Chapter 27- dysrhythmias Flashcards

(53 cards)

1
Q

what are the four intervals in the heart

A

-stimulation for the electrical impulse
-transmission to adjacent tissue
-contraction of atria then ventricles
-relaxation of atria then ventricles

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2
Q

what does automaticity mean

A

ability for the heart to generate electrical impulses

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3
Q

what does conductivity mean

A

ability of tissues to transmit electrical impulses

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4
Q

what is the pacemaker of the heart

A

SA node

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5
Q

what is an action potential

A

contraction of myocytes, controlled by voltage

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6
Q

what do sarcomeres, troponin, and Ca part of

A

neurons will stimulate muscle cells, which stimulate contraction

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7
Q

what is a sarcomere

A

contraction unit

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8
Q

what is troponin

A

binds to calcium, opening sites and causing contraction

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9
Q

what does calcium do

A

react to troponin

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10
Q

what electrolytes is the SA node dependent on

A

Na and K

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11
Q

what is automaticity

A

allows any part of the conduction system to start an impulse

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12
Q

what is the absolute refractory period

A

the cells can not respond to a new stimulus

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13
Q

what causes arrhythmias

A

irregular electrical impulse and or conduction

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14
Q

ectopic focus/ectopic beat and causes

A

impulse started by something other than SA
-hypoxia, ischemia, hypokalemia

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15
Q

what is an atria flutter

A

the atria is beating faster than the ventricles
-lub, lub, lub

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16
Q

what is atria fibrillation?

A

AV cannot keep up with SA – no p wave

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17
Q

what is a heart block

A

signal between atria and ventricles is messed up or blocked, emergency intervention is needed

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18
Q

the main concern when starting antidysrthmias

A

making the dysrhythmia worse or creating a new one

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19
Q

what is cardioversion

A

shock to the heart

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20
Q

what is defibrillation

A

emergency situation, VTAC and VFIB can be shocked

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21
Q

pacemaker

A

for tachy and brady arrhythmias to keep heart pace WNL
-some can do both if needed

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21
Q

what is ablation

A

Burnout problem areas

22
Q

MOA of antidysrhythmic drugs

A

-reduce automaticity
-slow conduction impulses
-prolong the refractory period

23
Q

when are antidysrhythmic drugs used

A

-treatment of atrial fibrillation or flutter
-maintaining NSR after ablation
-suppressing fast/irregular ventricle rate – isn’t filling with enough blood
-dysrhythmias that could be fatal — A fib causing clots

24
how do sodium channel blockers work
blocks the opening of sodium channels
25
what can sodium channel blockers treat
atrial dysthymia, supraventricular tachycardia -- bursts of high rapid HR which are normally idiopathic
26
side effects of sodium channel blockers
arrhythmias, bradycardia, hypotension -respiratory depression, dizziness, syncope, drowsiness, fatigue, confusion, anticholinergic
27
nursing concern for sodium channel blockers
interacts with anticoagulants and A fib pts need them
28
common sodium channel blockers
quinidine, lidocaine
29
how do beta blockers work
decrease conduction through the SA/AV node -decreases cardiac excitability, workload, and oxygen consumption
30
what are beta blockers used to treat
-dysrhythmia from SNS stimulation, a fib, and flutter, post-MI and CHF to prevent v-fib
31
side effects of beta blockers
BRADYCARDIA, HYPOTENSION, DIZZY, SYNCOPE -Av block, bronchospasm, dyspnea, drowsiness, fatigue, ED
32
nursing considerations of beta blockers
-taper off -when taken with Ca blocker can increase risk of heart block
33
common beta blockers
METOPEROL and ETGENELOL -propranolol, acebutolol, esmolo
34
what does potassium do for the heart
control contractility
35
what should potassium levels be
3.5-5
36
hypokalemia signs
ventricular dysrhythmias, muscle weakness, decreased deep tendon reflexes, weak peripheral pulses
37
issues with IV potassium
can burn, should be given in a Central line
38
hyperkalemia signs
dysrhythmias, v-fib, HB, cardiac arrest, muscle twitching, numbness in hands, feet and mouth
39
what can quickly bring down potassium levels
insulin/dextrose
40
how do potassium channel blockers work
prolongs action potential, slows repolarization, and prolongs the refractory period
41
when are potassium channel blockers used
given IV for life-threatening tacy dysrhythmias given PO for tachycardia, V/A fib, and A flutter
42
side effects of potassium channel blockers
bradycardia, hypotension, weakness, dizziness, worsened/new dysrhythmias, PULMONARY TOXICITY, HEPATOTOXICITY, blurred vision
43
who should not take potassium channel blockers
AV block, shock, hypotension, respiratory depression, and renal/hepatic impairment
44
common potassium channel blockers
AMIODARONE -dofetilide, ibutilide, sotalol
45
how do calcium channel blockers work
reduce automaticity of SA node and slow conduction of AV node
46
what can calcium channel blockers treat
supraventricular dysrhythmias (SA and AV) and tachycardia -emergency for A-fib, SVT
47
side effects of calcium channel blockers
BRADYCARDIA, HYPOTENSION, -flushed skin, MI, hepatotoxicity, peripheral edema
48
who shouldn't take calcium channel blockers
HB, sick sinus, HF, hypotension, renal/hepatic, PREGNANCY
49
nursing considerations with calcium channel blockers
grapefruit, monitor with beta-blockers and digoxin
50
what is sick sinus
irregular heart rhythm
51
how does adenosine work and when is it used
depression of conduction at the AV node, causing NSR -EMERGENCY MED
52
can digoxin treat dysrthmias
YES