Clinical Assessment Module 3 Pulm Flashcards
(232 cards)
Health History
what 7 things would you want to ask a patient about if they are presenting with pulm complaint
Chest pain-Wheezing in chest pain ….. No pain fibers in lungs themselves… what hurts is the chest space (parietal space) want to rule out chest pain…. Could be a pleurtis (between ribs and lungs)
Dyspnea- SOB, how does it affect your ADL’s (activity of daily living)
Wheezing
Cough-Cough, productive (wet), sputum (color), how often is the cough
Hemoptysis- coughing up blood (maybe TB or cancer)
Smoking history
Immunization history- flu pneumonia vaccinations
Techniques for exam
Inspection- Rate Rhythm Depth and Effort
Palpation
Percussion
Auscultation- And listen to lungs (normal or adventitious breath sounds)
What is Stridor
- Audible high-pitched wheeze
- Sign of upper airway obstruction in the larynx or trachea
- Something you notice when you walk in the room. Don’t need a stethoscope.
- Common with croup
- More on inspiration than expiration…. Obstruction in upper airways
What does Nasal Flaring represent?
- Represents an increased effort of breathing
- Trying to increase a way that air can get in from having low O2

what does intercostal retractions represent?
- Represents an increased effort of breathing
- Increased lung volume
- When fluid in lungs and body experiences hypoxemia
- Inspect patient without clothes and gown to really see the chest

Tripoding
what three conditions do you commonly see it with?
- Helps to open airway….
- Common in emphysema
- Epiglottitis common
- Haemophilus influenzae
Normal Breathing sound words to use (3)
Vesicular- Usually they are quiet and vesicular (heard more on inspiration)
Bronchial- louder and higher pitches (bronchial) more tracheal mostly on expiration
Bronchovesicular
Adventitious Sounds
Adventitious souds: abnormal sounds
- Crackles
- Wheezes
- Rhonchi
So, do you just listen to the back of the chest for lung sounds?
what are you missing?
NO NO NO NO! If you don’t listen to the front you ARE NOT even listening to the right middle lung….

where do you listen?

