chapter 6 fluid and electrolytes Flashcards

(36 cards)

1
Q

Equal solute concentrations, causes no fluid shifts

A

Isotonic fluid

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2
Q

lowe solute concentration

fluid shifts out

A

hypotonic

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3
Q

higher solute concentration

fluid shifts in

A

hypertonic

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4
Q

triggered by decrease blood volume and osmolarity

A

Thirst mechanism

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5
Q

Promotes reabsorption of water in the kidneys

A

Antidiuretic hormone

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6
Q

Increases reabsorption of sodium and water in the kidneys

A

Aldosterone

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7
Q

Stimulates renal vasodilation and suppresses aldosterone, increasing urinary output

A

Atrial natriuretic peptid

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8
Q

excess fluid in the interstitial space

A

edema

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9
Q

excess fluid in the intravascular space

A

hypervolemia or fluid volume excess

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10
Q

excess fluid in the intracellular space

A

water intoxication

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11
Q

manifestations as peripherial edema, periorbital edema, anasarca, cerebral edema, dyspnea, bounding pulse, tachycardia, JVD, HTN, polyuria, rapid weight gain, crackles, and bulging fontanelles

A

Fluid Excess

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12
Q

dehydration, hypovolemia or fluid volume deficit, can occur independently without electrolyte defects.
decrease in fluid level leads to increase in level of blood solutes
cell shrinkage
hypotension

A

Fluid deficit

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13
Q

positively charged electrolytes

A

cations

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14
Q

negatively charged electrolytes

A

anions

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15
Q

normal 135-145
most significant cation and prevalent electrolyte of extracellular fluid
controls serum osmolality and water balance
facilitates muscles and nerve impulses
main source is dietary intake
excreted thru the kidneys and GI tract

A

Sodium

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16
Q

excessive sodium
Hypertonic IV saline (3% saline)
causes fluid shifts

A

HYPERnatremia

17
Q

serum osmolarity decreases
anorexia, GI upset, poor skin turgor, dry mucous membranes, BP changes, pulse changes, edema, headache, lethargy, confusion, diminished deep tendon reflexes, muscle weakness, seizures, and coma

18
Q
normal 98-108
mineral electrolyte
Major extracellular anion
found in gastric secretions, pancreatic juices, bile, and csf
main source is dietary intake
excreted thru the kidneys
19
Q

normal 3.5-5
primary intracellular cation
electrical conduction, acid-base balance, and metabolism
can’t fluctuate much without causing serious issues

20
Q

normal 4-5
found in the bone and teeth
role in blood clotting, hormone secretion, receptor functions, nerve transmission, and muscular contraction
inverse relationship with phosphorus
synergistic relationship with magnesium
absorbed thru the GI tract (small intestines)

21
Q

normal 2.5-4.5
found in bones, small amounts in bloodstream
role in bone and tooth mineralizaton, cellular metabolism, acid-base balance, and cell membrane formation
excreted thru the kidneys

22
Q
normal 1.8-2.5
intracellular cation
stored in bone and muscle
cardiac rhythm
excreted thru kidneys
23
Q

reflects hydrogen concentrations

24
Q

chemicals that combine with an acid or a base to change pH

25
most significant in the extracellualar fluid forms from carbon dioxide reacting with water carbonic anhydrase causes carbonic acid to separate into hydrogen and bicarbonate carbonic anhydrase in the lungs allow for CO2 excretion and in the kidneys allows for hydrogen excretion
Bicarbonate-carbonic acid system
26
similar to the bicarbonate-carbonic acid system high concentrations in the intracellular fluid act as weak acids, and some act as weak bases primarily works in the kidneys by accepting or donating hydrogen
Phosphate system
27
primarily occurs in the capillaries acidity and hypoxia cause hemoglobin to release the oxygen hemoglobin then becomes a weaker acid, taking up extra hydrogen binding with oxygen makes hemoglobin more prone to release hydrogen hydrogen reacts with bicarb to form carbonic acid, which is converted to carbon dioxide and released into the alveoli
Hemoglobin system
28
most abundant buffering system proteins can act as an acid or a base by binding to or releasing hydrogen occurs in the intracellular and extracellular spaces hydrogen and CO2 diffuse across the cell membrane to bind with protein inside the cell albumin and plasma are the primary buffers in the intravascular space
protein system
29
manages pH by altering CO2 excretion speeding will excrete more CO2 decreasing acidity and vice versa uses chemoreceptors responds quickly, but is short lived
respiratory regulation
30
alters the excretion or retention of hydrogen or bicarb more effective because it permanently removes hydrogen responds slowest, but lasts the longest
renal regulation
31
results from a deficiency of bicarb or an excess of hydrogen
Metabolic Acidosis
32
bicarb and chloride results are added together and subtracted from the sodium normal 6-9
anion gap
33
results from excess bicarb or deficient acid or both
metabolic alkalosis
34
results from CO2 retention, which increases carbonic acid
respiratory acidosis
35
results from excess exhalation of CO2, which leads to carbonic acid deficits
Respiratory Alkalosis
36
respiratory and metabolic disorders resulting in an acidotic or alkalotic state both the respiratory and renal systems demonstrate an imbalance of acid or base
mixed disorders