Chronic Illnesses & Children Flashcards

1
Q

Chronic Illness is a condition that…

A

Interferes with daily functioning for 3+ mths in a year

Hospitalization for + 1mth in a year

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2
Q

What impact does Chronic Illness have on a child?

A

Developmental Aspects
Coping Mechanisms
Responses to Parental Behaviour
Types of illness or disability

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3
Q

What are the 3 adaption and coping STAGES?

A
  1. Shock & Denial
  2. Adjustment
  3. Reintegration and acknoledgment
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4
Q

What behaviours can we expect from someone in the Shock and Denial Stage?

A
Seeking Second Opinions
Minimizing Symptoms
Refusing to believe results
Delaying consent to treatment
Optimism despite diagnoses
Suspicion
Apathy/disinterest
Maladaptive which negatively affects treatment/life of child
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5
Q

What behaviours can we expect from someone in the Adjustment Stage?

A
Guilt
Anger
Over protection
Rejection & Rigidity
Overcompensation
Movement towards acceptance
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6
Q

What behaviours can we expect from someone in the Reintegration & Acknowledgment Stage?

A

Realism, realistic expectations
Becoming an expert
Getting on with life

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7
Q

What are the nursing interventions when caring for a child/family with a chronic illness

A

Provide support/accept families reaction i.e anger, denial..
Therapeutic communication
Education
Provided community resources
Normalization-promote independence
Self care practices: diabetes, meds, insulin admin etc.
Set Realistic future goals.
Assess families strengths and ability to adjust
Assess acceptance of child’s condition (phases)
Support families coping methods
Advocate for empowerment

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8
Q

Define Asthma.

List the S&S and what causes it.

A

Asthma is a common chronic airway disorder marked by:

  • Bronchoconstriction
  • Airway hyper responsiveness to various stimuli

S&S: proxysmal/persistant, DYSPENA, chest tightness WHEEZING, COUGH, and sputum.

Caused by: exposure to allergens, infections, stress, environmental changes

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9
Q

What is Status Asthmaticus? How is it diagnosed?

A

Severe bronchospasm that is not reversible by usual measures. If continues, hypoxemia worsens, more reduction in expiratory flow & effective ventilations decreases = increased acidosis.

Diagnoses: assessment of symptoms, medical history, allergy test, pulmonary function test (PFT’s spirometer, measurement FEVI, FVC=PEFR)

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10
Q

What triggers asthma?

A
Smoke( tabbacco, wood, oil)
Viral respiratory symptoms
Environmental Allergies 
Humidity & Cold Weather
Pollution
Exercise (SOBOE)
Aerosol Sprays
Medications (asprins)
Emotions ( anxiety attacks)
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11
Q

How is Asthma therapeutically managed?

A
Bronchodilators
Corticosteroids/Anti-inflammatory
Relaxation techniques
Manage the triggers
Exercise
Chest physiotherapy
Patient & Parent teaching
Hyposensitization
Airway management by:
      Short & long acting bronchodilators
      Anticholinergic (atrovent)
      Glucocorticoids
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12
Q

List the Controller medications used to manage asthma

A

Controllers:
Inhaled steroids (anti-inflammatory) - pulmicort, flovent
Long acting bronchodilators - servent (salmeterol)
Leukotriene-bronchodilators ( singulair)

Combination: long acting broncho and steroid - advair
( flovent + servent)
* works to prevent asthma episode
* Taken daily
* does not eliminate the need for reliever medications
* inhaled
* Prevents inflammation and controls symptoms
* Will not help during an acute episode

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13
Q

What are the Quick Relief rescue medications that are used to manage asthma? How do they work?

A

Fast acting bronchodilators ( short acting) - ventolin
Oral Steroids - prednisone/prednisolone
* Fast acting
* Relaxes bronchioles & relieves inflammation
* Relieves symptoms during acute episodes
* PRN

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14
Q

What are the goals of asthma therapy?

A

Control chronic and night time symptoms
Maintain the normal activity levels of a child
Prevent acute episodes
Avoid adverse effects of medications

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15
Q

List reasons for poor asthma control?

