Conditions Lecture Quiz 1 Flashcards

(55 cards)

1
Q

Occupational Science

A

A basic science; study of occupation; basic science that supports OT (an applied science)

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2
Q

Pharmacology

A

the study of drugs, their actions, dosage, therapeutic uses, and adverse effects

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3
Q

Medication Management

A

An IADL

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4
Q

Cerebral Palsy-developmental disability

A

• Definition: “A group of permanent disorders of the development of movement and posture, causing activity limitation, that are attributed to non-progressive disturbances that occurred in the developing fetal or infant brain. The motor disorders of cerebral palsy are often accompanied by disturbances of sensation, perception, cognition, communication, and behavior; by epilepsy, and by secondary musculoskeletal problems.”

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5
Q

Cerebral=

A

brain

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6
Q

Palsy=

A

weakness/affecting muscles

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7
Q

Etiology of CP

A

Injury or insult to the developing brain; prenatal, perinatal, postnatal; upper age limit for postnatal brain injury is 2-8; 70-80% have congenital CP in womb

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8
Q

Risk factors of CP

A

multiple births; prematurity and low birth weight are most common- high risk for infection, infection cuts off blood supply to brain; maternal infections; acquired post-birth- CVAs, infections (meningitis, jaundice)

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9
Q

4 types of brain injuries that often result in CP DX

A

periventricular leukomalacia, intraventricular hemmorrhage, hypoxic-ischemic encephalopathy, ceerebral dysgenesis

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10
Q

Periventricular leukomalacia

A

damage to white matter around ventricles, leave gaps in babies brain, form fluid-filled cyst, 32 weeks gestation

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11
Q

Intraventricular hemorrhage

A

Hemorrhage into ventricular system premature births b/c blood vessels really week. Level 1 and 2 are minimal, deficits don’t persist into adulthood; level 3 and 4 moderate to severe and lead to CP, 30 weeks gestation

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12
Q

Hypoxic-ischdmic encephalopathy

A

loss of oxygen to brain; damage to brain tissue

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13
Q

Cerebral Dysgenesis

A

brain malformation; baby brain not developed appropriately; associated to births 20 weeks gestation; mother may experience own trauma so go into labor early; infection

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14
Q

Acquired injury after birth- CP

A

cerebrovascular accidents; infections; poisoning; trauma-near drowning, strangulation, child abuse; closed head injuries-concussion, blunt force trauma

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15
Q

Diagnosis for CP

A

no definitive test; several factors considered- injury to developing brain, quality of movements is large factor; non-progressive; disorder in sensorimotor development; disorder originates from the brain; life-long disability

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16
Q

Prevalence of CP

A

most common motor disability in childhood; 1:323 children have it; 10,000 infants; highest prevalence among boys and AA children; 77% have spastic CP; many have co-occurring conditions: 41% with epilepsy and 7% with ASD

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17
Q

Common co-morbidities (two things exist at one time) with CP

A

swallowing difficulties; eye muscle imbalance; speech difficulties; epilepsy; blindness; deafness; urinary incontinence

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18
Q

Sings/Symptoms with CP

A

atypical motor performance: tone abnormalities, reflex abnormalities, postural abnormalities, delayed motor development

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19
Q

Reflex abnormalities with CP

A

retained primitive infant reflexes, delay in acquisition of righting and equilibrium reactions, hypertonicity, hypotonia

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20
Q

Types of CP

A

Spastic, athetoid, and ataxia; can see combinations but most often is Spastic and Athetoid

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21
Q

Spastic CP

A

upper motor neuron injury; deep tendon reflexes (knee jerk) present; hypertonicity; 80% of CP cases; types are subdivided anatomically according to parts of the body that are affected

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22
Q

Athetoid CP / dyskinetic

A

basal ganglia of the brain; involuntary uncontrolled motor movements (slow and writhing); in 10-20% of CP cases

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23
Q

Ataxia CP

A

cerebrum affected; unsteady, difficulty with balance when ambulating; least common; 5-10% of CP cases; hard to slow down, run more than walk

