Cranial Nerves: History + Exam Flashcards

1
Q

What associated symptoms should be asked about in the SOCRATES review of a headache?

A
n&v
altered consciousness
rash
pyrexia
neck stiffness
photophobia
visual loss, blurred vision
aura, seizures
tender scalp
malaise
rhinorrhoea, lacrimation
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2
Q

What conditions might be in the differential diagnosis if symptoms are relapsing and recurring?

A

migraine
epilepsy
MS

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3
Q

What exacerbating/relieving should be asked about in the SOCRATES review of a headache?

A

exacerbating: noise, stress, bending, standing up, coughing, sneezing, blowing nose, eating, combing hair, bright/flashing lights, certain foods/drugs, dehydration
relieving: analgesia, dark environment, lying down, rest/sleep

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4
Q

What qs are important to ask in the history of an episode of altered consciousness?

A

describe in own words

onset - gradual/sudden? time of day?
what were they doing at the time? - pain, infections, hot crowded room, emotional stress, prolonged standing?
how they felt before the episode?

associated symptoms

recovery time? amnesia, aggression, crying or weakness afterwards? feeling sad/crying after episode?
previous episodes? were they like this one?
was it witnessed? what did they say, can we contact them?

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5
Q

What associated symptoms should be asked about in the history of an episode of altered consciousness?

A

dizziness, nausea, vertigo, aura, tachycardia, sweating, weakness, paraestoesia, slurred speech, headache, tongue biting/incontinence, stiffening/jerking of limbs, awareness/responsiveness during episode, eyes open or closed, groans or crying?

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6
Q

What is the most important factor to ask about any neurological symptom?

A

course

sudden in onset? how long to reach peak of symptoms? getting better or worse? static/progressive/relapsing + remitting?

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7
Q

What conditions should be specifically asked about in the PMH?

A
head/spinal trauma 
metabolic/endocrine disorders e.g. diabetes
cancer, mets?
epilepsy
HTN
AF
heart diseases
previous episodes?
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8
Q

What medications should be specifically asked about in the drug history?

A
anticonvulsants
drugs that interact with anticonvulsants/lower seizure threshold
anticoagulants/anti-platelets
analgesics
antihypertensives
antidepressants
insulin
recreational drugs
over the counter drugs
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9
Q

Whats factors are important to ask about in the social history?

A

alcohol consumption
smoking
recreational drugs
occupation
social activities/hobbies/daily living/driving
home circumstances/independence/family support/housing

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10
Q

Why is alcohol important to ask about?

A
can cause:
seizures
neuropathy
ataxia
Wernick-Korsakoffe syndrome (lack of thiamine)
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11
Q

What conditions should be specifically asked about in the family history?

A
diabetes
cerebral haemorrhage
cerebrovascular disease/stroke
ischaemic heart disease 
migraine
epilepsy
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12
Q

What 4 things need to be tested in a neurological exam?

A
  1. walking
  2. cranial nerves
  3. motor system
  4. sensory system
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13
Q

What does a Parkinson’s gait look like?

A
stooped posture, rigidity
small shuffling steps
wrists, elbows, hips and knees flexed
no arm swing
resting tremor
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14
Q

What does a hemiplegic gait look like?

A

weakness on one side > weak arm hands, drags 1 leg behind

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15
Q

What does a scissoring gait look like?

A

knees can flex and knock together

restricted stiff movement

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16
Q

What can cause a scissoring gait?

A

any condition causing spasticity

e.g. cerebral palsy

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17
Q

What does a steppage gait look like?

A

foot drops and toes drag so hips and knees flex excessively (high stepping)
foot stamps

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18
Q

What can cause a steppage gait?

A

common perineal nerve palsy

= weakness of dorsiflexion > high step to avoid falling

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19
Q

How is gait examined?

A
  1. normal heel to toe walk - look for posture, balance, stride length, arm swing
  2. tandem walk test - look for same things
  3. Romberg test - stand feet together with eyes closed, dr stands close, +ve = fall
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20
Q

What does a +ve Romberg test indicate?

A

proprioception problem

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21
Q

What is the first step in a cranial nerve examination?

A

general inspection of patient’s head and neck

look for: scars, neurofibromas, facial asymmetry, ptosis, proptosis, skew deviation of the eyes, inequality of the pupils

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22
Q

How is CN I: olfactory nerve examined?

A

ask if they have had any alteration in their sense of smell/taste > only test further if they report problems

olfactory testing bottles/easily recognised scents e.g. soap, coffee
test each nostril separately, occluding the other one with your finger

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23
Q

How is CN II: optic nerve examined?