A

No $ for medications/devices
No understanding of meds / don’t take them correctly
Stopped taking controller b/c symptoms improved
Concerns re: side effects = stop taking
Poor perceiver of S&S
Severe asthmatic

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16
Q

What is involved in Acute Asthma Management?

assessment, history, interventions, eval, education

A

AIRWAY ** VS
ASSESSMENT: respiratory, cardio, other
HISTORY: what were they doing? what triggered attack?
INTERVENTIONS: O2, meds, iv access/hydration
EVALUATION/REASSESSMENT: improving, no wheezing, silent chest, Resp failure/arrest
EDUCATION: asthma symptoms and control; use of devices

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17
Q

Define Sickle Cell Anemia

A

Inherited hematologic disorder, leads to chronic hemolytic anemia & vaso-occlusive episodes

Most common among african decent

Defective hemoglobin molecule (Hb S) causes RBS’s to b/c sickle-shaped in hypoxic states leading to: hemolysis, impaired circulation, pain, inadequate oxygenation and possible tissue infarction

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18
Q

How is Sickle Cell Anemia diagnosed?

A

Newborn Screening ( not diagnostic, just recognized)
Cord Blood Testing
Genetic Testing

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19
Q

What is Sickle Cell Crisis?

A

Acute exacerbation of disorder

Patient presents with pain, swollen extremities

Dactolytic: sickle cell crisis in children ( hand/feet)

6 mths: for typical age of diagnoses b/c the hemoglobins are gone 9 hemoglobin F are replaced with A, so presents)

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20
Q

What triggers Sickle Cell Crisis?

A
Exercise past limit causing hypoxia
High altitude conditions
Dehydration
Infection
Extreme hot/cold
Anaesthesia (without adequate O2/)
Stress
Hypothermia
 *Patient in sickle cell crisis presents with pain*
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21
Q

What complications occur with Sickle Cell Crisis?

A

Splenic Sequestration
(enlarged due to pooling of blood in spleen –> hypovolemic shock)

Acute Chest Crisis
(sickling occurs in chest, impacting oxygenation/ventilation)

Can be life threateningconditons

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22
Q

What are the S&S with Sickle Cell Crisis?

A

Pain
Irritability
Swollen Extremities/Joints
Pale
Fatigue
Jaundice: sickle cells are destroyed which released bilirubin)
Anemia-due to bone marrow inability to keep up
* Sickle Cells last 10-20 days, normal cells last 120 days*

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23
Q

What therapeutic management is involved in sickle cell disease?

A
Hydration
Prophylactic antibiotics (penicillin)
Medications-
Fever
Pain mangament
Education
Psychosocial support child/family
Bone marrow/stem cell transplant
Splenectomy
Sports/extra circular activities**
   *freq. rest periods to lessen chance of sickling
   * Ensure dry after swimming
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24
Q

When caring for a child in sickle cell crisis what is the nursing management that takes place?

A
Pain Management
Oxygen/vitals
Promote Hydration
Monitor I &O
Administer antibiotics- post collection of cultures if febrile
Monitor for complications
Blood transfusion
Physiotherapy consult
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25
Q

Describe Cystic Fibrosis. What is it characterized by?

A

Genetically inherited autosomal recessive disease which affects the respiratory/digestive systems and sweat glands.

Characterized by:
Excessive thick mucous production due to failure of chloride channels to function with obstruct the airways, GI tract and reproductive tract.

Lungs & Pancreas

There is a high concentration of sodium and chloride in secretions rom sweat glands and lacrimal/salvia glands

Common among white ppl

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26
Q

How is Cystic Fibrosis diagnosed?

A
  • Sweat test
  • Newborns diagnosed 1st 2 weeks of life b/c or screening
  • Test absence/presence of enzymes in intestines
  • 3 day fecal fat analysis (assesses digestion)
  • CXR: lung assessment
  • PFT’s ( pulmonary function tests - when older)
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27
Q

What are the S&S of Cystic Fibroses?

A
Affects both lungs and digestive system
Repeated lung infections
Cough with thick mucous
Breathing difficulties
Poor weight gain
Bulky, greasy, foul smelling stool (steatorrhea)
Salty tasting skin
Treatment depends on state of disease/affected organs
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28
Q

What therapeutic management is involved with Cystic Fibroses?

A
Antibiotics (oral/inhaled/IV)
Bronchodilators
Mucolytics
Pancreatic enzymes
Vitamins
Chest Physiotherapy
O2
Diet (high calorie, protein, fat)
Physical activity
Immunizations
Education/support
lung transplantation, heart-lung transplant
Liver transplantation
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29
Q

What is Diabetes Mellitus? What are the 2 types

A

Disorder of carbohydrate metabolism due to absolute or relative deficiency in the hormone insulin.

Type 1 - Juvenile onset / IDDM

  • Genetic predisposition
  • Environmental trigger
  • Occurs post viral illness (autoimmune)
  • Progressive destruction of B-cells

Type 2 - Adult onset/NIDDM

  • Genetic predisposition
  • Environmental factors
  • Sedimentary lifestyle
30
Q

What is involved in therapeutic management for Diabetes Mellitus?

