class I and II resto Flashcards
most common cause of pulpal inflammation
microbial contamination
most common microbial source
caries
class I caries
lesions that occur in pits and fissures on the facial, lingual, and occlusal surface of M and PM, and lingual surface of max anterior teeth
class II lesion
lesions that occur in the proximal surfaces of posterior teeth
class V lesion
lesions that occur in smooth facial and lingual surfaces in the gingival third of teeth
how far deep should you go into dentin for cavity prep
0.5 mm
depth of amalgam in central area
2.0 mm
if the distance to the wall from the prep is >1.6 mm what should u do
convergent
if the distance to the wall from the prep is < or equal to 1.6 mm what should you do
divergent
what is extension for prevention
sacrificing sound enamel and dentin to place cavity margins into a “caries-immune” site
what does extension for prevention lead to
re-restoration cycle
why doesnt extension for prevention work
- resto is prone to failures
- does not guarantee caries-free
three preventative measures for controlling caries by preventive approach
- fluoride
- oral hygiene
- diet
HAP critical ph
5.5-6
FAP critical pH
4.5
two functions of fluoride
- form FAP crystals (acid resistant, decrease critical pH)
2. attract Ca+2 which attract PO4 ions (speed up remin)
which bacteria is abundant after cavitation
lactobacilli
what bacteria forms carious lesions
strep mutans
class V definition
lesions that occur on smooth facial and lingual surfaces in the gingival third of teeth
fissure caries disease process
- tip of triangle is towards surface
- follows enamel rods, hits dej, then goes down
smooth surface caries disease process
- base of triangle is towards surface
- follows enamel rods, hits dej, then goes down more
when do you do class v with amalgam
- moisture control is a problem
- high caries risk
- all sub gingival cases
occlusal boundary for class v cavity outline is
height of contour
stages of ecological hypothesis
- dynamic stability
- acidogenic
- aciduric
