Child with elimination problem Flashcards

1
Q

Possesting

A

Non-forceful return of small amounts of milk in accompaniment of return of swallowed air (wind)
Occurs in nearly all babies from time to time

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2
Q

Regurgitation

A

Larger, more frequent returns or losses of milk, may indicate presence of GORD

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3
Q

Vomiting

A

Forceful ejection of gastric contents

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4
Q

Red flags in vomiting

A

Bile stained vomit - intestinal obstruction
Haematemesis
Projective vomiting - pyloric stenosis
Vomiting after paroxysmal coughing - whooping cough
Abdo pain
Abdo distension
Hepatosplenomegaly
Blood in stool - intussusception
Severe dehydration/shock
Bulging fontanelle or seizures
FTT

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5
Q

Causes of vomiting in infants

A

GORD
Feeding problems
Infection
Gastroenteritis
Mengingitis
Pyloric stenosis
Intussusception
Hirschsprung’s disease

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6
Q

Causes of vomiting in preschool children

A

Gastroenteritis
Infection
Respiratory tract/otitis media
Appendicitis
Intestinal obstruction
Raised ICP
Coeliac disease
Renal failure

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7
Q

Causes of vomiting in School-age or adolescents

A

Gastroenteritis
Infection
Peptic ulceration
Appendicitis
DKA
ED

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8
Q

Pyloric stenosis definition

A

Hypertrophy of pyloric muscle causing gastric outlet obstruction
Presents at 2-7w

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9
Q

S+S of Pyloric stenosis

A

Vomiting which increases in frequency and forcefulness over time
Hunger after vomiting until dehydration leads to a loss of interest in feeding
Weight loss

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10
Q

Diagnosis

A

Hypochloraemic metabolic alkalosis with low plasma sodium and postassium occurs as a result of vomiting
Feed test
Gastric peristalsis may be seen
Pyloric mass - feels like an olive
Stomach over distended with air
USS is helpful

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11
Q

Management of pyloric stenosis

A

Correcet any fluid and electrolyte disturbance with IV fluids
Once hydrated, acid-base balance and electrolytes are normal
Can treat with pyloromyotomy - involves division of hypertrophied muscle down to mucosa

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12
Q

RF for GORD

A

Predominantly fluid diet
Mainly horizontal posture
Short intra-abdominal length of oesophagus
Prematurity
FHx
Obesity
Neurodisability
Repaired congenital diaphragmatic hernia/oesophageal atresia

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13
Q

Severe reflux

A

More common in:
Children with CP or other neurodevelopment disorders
Preterm infants especially if there is co-existent bronchopulmonary dysplasia
Following surgery for oesophageal atresia or diaphragmatic hernia

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14
Q

S+S of GORD

A

Recurrent regurg
Vomiting
Putting weight normally
Otherwise well
Mess, smell and frequent changing of clothes can be frustrating for parents and carers

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15
Q

GORD investigations

A

Usually diagnosed clinically and no investigations are normally required
24h oesophageal pH monitoring to quantify degree of reflux
24h impedance monitoring
Endoscopy with oesophageal biopsies to identify oesophagitis
Contrast studies to exclude underlying anatomical abnormalities in GIT

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16
Q

Mx of GORD

A

Uncomplicated GORD - parental reassurance and adding thickening agents to feeds + positioning infant prone after feeds
Usually resolves within a year - increased length of oesophagus, increased strength of LOS, weaning
More significant - acid suppression - either H2RA (ranitidine) or PPIs (omeprazole)
Or drugs that enhance gastric emptying (domperidone)
Surgery reserved for those with complications or unresponsive to intestinal medical treatment or oesophageal strictures - Nissen Fundoplication

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17
Q

Intestinal obstruction S+S

A

Bile stained vomit - Bowel obstruction until proven otherwise
Colicky abdo pain
Vomiting = early feature
Abdo distension
Absolute constipation + pain = large bowel obstruction

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18
Q

Radiological features of bowel obstruction

A

SBO - 80% of all mechanical intestinal obstruction
Dilated loops of small bowel proximal to obstruction
Gas fluid levels if AXR if erect

LBO - 20% bowel obstructions
Colonic distension
Collapsed distal colon - few or no air-fluid levels in large bowel as water is reabsorbed
Rectum has little or no air

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19
Q

Mx of intestinal obstruction

A

Immediate stabilisation: NBM, wide bore NGT, IV fluids and AXR
Surgery to remove the obstruction

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20
Q

Encopresis

A

Very loose, smelly stool passed without sensation or awareness

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21
Q

RF for constipation and encopresis

A

Cerebral Palsy
Downs syndrome
ASC
Safeguarding including FII (fabricated or induced illness)

