Chapter 41- Alterations in Musculoskeletal Function Flashcards

(97 cards)

1
Q

Who has highest incidence of fractures?

A

young males 15-24 y/o and older persons > 65 y/o

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2
Q

Most common fracture sites of healthy bones in young people and why

A

tibia, clavicle, lower humerus from trauma

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3
Q

Most common fx sites in older adults and why

A

upper femur, upper humerus, vertebrae, pelvis d/t osteoporosis

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4
Q

complete fracture

A

bone broken entirely

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5
Q

incomplete fracture

A

bone damaged but still in one piece

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6
Q

open fractures

A

formerly compound; skin is OPEN; can be complete or incomplete

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7
Q

closed fractures

A

If skin is not open; complete or incomplete

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8
Q

comminuted fracture

A

bone breaks into more than 2 fragments

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9
Q

linear fracture

A

fracture line runs parallel to long axis of bone

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10
Q

oblique fracture

A

direction of fracture line slanted angle to shaft of bone

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11
Q

spiral fracture

A

fracture line encircles the bone

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12
Q

transverse fracture

A

fracture line occurs straight across the bone

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13
Q

who do incomplete fractures occur most commonly in

A

children; more flexible growing bones

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14
Q

greenstick fracture

A

perforates one cortex and splinters the spongy bone
think young tree branch that’s bent sharply; outer surface disrupted but inner remains intact
bends/cracks
most common in children

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15
Q

where do greenstick fxs usually occur

A

metaphysis or diaphysis of tibia, radius, ulna

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16
Q

Torus fx

A

cortex buckles but doesn’t break

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17
Q

Bowing fractures

A

when longitudinal force applied to bone= bending of bone; usually occurs in pair (one bowed, one fx)

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18
Q

Why is bowing fx hard to correct

A

resists reduction/correction because force to correct must equal force that caused; bowed bone interferes w/ reduction of fx bone

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19
Q

Who does bowing fx occur in and what bones

A

children; paired radius-ulna or fibula-tibia

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20
Q

Pathologic/insufficiency/fragility fracture

A

break at site or preexisting abnormality resulting from force that wouldn’t fx normal bone; CAN occur with NORMAL weight bearing/activity

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21
Q

What causes pathologic fx

A

any disease that weakens a bone (esp. cortex)

Ex: osteoporosis, cancer, infection, RA, Paget disease, osteomalacia, rickets, hyperparathyroidism, radiation

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22
Q

stress fx

A

repeated strain like in sports’ normal or abnormal bone. Forces on bone cumulative eventually causing fx; tiny cracks in a bone

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23
Q

Fatigue fx

A

repetitive sometimes abnormal stress/torque applied to bone w/ normal ability to deform and recover
Usually in individuals who engage in a new/different activity that is both strenuous and repetitive
Muscle strength faster than bone strength

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24
Q

transchondral fx

A

fragmentation and separation of portion of articular cartilage (It allows the bones to glide over each other with very little friction.)
single or multiple sites; cartilage alone or cartilage + bone

