digestive tract disorders part 3 Flashcards

1
Q

Inflammation of the lining of the stomach

A

Gastritis

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2
Q

Cause of GAstritis

A

Mucosal barrier that normally protects the stomach from autodigestion breaks down
Hydrochloric acid, histamine, and pepsin cause tissue edema, increased capillary permeability, possible hemorrhage
Helicobacter pylori thought to be prime culprit
dietary indescretions, reflux of duodenal contents, use of aspirin, steroids, NSAIDS, alcohol, caffeine, cigarettes, food allergies, infection

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3
Q

S/S of Gastrtis

A

Nausea may or may not be present, vomiting, anorexia, a feeling of fullness, and pain in the stomach area,
pressure,
client may see blood in their emesis or note darkening of their stool color

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4
Q

medical diagnosis of gastritis

A

Gastroscopy
Laboratory studies to detect occult blood in the feces, low blood hemoglobin and hematocrit, and low serum gastrin levels ( may detect anemia) ; H. pylori can be confirmed by breath, urine, stool, or serum tests, or by gastric tissue biopsy

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5
Q

Medical treatment of gastritis

A

Oral fluids and foods withheld until the acute symptoms subside; IV fluids administered
Medications to reduce gastric acidity and relieve nausea
Analgesics for pain relief and antibiotics for H. pylori
Surgical intervention may be needed
some clients may wish to avoid spicy food/ high fats and caffeine
avoidance of alcohol and NSAIDS

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6
Q

Nursing assessement of Gastritis

A

Patient’s present illness
Pain, indigestion, nausea, and vomiting (observe color)
Determine the onset, duration, and location of pain
Note factors that trigger or relieve the symptoms
Diet, use of alcohol and tobacco, activity/rest patterns
Patient’s general appearance for signs of distress
Compare vital signs, height, weight to previous readings
Note the skin color and check turgor
Inspect abdomen for distention; palpate for tenderness
Auscultate abdomen for increased bowel sounds

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7
Q

nursing interventions of gastritis

A

Pain
Imbalanced Nutrition: Less Than Body Requirements
Deficient Fluid Volume
Ineffective Coping

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8
Q

Loss of tissue from lining of the digestive tract
Classified as gastric or duodenal
most occur in the duodenum by may develop at the lower end of the esophagus

A

Peptic Ulcer

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9
Q

Cause of peptic ulcer

A

Contributing factors: drugs, infection, stress
Most ulcers are caused by the microorganism H. pylori
when the normal balance between factors that promotes mucosal injury and factors that protect the mucosa is disrupted

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10
Q

S/S of peptic ulcer

A

Burning pain that occur 1-7h after meals , abdominal pain
Nausea, anorexia, weight loss , disrupted sleep,
single greatest risk is development of PUD is infection w/ gram negative bacterium H pylori

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11
Q

complications with peptic ulcer

A

Hemorrhage, perforation, or pyloric obstruction

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12
Q

medical diagnosis of peptic ulcer

A

Barium swallow examination, gastroscopy, and esophagogastroduodenoscopy
H. pylori can be detected by antibodies in the blood or stool so we need to eradicate the bacteria, reduce the acid levels, and by a breath test

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13
Q

medical treatment of peptic ulcer

A

Drug therapy
Diet therapy
Managing complications

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14
Q

Begins in the mucous membranes, invades the gastric wall, and spreads to the regional lymphatics, liver, pancreas, and colon
No specific signs or symptoms in the early stages
enlarged mass or ulcerating lesion that expands or penetrates several tissue layers

A

stomach cancer

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15
Q

The most common type of stomach cancer that rises from the granular cells in the inner layer of the stomach

A

adenocarcinoma

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16
Q

cause of stomach cancer

A

H. pylori infection, pernicious anemia, chronic atrophic gastritis, and achlorhydria, type A blood, and a family history
Cigarette smoking, alcohol abuse, and a diet high in starch, salt, pickled foods, salted meats, and nitrates

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17
Q

s/s of stomach cancer

A

Late signs and symptoms are vomiting, ascites, liver enlargement, and an abdominal mass, pain
early signs, feeling of fullness, Indigestion, heartburn, n/v, anorexia, weight loss, fatigue, and anemia, stool contain occult blood

18
Q

medical diagnosis of stomach cancer

A

Gastroscopy, endoscopic ultrasound, upper GI series, CT, PET scan, MRI, laparoscopy
Laboratory studies include hemoglobin and hematocrit, serum albumin, liver function tests, and carcinoembryonic antigen

19
Q

medical treatment of stomach cancer

A

Surgery, chemotherapy, and radiation therapy

20
Q

Preoperative care of the patient with stomach cancer

A

Inform about the nasogastric tube and IV fluids; teach coughing, deep breathing, and leg exercises
Identify/support patient’s coping methods
Include sources of support, such as family members or a spiritual counselor, in the preoperative care

