d 3 Flashcards

1
Q

Pyloric stenosis

what and s/s

A
  • A narrowing of the outlet from the stomach to the small intestine (called the pylorus) that occurs in infants, usually 2 – 8 weeks of age

s/s
- Forceful vomiting – projectile (Non-bloody and non-bilious)
- Hungry after emesis and feed vigorously
- Decreased urine output
- Weight loss despite ++feeding

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2
Q

pyloric stenosis

diagnosis, managment

A

Diagnosis
- Electrolyte imbalance and metabolic alkalosis
- Visible gastric peristalsis left to right following feeding
- Diminished or absent bowel sounds
- Curdled milk appearance in emesis
- Signs of dehydration
- Ultrasound/upper GI series (if olive not felt)

Management:
- NPO
- IV access/blood work
- Replace fluids and electrolytes to correct dehydration and metabolic abnormalities
- Ensure adequate urine output
- Consider NG tube / Surgical consult
- Treatment is surgical repair

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3
Q

intussusception

what? classic triad?

A
  • A loop of bowel has slipped into another section of bowel –telescoped
  • Swelling, reduced blood flow, obstruction, and tissue damage

Classic triad
- Vomiting
- Bloody, mucoid (“currant jelly”) stools
- Intermittent colicky abdominal pain – with distention and tenderness

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4
Q

intussusception

managment and complications

A

Management
- NPO
- IV – rehydrate
- May require NG tube (decompress intestine)
- Radiologist/surgeon will attempt to reduce with an air enema if child stable
- Admit for observation if successful // Prepare for OR if not successful-surgical emergency

Complications
- Necrosis of bowel
- Perforation of bowel
- peritonitis
- Shock
- Sepsis
- Recurrent intussusception

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5
Q

volvulus

Cause? s/s Complications? Managment?

A

Caused by
- malrotation of bowel –> Bowel twists on itself
- Mid gut volvulus most common type

s/s
- bilious vomiting
- abdominal pain
- constipation

complications
- Surgical emergency
- Severe dehydration
- Electrolyte imbalance
- Prolonged vomiting can lead to shock

Management:
- IV rehydration
- Gastric decompression (consider NG)
- Surgical consult
- Emergency surgery

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6
Q

Blunt trauma // splenic injury

Causes? Signs of splenic injury? Tx

A
  • Causes: falls, MVCs, NAI, etc.
  • Can injure Liver, spleen and fluid filled loops of small bowel most commonly injured
  • Most common solid organ injury is spleen

Classic sign:
- LUQ pain and referred pain left shoulder

Treatment
- If patient has stable VS will have imaging to confirm
- Treatment is conservative if stable: admit for observation
IV boluses, CBC, type and cross match and analgesia

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7
Q

Blunt trauma complications

why conservatice tx, complications

A

Why conservative treatment?
- Spleen filters blood and removes old/damaged cells
- Can live without it but
- immuno-compromised

Complications
- Medical and Surgical Medical Emergencies
- Severe dehydration
- Hemorrhagic shock from GI bleed or abdominal injury
- Bowel obstruction // intussusception // malrotation and volvulus
- hernia
- post-operative adhesions –> necrosis of bowel
- Peritonitis
- Sepsis

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8
Q

sickle cell disease

A

Definition
- Chronic disease with acute exacerbations, Caused by mutation in DNA resulting in absence of normal HbA

RBC effect
- Healthy RBCs are flexible, biconcave discs with lifespan of 120 days
- SCD RBCs polymerize, forming microtubules making cells crescent-shaped and friable with 10–20-day lifespan
- They get stuck in small vessels and occlude them → hypoxia
- Leads to vaso-occlusion

Genetics
- Autosomal recessive trait

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9
Q

sickle cell presentation, pain and managment

result of what 3 factors

A

Presentation
- Pallor/Jaundice
- Often anemic d/t immature cell death

Result of three factors
1. Ischemia due to occlusion of vessels by misshaped RBCs
2. Endothelial damage
3. Local inflammation

Pain
- Vaso-occlusive crisis is hallmark of SCD
- Debilitating pain
- Hydromorphone is not even effective
- Most common reason to seek care

Management
- Penicillin prophylaxis
- Hydroxyurea
- Folic acid supplement
- Transfusions/Chelation

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10
Q

racism in SCD

A

Racism
- Sickle cell disease predominantly impacts people of African descent
- Racism in the care of patients living with SCD is well documented

For the child
- Medical nonadherence, mistrust of HCPs, poorer psychical and mental health outcomes across the lifespan
- Pain is not consistently validated; often undertreated and experience longer delays in medication administration than white children
- Lower-quality care for children across

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11
Q

Cancer

mutation results in?

A

What is Cancer?
- Genetic mutation
- Permanent DNA alteration
- Begins in single cell (clonal)

Mutation results in
- Lack of differentiation
- Loss of contact inhibition
- Unregulated growth
- Cellular immortality

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12
Q

cancer treatment modalities

A

Treatment Modalities
- Chemotherapy
- Biotherapy
- Immunotherapy
- Radiation
- Surgery
- HSC (stem cells)
- bone marrow transplants

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13
Q

pediatric palliative care differences

A
  • Same definition as palliative care but caters to unique needs of the child and their family

Differences include:
- Prognosis, life expectancy & functional outcome often unclear
- Greater use of intensive disease-modifying or life-sustaining treatments
- Focus on growth & development at the same time as death
- Greater physical & emotional burden for families
- informational, recreational and educational needs change as child grows
- Greater number of congenital anomalies

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14
Q

pain and sx managment

medical

A

Analgesics
Antacids
Antibiotics/Antivirals
Anticholinergics
Anticonvulsants
Antispasticity
Benzodiazepines
Chemotherapies/Radiation Therapies
NSAIDs
Surgeries
Opioids

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15
Q

pain managment

non pharm interventions

A
  • Arts & Crafts
  • Bath
  • Change of position
  • Change of location
  • Distraction
  • Essential Oils
  • Hot/Cold Compresses
  • Massage/Acupuncture
  • Music
  • Physical activity
  • Play (one-on-one, with peers)
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16
Q

PPC important considerations

A
  • How old is the child? What developmental stage is the child at?
  • How much does the child understand about whatis happening to them?
  • Who does the child want by their side
  • Who do they consider to be family?
  • How do they show signs of pain or distress?
  • What is important for the parents of the child with regards to care? What do they want to avoid?
  • Does the child or family have any special requests or final wishes?
  • Does the child and family follow certain religious, cultural, or spiritual practices or beliefs?
17
Q

death in the child

neuro, msk carduac

A

Neurological
- decreased wakefulness
- decreased sensitivity to touch or stimulus
- increased pain,
- increased seizure activity

Cardiac
- tachycardia,
- Bradycardia
- decreased circulation
- pale or mottled skin color

Musculoskeletal
- increased/decreased muscular tone
- Spasms
- incontinence

18
Q

death in a child

GU and and resp

A

Respiratory
- Dyspnea
- Tachypnea
- increased exertion
- increased secretions

Gastro-urinary
- decrease appetite + poor food tolerance
- increased nausea
- Vomiting
- decreased urine production
- Constipation
- diarrhea