Crawford - Ill-Appearing Flashcards

(34 cards)

1
Q

What is done in primary survey?

A

ABCDE = airway, breathing, circulation, disability, expose

Address only what’s going to kill them in the next 5 minutes?

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2
Q

What is Sellick’s maneuver in airway treatment?

A

pinch cricoid cartilage to decrease gastric insufflation

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3
Q

Two components of breathing that need to be assessed in primary survey?

A

oxygenation: O2 sat
ventilation: end tidal CO2

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4
Q

How to optimize oxygen content in hypoxic patient?

A
  • supplemental oxygen
  • increase PEEP with mechanical ventilation or needle decompression
  • increase hemoglobin
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5
Q

What treatments can keep BP low even if circulation needs to increase?

A

beta blockers

pacemaker

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6
Q

Hypotension following injury is due to _________ until proven otherwise.

A

hypovolemia

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7
Q

CO = ____ x _____

A

SV x HR

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8
Q

What medications will increase heart rate? (chronotropy)

A

Non-selective adrenergic agonists - epinephrine

Anticholinergics - atropine

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9
Q

What medications will increase stroke volume? (inotropy)?

A

Adrenergic agonists – epinephrine, dobutamine

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10
Q

What medications will increase stroke volume without increasing heart rate? (pure inotropy)

A

Digoxin

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11
Q

What medications will constrict blood vessels?

A

Adrenergic agonists
Epinephrine
Norepinephrine
Vasopressin

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12
Q

What medications will dilate blood vessels?

A

Nitroglycerin
Nitroprusside
Calcium channel blockers

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13
Q

What is assessed in the D (disability) of the primary survey?

A

LOC: determine cause (hypoxia, cerebral injury, med/drug), GCS

Neurologically intact?: sensation and movement along dermatomes

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14
Q

What is important during E (exposure) step of primary survey?

A

Keep patient warm to prevent hypothermia (trauma triad)

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15
Q

Normal urine output that indicates adequate renal perfusion?

A

0.5 - 1 mL/kg/hr

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16
Q

End organ signs of shock

A
Altered mental status
High HR and RR
Low urine output
Low arterial pH
Low BP (specifically MAP)
17
Q

Basic pathophysiology of shock and why it occurs

A

inadequate tissue perfusion; not enough oxygen

3 components: blood (fluid), heart (pump), blood vessels (pipes)

18
Q

“warm” vs “cold” shock

A

early/compensated shock is warm - sympathetic activation (vasoconstriction, high HR and RR)

later decompensated shock patient becomes cold/clammy and hypotensive

19
Q

Different classifications of shock

A

Distributive
Hypovolemic
Obstructive
Cardiogenic

20
Q

Causes of obstructive shock

A

Massive PE
Tension pneumothorax
Cardiac tamponade
Constrictive pericarditis

21
Q

BP = _____ x ______

A

BP = SVR x CO

22
Q

Pathophysiology of Low SVR

A

things that cause vasodilation - septic, neurogenic, and anaphylactic shock

BVs are big and leaky, so blood sits in periphery and heart overworks to compensate

23
Q

Signs of low SVR

A

WARM
pink
bounding pulses
hyper dynamic heart (fast and pounding)

24
Q

Signs of high SVR

A

Cool extremities
Pale
Poor cap refill
Thready pulses (narrow pulse pressure due to high diastolic BP)

25
Stroke volume depends on what 3 factors? How do they cause shock?
preload = hypovolemic shock if too low afterload (aortic resistance) = obstructive shock if too high contractility = cardiogenic shock
26
Management priorities in hypoperfused states
1) Volume: Fluids to get to CVP 10 2) BP: Vasopressors to get to MAP 60 and SBP 90 3) Flow: inotropes for improved signs of perfusion
27
Why must you improve circulating volume before adding a vasopressor?
Vasopressors risk tachycardia and increased myocardial oxygen consumption if used too soon
28
Signs of 15-35% blood loss
Increased HR and DBP (narrow pp), prolonged cap refill, flat neck veins
29
Symptoms of cardiogenic shock
- Skin: cool, moist, pale with mottling - CHF signs: JVD, HJR, APE, pedal edema - Heart: murmurs, S3 or S4 - Pulse: rapid and thready/weak - Decreased BP - Decreased urine output
30
Treatment of cariogenic shock
Increase oxygen: mechanical ventilation, reperfusion Maximize cardiac output: dysrhythmics, pacing, cardioversion, vasopressors, inotropes Decrease afterload: intra-aortic balloon pump
31
Vascular changes in anaphylactic shock
- Diffuse vasodilation from histamine and bradykinin release | - Bronchoconstriction
32
Treatment of anaphylactic shock
1) ABCs 2) Epinephrine 3) Steroids, H2 blockers, Albuterol, etc.
33
What is septic shock?
multiple organ system inflammation causing hypoperfusion SIRS -> sepsis -> severe sepsis -> septic shock SIRS criteria + source of infection + hypotension
34
Septic shock treatment
Prompt volume replacement Early antibiotic administration If MAP