Diagnosis and Microbiology Flashcards

(60 cards)

0
Q

How can you classification the severity of bone loss?

A

Mild: 25% of root length (1-2mm CAL)
Moderate: 25-50%(3-4mm CAL)
Severe: more than 50% (6+mm CAL)

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1
Q

How can you categorise distribution?

A

Generalised : PPD or more than 3mm in more than 30% of sites
Localised : PPD. Of less than 3mm in less than 30% of sites

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2
Q

How can you assess the shape of bone loss?

A

Horizontal or vertical

Horizontal is entire width of interdental bone loss
Vertical : adjacent to tooth surface

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3
Q

What is the host response to perio antigens?

A

PMN complement and in Late stages you see T cels B cells and very late is plasma infiltrate

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4
Q

What is the purpose of neutrophil in pDl?

A

They act as surveillance and maintains integrity of PDL

Adhere, chemo taxis, phagocytosis, generate super oxide

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5
Q

What is the PDL like in health?

A

Very little plaque

Minimal GCF
Few PMN

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6
Q

What happens in the initial gingival lesion?

A

2-14 days
Early plaque: gram pos bacteria
Increases PMN

Vasculitis with appearance of IGs and complement

JE begins to proliferate and FEW Plasma cels

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7
Q

Wat happens in the established gingival lesion?

A

Grame neg and pos bactera

PLASMA CELLS. IGs predominate

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8
Q

What happens in the advanced gingival lesion?

A

Connective tissue attachment loss

> 50% PLASMA cells

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9
Q

What is he microbial aetiology of perio?

A

Non specific vs specific vs ecological plaque

Non specific : caused by mixed microbial bass of bactera

Specific: one group or a single organism causes perio

Ecological: changes in environmental conditions lead to an ecological shift favouring pathogenic organisms

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10
Q

How does tissue destruction occur in perio?

A

Directly by bacteria eg collagenases,muster oxide, toxins

Indirectly via host response; humoral immunity and cellular

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11
Q

Which interleukin is key in perio destruction for bone manage?

A

IL1 released by macrophages and CT cell

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12
Q

Which factors make you a susceptible host?

A

Smoking:
Syestmic condition eg HIV, diabetes have four fold increased risk since they have decreased neutrophils
Genetic familial
Stress
Leukaemia
Overhangs, crowding, dentures, weak contacts, enamel pearls

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13
Q

Which OH indices are there?

A

Greene and Vermillion

Quigley and Hein 1962 mod Turesky 1970

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14
Q

What does the Green and vermillion show?

A

Soft and hard depsotos

0: nothing
1: soft debris 2/3 band of calculus sub

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15
Q

What did the Turesky show?

A

0: nothing
1: flecks
2: continuous band of 1mm
3: less than 1/3
4. Less than 2/3
5: >2/3

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16
Q

Which gingival index is there?

A

Loe 1967

0: no inflam
1: change in colour and texture
2: inflam and bleeding from probing
3: overt inflam and spon bleeding

Muhlemann and Son 1971

1: no bleeding on probing
2: bleeding within 15seconds

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17
Q

How can we screen for perio?

A

BPE

3: 6PPC that sextand
4: whole mouth
Each sextand must have 2 teeth or add to next sextant

  • furcation but must be added to score
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18
Q

What are the scores for BPE?

A

1: healthy
2: BOP/calculus/overhand : OHI and scale
3: part black band gone 3.5-5.5 pocket depth :.OHI scale, RSD
4: black band gone: OHI Scale, consider surgery

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19
Q

What factors will affect picket depth?

A
Size of probe
Force applied
Contour of tooth
Angulation of probe
Presence of calcius
Presence of inflammation
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20
Q

Which mobility is normal?

A

<0.2mm

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21
Q

What are mobility indices?

A

Grace and smales

Miller

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22
Q

What is the grace and smales mobility index?

A

0: no apparent mobility
1: single tooth where mobility is les than 1
2: mobility 1-2
3: >2 horizontal /vertical movement

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23
Q

What is the miller index?

