Cranial Nerves Flashcards

0
Q

Why do you not use ammonia to test for olfaction?

A

it can be recognized by nasal epithelium and does not require an intact olfactory pathway.

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1
Q

cranial nerve nuclei are found mainly where?

A

at the level of the brainstem

sensory nuclei develop within the dorsal/alar plate (of neuro tube), motor nuclei develop within the basal plate

in the hindbrain, the alar plate lies lateral to the basal plate in the floor of the 4th ventricle

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2
Q

Is CN I a peripheral nerve?

A

not really. its a central nervous system tract terminating in the olfactory bulb

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3
Q

How do you perceive a particular smell?

A

true olfactory nerves (second order ganglion nerve cells) terminate in the mucous membrane that lines the nasal cavity. to perceive a smell, molecules must dissolve into the mucous overlying the cribriform plate and supply chemical stimulation to the nerve endings at that location.

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4
Q

anosmia

A

complete loss of smell
usually not indicative of a cortical lesion, but mainly associated with viral infections, allergic rhinitis, aging, or head trauma, rhinorrhea (CSF drainage), and backwash meningitis

sense of smell may return eventually

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5
Q

lesion of what gyrus can cause hallucinations of smell?

A

lesion of the uncinate gyrus of the anterior temporal lobe
hallucinations of smell can be associated with deja vu

termed uncinate fits or seizures

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6
Q

hyposmia

A

decreased sense of smell

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7
Q

hyperosmia

A

increased sense of smell

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8
Q

parosmia

A

perversion of smell

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9
Q

cacosmia

A

abnormally disagreeable smell

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10
Q

anosmia in both nostrils

A

common causes: blocked nasal passage, common cold (MC), trauma, and a relative loss with normal aging.

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11
Q

Is CN I a true nerve?

A

No, its a fiber tract of the brain.

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12
Q

Is CN II a true nerve?

A

no, it is a fiber tract of the brain

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13
Q

Meyers loop?

A

fan-like radiating portion of optic nerve that curves around the inferior horn of the lateral ventricle

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14
Q

geniculocalcarine tract?

A

contains 4th order neurons of optic tract from the LGN and passes to the occipital (calcarine) cortex

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15
Q

Central connections of optic nerve

A
  1. from pretectal region –> Edinger westphal nucleus via posterior commisure
  2. from superior colliculi –> other cranial and spinal nuclei via tectobulbar and tectospinal tracts
  3. from occipital cortex–> other cortical and subcortical areas
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16
Q

Pretectal area of CN II

A

simple and consensual light reflexes

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17
Q

connections from superior colliculi from CN II

A

involuntary musculoskeletal refexes

reflex movements of the eye and head after optic stimulation

18
Q

genniculocalcarine tract (from LGN) from CN II

A

visual perception

19
Q

through the tectobulbar and tectospinal tracts of CN II

A

through cranial and spinal nuclei–> involuntary reflexes (ex. accomodation)

through pontine nuclei–> corticopontine tract–> postural reflexes

20
Q

macula

A

retinal area of central vision

21
Q

fovea centralis

A

small central pit composed of closely packed cones, where vision is sharpest and color discrimination most acute.

22
Q

rods and cones of retina react specifically to what?

A

physical light

23
Q

Cones are responsible for what?

A

sharp vision and color discrimination

24
rods are responsible for what?
low intensity light vision and night vision
25
retrobulbar neuritis
lesion of optic nerve or tract | MC cause is multiple sclerosis
26
optic or bulbar neuritis
lesion which includes various forms of retinitis
27
papilledema (AKA "choked disc")
commonly seen symptom of increased intracranial pressure due to brain tumors, abscesses, hemorrhage, hypertension, and other causes
28
optic atrophy
decreased visual acuity and change in color of optic dis to light pink, white, or gray
29
primary optic atropy
caused by processes that involve the optic nerve and do not produce papilledema
30
secondary optic atrophy
a sequal of papilledema
31
primary (simple) optic atrophy
may be due to tabes dorsalis, multiple sclerosis, or hereditary
32
secondary optic atrophy
may be due to neuritis, glaucoma, or increased intracranial pressure
33
Foster Kennedy Syndrome
may be caused by tumors at the base of the frontal lobe and is characterized by ipsilateral blindness and anosmia (with atrophy of optic and olfactory nerve) and contralateral papilledema
34
amaurotic familial idiocy (AKA Tay-Sachs disease)
cerebromacular degeneration with severe mental deficiency occurring in Jewish families and is associated with blindness, optic atrophy, and dark cherry red spot in place of the macula lutea
35
Argyll Robertson pupil
reacts only to accommodation neither a direct or indirect reaction to light occurs mostly as a diabetic complication
36
Holmes-Adie Syndrome
tonic pupillary reaction and the absence of one or more tendon reflexes. pupil is said to be "myotonic", with a very slow, almost imperceptible contraction to light and in near vision, a slower dilation upon removal of the stimuli. People with this syndrome have an abnormal sensitivity to weak solution (~25% methacholine): instilled into the conjunctival sac is demonstrable in affected eyes; tonic pupils constrict, whereas pupils of normal eyes remain unaffected (ADLER-SCHEIE TEST)
37
heterotropia
deviation of bilateral eye alignment
38
Exotropia
deviation of eyes outward/lateral
39
Esotropia
deviation of eyes inward/medial
40
hypertropia
deviation of eyes upward
41
hypotropia
deviation of eyes downward
42
yoked movement
the medial longitudinal fasciculus (MLF) primary function is to coordinate eye movements by interconnecting the nuclei of CN III, IV, and VI