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22
Q

Complications of constipation and encopresis

A

Anal fissure
Haemorrhoids
Rectal prolapse
Megarectum - rectum so dilated there is no sensation
Faecal impaction and soiling requiring disimpaction regime - obstruction
Volvulus
Distress and psychosocial issues (miss school)

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23
Q

Presentation of constipation

A

Less than 3 stools a week
Hard stools, difficult to pass
Rabbit dropping stools
Straining and painful passage
Abdo pain
Retentive posturing
Rectal bleeding
Loss of sensation of need to open bowels

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24
Q

Causes of functional constipation

A

Habitually not opening bowels
Low fibre diet
Poor intake and dehydration
Sedentary lifestyle
Psychosocial problems such as a difficult home or school environment (safeguarding)

25
Red flags for constipation
Not passing meconium within 48h (CF or Hirschprungs) Onset from birth to few weeks of life - Hirschsprungs Distension and vomiting FHx of Hirschsprungs Ribbon stool pattern - anal stenosis Leg weakness or motor delay Abnormal appearance of anus Abnormal lumbosacral and gluteal region
26
Amber flags for Constipation
FTT Developmental delay Coeliac HYpothyroidism CMPA Safeguarding
27
Mx of constipation primary care
Correct any reversible factors e.g high fibre diet and good hydration Start laxatives - 1st line = movicol Faecal impaction disimpaction regimen with high dose laxatives at first Encourage and praise visiting toiler - bowel diary, scheduling visits and star charts Foot stool - for positioning
28
Mx of constipation secondary care
Manual evacuation Polyethylene glycols via NG for whole gut lavage ACE - anterograde colonic enema - enema through appendix or coecal junction Psychosocial + behavioural therapies
29
Disimpaction regime
Escalating dose of macgrocols (movicol/laxido) If fails add stimulant (Senna) If macrogol not tolerated use stimulant with lactulose/docusate Avoid suppositories/enema in primary care
30
Daytime enuresis
Lack of bladder control during the day in a child who is old enough to be continent (over age of 3-5) Nocturnal enuresis is normally present
31
Causes of enuresis
Lack of attention to bladder sensation - may be a developmental, psychogenic problem, may occur in otherwise normal children who are too preoccupied Detrusor muscle instability: sudden, urgent urge to void which is induced by sudden bladder contractions Bladder neck weakness Neuropathic bladder - enlarged and fails to empty properly, assoicated w/ spina bifida UTI Ectopic ureter - constant dripping and child is always damp
32
RF for enuresis
FHx Males Obesity Developmental delay Constipation Faecal impaction Psychological or behavioural disorders Sleep apnoea and upper airway obstruction symptoms
33
Investigations for enuresis
MCS urine sample USS may show bladder pathology XR of spine may show vertebral anomaly MRI spine - tethering of cord
34
Mx of enuresis
Affected children with no neuro cause - star charts, bladder training and pelvic floor exercises Treat any constipation first , then daytime, then nighttime enuresis Small portable alarm with pad in pants - activated by urine - can be used for lack of attention to bladder sensation Anticholinergic drugs - oxybutynin - may be helpful if other measures fail, decrease bladder contractions
35
Causes of secondary enuresis
Emotional upset UTI Polyuria from osmotic diuresis in DM or renal contracting disorder (e.g sickle cell disorder or CKD) Faecal retention severe enough to reduce bladder volume and can cause bladder neck dysfunction
36
Causes of nocturnal enuresis
Genetically determined delay in sphincter competence Small children need to be stress free and have a measure of parental approval in order to learn night-time incontinence Emotional stress can interfere and cause secondary enuresis Underlying stress
37
Mx of nocturnal enuresis
Explain problem is common and beyond control - parents should stop punitive procedures Star chart - wet beds treated as matter of fact and not punished >5y - alarm - rings when start weeing so get up and go toilet - reward system Desmopressin - rapid/short term solution e.g camp Recurs - restart desmopressin plus alarm
38
Acute gastroenteritis
3 or more partially formed or watery stools per day foe less than 14 days
39
Dysentry
loose stool with blood and mucus, pyrexia and abdo cramps
40
Causes of gastroenteritis
Organisms or toxin related Rotavirus - 60% Campylobacter - severe abdo pain Shigella/Salmonella produce dysenteric type of infection with blood and pus in stool, pain and tenesmus Cholera and enterotoxigenic E. Coli infection - profuse, rapidly deteriorating diarrhoea
41
Investigations for gastroenteritis
Usually none Stool culture - if septic, blood or mucus, I.C child, recent travel not improved in 7 days U&Es - creatinine, glucose if IV fluid needed or signs of hypernatraemia Blood culture for abx