24
Q

Spastic CP characterizations

A

characterized by hypertonicity; retained primitive reflexes in affected areas of the body; slow, restricted movement; contractures: permanent shortening of a muscle/joint-address with splint, adaptive devices

25
Types of Spastic CP
monopleagia, diplegia, hemiplegia, triplegia, quadriplegia
26
Spastic Hemiplegia
involves one entire side of the body; lack of righting and equilibrium reactions on involved side; early signs=asymmetrical hand use during first year or dragging one side of body; prominent 8-12 months
27
Spastic Diplegia
involves both LEs with mild deficits in UEs; LE equilibrium reactions delayed or missing; results in spinal and hip deformities-no strength/endurance; early signs=inability to sit independently and pulling self with arms only, legs stiffly extended should be able to sit alone at 6 months; most children will learn to walk with assistive devices
28
Spastic Quadriplegia
entire body involved; typical presentation = spasticity in flexor muscles of UE and extensor muscles in LE; presence of reflexes; oral musculature affected; likely will have feeding tube; small % will learn to walk
29
Why are postural deformities closely monitored?
incase end up in fixed position, respiration will be affected/lungs
30
Athetosis CP
involuntary movemnt, often fluctuating tone-head, neck, trunk and extremities; not present during sleep; hypermobility present due to fluctuating hypotonicity
31
Ataxia CP
characterized by wide base of support; staggering gait; controlled movements appear clumsy; intention tremors may be present; if cued and thought about too longtremoring starts; hypotonicity
32
Associated disorders with CP
cognitive impairment; seizure disorder; visual/hearing impairments; oral motor deficits; gastrointestinal difficulties; pulmonary complications
33
CP medical management/prognosis
normalizing tone; decreasing spasticity-balcofen, botox-blocks acetylcholine from being released and gives temporary paralysis; orthotics and splinting; average lifespan
34
Gross motor function classification system-CP Test
Level 1: Walks without limitations Level 2: cannot run/jump, may use assistive devices Level 3: stands with no support, sit with little support; use assistive devices for walking Level 4: Walks with use of assistive devices; some support in sitting Level 5: support needed to sit, stand or maintain head/neck control
35
OT Role with CP
varies with age; younger children - gross motor/ fine motor/ speech; school-age children: environment, management of school supplied, continued work on gross motor fine motor and speech; adults: IADLs, ADLs, advocacy
36
Autism Definition
definition: Autism is a complex developmental disability that is the result of a neurological disorder that affects the functioning of the brain, impacting development in the areas of social interaction and communication skills
37
Autism Etiology
o Researchers believe that certain factors are more likely to cause Autism however there is no single documented/proven cause o It is diagnosed based on observed behaviors o Postmortem research, PET scans and MRI’s have revealed structural differences in the brains of those with Autism ♣ Decreased activation of the frontal lobe ♣ Decreased number of Purkinje cells within the cerebellum ♣ Abnormal structure of the amygdala
38
Hypothesized causes of Autism
Vaccinations, genetics, environmental factors-toxin exposure such as lead, diet-gluten
39
Autism abnormalities in brain structure
o Increased Head circumference: typically between ages 3-4 is noted; pediatricians measure based on age o Cerebellum: structural differences; role in response to sensory discrimination o Purkinje cells: neurons found within cerebellum; these individuals have fewer cells in general; near surface important for coordinating and sending messages to other areas of brain o Inferior olives: bonded to the purkinje cells; happens 28-30 weeks gestation; when a purkinje cell dies, the olive dies with it; used for timing and learning of movements o Frontal lobe: increased in size; responsible for executive functioning; decreased activation o Limbic system: emotional control regulation is impacted; amygdala and hippocampus are abnormal in their structure
40
Autism Genetics
o Genes are the brains written instructions for function o Sibling studies: monozygotic (identical) twins (95% chance of other) (10% in fraternal twins) o Second child having ASD is 3-7% if the first is male with ASD, 7-14% if the first child is female with ASD o Higher rate of speech and language disorders o Relatives more commonly display traits of ASD (anxiety, aloofness)