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25
typical sites transchondral fx and who affected more
distal femur, ankle, patella, elbow, wrist Adolescents
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direct/primary healing
intramembranous bone formation occurs when adjacent bone cortices are in contact with one another like with SURGICAL fixation devices ***NO callus formation
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indirect/secondary healing
both intramembranous and endochondral bone formation, development of CALLUS, and eventual remodeling of solid bone
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Endochondral bone formation
bone formation that begins with an underlying cartilage scaffold
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When is secondary/indirect healing mostly seen
non-operative fracture treatment; fracture treated w/ cast
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how are calluses formed
bone tissue adjacent to fx dies, osteoblasts synthesize collagen and matrix, becomes mineralized= callus
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s/s fx
unnatural alignment (deformity), swelling, muscle spasm, tenderness, pain/impaired sensation, decreased mobility
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INITIAL sx following fx
numbness at fx site d/t nerve trauma x several mins then subsequent pain is severe
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s/s pathologic fx
angular deformity, painless swelling, generalized bone pain
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s/s transchondral fx
asx painful during ROM limited RPM and crepitus
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dislocation
displacement 1 or more bones in joint in which opposing joint surfaces entirely lose contact with one another
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subluxation
contact between opposing joint surfaces partially loss (partial dislocation)
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who is more at risk for dislocation and subluxation
< 20 y/o and generally associated w/ fx
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s/s dislocation or subluxation
pain swelling limited ROM joint deformity
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what causes pain in fx
muscle spasms overriding of fx segments damage to adjacent soft tissue
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what causes pain in dislocation or subluxation
effusion of inflammatory exudate into joint nerves tendon/ligament injury muscle contraction
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tendon
connects skeletal muscle to bone or another structure; meant to connect skeleton for movement area of attachment= enthesis
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ligament
connects bone to bone where they meet in JOINT, meant to keep things held together/stabilized
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strain/where common
tearing/stretching of muscle or tendon most common in hands, feet, knee, upper arm, thigh, ankle, heel (akilles)
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sprain/where common
ligament tears most common in wrist, ankle, elbow, knee
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avulsion/who commonly affects
complete separation of tendon/ligament from bony attachment young athletes- sprinters, hurdlers, distance runners
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s/s tendon/ligament injury
soft tissue swelling change in tendon/ligament contour disclocation/subluxation bones SHARP localized pain tenderness persists over distribution of tendon/ligament movement/weight bear increases pain
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s/s lateral epicondylopathy/tennis elbow & medial epicondylopathy/golfer's elbow
localized to one side of joint local tenderness, more pain with ACTIVE ROM than passive tissue degradation loss of grip, strength, pain
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bursitis
inflamed sacs of synovial fluids (meant to separate/lubricate/cushion structures) middle age d/t trauma shoulder, hip, knee, elbow (less often spine, wrist, foot, ankle)
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*rhabdomyolysis/myoglobinuria
rapid breakdown muscle that causes release intracellular contents like myoglobin into extracellular space & bloodstream
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*s/s rhabdomyolysis
muscle pain and weakness dark urine
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**labs in rhabdomyolysis
#1= high CK (normal 5-25 women, 5-35 men) 5-10x upper limit myoglobin in urine high K (norm 3.6 to 5.2) high phosph. low BUN
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*complications rhabdomyolysis
ARF disseminated intravascular coagulation (DIC) cardiac arrythmias
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*when is ARF likely in rhabdomyolysis
CK > 15,000
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*s/s hyperkalemia (> 5.2)
Abdominal (belly) pain and diarrhea. Chest pain. Heart palpitations or arrhythmia (irregular, fast or fluttering heartbeat). Muscle weakness or numbness in limbs. Nausea and vomiting
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s/s hypernatremia (> 145)
excessive thirst lethargy confusion muscle twitching or spasms
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s/s hyponatremia ( < 135)
Nausea and vomiting Headache Confusion Loss of energy, drowsiness and fatigue Restlessness and irritability Muscle weakness, spasms or cramps Seizures Coma
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s/s compartment syndrome
6 P's: pain pressure pallor paresthesia paresis (impaired function) pulselessness
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what is and s/s malignant hyperthermia
prolonged release intracellular Ca hypermetabolism: high body temp, muscle rigidity, rhabdomyolysis respiratory acidosis (high tidal end Co2) tachycardia masseter & skeletal muscle spasm
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osteoporosis
most common bone disease decreased bone mineral density, thinner more porous, increased risk of fractures bc alterations in bone microarchitecture disrupted bone resorption & formation NOT consequence of aging (but is risk)
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most common fx site osteoporosis
spine, femoral neck, wrist
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causes osteoporosis
1. postmenopausal 2. secondary 3. regional (confined to segment skeleton, no known cause) endocrine disorders (thyroid, growth, parathyroid, cortisol like cushing) meds (PPO, glucocorticoids... ) menopause (decreased estrogen) low vit D & Ca family hx obesity smoking diet
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s/s osteoporosis and common fx sites
pain & bone deformity d/t fx kyphosis, decreased height fx long bones (femur), distal radius, ribs, vertebrae
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*osteomalacia
ADULT bone metabolic disease characterized by inadequate delayed mineralization of osteoid in mature compact and spongy bone It is a disorder of decreased mineralization, which results in bone breaking down faster than it can re-form NO mineral calcification and deposition a problem with vitamin D, which helps your body absorb calcium
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cause osteomalacia
***low vit D = decreased Ca absorption > low phos. HIV, liver, cancer, kidney, nutrient disorders can cause low D > osteomalacia
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*s/s osteomalacia
diffuse muscular/skeletal pain and tenderness especially in HIPS= hesitant to walk waddling gait (muscular weakness) facial deformities bowed legs, knocked knees fragility fx: bone fx, vertebral collapse w/ min trauma low back pain eaerly UREMIA
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*lab osteomalacia
high BUN/Cr norm/low Ca high phos. high alkaline phos. & PTH
