Chapter 2 Aphasia Flashcards

(60 cards)

1
Q

Adult language impairment include

A

 Language Development Through the Lifespan
 Aphasia
 Right Hemisphere Damage
 Traumatic Brain Injury
 Dementia

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2
Q

Language impairment through life span

A

Unless there is neuropathy, adults continue to
refine communication skills.
 Use
 *Adults are skilled conversationalists
 *Narratives improve until the seventies
 Content
 *Some words fade and others are added
 * Deficits in accuracy and the speed of word
retrieval/naming
 Form
 *Continue to acquire some aspects of syntax
 *Complex sentence construction declines with
advanced ag

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3
Q

Aphasia

A

*Literally “without language”
 Affects over 1 million people in the U.S
 My affect listening, speaking, reading, writing
 Range In Severity
 *Related to cause, location/extent/age of
brain injury, age/general health of patient
 Patterns of behavior can be used to
categorize by type/symptom

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4
Q

Expressive language Aphasia
Impairments characteristics

A

*Reduced vocabulary
 *Omission/addition of words
 *Stereotypic Speech
 *Delayed or reduced output of speech
 *Hyperfluent speech
 * Word Substitutions

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5
Q

Language Comprehension deficits Aphasia

A

*Impaired interpretation of linguistic deficits

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6
Q

Concomitant Deficts

A

*Hemiparesis *Agraphia
 *Hemiplegia *Alexia
 *Hemisensory *Anomia
 Impairment *Jargon
 *Hemianopsia *Neologism
 *Dysphagia *Paraphasia
 *Agrammatism *Verbal Stereotype

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7
Q

Sensory Involvement Aphasia

A

Touch, Vision and Auditory Comprehension
can be affected after a CVA.
 Hemisensory Impairment-inability to sense
pain or touch on one side of the body
 Loss of Vision can also occur after a CVA
 Damage to either the optic nerve/tract or
damage to the Occipital Lobe is called
HEMIANOPSIA.

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8
Q

 Damage from a CVA

A

HEARING ACUITY is NOT affected by a CVA.
 Damage from a CVA occurs in the TEMPORAL
LOBE OF THE CORTEX which serves to
interpret auditory signals and make sense out
of them, therefore, it’s Auditory
Comprehension that is affected.

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9
Q

DYSPHAGIA-

A

Swallowing Disorder

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10
Q

Cognition Aphasia

A

The cognitive function of the two halves of
the brain is responsible for integrating,
processing and computing information.
 Could affect behaviors such as problem
solving, memory, judgment, reasoning,
perception and imagination.
 fMRI (functional magnetic resonance imaging)
 PET scan (Position Emission Tomography)

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11
Q

ANOMIA

A

-Difficulty in naming things, objects
and people.

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12
Q

 PARAPHASIA

A

word substitution problem
 *Phonemic Paraphasia-word substitution
based on phonemic similarity (ie “tar” for
“car” or “hiss” for “kiss”)
 *Verbal paraphasia-substituted words have
similar meanings similar to correct words (ie
A woman referred to her husband as “wife”)

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13
Q

*NEOLOGISM

A

creation of a new word often
meaningless. (ie “ponty” for “chair”)

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14
Q

AGRAMMATISM-

A

-omission of certain grammatical elements

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15
Q

JARGON

A

-relatively fluent but irrelevant or meaningless speech

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16
Q

VERBAL STEREOYPES-

A

expression is repeated over and over.

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17
Q

AGRAPHIA

A

writing problems

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18
Q

ALEXIA

A

-reading problems

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19
Q

AGNOSIA

A

difficulty in understanding sensory information
 *Auditory Verbal Agnosia
 *Visual Agnosia

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20
Q

Causes of Aphasia
CEREBRAL VASCULAR ACCIDENT (CVA)

A

ISCHEMIC- are caused by a blocked or
interrupted blood supply to the brain.
 –Cerebral arteriosclerosis(hardening of
the arteries.
 –Embolism
 –Thrombosis

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21
Q

Causes of Aphasia
HEMORRHAGIC

A

-are caused by bleeding in
the brain due to ruptured vessels
 –Intercerebral (within the brain)
 –Extracerebral (within the meninges)
 *Aneurysm
 *Arteriovenous Malformations

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22
Q

Causes of Aphasia
TRANSIENT ISCHEMIC ATTACK (TIA)