Crackles
- Intermittent, Brief
- Nonmusical
- Suggestive of pneumonia or heart failure
- Mostly inspiration
- stepping on dry leaves (crackling tissue paper)
- Loud crackles everywhere — inspect chest (may be hairy or clothes)
Wheezing
- Relatively high-pitched sound with hissing or shrill quality
- Suggestive of narrowed airways, as in asthma, COPD asthma or bronchitis
- Constricted airway- airway smaller as air goes through=wheezing
- You sometimes can here it without auscultation
- Inspiration and expiration
Rhonchi
- Relatively low-pitched sound with snoring quality
- Suggestive of secretions in large airways
- Can ask them to cough and it can maybe clear it
- Usually heard more on expiration
Speical tests: list them (4)
TACTILE FREMITUS
BRONCHOPHONY
EGOPHONY
WHISPERED PECTORILOQUY
Quick info Hoffman listed off :
- See Bates Table 8-5 (p 328)
- Fremitus- transmission of vibration through chest
- If weird stuff going on then you have increased fremitus
- If you hear more profound on one side then that can be where the consolidation is
- Listen anterior and posterior!
- Bronchophony/ egophony/ whispered pectoriloquy = do only one to better help understand consolidattion in lungs of the pt
- Fluid transmits vibration more then air
Tactile Fremitus
when is it increased? when is it decreased?
presence of fluid transmits better through liquid than air
Technique: Use the ulnar surface of the hand and ask the patient to repeat the words “ninety-nine” as the entire posterior thorax is covered
Findings:
Normal lung transmits a palpable vibratory sensation to the chest wall; this is called fremitus
Fremitus becomes more pronounced over areas of consolidation, as in pneumonia (when the normally air-filled parenchyma becomes fluid-filled)
Fremitus is decreased or absent when the transmission of vibrations is impeded by a thick chest wall, an obstructed bronchus, COPD or pleural changes (eg, effusion or air)
Bronchophony
Techniques: Ask the patient to repeat the word “ninety-nine” each time the lung fields are auscultated
Findings:
Normally the lung sounds transmitted through the chest wall are muffled and indistinct
Bronchophony is when the spoken words are louder or more clear as would be the case over consolidation, as in pneumonia
Egophony
Techniques: Ask the patient to repeat the vowel “E” each time the lung fields are auscultated
Findings: Normally the spoken “E” is heard as “E”
If “E” sounds like “A,” egophony is present, seen in lobar consolidation from pneumonia
Whispered Pectoriloquy
Techniques: Ask the patient to whisper “one-two-three” while the lung fields are auscultated
Findings: Normally the whispered words are heard faintly and indistinctly, if at all
Louder, clearer whispered words are called whispered pectoriloquy and represent consolidation, as in pneumonia
CASE 1:
- 14 Y.O. Female w/ no medical Hx presents c/o SOB.
- HPI: Onset 2 weeks prior with SOB and cough when going outside in the cold. Lasts 1-2 hours. Seems to be getting worse. Can’t play outside. Now doesn’t want to go to school.
- Meds – None
- Exam – WD/WN/WF. Sitting on exam table. Anxious.
–V.S. – P-94, RR-32, BP-117/72, T-37, SaO2-97% RA
what do we think?
•Further exam:
- Heart - reg, no murmur
- Lungs – diffuse expiratory wheezes, prolonged expiratory time
- Percussion, egophany, fremitus normal, no whispered pectoriloquy
- Peak flow = 100 L/min (want to see at least 200, so this is bad)
- Abdomen – benign
- Extremities – pulses 2+, No edema
- DDX – COPD, Bronchitis, Asthma, Anxiety/Panic
- Other diagnostics:
- CXR
- PFTs (reversibility)
What we discussed:
(Wheezes start in expiratory and if bad enough go to inspiration
No consolidation because of no whispered pertorlioquy
Bad peak flow—cant breathe out
Such thing as a cardiac wheeze
If someone is faking a wheeze then you hear it more up top vs. where it should come from lower.)
WHAT IS IT: Asthma
Asthma
what are the four categorizations?
what do you treat with?
what airway size does this effect?
why is it harder to exhale and not inhale (as much)?
- Categorization (tells us the treatment)
- Intermittent- comes at certain time with certain stressor
- Mild persistent
- Moderate persistent
- Severe persistent
- Tx:
- Beta agonists- use 10-20 min before going outside
- Short vs. long acting
- Steroids (reduce inflammation—make lumen wider)- then move onto steroids once under control
- Inhaled vs. systemic
- Leukotriene receptor antagonists
- Beta agonists- use 10-20 min before going outside
NOTES:
Airway becomes inflamed but smaller in the middle (becomes thicker) so muscles are are squishing in on airway —- resistance of flow goes up exponentially while inflamed (constricted) airway
Harder to exhale–
Radial traction=
Inhale-diaphragm drops chest wall out and enlarge chest= when that happens the lumen get a tiny bit bigger this is radial traction (pulling from center out)
Exhale-diaphragm rises chest wall closes and diameter closes in pushed towards center (when we exhale you have more constriction) that’s why you hear wheezes on exhale and when its really bad it is on inhale because they can barely exhale
- 62 Y.O. male w/ Hx heart Dz, Diabetes, HTN, presents c/o malaise.
- HPI: Onset 2 days prior with fatigue and non-productive cough. Yesterday developed increasing malaise and fever of 100.4. Admits to chills.
- Meds – metformin, metoprolol, lisinopril, aspirin, atorvastatin
- Exam – WD/WN/WM. Sitting on exam table. Appears ill.
–V.S. – P-94, RR-28, BP-133/85, T-38.2, SaO2-93% RA
– What do you think? –
- Pulse- high- metoprolol
- RR-high– increase o2 with infection
- Temp- fever
- O2- low- low o2 stat suggest pulmonary
- Maybe infection
Further exam:
- Heart - reg, no murmur
- Lungs – Diminshed over LLL
- Percussion dull at L base, no egophany or whispered pectoriloquy, fremitus decreased over LLL
- Abdomen – benign
- Extremities – pulses 2+, No edema
DDX – COPD, Bronchitis, pneumonia, pleural effusion, heart failure
Other diagnostics:
- CXR- yes with this case you should get it … required for pleural effusion diagnosis
- CBC (white count elevated with neutrophils will tell us infection) , sputum Cx, EKG, Troponin-I
COPD- basically asthma on steroids
Bronchitis- no because this is viral so don’t expect fever and malaise
Pneumonia- no because he doesn’t have signs of consolidation
Pleural effusion- possibly
Heart failure- fluid can back up into the lungs but then you would expect crackles and no fever
What is it?
Pleural Effusion - ? empyema
Pleural Effusion vs empyema
what test shoul you do?
what are the two treatment plans depending on the fluid type?
•Tx:
- Thoracentesis with analysis
- Cell count, cytology, culture, chemistries
- ? Antibiotics – yes due to infection
- ? Diuretics– if infection NO – if from heart failure (yes!)
NOTES:
Have to always sample it
Empyema is an exudate
Transudate- fluid has been transmitted through a barrier
Exudates- dumped by something (dead cells dump blood, cancer cells dump, bacterial infections dump puss)
- 62 Y.O. male w/ Hx heart Dz, Diabetes, HTN, presents c/o malaise.
- HPI: Onset 2 days prior with fatigue and cough productive of green sputum. Yesterday developed increasing malaise and fever of 100.4. Admits to chills.
- Meds – metformin, metoprolol, lisinopril, aspirin, atorvastatin
- Exam – WD/WN/WM. Sitting on exam table. Appears ill.
–V.S. – P-94, RR-28, BP-133/85, T-38.2, SaO2-93% RA
– What do you think? –
Green sputum- has productive cough compared to last guy
Pulse- HIGH
RR- high
BP-ok
Temp-high
O2- low
Productive cough usually tells you RESPIRATORY
- Further exam:
- Heart - reg, no murmur
- Lungs – Inspiratory crackles over LLL
- Percussion normal, has some egophany and whispered pectoriloquy, fremitus increased over LLL
- Abdomen – benign
- Extremities – pulses 2+, No edema
- DDX – COPD, Bronchitis, pneumonia, heart failure
- Other diagnostics:
- CXR- YES!!!! NECESSARY TO CONFIRM THE DIAGNOSIS
- CBC (elevated white count), sputum Cx (if you can YES), EKG, Troponin-I
NOTES: HE HAS CONSOLIDATION= CRACKLES
WHAT IS IT: Pneumonia – Likely acute bacterial
Pneumonia – Likely acute bacterial
Tx:
- Antibiotics – Consider appropriate choice- CAP vs. HCAP
- Fluids
- Bronchodilators (if wheezing) /mucolytics (if something needs to be broken up)
- Chest physiotherapy (tap on the chest)
- Flutter valve (helpful to also break things up)
- Oxygen (no O2 with stats in 90’s)
NOTES: COMMUNITY AQUIRED PNEUMONIA (CAP) VS. HEALTH CARE ACQUIRED PNEUMONIA (HCAP)
Has acute infection should have fluids


































