21
Q

Postoperative care of the patient with stomach cancer

A

Assessment
Comfort, appetite, and nausea and vomiting
Monitor weight changes and determine dietary preferences
Identify the patient’s support system and coping strategies
Interventions
Pain
Imbalanced Nutrition: Less Than Body Requirements
Ineffective Coping

22
Q

is a condition where ingested foods bypass the stomach too rapidly and enter the small intestine largely undigested. It happens when the small intestine expands too quickly due to the presence of hyperosmolar (having increasedosmolarity) contents from thestomach. This causes symptoms due to the fluid shift into the gut lumen with plasma volume contraction and acute intestinal distention

A

Dumping syndrome

23
Q

“Early” dumping begins concurrently within ? min from ingestion of a meal

A

15 to 30 minutes

24
Q

“Late” dumping happens ? hours after eating.

A

one to three hours

25
S/s of early and late dumping syndrome
Symptoms of early dumping include nausea, vomiting, bloating, cramping, diarrhea, dizziness, and fatigue. Symptoms of late dumping include weakness, sweating, and dizziness. Many people have both types. The syndrome is most often associated with gastric bypass (Roux-en-Y) surgery.
26
In addition, people with this syndrome often suffer from What?, because the rapid "dumping" of food triggers the pancreas to release excessive amounts of What? into the bloodstream. This type of hypoglycemia is referred to as ?
low blood sugar, or hypoglycemia insulin "alimentary hypoglycemia."
27
Strangulated hernia, tumor, paralytic ileus, stricture, volvulus (twisting of the bowel), intussusception (telescoping of the bowel into itself), and postoperative adhesions is the cause of what
Intestinal Obstruction
28
s/s of Intestinal Obstruction
Vomiting (possibly projectile), abdominal pain, and constipation Blood or purulent drainage passed rectally Abdominal distention, especially with colon obstruction
29
complications of intestinal obstruction
Fluid and electrolyte imbalances and metabolic alkalosis | Gangrene and perforation of the bowel
30
Nursing assessment of intestinal obstruction
Symptoms, including pain and nausea Onset and progression of symptoms Hernia, cancer of the digestive tract, and abdominal surgeries Ask when the patient’s last bowel movement was and if the characteristics were nor
31
cause of appendicitis
Inflammation of the appendix | A ruptured appendix allows digestive contents to enter the abdominal cavity, causing peritonitis
32
S/S of appendicitis
Pain at McBurney’s point, midway between the umbilicus and the iliac crest Temperature elevation, nausea, and vomiting Elevated WBC count (10,000-15,000/mm3 ) Peritonitis: absence of bowel sounds, severe abdominal distention, increased pulse and temperature, nausea/vomiting; rigid abdomen
33
medical treatment of appendicitis
Nothing by mouth A cold pack to the abdomen may be ordered Laxatives and heat applications should never be used for undiagnosed abdominal pain Immediate surgical treatment indicated Ruptured appendix: surgery may be delayed 6-8 hours while antibiotics and IV fluids given
34
nursing assessment of appendicitis
Location, severity, onset, duration, precipitating factors, and alleviating measures in relation to the pain Previous abdominal distress, chronic illnesses, surgeries; record allergies and medications Temperature; abdominal pain, distention, and tenderness; presence and characteristics of bowel sounds
35
Preoperative interventions of appendicitis
Semi-Fowler or side-lying position with the hips flexed Until physician determines the diagnosis, analgesics may be withheld If rupture suspected, elevate patient’s head to localize the infection
36
Postoperative interventions of appendicitis
Administer antibiotics, intravenous fluids, and possibly gastrointestinal decompression Assist the patient in turning, coughing, and deep breathing; incentive spirometry Splint the incision during deep breathing Early ambulation Assess abdominal wound for redness, swelling, and foul drainage Wound care as ordered or according to agency policy
37
is a procedure in which a needle or catheter is inserted into the peritoneal cavity to obtain ascitic fluid for diagnostic or therapeutic purposes…Ascitic fluid may be used to help determine its etiology as well as to evaluate for infection or presence of cancer.
Paracentesis
38
Inflammation of peritoneum caused by chemical or bacterial contamination of the peritoneal cavity
Peritonitis
39
s/s of peritonitis
Pain over affected area, rebound tenderness, abdominal rigidity and distention, fever, tachycardia, tachypnea, nausea, and vomiting
40
medical treatment of peritonitis
Gastrointestinal decompression, intravenous fluids, antibiotics, and analgesics Surgery to close a ruptured structure and remove foreign material and fluid from the peritoneal cavity
41
assessment of peritonitis
Onset, location, and severity of the pain and any related symptoms Record a history of abdominal trauma, including surgery Record vital signs Inspect abdomen for distention and auscultate for the presence of bowel sounds