CV Flashcards

(17 cards)

1
Q

How do TAAs present?

A
  • dilation of thoracic aorta = asymptomatic
  • chest or back pain
  • trachea/L bronchus compression > cough, shortness of breath, stridor
  • same nerve compressions as pericardial effusion
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2
Q

How do ruptured aortic aneurysms present?

A
  • severe chest/back pain (TAA)
  • abdo pain radiating to back/groin (AAA)
  • shock (hypotension, tachy)
  • collapse
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3
Q

What is the aetiology of AAAs?

A
  • coarctation of the aorta
  • Marfan’s syndrome
  • aortic surgery
  • 3rd trimester pregnancy
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4
Q

How are ruptured aortic aneurysms treated?

A
  • permissive hypotension to decrease blood loss
  • immediate vascular review and emergency surgery
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5
Q

How does a DVT present?

A
  • unilateral calf pain and tenderness along deep vein line
  • swelling >3cm
  • erythema
  • pitting oedema
  • superficial veins distended
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6
Q

What are risk factors for DVT?

A
  • immobility
  • recent surgery
  • long haul travel
  • pregnancy
  • oestrogen (HRT and cocp)
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7
Q

What is syncope?

A
  • transient loss of consciousness
  • due to global cerebral hypoperfusion
  • rapid onset, short duration, spontaneous complete recovery
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8
Q

What are the causes of reflex syncope?

A
  • vasovagal: triggered by emotion, pain, stress (fainting
  • situational
  • carotid sinus syncope
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9
Q

What are the causes of orthostatic syncope?

A
  • primary autonomic failure: Parkinson’s
  • 2º autonomic failure: diabetic nephropathy
  • drug-induced: diuretics, vasodilators
  • volume depletion: haemorrhage, diarrhoea
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10
Q

What are the causes of cardiac syncope?

A
  • arrhythmia
  • structural
  • PE
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11
Q

How is syncope investigated?

A
  • CV exam
  • postural BP readings
  • ECG
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12
Q

What BP drop is significant for postural hypotension?

A
  • lying to standing
  • fall in systolic >20 or diastolic >10
  • systolic falls below 90mmHg after standing
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13
Q

What are the 3 acute coronary syndromes?

A
  • STEMI
  • NSTEMI
  • Unstable angina: prolonged, severe angina, usually at rest possibly with ECG changes
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14
Q

How does ACS develop?

A
  • build up of fatty plaques in artery walls
  • gradual narrowing > less blood and less oxygen reach heart leading to angina
  • plaque rupture leading to occlusion
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15
Q

What are non modifiable risk factors for ACS?

A
  • age
  • male
  • FHx
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16
Q

What are modifiable risk factors for ACS?

A
  • smoking
  • diabetes
  • hypertension
  • hypercholesterolaemia
  • obesity