A
  • mini stroke
     * warning sign that the person is at
    increased risk of a future stroke
     * symptoms usually disappear completely
    within 24 hours
     * 30% of people have damage evident on
    sensitive brain imaging techniques such as
    MRI after a TIA.
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23
Q

Causes of Aphasia
Lesion

A

 Lesion-an injury that leaves an area of
cortical tissue incapable of functioning in a
normal way.
 How can you get brain damage
 1. CLOSED HEAD INJURY
 2. OPEN HEAD INJURY
 3. CONTRECOUP INJURY

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24
Q

Causes of Aphasia
NEOPLASM-

A

NEOPLASM-tumor
 *Intercranial (within the brain)
 *Metastic (grown elsewhere but migrated and
attached to the brain tissue and still growing)

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25
Stroke causes
Most victims of a stroke are middle age and beyond  Risk of stroke increases with:  *Smoking, alcohol use, poor diet, lack of exercise, high blood pressure, high cholesterol, diabetes, obesity, previous strokes  First signs  *Loss of consciousness, headache, weak/immobile limbs, slurred speech  One third will die from a stroke or soon after  Those that survive may need services after acute care stay
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 FLUENT APHASIAS
*Word substitutions, neologisms, and often verbose verbal output  *Often posterior lesions in the left hemisphere  *Wernicke’s Aphasia  *Anomic Aphasia  *Conduction Aphasia  *Transcortical Sensory Aphasia  *Subcortical Aphasia
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Wernicke’s Aphasia
Poor Auditory Comprehension  Impaired repetition skill  Intact grammatical structures  Severe word finding problems  Poor visual comprehension  Seldom are paralyzed because damage is not in motor area  Rapid rate of speech with normal prosodic features and good articulation  Incessant, effortlessly produced, flowing speech with normal phrase length  Empty speech  Writing problems  Verbal paraphasias and neologisms  Reading comprehension problems  Circumlocutions  Reduced ability to comprehend the speech of others  Speech often lacks content  Have a hard time monitoring themselves  Generally poor communication in spite of fluent speech  Good articulation
28
Anomic Aphasia (fluent)
Damage at the convergence of the parietal- temporal-occipital cortex  Fluent spontaneous speech marred by word retrieval difficulties  Mild to moderate auditory comprehension problems  Severe anomia in both speech and writing  When a word is furnished-patient usually recognizes it immediately  Intact repetition  Normal oral reading skills and good reading comprehension  Unimpaired articulation
29
Conduction aphasia (fluent)
Caused by lesions in the region between the Broca’s and Wernicke’s area.  Disproportionate impairment in repetition  Paraphasic speech  Marked word-finding difficulties  Empty speech  Reading and Writing are usually good  Good syntax, prosody, and articulation  Auditory comprehension ranges from mild to moderate  Naming deficits ranges from mild to severe
30
Transcortical sensory aphasia (fluent) TSA
 Lesion in the temporo-parietal region  Rarest of the fluent aphasias  Good repetition skills but poor comprehension of repeated words  Normal automatic speech (counting)  Paraphasic and empty speech  Echolalia of grammatically incorrect forms  Difficulty in pointing, obeying commands, or answering yes/no questions  Good reading outloud but poor comprehension of material read  Writing problems that parallel those in expressive speech.  Impaired auditory comprehension of spoken language  Severe naming problems
31
Subcortical Aphasia (fluent)
Caused by lesions in the basal ganglia and surrounding structures  Intact repetition skills  Word-finding problems  Articulation problems  Semantic paraphasia  Normal comprehension for routine conversation; may be defective for complex material  Fluent speech, which may include some pauses and hesitations  Prosodic problems  Relatively preserved writing skills
32
Broca's aphasia (nonfluent)
Damage in the Posterior-Inferior Frontal Gyrus of the Left Hemisphere  Nonfluent, effortful, slow, halting and uneven speech  Misarticulated or distorted sounds  Agrammatic or telegraphic speech  Impaired repetition of words and sentences  Impaired naming  Better auditory comprehension of spoken language  Poor oral reading and poor comprehension of material read  Monotonous speech  Apraxia of speech  Dysarthria  Writing Problems  Limited word output, short phrases and sentences  Telegraphic speech
33
Transcortical Motor aphasia TMA (nonfluent)