A
EDUCATION!!!
Understand the disease
Insulin therapy ( action, dose, admin)
Blood glucose monitoring
Nutrition/physical activity
Illnesses
Complications
Self-Care habits
31
Q

What Complications can occur with Diabetes Mellitus?

A

HYPOGLYCEMIA

  • insulin overdose
  • missed meal
  • overexertion

HYPERGLYCEMIA/DIABETIC KETOACIDOSIS

  • 20% of cases are new diagnoses diabetics
  • Failure to take insulin
  • insulin pump failure
  • acute stress from trauma, febrile/viral, severe stress
32
Q

What are the Presentations of Diabetes Mellitus?

A

Serum glucose > 11 mmol/L

pH

33
Q

What is Diabetic Ketoacidosis -Diabetic Crisis (DKA)?

A

Complete state of insulin defiancy

Requires:

  • Assessment
  • Administration of insulin
  • Fluids to overcome dehydration & electrolytes replacement
34
Q

What are the Goals for Diabetic Ketoacidosis -Diabetic Crisis (DKA)?

A
  1. Rehydration:
    * Not in shock: 0.9 normal saline, 3-5 ml/kg/hr x 12hrs then re-assess
    * Correct dehydration over 48 hrs
    * Reverse shock if present
  2. Correct pH and Administer Insulin:
    * infusion of 0.1 U/Kg/hr
  3. Restore Potassium Balance
    * add to maintenance fluids of serum K + is 3-5 mmol/ L per hour
  4. Hermiation causes death in DKA (31)
35
Q

Define Inflammatory Bowel Disease. Describe the 2 types

A

Inflammation of the GI tract.

2 types:

  1. Crohn’s Disease: inflammation in any segment of the GI tract from the mouth to anus; most common is the terminal ileum
  2. Ulcerative Colitis: inflammation of large intestine ONLY

Periods marked by remission & exacerbation

36
Q

Describe the Causes/Diagnoses of Crohns Disease

A
Causes:
  * genetic predisposition
  * Autoimmune process
  * Environmental factors
  * Common in caucasian, jews
Diagnoses: 
   * Colonscopy
   * No Cure
37
Q

Describe the Causes/Diagnoses of Ulcerative Colitis

A
Causes:
Genetic predisposition
Bacterial origin, allergic reactions
Autoimmune  process
Environmental
Common in caucasian, jews

Diagnoses:
Sigmoidoscopy: edematous, friable mucosa, abscesses may be present
Only cure - colectomy
Long-term cancer risk

38
Q

What are the symptoms of inflammatory bowel disease?

A
Abdominal Pain
Loss of appetite
Diarrhoea (bloody)
Nausea
Vomiting
Weight loss (food avoidance, malabsorption)
Urgency - stools (UC)
Mouth ulcers
Rashes
Joint Pain
39
Q

What is the Therapeutic Management for inflammatory bowel disease?

A
Pharmacological agents - anti inflam, steroids, immunomodulators
Enternal/parenteral nutrition
Nutrition -low fibre, high-protein/calorie diet with vit/iron
Surgery (UC)
Fluid/Electrolyte  balance
Monitor Weight
Rest
Parent/parent teaching
Psycho-education
Manage complication
40
Q

What does Acute Management for inflammatory bowel disease look like?

A
Pain management
blood transfusion (UC)
I & O
Hemodynamic stability
Skin integrity/care
Manage complications
41
Q

What are the definitions of pain?

A

Its what the patient says it is & exists when the patient says it does.
Its complex and subjective
If has emotional, behaviour & physiological components
An unpleasant sensory & emotional experience associated with actual or potential tissue damage or described in terms of damage.

42
Q

What are the functions of pain?

A

Adaptive function
Protective mechanisms
Past experiences define pain experience

43
Q

What are the child factors affecting pain?

A

Age, development/cognitive level
Culture
Previous experience of pain
Parental presence

44
Q

What are the goals of pediatric pain?

A
Decrease pain and suffering
Minimizing side effects
Increase comfort & well being
Enhances coping abilities
Educate children/families to communicate about pain
45
Q

What are the benefits of effective pain management?

A
  • Relief & comfort
  • less complications related to stress
  • Early mobilization & healing
  • Increases participation in needed diagnostic & therapeutic interventions
  • Reduced negative impact on future medical encounters -memory
  • reduced stress for family members
  • shortened hospital stays & reduced costs
46
Q

What is does the pain assessment QUEST stand for?

