CHAPTER 16: MANAGEMENT OF PATIENT WITH NEUROLOGIC TRAUMA Flashcards

(44 cards)

1
Q

describes an injury that is the result of an external
force and is of sufficient magnitude to interfere
with daily life and prompts the seeking of
treatment.

A

Traumatic Brain Injury (TBI) or craniocerebral trauma

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2
Q

Classifications of Head Injury

A

Primary Injury
Secondary Injury

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3
Q

consequence of direct contact to the head/brain
during the instant of initial injury, causing extracranial
focal injuries (e.g., contusions, lacerations, external
hematomas, and skull fractures)

A

Primary Injury

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4
Q

evolves over the ensuing hours and days after the
initial injury and results from inadequate delivery of
nutrients and oxygen to the cells

A

Secondary Injury

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5
Q

Minor injury but the scalp bleeds profusely because blood vessels constricts poorly; May result in an abrasion, contusion, laceration

A

Scalp Injury

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6
Q

Break in the skull due to forceful trauma; may occur with or without brain damage.

A

Skull Fracture

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7
Q

Types of Skull Fracture

A

Simple Linear
Comminuted
Depressed
Basilar

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8
Q

Skull Fracture: Break in the continuity of the bone

A

Simple Linear

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9
Q

Skull Fracture: Splintered on multiple fracture line

A

Comminuted

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10
Q

Skull Fracture: Bone fragments are embedded into brain tissue

A

Depressed

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11
Q

Skull Fracture: Fracture at the base of the skull

A

Basilar

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12
Q

What are the clinical manifestations of basilar fracture?

A

Battle’s Sign - mastoid bruising (ilalim ng ears)
Racoon’s Eye - ecchymosis in the eyes
Otorrhea - CSF escape in the ears
Rhinorrhea - CSF escape in the nose

Rationale: Basilar fracture tends to traverse to the paranasal sinus thus produce hemorrhage in the nose, pharynx & ears

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13
Q

Mastoid bruising

A

Battle’s Sign

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14
Q

Ecchymosis in the eyes

A

Racoon’s eye

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15
Q

CSF escape in the ears

A

Otorrhea

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16
Q

CSF escape in the nose

A

Rhinorrhea

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17
Q

Medical Management for non-depressed

A

Close monitoring for 24 hrs; mgh; no surgery

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18
Q

Medical Management for depressed

A

require surgery with elevation of the skull and débridement, usually within 24 hours of injury.

19
Q

Mechanisms of Brain Injury

A

Closed (Blunt) Injury
Open Brain Injury

20
Q

head accelerates & then rapidly decelerates or collide with another object & brain tissue is damaged but there is no opening through skull or dura

A

Closed (Blunt) Injury

21
Q

an object penetrates the skull, enters the brain, damages the soft brain tissue or when blunt trauma is severe that it opens the scalp, skull and dura to expose the brain

A

Open Brain Injury

22
Q

Type of Brain Injury: the brain is BRUISED and damaged in a specific area because of severe acceleration–deceleration force or blunt trauma

23
Q

Type of Brain Injury: Hematomas are collections of blood in the brain that may be epidural (above the dura), subdural (below the dura), or intracerebral (within the brain)

A

Intracranial Hemorrhage

24
Q

Collection of blood in the epidural (extradural) space between the skull and the dura mater.

A

Epidural Hematoma

25
a collection of blood between the dura and the brain, a space normally occupied by a thin cushion of fluid
Subdural Hematoma
26
Medical Management of Intracerebral Hemorrhage and Hematoma
➢control of ICP ➢careful administration of fluids, electrolytes, ➢antihypertensive medications
27
Surgical Management of Intracerebral Hemorrhage and Hematoma
> craniotomy or craniectomy permits removal of the blood clot and control of hemorrhage
28
temporary loss of neurologic function with no apparent structural damage of the brain. ; mechanism of injury is usually blunt trauma from an acceleration–deceleration force, a direct blow, or a blast injury
Concussion
29
results from widespread shearing (tearing) and rotational forces that produce damage throughout the brain—to axons in the cerebral hemispheres, corpus callosum, and brainstem.
Diffuse Axonal Injury
30
3 Cardinal Signs of Brain Death
Coma the absence of brainstem reflexes Apnea
31
Test to Confirm Brain Death
cerebral blood flow studies electroencephalogram (EEG) transcranial Doppler brainstem auditory-evoked potential,
32
A tool for assessing a patient's response to stimuli
Glasgow Coma Scale Scores range from 3 (deep coma) to 15 (normal)
33
What does the GCS contain?
Eye-opening response Best verbal response Best motor response
34
What is used to assess LOC at regular intervals?
GCS Rationale: changes in the LOC precede all other changes in vital and neurologic signs.
35
What is monitored at frequent intervals to assess the intracranial status?
Vital Signs
36
Vital Signs: Signs of increasing ICP include:
✓ slowing of the heart rate (bradycardia), ✓ increasing systolic blood pressure ✓ widening pulse pressure (Cushing reflex) ✓ respirations become rapid ✓ blood pressure may decrease ✓ pulse slows further ✓ temperature is maintained at less than 38°C ✓ Tachycardia and arterial hypotension may indicate that bleeding is occurring elsewhere in the body
37
Which areas do SCI commonly occur?
C5, C6, C7, T12, L1 Rationale: This area has a greater range of mobility in the vertebral column in this areas
38
Causes of SCI
MVA Gun shots Falls Sports injuries
39
SCI Classification: result of the initial insult or trauma, usually permanent
Primary Injury
40
SCI Classification: resulting from SCI include edema and hemorrhage
Secondary injury
41
TYPE of SCI: ➢signifies loss of both sensory and voluntary motor communication from the brain to the periphery, resulting in paraplegia or tetraplegia
Complete Spinal Cord Lesion
42
TYPE OF SCI: ➢denotes that the ability of the spinal cord to relay messages to and from the brain is not completely absent
Incomplete Spinal Cord Lesion
43
➢complete injury in the thoracic area causes complete paralysis in the legs but the arms can still function
paraplegia
44
➢paralysis of all four extremities; formerly called quadriplegia
tetraplegia/quadriplegia