A

1: 1mm
3: >1 and axial

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24
How can we measure furcation?
Used curved nabers probe 1: 1/3 but not total width: cul de sac 3: through
25
What is the picket probing deoth?
Gingiva margin to base of pocket
26
How much force do you use for probing?
25g 0.25N 0.5mm diameter
27
How do you measure recession?
CEJ to gingival margin
28
What is the clinical attachment loss?
CEJ to base of pocket
29
What are the changes from the 1989 to 199 classification?
1999 has: perio endo , periodontal abscess, development conditions, gingival diseases Adult perio: chronic perio Early onset: aggressive Necrotising ulcerative perio: necrotising perio
30
Why does smoking cause perio?
Reduced blood flow to gingiva Impaired WBC Imparted owing healing Increased inflam cytokines
31
Why does diabetes cause perio?
Imparted healing and poor response to therapy | Thickening of lumen of blood vessel
32
What are the FGDP guidelines re radiograph?
Pocketing of less than 6mm horizontals Pocketing or more then 6mm vertical BW (PA) Irregular pockets : BW horizontal or vertical (PA) Concurrent problems: DP/PA Perio endo: PA
33
Pocketing can be true or false, what does this mean?
True: apical migration of JE | False; gingival enlargements and no apical migration
34
How long does it take the Aquired pellicle to form?
Within minutes and made from salivary glycoproteins
35
What are the early colonisers of plaque?
0-7 days they form and are mainly gram pos 3/4 hours see strep and actinomycosis These bind to saliva and provide anaerobic environment
36
How long does it take the late colonisers?
After 7 days of plaque they come
37
Which study showed the relationship between plaque and gingivitis?
Loe et al 1965
38
Which study showed the chronology between plaque and gingivitis?
Lang et al 1973
39
Which teeth mainly become affected with increasing age by perio?
Posterior teeth
40
What are the sublingual plaque retentive factors?
``` Calculus Furcation Rough cementum Iatrogenic: overhangs, defective margins Root grooves Resorption Boney pockets ```
41
What are the supragival factors that affect plaque?
Caries Overhangs Exposed root surface Unpolished fillings
42
How long does it take gingivitis to set in?
48 hours
43
Which are the strong association organisms?
Aa PG Tanerella forsythia Red
44
What are the moderate association,
Treponema denticola Fusoform nuclear up Peptostreptococcus micros Orange
45
How do you mange boney defects?
Non surgical debridement or surgical
46
What are the types of bone loss you can have?
Horizontal Vertical Interdental
47
How can vertical be classified?
1/2/3/4 wall defects 4 is known as Circumfrential By the number of remaining walls
48
What are the surgical treatment options for vertices bone defects?
Conventional flap and curettage Obturate defects eg bone graft or synthetics GTR
49
What are the type of bone grafts available?
Autogenous self Homogenous same species Isogenous twin Heterogenous: freeze dried/diff species
50
Where are the sources for Autogenous bone grafts?
Adjacent bone Edentulous ridge Iliac crest
51
What are the options for artificial bone?
``` Biodegradable ceramics HAP Acrylic PLaster of Paris Metals Epoxy resin ```
52
When are synthetic bone grafts used?
1 operation site It is cosly and cannot always predict outcome + little tissue reaction
53
What is GTR?
Placing a barrier to epilthlil migration prior to completion of sugery allowing new CT attachment It is more effective in reducing probing depths than open flap debridement improves CA and reduced PPD, less recession
54
Which materials can be used in GTR?
Gortex Collagen eg Biomend PLA (poly lactic acid) eg resolut Growth factors eg Emdogain
55
What is the difference between first generation and second generation bio absorbable membranes?
First generations : no second surgery, requires stablisstion sutures,better than Gortex Second : no second surgery, no stablisstion sutures, free flow or custom made by operator
56
What is the advantage of using collagen?
Natrual haemostat since promotes platelet plugging Promotes early wound stabilisation, matriation, chemo tactic for fibroblasts BIOMEND
57
What is Emdogain?
Improves PPD compared to flap surgery It is an enamel matrix protein Place onto exposed root surface 2 compoantsn: Emdogain and perio Glas
58
How do you use Emdogain?
Raise flap Curettage area Apply pre gel EDTA conditioner which removed smear later on exposed root for 2mins Rinse and apply Emdogain from apical region and work up Close flap No robing 6 months
59
What is GCF?
In health it is a transudate | In disease becomes an exudate