Chapter 59 Flashcards

1
Q

Risk for Cholecystitis

A
Fat fertile fourth female 
Familial or genetic tendency
Obesity
T2DM
Dyslipidemia
Insulin resistance 
Age
Female
Crohns 
Rapid wt loss
American Indian 
Mexican American 
Increased cholesterol 
Hormone replacement therapy 
Cholesterol lowering drugs 
Prolonged TPN
Gastric bypass
Sickle cell
Pregnant 
Diet- high fat, high calories, low fiber, high white carbs
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2
Q

Physical assessment Cholecystitis

A
Flatulence 
Dyspepsia
Eructation
Anorexia
N/V
Upper abd pain
Feeling full
Rebound tenderness (Blumbergs sign)
Fever
Jaundice
Clay color stools
Dark urine
Steatorrhea 
Pruritis
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3
Q

Diagnostic Cholecystitis

A
Increased WBC
Elevated alkaline phosphatase, AST, LDH
Serum bilirubin elevated
Increased glucose 
Xray to see gallstones 
US of RUQ is best initial diagnostic 
HIDA scan 
ERCP
MRCP
MRI with contrast
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4
Q

Nonsurgical management

A
Avoid fatty foods
IV therapy for hydration 
Opioids- morphine or dilaudid 
Toradol for mild pain 
antiemetic 
IV ABT
Oral bile acid dissolution- Actigall- dissolves stones- US for first 6 months- report D/V and sever abd pain- take with food or milk
Lithotripsy or biliary cath
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5
Q

Surgical management Cholecystitis

A

Laparoscopic Cholecystectomy- “gold standard”, not common complications, low death rate, rare injuries, recovery is quicker, postop pain less severe- assess oxygen and educate on deep breathing exercises
NOTES- remove gallbladder through vagina

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6
Q

Risk acute pancreatitis

A
Biliary tract disease
Trauma
Pancreatic obstruction
Metabolic disturbance
Renal disturbance
Hereditary
Ulcers
Viral infections
Alcohol
Drug toxicity
Cigarette smoking
Cystic fibrosis
Gallstones
Abd surgery
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7
Q

Physical assessment acute pancreatitis

A

Diagnosed with severe abdominal pain in mid epigastric or LUQ
Sudden onsets and radiates to back, left flank, left shoulder
Fetal position
Jaundice
Blue discoloration on abdomen and periumbilical area(Cullen’s sign)
Blue discoloration of flank(Turner Gray Syndrome)
Bowel sounds

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8
Q

Laboratory/ Imaging assessment

A
Increased amylase, lipase, trypsin, elastase, glucose, bilirubin, ALT, AST, leukocytes, BUN
Decreased calcium and magnesium, albumin
Amylase increases within 12-24 hours 
Calcium may be decreased for 7-10 days, poor prognosis < 8
US most sensitive 
CT with contrast 
Xray for gallstones 
ERCP for pancreatic stones
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9
Q

Interventions acute pancreatitis

A
Decrease inflammation
Treat complications 
ABCs
Pain management 
Hydration- isotonic 
Fasting and rest-NPO
NG for more severe patients 
Morphine 
Histamine receptor antagonists- Zantac 
Proton pump inhibitor- Prilosec 
Side lying position
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10
Q

Chronic pancreatitis

A
Intense abd pain- burning
Tenderness
Ascites
LUQ mass
Respiratory compromise 
Steatorrhea 
Weight loss
Jaundice
Dark urine
Polyuria, polydipsia, polyphagia
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11
Q

Interventions chronic pancreatitis

A

Manage pain- opioids
Nutrition- pancreatic enzyme replacement therapy to prevent malnutrition, pancrelipase take with meals, monitor stools to evaluate effectiveness- should become less frequent and fatty, drugs to decrease gastric acid to destroy lipase, 4-6000 calories, increase protein, avoid fat foods and alcohol
Prevent recurrence- avoid irritants such as caffeine, bland low fat meals, pancreatic enzyme replacement before meals, skin care, managing DM

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12
Q

Pancreatic CA assessment

A
Jaundice
Clay color stool
Dark urine
Abd pain
Weight loss
Anorexia 
N/V
Glucose intolerance
Splenomegaly
Flatulence
GI bleed from pressure on portal vein 
Ascites
Leg or calf pain 
Weakness
Back pain
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13
Q

Interventions pancreatic CA

A
Prevent spread
Decrease pain- opioids 
Palliative tx
Chemo or radiation
Biliary stent if obstruction
Surgery if small tumor <3 cm
Whipper procedure for CA of the head
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