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Autism environment
o External influences that may cause damage to a person’s internal system if exposed during a period of critical development ♣ Exposure to viruses, infections, drugs/alcohol, high levels of mercury
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Autism psychologic abnormalities
o Gastrointestinal disorder | ♣ Reflux, gastritis, persistent gas, diarrhea and constipation
43
Autism signs/symptoms
``` o Difficulty with social communication o Restricted and repetitive behaviors o Motor abnormalities o Sensory processing deficits o Feeding disorders o Co-occurring medical conditions- ADHD, anxiety, epilepsy, down syndrome ```
44
Autism prevalence and incidence
o ASD impacts approximately 1% of the world’s population o 1 in every 59 children o Fastest growing developmental disability o Occurs in all racial, ethnic, and socioeconomic groups o 4.5 times more common in boys (1 in 42) than girls (1 in 189) o there has been a 120% increase since 2000
45
Autism Meet diagnostic criteria
Must meet A, B, C, and D
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Autism Diagnostic Criteria A
o A. Persistent deficits in social communication and social interaction across contexts, not accounted for by general developmental delays, and manifest by all 3 of the following: ♣ Deficits in social-emotional reciprocity-initiate and respond ♣ Deficits in nonverbal communicative behaviors used for social interaction ♣ Deficits in developing and maintaining relationships
47
Autism Diagnostic Criteria B
o B. Restricted, repetitive patterns of behaviors, interests or activities as manifested by at least two of the following: ♣ Stereotyped or repetitive speech, motor movements or use of objects ♣ Excessive adherence to routines, ritualized patterns of verbal or nonverbal behaviors, or excessive resistance to change ♣ Highly restricted, fixated interests that are abnormal in intensity of focus ♣ Hyper-or hypo-reactivity to sensory input or unusual interest in sensory aspects of the environment
48
Autism Diagnostic Criteria C
o C. Symptoms must be present in early childhood (but may not become fully manifest until social demands exceed limited capacities) o D. Symptoms together limit and impair everyday functioning o E. These disturbances are not better explained by intellectual disability or global developmental delay
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Autism Diagnostic Criteria D
Symptoms together limit and impair everyday functioning
50
Autism diagnostic criteria e
o E. These disturbances are not better explained by intellectual disability or global developmental delay
51
DSM V
♣ DSM-5, p. 51 ♣ ” Note: Individuals with a well-established DSM-IV diagnosis of autistic disorder, Asperger’s disorder or pervasive developmental disorder not otherwise specified should be given the diagnosis of autism spectrum disorder. Individuals who have marked deficits in social communication but whose symptoms do not otherwise meet criteria for autism spectrum disorder, should be evaluated for social (pragmatic) communication disorder.” ♣ A comprehensive diagnostic report should specify the following: ♣ With or without accompanying intellectual impairment ♣ With or without accompanying language impairment ♣ Associated with a known medical condition, genetic condition or environmental factor ♣ Associated with another neurodevelopmental, mental or behavioral disorder
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Autism Psychiatric differential diagnosis (others)
o Rett Syndrome (gene mutation disorder diagnosed by blood testing)-muscle atrophy o Selective mutism o Social (pragmatic) Communication Disorder o Intellectual Disability o Stereotypic Movement Disorder o Attention deficits/Hyperactivity Disorder o Schizophrenia
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Severity level 1 of ASD
♣ Still inflexible but do not have coping skills to manage unexpected changes; can communicate but still lack socio-emotional reciprocity; can be employed
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Severity level 2 of ASD
♣ Can speak in full sentences – not functional for formal communication; can cope in majority of context
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Severity level 3 of ASD
♣ Deficits in social communication; extreme inflexibility; extreme difficulty with coping skills; limited words in communication; no basic imitation – required/building block for communication; deficits affect them across all contexts (school, home, community)