A
  1. visual acuity
    - snellen chart if available
    - if not: cover one eye, ask them to state how many fingers you’re holding up/different font sizes from a newspaper
    - wear glasses if they usually do
    - if no glasses: pinhole test > won’t correct acuity in people with an optic nerve issue
  2. visual fields
    - sit 1m opposite pt with eyes at same level
    - cover one of own eyes, pt covers eye opposite
    - ask pt to stare into your other eye
    - bring object/finger from point outside visual field diagonally into visual field
    - ask them to signal when they first see it
    - repeat in all 4 quadrants for each eye
  3. visual inattention/visual extinction
    - hold hands up in periphery of pts vision
    - move tip of index finger up and down on one side at a time, then on both sides together
    - ask pt to report which side is moving
    - visual inattention/extinc = can detect if one is moving individually, ignores one side when both move at same time
  4. direct and consensual light reflexes
    - pt looks straight ahead
    - bring light in from side so pt doesn’t focus on it
    - shine light into eye and look for constriction of that pupil (direct light reflex) and the contralateral one (consensual light reflex) = both constrict
  • check relative afferent pupillary defect
  • swing flashlight between eyes > should remain constricted
  • if one dilates = RAPD (problem in ipsilateral optic nerve)
  1. accommodation
    - pt focuses on a distant point, then your finger, held - 30cm in front of their nose
    - normal = constriction of both pupils
  2. ophthalmoscopy, testing colour vision, assessing size of blind spot
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24
Q

On fundoscopy, what might a lost red reflex indicate?

A

cataracts

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25
Q

A lesion in the left optic nerve will cause what kind of visual field defect?

A

left eye vision loss (ipsilateral)

26
Q

A lesion in the optic chiasm will cause what kind of visual field defect?

A

bitemporal hemianopia

outer fields of both eyes

27
Q

A lesion in the right optic tracts will cause what kind of visual field defect?

A

left homonymous hemianopia (contralateral)

left field of both eyes lost

28
Q

A lesion in the left optic radiations will cause what kind of visual field defect?

A

right homonymous hemianopia with macular sparing (contralateral)
(right field of both eyes lost with circle in middle spared)

29
Q

What are the 3 symptoms of Horner’s syndrome?

A

ptosis (drooping of eyelid)
miosis (constriction of pupil)
anhydrosis
= on affected side

30
Q

What happens to the eye in 3rd nerve palsy?

A

ipsilateral:
eyeball deviated out + down (superior rectus palsy)
mydriasis (pupils dilated)
ptosis > eye looks closed

31
Q

What happens to the eye in 4th nerve palsy?

A

ipsilateral eye elevates (superior oblique palsy)

contralateral head tilt to lessen diplopia

32
Q

If both eyelids are closed, what condition is it more likely to be?

A

something affecting the muscles

myasthenia?

33
Q

How are CN III: oculomotor, IV: trochlear and VI: abducens nerves examined?

A
  • ask pt to keep head still and follow finger with their eyes (midway between you and pt)
  • trace a large H shape and central I shape
  • take pts eyes to limit of their gaze as you do
  • ask if they experience diplopia at any point
  • observe for dysconjugate eye movements, nystagmus
34
Q

How is CN V: trigeminal nerve examined?

A
  1. sensation in skin
    - demonstrate stimulus (cotton wool) on sternum withers open, check they can feel it
    - pt closes eyes, check they can feel it on each dermatome of CN V
    - can they feel it, is there a difference in the sensation between the 2 sides of the body?
  2. motor component
    - clench teeth + palpate contraction of masseter and temporalis muscles
    - pt to open mouth against resistance: look for jaw deviation > towards side of paralysed pterygoid
  3. jaw jerk
    - ask pt to half open mouth
    - put index finger vertically over midline of lower jaw
    - tap on index finger with tendon hammer
    - sudden brisk closure > UMN lesion above pons
  4. corneal reflex - not usually done
    - touch eye with cotton wool, normal = involuntary blinking
35
Q

What does CN V innervate?

A

sensory: facial + corneal sensation
motor: jaw closure, mastication

36
Q

What are the 3 branches of CN V called?

A

ophthalmic
maxillary
mandibular

37
Q

On opening against resistance, the pt’s jaw deviates to the right. What is this a sign of?

A

right paralysed pterygoid

= CN V palsy

38
Q

Pt presents with a brisk jaw jerk. What might this indicate?

A

UMN lesion above pons

39
Q

How is CN VII: facial nerve examined?

A

look for asymmetry/appearance of face, especially forehead, obicularis oculi muscles

  1. screw up eye, don’t let them be opened by you
  2. puff out cheeks, maintain on your resistance
  3. raise eyebrows > wrinkles indicate intact nerve supply, no elevation on side of UMN lesion
  4. purse lips
  5. show their teeth, count teeth on each side, weak side = more covered
40
Q

What kind of UMN supply do muscles of the upper face have compared to muscles of the lower face? What affect does this have on the impact of a lesion?

A
upper face (frontal, obicularis oculi): bilateral UMN supply, can be spared in UMN lesions e.g. stroke
= forehead sparing on side of lesion always 

lower face: unilateral UMN supply, weakened on the side of the UMN lesion
= BELL’S PALSY

41
Q

Pt presents with unilateral loss of taste and hyperacusis on the same side. What does this indicate?