Lesions in the anterior superior or frontal lobe  Initial speechlessness  Echolalia and Perseveration  Absent or reduced spontaneous speech  Nonfluent, paraphasic, agrammatic, and telegraphic speech  Intact repetition skill  Slow difficult reading outloud  Good comprehension of simple conversations  Seriously disturbed writing  Unfinished sentences
34
Global Aphasia (nonfluent)
Extensive lesions affecting all language areas  Most severe form of nonfluent aphasia  Profound impaired language skills  Greatly reduced fluency  Impaired repetition  Impaired reading and writing  Auditory comprehension limited to single words  Impaired naming  Perseveration
35
Additional types of aphasia
 Some aphasias may affect primarily one modality  Alexia with Agraphia-Reading with writing impairment  Alexia without Agraphia-Reading impairment  Pure Agraphia- Severe writing disorder  Pure Word deafness-Lack of auditory comprehension with error free spontaneous speech  Crossed Aphasis-Aphasia accompanying right hemisphere damage.
36
Lifespan issue of aphasia
May receive services for at least the first few months  Families are often frightened and confused  May exhibit perserveration, disinhibition, emotional problems  Course/extent of recovery difficult to predict  Spontaneous recovery  The earlier the treatment, the better the recovery  Loss of Language ability can lead to social isolation.
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Aphasia Assessment
Assessment occurs in multiple phases as client recovers  Medical History  Interview with client and family  Oral Peripheral Examination  Hearing Testing  Direct Speech and Language Testing  Counseling is ongoing Formal testing postponed until patient is stable  Address  *Overall communication skills  *Expressive language  *Receptive language  *All modalities across all aspects of language  Standardized tests are available  Observation/interpretation of client behavior Definitive diagnosis difficult early on  SLP  *Identifies changes in language performance and behavior  *Genetic history and health information  *Observation in different environments  *A few language tests exist  * Scales can be used for rating loss  *Aphasia assessments can be used.
38
Aphasia screening
 Aphasia Language Performance Scales (ALPS)  Sklar Aphasia Scale (SAS)  Bedside Evaluation and Screening TestSecond Edition (BEST-2)
39
Standardize test for aphasia
The Boston Diagnostic Aphasia Examination  The Western Aphasia Battery  The Minnesota Test for Differential Diagnosis of Aphasia  The PORCH Index of Communicative Ability  Bilingual Aphasia Test
40
Functional assessment tools for aphasia
Functional Communication Profile  Communicative Abilities in Daily Living  The Communicative Effectiveness Index  Functional Assessment of Communication Skills for Adults  Reading Comprehension Battery for Aphasia
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what do test for aphasia do and evaluate?
 1. Severity of disorder  2. Classification  3. Strengths and Weaknesses  4. Competence of test- accountability  5. #’s to tell other professionals-data to report  6. Changes-want to show if patient gets better  7. Watch patient take test-what strategies are they using  8. To plan treatment  9. We use tests because they are there.  10. Provide Information and Answer Questions
42
specific speech and language skills to be assessed
Assessment of:  *Repetition Skills  *Naming Skills  *Auditory Comprehension of Spoken Language  *Comprehension of single words  *Comprehension of sentences and paragraphs  *Reading Skills  *Writing Skills  *Gestures and Pantomime  *Automated Speech and Singing
43
Aphasia intervantion
 A GOOD ASSESSMENT LEADS TO GOOD THERAPY!!!!  THE OVERALL GOAL OF INTERVENTION IS TO AID IN THE RECOVERY OF LANGUAGE AND TO PROVIDE STRATEGIES TO COMPENSATE OF PERSISTENT LANGUAGE DEFICITS. Determine by assessment and client/family needs  Cross Modality generalization  Conversational techniques  “Bridging” between hemispheres  Multimodality stimulation  AAC  Neural plasticity  Involve family members
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Right hemisphere disorder
 Group of deficits resulting from right cerebral hemisphere injury  Characteristics  *Neglect information from left side  Unrealistic denial  Impaired judgment and self-monitoring  Lack of motivation  Inattention
45
Right Hemisphere damege
 Cognitive deficits result in communication problems  Linguistic deficits  *Pragmatics most impaired  *Incorrect Interpretations  *Misinterpret contextual information  *Understanding may be concrete  *Difficulty with naming, writing, repetition  Paralinguistic deficits  *Aprosodia
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assessment for RHD
Visual scanning and tracking  Auditory and visual comprehension  Direction following  Response to Emotion  Naming and Describing  Writing  Observation is essential for pragmatics  Portions of aphasia batteries, standardized measures for RHD, and nonstandardized measures.