42
Mx of gastroenteritis
Antidiarrhoeals not used - ineffective, may prolong bacteria can be S/E Abx rarely needed - sepsis, extra-intestinal spread, malnourished or I.C Abx for: C. diff associated w/ pseudomembranous colitis Cholera Shigella Giardiasis
43
Mx of hypernatraemic dehydration
Difficult to manage ORS If IV fluids too quick - rapid reduction in plasma sodium conc. and osmolarity will lead to a shift in water = cerebral oedema - give fluid over 48h Nutrition - developing countries can lead to malnutrition, may be zinc deficiency and supplementation may be helpful
44
Advice to parents for gastroenteriris
Most managed at home Contact HCP if dehydration sx develop Child is not clinically dehydrated - drink plenty of fluids Don't drink fruit juices and carbonated drinks Give ORS to increased risk Advise that usual diarrhoea is 5-7d Usual vomiting is 1-2d Doesn't resolve within time period contact HCP
45
Importance of recognising UTI in children
Up to 50% have structurally abnormality in UT Pyelonephritis may damage growing kidney by forming a scar, predisposing to HTN and CKD if scarring is bilateral
46
S+S of UTI
classic tria: dysuria, frequency + loin pain in older children fever vomiting lethargy irritability poor feeding FTT abdo pain jaundice haematuria offensive urine
47
Investigations for UTI
Clean catch urine is recommended Not available: urine collection pads or invasive: catheter samples or suprapubic aspiration Infants <3m referred to specialist + MCS sample Use dipstick testing in children >3m + <3y
48
Indications for culture
Suspect acute pyelonephritis High to intermediate risk of serious illness Infants under 3m Positive for leukocyte esterase or nitrite Infants + children with recurrent UTI Infection doesn't respond to treatment within 24-48h Clinical sx + dipstick tests don't correlate
49
Mx of UTI in infants <3m
referral to paeds, treat with parenteral abx use cephalosporin - cefotaxime or ceftriaxone + abx against listeria (amoxicllin)
50
Mx of UTI in 3m-15y
Start abx if leukocyte esterase, nitrates or both are positive Lower: 1. Trimethoprim/Nitrofurantoin 2. Nitrofurantoin/amoxicillin/cefalexin Upper: 1. Oral: cefalexin/co-amoxiclav 1. IV: co-amoxiclav/cefuroxime/ceftraixone
51
Mx of UTI pregnant women 12+
1. Nitrofurantoin 2. Amoxicillin/cefalexin 3. Consult microbiologist
52
S+S of dehydration
Unwell Irritable/lethargic Reduced U/O Pale or mottled skin cold extremities sunken eyes dry mucous membranes/skin turgor tachycardia/tachypnoea low BP Slow crt
53
Hyponatraemic dehydration
Children with diarrhoea drink large quanities of water = greater net loss of sodium than water, leading to fall in plasma sodium Leads to a shift of water from extracellular to intracellular compartements Increase in intracellular volume leads to a greater degree of shock per unit water loss
54
Isonatreamic dehydration
total body deficit of sodium + water losses of sodium + water are proportional and plasma sodium remains within normal range (isonatraemic)
55
Hypernatraemic dehydration
More water loss than to sodium relatively and plasma sodium concentration increases Occurs from high water losses ECF becomes hypertonic with respect to ICF Water shifts into ECF Harder to spot as signs of ECF depletion are less per unit of loss Dangerous as fluid is drawn out of brain and cerebral shrinkage within a rigid skull - jittery movements, increased muscle tone with hyperreflexia, altered consciousness, seizures and multiple, small cerebral haemorrhages
56
Rehydration
ORS mainstay - correction of deficit, maintenance and ongoing losses No dehydration - usual feeds/bottle, ORS supplement (if refuse/don't like taste aren't too dehydrated) no fruit juices or carbonated drinks With signs but at home: <5y 50ml/kg for deficit over 4h + maintenance ORS >5y 200ml ORS after each loose stool plus normal fluid intake Shock: IV therapy, rapid infusion of 0.9% sodium chloride solution
57
Causes of chronic diarrhoea
Toddler diarrhoea - commonest cause of persistent loose stools per day for 4+ weeks Coeliac disease Lactose intolerance - primary (rare) and secondary (after acute gastroenteritis) Functional GI disorders - toddler diarrhoea and IBS IBD Food allergy and intolerances CF Small intestinal bacterial overgrowth Malignancy
58
Toddler diarrhoea
Stools are varying consistency Sometimes well formed and sometimes explosive and loose Presence of undigested vegetables - "peas and carrots diarrhoea" Affected children are gen. well Underlying maturational delay in intestinal motility which leads to intestinal hurry Usually grow out of it by 5, achieving foecal continence Relieve symptoms can be achieved by making sure child's diet has fat and fibre (slows gut transit) Excessive consumption of fresh fruit juice can exacerbate