A

LMN lesion

42
Q

What can trigger trigeminal neuralgia?

A

cold, touch, shivering, shaving, eating, drinking

43
Q

What is Bell’s palsy?

A

lower VII lesion, all facial muscles of affected side are weak
can be due to a UMN or LMN lesion

44
Q

A pt presents with facial drop/asymmetry. Is the lesion in the brain or the nerve itself?

A

if in the brain > UMN lesion e.g. stroke

if in the nerve itself > LMN lesion

45
Q

How does Bell’s palsy due to a UMN lesion present?

A

forehead sparing > can close eye, wrinkles seen
affects the CONTRALATERAL side
facial muscle weakness in lower half of face

due to bilateral UMN innervation

46
Q

How does Bell’s palsy due to a LMN lesion present?

A

all facial muscles affected, no forehead wrinkles, facial droop
can’t close eye, see whites of eye
affects IPSILATERAL SIDE

47
Q

How is CN VIII: vestibulocochlear nerve examined?

A
  1. cover opposite ear with hand, whisper a number to the pt, ask them to repeat it

abnormality suspected?
2. Rinnes and Weber’s tests to determine if it’s a sensory or conductive defect

  1. dizziness = common symptom
    - vestibule ocular reflex/Hall pike and Unterberg could be done
    • vor: pt focus on your nose, jerk head fast to L/R for them, their eyes should stay focussed on your nose
    • peripheral problem = eyes move in same direction as jerk then do a corrective jerk/nystagmus
48
Q

How are CN IX: glossopharyngeal and CN X: vagus nerves examined?

A
  1. ask pt to open their mouth wide, is the uvula in the midline at rest?
  2. ask pt to say ‘aah’, note asymmetry of movement
    > uvula will deviate away from the CN IX/X palsy
  3. does pt have any difficult swallowing?
    (gag reflex doesn’t need to be performed routinely)
  4. observe drinking a sip of water > problem if they cough/choke
  5. ask pt to cough > bovine (non-explosive, weak) cough = vagal palsy
  6. note any hoarseness of the voice
49
Q

A weak bovine cough indicates a lesion in which CN?

A

X

due to inability to close glottis

50
Q

Pt presents with uvula deviation to the left. Which nerve has been affected?

A

right vagus

pulling muscles so the working muscles pull it to their side

51
Q

How is CN XI: accessory nerve examined?

A
  1. test trapezius: shrug shoulders against resistance
    > look at back for asymmetry/scapula winging
  2. test sternocleidomastoid: turn head against resistance, palpate during
52
Q

What does CN XI innervate?

A

trapezius + sternocleidomastoid

53
Q

Pt presents with winging of the scapula. Why might this happen?

A

weakness of trapezius due to VIth nerve palsy

54
Q

How is CN XII: hypoglossal nerve examined?

A
  1. ask pt to open mouth and observe tongue for fasciculation
  2. protrude tongue, any deviation?
    > deviation occurs towards the side of a lesion
  3. ask pt to push tongue into their cheek against the resistance of your finger, assess power
55
Q

What signs are seen in CN XII palsy?

A

ipsilateral atrophy/wasting

deviation towards side of lesion

56
Q

What is Rinne’s test? What are the findings in conductive hearing loss compared to sensorineural hearing loss?

A
  • tuning fork held on mastoid until sound is no longer heard
  • then held near external acoustic meatus, sound should continue to be heard as air > bone conduction

conductive hearing loss: bone conduction > air conduction, sound not heard at the EAM

sensorineural hearing loss: air + bone conduction decreased by similar amount

57
Q

What is Weber’s test? What are the findings in conductive hearing loss compared to sensorineural hearing loss?

A
  • tuning fork held against forehead in midline
  • vibrations normally perceived equally in both ears

conductive hearing loss: sound louder in abnormal ear
sensorineural hearing loss: sound louder in normal ear
sensitivity increased by pt blocking external ear canals with fingers

if +ve > do rinne’s test

58
Q

How is meningism tested for? What is it’s pathognomonic feature? What can it indicate?

A
  1. ask about neck pain > whilst lying, touch chin to chest
  2. you support head with hands, move back and forth > check for stiffness and pain (incl abdominal pain)

= won’t affect SIDE TO SIDE movement (must be MSK)

indicates meningitis, SAH

59
Q

How does an UMN lesion present?

A

everything INCREASES
part of the motor system in the brain + spinal cord

increased spastic tone
pyramidal pattern of weakness
increased reflexes

60
Q

What 2 types of increased muscle tone are there?

A
  1. spasticity
    - first increase then decrease in tone
    - typical for UMN lesions
  2. rigidity
    - increased tone over entire radius of joint movement
    - present in parkinson’s
61
Q

How does an LMN lesion present?

A

everything DECREASES
in the PNS > horn, roots, plexus

decreases muscle mass (atrophy + fasciculations - check tongue)
decreased tone
peripheral pattern of weakness
decreased reflexes