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Intervention for RHD
 Visual and auditory recognition  Respond appropriately in conversation  Track increasingly complex information  Use time restraints  Sequencing and explaining actions  Synthesize skills within conversation  Target nonlinguistic markers.
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Traumatic Brain Injury
Disruption in normal functioning caused by a blow or jolt to the head or penetrating injury  Leading Causes  *Falls (28%)  *Motor Vehicle Accidents(20%)  *Blows to the Head (19%)  *Assaults (11%)  1.4 million people sustain TBI annually  *Males sustain TBI more often than females. Affects orientation, memory, attention, reasoning/problem solving, executive function  Most disturbed language area is pragmatics  Deficits may also include  **Speech  **Voice  **Swallowing  **Psychosocial/personality changes  Severity related to initial levels of consciousness and post-traumatic amnesia
49
Lifespan issues with TBI
Most are young, results of vehicular accident  Several Stages of recovery  Initially, nonresponsive and requires full assistance  Gradually respond to stimuli and recognize some individuals  Confusion and Agitation  Inappropriate, incoherent, emotional language  Later, can remain alert and hold short conversation  Oriented to person and place, not time  Inappropriate, unaware, unrealistic, and uncooperative  In later stages of recovery, can initiate and carry out tasks  May consistently behave in a socially appropriate manner.  Periodic depression and irritability  Most will have lingering deficits, especially pragmatics
50
Assessment for TBI
SLP  *Cognitive-communication abilities  *Swallowing  *Assessment varies with stages of recovery  *Few comprehensive tools  *Sampling essential for pragmatics
51
Intervantion for TBI
Cognitive Rehabilitation  *Restorative Approach  *Compensatory Approach  Early Stages  *Orientation, sensorimotor stimulation, recognition  Middle Stages  *Reduce confusion, improve memory and goal directed behavior  Late Stages  *Comprehension of complex information and directions, conversational and social skills
52
Dementia
 Intellectual Decline due to neurological causes  *Additional deficits  *Poor reasoning/judgment, impaired abstract thinking, inability to attend to relevant information, personality changes  *Language functions most dependent on memory are affected  *Fewer than 15% of elderly experience dementia  *20% respond to treatment
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Types of dementia
CORTICAL DEMENTIA and SUBCORTICAL DEMENTIA
54
CORTICAL DEMENTIA
Visuospatial deficits, memory problems, judgment and abstract thinking disturbances, and language deficits in naming, reading and writing and auditory comprehension  *ALZHEIMER’S  *PICK’S
55
SUBCORTICAL DEMENTIA
Deficits in memory, problem solving, language, neuromuscular control  *Multiple sclerosis  *AIDS-related encephalopathy  *Parkinson’s  *Huntington’s
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Alzheimer's dISEASE
 Cortical pathology  Affects 13% of those over 65  50% of those over 85  Affects primarily memory, language, or visuospatial skills  Cause unknown  Genetic and environmental  Presence of neurofilaments and plaques  Extensive damage to hippocampus and cortex
57
Alzheimer's severity
Mild dementia  Name recall difficulty, occasional disorientation, memory loss  Later stages  Paraphasia, delayed responding, reduced vocabulary and syntactic structure, pronoun confusion, topic digression, inability to shift/return to topic, reading/writing errors  Most severe form  Naming errors, generic terms, syntactic errors, minimal comprehension, jargon, echolalia, mutism
58
Lifespan issues with Alzheimer's
Often unaware or ignores early signs  No cures  Drug therapy may help  Early stages  Memory loss  As disease progresses, memory loss increases and vocabulary decreases  Most advanced stages  All intellectual functions are severely impaired  Almost all reside in nursing homes
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Assessment for Alzheimer's
 Definitive diagnosis difficult early on  SLP  Identifies changes in language performance and behavior  Genetic history and health information  Observation in different environments  A few language tests exist  Scales can be used for rating loss  Aphasia assessments can be used
60
Intervention for Alzheimer's
Goal  Maintain client at highest level of functioning and help others maximize client’s participation  Emphasize intact abilities  Compensate for deficient abilities  Target memory or word retrieval  Coherent verbal responses  Longer, more complex utterances with memory aids  Stimulating cognitive processes plus pharmacological treatment is best