A
Q-Question to the child ( 3 + can report pain)
U- Use pain rating scales
E- Evaluate the patients behaviour
S- secure parental input
T- Take cause into account
T-Take action
47
Q

List the different types of pain scales with their correlating ages

A
  1. Neonatal Infant Pain Scale (NIPS)-Preterm 2mths
  2. Premature Infant Pain Scale (PIPP) Pre/full term neonates
  3. FLACC Tool- 2mths-7yrs, non-verbal,cognitively impaired
  4. Faces Pain Scale Revised (FPSR) 5-12 yrs
  5. Non Communicating Children’s Pain Checklist-post preoperative Version (NCCPC-PV) 3-18 yrs who are unable to talk b/c of cognitive impairment or disabilities. Post-op or procedural pain in hospital.
  6. Numerical Rating Scale (NRS) 0-10. 7yrs & later
48
Q

What are the behavioural measures used to assess pain?

A

Used for infants to preschoolers of age 2 yrs
Assessment based on: vocalizations, facial expressions & body movements & positioning
Most reliable for short, sharp, procedural pain
Less reliable for recurrent of chronic pain & older kids
May need to utilze parents
Ex. FLACC, CHEOPS, TPPS, PPRS

49
Q

What does the FLACC test?

A

Tool used to assess child’s behaviour.
Scoring 0-2 ( 0 = good results, 2 = poor results)

Face
Legs
Activity
Cry
Consolability
50
Q

What are the physiological measures used to assess pain?

A

HR, R, BP, O2
Shallow Respiratory Effort
Palmar sweating
Transcutaneous O2
Increased muscle tone
*Physiological measures usually accompany pain
* Not able to distinguish physical response to pain & other stressors to the body
* Useful in infants & non verbal children

51
Q

List the self report measures used for assessing pain.

A

Children can report pain as young as 3yrs
The ability to measure, classify, seriate is possible by 7+ yrs
Ask location/pain quality

Tools used:

OUCHER= number scale for older kids & picture scale for younger kids

Pain thermometers

Numeric Pain Ratings: 8+ “0-10” easy self report.

Word scales & Verbal Analog Scale (VAS): child classifies pain out of 4 categories “ none - alot”

Faces Pain Scale: six cartoon faces- smiling (no pain) - tearful face (worst pain)

(must be culturally valid)

52
Q

What are the Non-opioids used in pain management?

A

Non-Opioids
Acetaminophen: mild to moderate pain (PO, NG, PR)
*10 mg/kg dosage
*Analgesia / antipyretic
*Hepatotoxic if lg doses of given phentoin, pheno, rifampin

NSAIDS: asa, ibuprofen, naproxen, ketorolac (IV)

  • inhibits prostaglandin synthesis
  • Analgesic, anti-inflammatory & antipyretic
53
Q

What Opioids used in pain management? What do they do?

A

Morphine (gold standard)

  • onset 1-3 minutes IV; 30-60 PO
  • Peak effect - 20min IV
  • Duration: 1-3 hrs

Fentanyl

  • Onset 1-3 minutes IV
  • Duration: 1-3hrs

Hydromorphone:
* 5-7 times more potent than morphine

54
Q

What are the side effects of Opioids on the different systems? ( cardio, resp, GI etc)

A

CSN: Analgesia, drowsiness, and sedation, miosis
(constricted pupils), euphoria/ dysphoria, seizures

Cardio: Hypotension (morphine), Bradycardia (fentanyl, morphine), Tachycardia ( meperidine)

Respiratory: Decreased resp rate, tidal vol, SaO2

G.I: Decreased peristalsis-constipation, nausea vomiting

Genitoury: Urinary retention

Skin: Uticarcia, pruitis, wheals, flushing, diaphoresis

55
Q

What are some of the complications that arise from taking opioids?

A

HR/BP: changes-know the patients baseline & monitor VS per NSG guidelines

Nausea/Vomiting: give antiemetics - watch for sedation

Constipation: reduced peristalsis, watch for BM, give colace/lactulose

Urinary Retention: blocks innervation of bladder, monitor I&O, check for distention, catheter if necessary.

Pruitis: from histamine release, itching, rubbing, flushing, give benadryl.

Sedation: Sedation scale q1h, and potentiating medications

56
Q

What are the guidelines with Opioid safety?

