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chap 6 symp Flashcards

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1
Q

Raynaud phenomenon

A

Scleroderma: Episodic vasoconstriction of the a’s of the extremities.

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2
Q

Synovitis

A

Sjogren syndrome

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3
Q

Obliterative phlebitis

A

IgG4 Related Disease: inflamm results in pemanent closure of vessel

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4
Q

increased incidence of tumors

A

Ataxia-telangiectasia synd

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5
Q

women aged 50-60

A

Sjogren syndrome, Scleroderma

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6
Q

Coronary a. dz

A

SLE: can lead to angina or MI.

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7
Q

adverse side effects of HAART

A
  • Lipoatrophy (loss of facial fat)
  • Lipoaccumulation (excess fat deposition centrally)
  • Elevated lipids
  • Insulin resistance
  • Peripheral neuropathy
  • Premature cardiovascular kidney and liver disease
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8
Q

Dryness of the nasal mucosa and epistaxis.

A

Sjogren syndrome

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9
Q

Peripheral neuropathy

A

Sjogren syndrome

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10
Q

edema, erythema, scaliness, follicular plugging, and skin atrophy surrounded by an elevated erythematous border; scarring

A

SLE: Chronic discoid lupus erythematosus

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11
Q

Wide spread damage to small blood vessels

A

Scleroderma

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12
Q

increased PTT

A

SLE

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13
Q

Dysphagia

A

Scleroderma: from esophageal fibrosis that leads to hypomotility

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14
Q

prone to developing AI dis’s

A

Brutons Agammaglobinemia, Isolated IgA deficeincy

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15
Q

Progressive interstitial and perivascular fibrosis in the skin(early) and multiple organs like the GI tract, kidneys, heart, muscles, and lungs later

A

Scleroderma

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16
Q

Renal:
• Mild proteinuria
• Malignant hypertension

A

Scleroderma

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17
Q

middle-aged and older men

A

IgG4 Related Disease

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18
Q

Neuropsychiatric symptoms

A

SLE Due to non-inflammatory occlusion of small vessels

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19
Q

increased serum IgG4

A

IgG4 Related Disease

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20
Q

Diffuse pulmonary fibrosis

A

Sjogren syndrome

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21
Q

Keratoconjunctivitis

A

Sjogren syndrome: blurring of vision, burning, and itching.

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22
Q

defect in ability of helper T cells to deliver activating signals to B cells and macrophages.

A

Hyper IgM synd

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23
Q

susceptable to recurrent pyogenic infections. and to Pneumocystis jiroveci pneumonia.

A

Hyper IgM synd

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24
Q

recurrent infection that leads to early death.

A

Wiskott-Aldrich synd

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25
Respiratory difficulty
Scleroderma
26
make IgM antibodies can’t produce IgG, IgA, and IgE antibodies.
Hyper IgM synd
27
Inflammation of the serosal membranes
SLE - acute: fibrinous exudate - later: thickened
28
eczema
Wiskott-Aldrich synd
29
loss of T cell imunity
DiGeorge Synd
30
skin rash widespread, superficial, and non-scarring
SLE: Subacute cutaneous lupus erythematosus
31
Splenomegaly, capsular thickening, and follicular hyperplasia.
SLE
32
congenital defects of <3 and great valves
DiGeorge Synd
33
Valvular (libman-sacks) endocarditis
SLE - Uncommon now due to the use of steroids. - Nonbacterial verrucous endocarditis.
34
immunodeficiency
Ataxia-telangiectasia synd
35
telangiectasia , , and.
Ataxia-telangiectasia synd (vascular malformations)
36
* Calcinosis * Raynaud phenomenon * Esophageal dysmotility * Sclerodactyly * Telangiectasia
CREST Synd
37
butterfly malar rash
SLE: on cheeks and forehead; may have on ext's and trunk; also on uninvolved skin
38
false + for syphilis
SLE: d/t antiphospholipids abs
39
Recurrent bronchitis and pneumonitis
Sjogren syndrome
40
hypocalcemia and tetany
DiGeorge Synd
41
Increased number of infections
SLE: d/t immune system dysfunction or tx with immunosuppressive drugs.
42
Parotid gland enlargement
Sjogren syndrome
43
normal levels B cells, but low Ig's
CVID
44
esp involves skin, joints, kidney, and serosal membranes.
SLE
45
have inherited dificicnces of early compliment components
SLE --> impaired removal fo immune complexes
46
low levels of both serum and secretory IgA.
Isolated IgA deficiency
47
Chronic interstitial fibrosis and secondary pulmonary HTN.
SLE
48
mixture of the features of SLE, systemic sclerosis, and polymyositis.
Mixed Connective Tissue Disease
49
B cells are absent or markedly decreased in the circulation
Brutons Agammaglobinemia
50
susceptable to recurent infections: • Bacterial infections of the respiratory tract: Haemophilus influenzae, Streptococcus pneumoniae, or Staphylococcus aureus. • Viruses in the bloodstream or mucosal secretions: Enteroviruses, such as echovirus, poliovirus, and coxsackievirus. • Giardia lamblia (intestinal protozoan).
Brutons Agammaglobinemia
51
skin thickening
Scleroderma
52
Thrombocytopenia
Wiskott-Aldrich synd
53
assoc w autoimmune hemolytic anemia, thrombocytopenia, and neutropenia.
Hyper IgM synd
54
storiform fibrosis
IgG4 Related Disease (whirled like storage pattern)
55
Affects Hispanic and African American women more than white men
SLE
56
Infants: Extensive diaper rash.
SCID
57
Infants: Failure to thrive.
SCID
58
Renal: Hematuria, red cell casts, proteinuria, and sometimes present as classic nephrotic syndrome.
SLE
59
Xerostomia
Sjogren syndrome: difficultly in swallowing solid foods, decrease in taste, cracks and fissures in the mouth, dryness of the buccal mucosa.
60
Myocarditis
SLE:(inflammation of the myocardium) can lead to tachycardia and EKG abnormalities.
61
Abnormal gait
Ataxia-telangiectasia synd
62
T cell–mediated reactions are normal
Brutons Agammaglobinemia
63
has anas and antiphospholipids abs
SLE
64
Myocardial fibrosis
Scleroderma: leading to arrhythmias or cardiac failure
65
susceptible to recurrent, severe infections: | •Candida albicans, Pneumocystis jiroveci, Pseudomonas, CMV, varicella, and many bac
SCID
66
Pleuritic and pleural effusions
SLE
67
recurrent sinopulmonary infections and diarrhea
Isolated IgA deficiency
68
neurologic deficits
Ataxia-telangiectasia synd
69
Infants have oral candidiasis
SCID: (prominant thrush)
70
abn mouth, ears, face
DiGeorge Synd
71
acute necrotizing vasculitis
SLE
72
Plasma cells are absent throughout the body
Brutons Agammaglobinemia, CVID
73
Renal: renal tubular acidosis, uricosuria, phosphaturia
Sjogren syndrome
74
Non-erosive synovitis w lttle deformity
SLE
75
immune destruction of the lacrimal and salivary glands by lymphocytes.
Sjogren syndrome
76
Valve prbs
SLE(mostly to mitral and aortic) can lead to stenosis and/or regurgitation.
77
Serum levels of all classes of immunoglobulins are depressed.
Brutons Agammaglobinemia, CVID

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