull, aching

A

muscle pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

dull aching

A

ligament, joint capsule

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

sharp shooting

A

Nerve root

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Sharp bright, lightening like

A

Nerve

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Burning, pressurelike, stinging, aching

A

sympathetic nerve

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Deep nagging dull

A

Bone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Sharp, severe, intolerable

A

fracture

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Throbbing, diffuse

A

Vasculature

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Locking (joint) vs. Pseudolocking

A

Locking: joint cannot be fully extended i.e meniscal tear in the knee
Pseudolocking: does not extend one time, does extend next time i.e. loose body

17
Q

spasm locking/ giving way

A

Spasm locking: can no be put through a full ROM because mm spasm or because mvmt was too fast

Giving way: by reflex inhibition or weakness of mm due to anticipating pain or instability

18
Q

Laxity vs hypermobility

A

Laxity: excessive ROM but can control movement and no pathology

Hypermobility: has pathological component, can not control at end range, instability of joint

19
Q

Static flexibility and dynamic flexibility

A

Static flexibility: ROM available

Dynamic flexibility: stiffness, ease of movement

20
Q

translational instability vs anatomical instability

A

translational instability: AKA pathological or mechanical instability, loss of control of the small arthrokinematic joint movement (spin, slide, roll, translation)

Anatomical instability: AKA clinical or gross instability, mechanical instability or pathological hypermobility, excessive or gross physiological movement in the joint where patient become apprehensive a the end of ROM because of subluxation or dislocation

21
Q

Functional instability

A

either or both type, arthrokinematic or osteokinematic

22
Q

Voluntary instability vs involuntary instability

A

Voluntary instability: initiated by mm contraction

Involuntary instability: initiated by positioning

23
Q

circle concept of instability

A

one side joint dislocation/injury affect the other side

24
Q

myotome, dermatome, sclerotome

A

myotome: mm supplied by a single nerve root
Dermatome: area of skin “””
sclerotome: area of bone or fascia “”””

25
Radicular or radicular pain
form of referred pain, sharp shooting pain felt in a dermatome, myotome, sclerotome due to direct involvement or damage to spinal nerve or nerve root
26
Radiculopathy
radiating paresthesia, numbness or weakness but not pain
27
myelopathy
is a neurogenic disorder involving the spinal cord or brain and resulting an upper motor neuron lesion (can affect both upper and lower limb)
28
myofascial hypomobility
results from adaptive shortening or hypertonicity of the muscles or from posttraumatic adhesions or scarring
29
pericapsular hypomobility
a capsular or ligamentous origin may result from adhesions, scarring, arthritis, arthrosis, fibrosis, or tissue adaptation
30
Pathomechanical hypomobility
as a result of joint trauma (micro or macro) leading to restriction in one or more directions
31
abnormal end feel: early mm spasm, late mm spasm
early mm spasm: protective spasm from injury | late mm spasm: due to instability or pain
32
Abnormal end feel: hard capsular/soft capsular
``` hard capsular: a thicker stretching quality to it (such as frozen shoulder) Soft capsular (boggy): edema, synovitis ```
33
abnormal end feel: Bone to bone/empty/springy block
Bone to bone: osteophytic formation Empty: acute subacromial bursitis, can't achieve due to pain Springy block: meniscus tear
34
postural or tonic muscles
responsible for maintaining upright posture, tendency to become tight and hypertonic with pathology Pec, psoas, gastroc/soleus
35
phasic muscles
tend to become weak and inhibited with pathology | Rhomboids, abs, tib ant
36
SOAP means
Subjective Objective Assessment Plan