A
Controlled substance in Canada
Swift check of narcotic  count
2 RN's to sign dose and waste
Monitor: LOC, SAO2, Resp
Following must be present: self inflating bag and max. O2,suction, Narcan (naloxone) on hand, opioid antagonist.
57
Q

What are the Non-Pharmacological interventions

A
Distraction
Heat & cold
Relaxation
guided imagery
Touch
Containment ( bundling)
Positioning
Non-nutritive sucking
Kangaroo holding
58
Q

Explain the Bromage Sedation Scale:

A

0-Alert
1- Occasionally drowsy, easy to arouse
2- Somnolent, different to arouse
S- Normal sleep, easy to arouse
* producing a calming or sedating effect via admin of drugs
* used for painful diagnostic & therapeutic procedures
* Level of sedation ranges from anxiolysis to general anesthesia*
* Given with analgesia

Goals:
maintain patient safety
minimize variations in vital signs
Enhance cooperation

59
Q

Explain the etiology in childhood cancers

A

*Leading cause of death from disease in children 80% in last decade

60
Q

What are the factors involved in childhood cancers?

A

Genetic basis for some types
*retinoblastoma, neuroblastoma, & wilms tumor

Chromosome abnormalities

Immunodeficient child more likely to devel. various cancers

Environmental Carcinogens

Drug exposure as risk for cancer

61
Q

What are the types of childhood cancers?

A

Cancer of the blood & lymph system
Nervous system
Bone
Other solid tumors

62
Q

Describe Leukemia

A
Peaks in children 2-5yrs
Starts in the stem cell of bone marrow
  * Lymphoid stem cells (ALL)
  * Myeloid stem cells ( acute meyleblastic leukemia)
Some symptoms reflect bone marrow failure:
* Anemia, pallor
* Fever
* brusing, petechiae
* Bone Pain
63
Q

Describe Lymphoma

A

Originate in the lymphoid & hematopoietic systems

Hodgkin Lymphoma:
* older children
* Reed-Sternberg cells
* Primarily involves lymph nodes
* Usually metastasizes to non-nodal or extra lymphatic sites 
       (spleen, liver, bone marrow)

Non-Hodgkin Lymphoma

  • Classified as B-cell, T-cell & NK-cell lymphomas
  • Usually diffuse disease rather than nodular
  • Highly aggressive

Painless enlargement of lymph nodes
Low grade fever, night sweats, anorexia, weight loss, fatigue

64
Q

What are the Clinical Manifestations of Cancer in Children?

A
Pain
Fever
Skin Changes
Anemia
Abdominal/mediastinal mass
Swollen lymphonodes 
Fatigue
Anorexia
Weight loss
Drenching night sweats
Rash
65
Q

What are the diagnostics involved in childhood cancers?

A
Labs
bone marrow aspirate & biopsy
Lumbar puncture
Imaging
Review of symptoms
Physical
66
Q

List the therapy involved in childhood cancers

A
Surgery
Chemo
Radiation
biotherapy 
bone marrow transplant
hematopoietic stem cell transplant
67
Q

What are the side effect of Chemo/Radiation?

A
Infection/neutropenia
Hemorrhage
Anemia
Nausea & vomiting
Altered nutrition
Mucosal ulceration
Fatigue
Neurological problems
Alopecia/skin changes
steroid effects - moon face & mood swings
68
Q

What complications arise with childhood cancers?

A

Mucositis
Fever/neutropenia
Septic shock = organ failure
Tumor lysis syndrome

69
Q

What is involved in the nursing care for a child with cancer?

A
Education re: treatment & disease
Administer chemotherapy
Manage pain/side effects
Prevention on infections
Manage complications of disease
Assess and support coping & child of family

Cessation of Therapy:

  • Importance of follow up after completion of therapy
  • Genetic counseling ( when child reaches adulthood)
  • Pretreatment of sperm bank offered to adolescent boys
70
Q

What should the nurse be aware of with a childhood cancer survivor?

A

There are long term effects of treatment

Psychosocial, cognitive, emotional & physical development may be affected by treatment and the disease

71
Q

What is involved in the bereavement /end of life care in caring for a child with cancer?

What are the goals?

A

Approaches that improve quality of life thru prevention & Relief of suffering
Multidisciplinary approach
Focus on symptom control/support
Treatment options
Decisions making at end of life ( parents, HC team, * child)

Goals:
 * support child/family/care team
 * Advocate wises/empower decisions
 * Collaborate, Communicate, Coordinate
 * Provide consistency/continuity
 * Provide expertise/educate
 * What is he child's awareness of dying?
        Their awareness & reactions
         Developmental age
         Previous knowledge
         Be honest!
72
Q

Describe Grief and Mourning

A

Grief: emotional response to loss, characterized by sadness, sorrow, guilt, anger,depression,shock, anxiety…
Multifaceted- physical, emotional, cognitive changes

Mourning: cultural response to grief

Bereavement: state of having suffered loss

Bereavement-is what happens
Grief- is what one feels in reaction is bereavement
Mourning-is what one does to express grief