Chapter 39 Shock Flashcards
Hypovolemic Shock (fxn)
total body fluid decreased (in all fluid compartments)
hemorrhage
dehydration
Cardiogenic (fxn)
direct pump failure. fluid volume not affected
myocardial infarction
valvular problems (stenosis, incompetence)
myopathies
dysrhythmias
cardiac arrest
Distributive (fxn)
fluid shifted from central vascular space. total body fluid volume normal or increased
neural induced loss of vascular tone (head trauma, anesthesia, opioids, sedatives)
chemical induced loss of vascular tone (sepsis, anaphylaxis, capillary leak)
Obstructive (fxn)
cardiac function decreased by noncardiac factors. total body fluid volume not affected. central volume decreased pulmonary HTN tension pneumothorax pericarditis thoracic tumor tampnade
Hypovolemic (site of origin)
central vascular volume decreased. total body fluid may or may not be decreased
hemorrhage
dehydration
fluid shifts (trauma, burns, anaphylaxis)
Cardiogenic (site of origin)
direct pump failure, indirect pump failure, decreased CO, total body fluid not decreased valvular problems (stenosis, incompetence) MI myopathies dysrhythmias cardiac arrest tamponade pericrditis pulm. HTN pulm. emboli
Vasogenic (site of origin)
loss of vascular tone, total body fluid not decreased.
neurogenic (head trauma, vasovagal response, drugs affecting the CNS: anesthesia, opioids, sedatives)
vessel dilation (anaphylaxis, inflammation)
Cardio manifestations of shock
Decreased CO, increased pulse
Thready pulse, decreased BP
narrowed pulse pressure, postural hypotension
Low central venous pressure, flat hand and neck veins in dependent positions
Slow cap refill, diminished peripheral pulses
Septic (site of origin)
loss of vascular tone, eventual reduced CO, seen as a more intense type of vasogenic shock
infection
Respiratory manifestations of shock
Increased RR, shallow depth
Decreased paco2
Decreased pao2
Cyanosis esp around lips and nail beds
Neuromuscular manifestations of shock
Early: anxiety, restlessness, increased thirst
Late: decreased CNS activity (lethargy to coma), generalized muscle weakness, diminished or absent deep tendon reflexes, sluggish pupillary response to light
Renal manifestations of shock
Decreased urine output
Increased specific gravity
Sugar and acetone present in urine
Integumentary manifestations of shock
Cool to cold
Pale to mottled to cyanotic
Moist, clammy
Mouth dry; paste like coating present
Gastrointestinal manifestations of shock
Decreased motility
Diminished or absent bowel sounds
Nausea and vomiting
Constipation
Causes of hypovolemic Shock
Body fluid depletion
Hemorrhage (trauma, GI ulcer, surgery, inadequate clotting: hemophilia, liver dx, malnutrition, bone marrow suppression, cancer, anti coagulation therapy)
Causes of cardiogenic shock
Direct pump failure MI cardiac arrest Ventricular dysrhythmias (fibrillation,tachycardia) Cardiac amyloidosis Cardiomyopathy (viral, toxic) Myocardial degeneration
Causes of distributive shock
Decreased vascular volume or tone: overall Neural induced (pain, anesthesia, stress, spinal cord injury, head trauma) Chemical induced (anaphylaxis, sepsis, capillary leak: burns, extensive trauma, hepatic dysfunction, hypoproteinemia)
Initial stage hypovolemic shock
Decrease in map of 5-10mmhg
Increased sympathetic stimulation (mild vasoconstriction, increase in heart rate)
Non progressive stage of shock
Decrease in map of 10-15mmhg Continued SNS stimulation (mod vasoconstriction, increased HR, decreased pulse pressure) Chemical compensation (renin, angiotensin, aldosterone, anti diuretic: increased vasoconstriction, decreased urine output, stimulation of thirst reflex) Some anaerobic Metabolism in non vital organs (mild acidosis, mild hyperkalemia)
Progressive stage of shock
Decrease in map of >20mmhg
Anoxia to nonvital organs
Hypoxia of vital organs
Overall metabolism is anaerobic (moderate acidosis, moderate hyperkalemia, tissue ischemia)
Refractory stage of shock
Severe tissue hypoxia with ischemia and necrosis
Release of myocardial depressant factor from pancreas
Buildup of toxic metabolites
Multiple organ dysfunction syndrome
Death
Detecting and Treating Impending Shock #1
monitor early responses (normal BP, narrowed pulse puressure, mild orthostatic hypotension, slight delayed capillay refill, pale/cool skin or flushe skin, slight tachypnea, nausea, vomiting, thirst, weakness.
monitor possible sources of fluid loss: hest tube, nasogastric drainage, diarrhea, vomiting, increased abdominal girl, extremity girth, hematemesis, hematochezia.
monitor circulatory status (BP, skin color, skin temp, heart sounds, HR, rhythm, presence/quality periph pulses, capillary refill.
monitor inadequate tissue oxygenation (apprehension, increased anxiety, change in mental status, agitation, oliguria, cool mottled periphery
Detecting and Treating Impending Shock #2
monitor pulse ox monitor lab values: Hgb, Hct, clotting profile, ABG, lactate level, electrolyte level, cultures, chemistry profile note bruising, petechiae, mucous membr. note color, amt, consistency, frequency of stools, vomitus, and nasogastric drainage test urine for blood, protein place pt. in supine, legs elevated administer IV or oral fluids insert and maintain large bore IV access admin O2/mech ventilation
Care of Pt. in Hypovolemic Shock
ensure patent airway start IV catheter administer O2 elevate feet, keep head flat or elevated 30* examine pt. for bleeding if overt bleeding, apply direct pressure administer drugs as prescribed increase rate of IV fluid delivery do not leave pt.