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rickets
like osteomalacia but in children
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Paget Disease/osteitis deformans (PBD)
second most common bone disease after osteoporosis state of increased metabolic activity in bone characterized by localized abnormal and EXCESSIVE bone remodeling Enlarged softened bone= bowing deformity, fx, neuro problems
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most commonly affected bone paget disease
vertebrae, skill, sacrum, sternum, pelvis, femur
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risk paget disease
men > 55, women > 40
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s/s & labs paget disease
asx often irregualr bone trabeculae w/ thick/disorganized pattern HA frontal, occipital skull thickness/asymmetrical altered mentation/dementia from compression on brain by thick skull sensory abnormalities, deaf, motor function, optic nerve, lacrimal duct all from skull compression on nerves thick mandible/maxilla stress fx BLE spastic quadriplegia elevated serum alkaline phosphatase or abnorm. xray high PINP
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*osteomyelitis, most common pathogen, & two types
bone infection caused usually by bacteria ***staph. aureus (esp. in human bite). Can be fungi, parasite, virus also 1. hematogenous 2. contiguous
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hematogenous osteomyelitis & s/s
pathogens through blood > bone infect. *Children fever pain voluntary immobility of affected limb kids: cutaneous, sinus, ear, dental infections primary source adults: spine, pelvis, small bones
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contiguous osteomyelitis & s/s
infection spreads to adjacent bone often caused by open fx, penetrating wounds, surgery, metabolic/vascular disease, age, lifestyle *males
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*s/s osteomyelitis
insidious onset, vague sx fever malaise anorexia & wt loss pain in/around infected areas maybe/maybe not edema recent infection or instrumentation precedes abscesses (Brodie abscesses) lymphadenopathy in chronic= silent between exacerbations
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Brodie abscesses
abscesses in subacute or chronic osteomyelitis circumscribed lesions 1-4cm ends of long bones and surrounded by dense ossified bone matrix evidence pathogen less virulent or immune system somewhat successful
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*labs osteomyelitis
elevated WBC C-reactive protein (CRP)
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osteoarthritis (OA) what is it? patho? affected?
most common age-related disorder of synovial joints ***loss/damaged cartilage & bone spurts *inflammation, new bone of joint margins, subchondral bone change, variable degrees synovitis, thickening capsule joint LOAD-BEARING AREAS= hips, hands, spine > 40 y/o, women stress on joints
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*clinical manifestations osteoarthritis
5th/6th decade of life #1- pain in 1+ joints using w/ weight/load bear or use; RESTING RELIEVES *stiffness < 30 min crepitus, creeking, grating swelling/enlargement tender limited ROM muscle waste partial dislocation deformity limp synovitis (inflam. joint lining) nocturnal pain unrelieved and w/ paresthesia referred pain like sciatic nerve back of thigh brachial neuralgia (pain in arm) w/ neck movement lack of sleep= more stress chronic pain
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heberden and bouchard nodes
bone enlargement in hands w/ osteoarthritis
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*rheumatoid arthritis (RA)
chronic, systemic, inflammatory autoimmune disease, joint swelling and tenderness and destruction of synovial joints leading to disability thickened synovial tissue invades bone and acts like localized tumor & bone destruction spreads to surrounding ligaments, tendons IgM IgG antibodies , T & B cells
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synovial membrane
A layer of connective tissue that lines the cavities of joints, tendon sheaths, and bursae Inflamed in RA
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*clinical manifestations rheumatoid arthritis
insidious usually, 15% acute general systemic of inflammation: fever, fatigue, weakness, anorexia, weight loss, general aching/stiffness joints painful, tender, stiff (pain d/t pressure from swelling in early stage) later pain from from sclerosis of subchondral bone & new bone formation stiffness d/t synovitis warm joints, boggy synovial membrane *skin over joint ruddy, cyanotic hue, thin and shiny lost ROM/mobility
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2 complications chronic RA from excessive inflammatory exudate in synovial cavity
1. cysts in articular cartilage or subchondral bone 2. rupture of cyst or synovial joint
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rheumatoid nodules
30% extra-articular manifestations elbows & fingers usually risk of invading skin, cardiac valves, pericardium, pleura, lung, spleen
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Caplan syndrome
Can occur w/ RA pulmonary nodules & pneumoconiosis (chronic inflam. lungs from inhalation of dust) swelling (inflammation) and scarring of the lungs.
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*Ankylosing Spondylitis
most common of group of inflammatory arthropathies known as spondyloarthropathies (SpAs) chronic autoimmune inflammatory joint disease characterized by stiffening and fusion (ankylosis) of spine & sacroiliac joints
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Difference between RA and AS
1. RA inflammation is synovial membrane so synovial joints 2. AS excessive bone formation occurs and the inflammation is at enthesis (where ligaments, tendons, joint insert into bone) and end results sacroiliac & vertebral column fuse at joint
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Who does AS occur in
MEN > women Later adolescence, peaks 20 y/o secondary affects older groups & associated w/ inflammatory diseases associated with HLA
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*s/s AS
bamboo spine (square appearing vertebral body) low back pain & stiffness (esp. early AM) insidious pain then persistent **worse after rest, alleviated by activity loss of ROM in AM d/t inflammation and muscle spasm kyphosis costovertebral= pain worse inspiration sitting on hard seat unbearable limp elevated ESR, CRP IgA kidney disease
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*fibromyalgia
chronic musculoskeletal syndrome characterized by diffuse joint and muscle pain, fatigue, and increased sensitivity to touch called tender points
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s/s fibromyalgia (FM)
*diffuse chronic pain (starts in one location like neck/shoulders, then more generalized) fatigue (esp. arising from sleep & midafternoon) HA irritable bowel sensitivity to cold non-restorative sleep anxiety & depression widespread tenderness in all body regions, increased sensitivity to numerous stimuli: heat, cold, electrical, light, sound
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Who does FM affect
women 30-50 y/o
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*epicondyle
bony prominence at end of bone where tendons or ligaments attach
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epicondylopathy
when force causes microtears in tissue; a painful irritation of tendon insertions of the forearm muscles. repetitive load-bearing activities or acute injuries involving flexion, extension, pronation, supination of elbow and forearm - lateral (tennis elbow)= overstretching ECRB tendon & forearm muscles - medial (golfer's elbow)= forearm muscles responsible for forearm flexion and pronation
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why are really old and really young at risk for fx
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nonunion
failure of bone ends to grow together