DMS EKG I Flashcards

(47 cards)

1
Q

What are the anterior leads?

A

V2
V3
V4

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2
Q

What are the inferior leads?

A

II
III
aVF

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3
Q

What are the left lateral leads?

A

I
aVL
V5
V6

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4
Q

What are the right leads?

A

aVR

V1

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5
Q

What happens when the wave of depolarization meets the myocardial cell?

A

releases Ca causing it to contract

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6
Q

Which leads should you look at for atrial enlargement?

A

II & V1

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7
Q

R atrial enlargement

A

look at II & V1

inc. height of 1st portion of P wave

possible R axis deviation of P wave

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8
Q

L atrial enlargement

A

look at II & V1

sometimes have inc. height of last portion of P wave

inc. P wave duration

aka P mitrale

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9
Q

R vent. hypertrophy

A

R axis dev

ratio of R:S height >1 in V1
<1 in V6

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10
Q

L vent. hypertrophy

A
  1. R wave in V5/V6 + S wave in V1/V2 >35
  2. R wave in V5 > 26
  3. R wave in V6 > 18
  4. R wave in V6 > V5
  5. R wave in aVL > 13
  6. R wave in aVF > 21
  7. R wave in I > 14
  8. R wave in I + S wave in III > 25
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11
Q

4 questions to ask about arrhythmias

A
  1. Are normal P waves present?
  2. Are the QRS complexes narrow or wide? (0.12s normal)
  3. What is the relationship btwn the P waves & the QRS complexes?
  4. Is the rhythm regular or irregular?
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12
Q

Paroxysmal Supraventricular Tachycardia (PSVT)

A

P waves retrograde if visible in II/III

150-250 bpm

Carotid massage - slows or terminates

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13
Q

A Flutter

A

Saw-toothed
2:1, 3:1, 4:1
Best seen in II & III

atrial rate: 250-350 bpm
vent. rate slower

Carotid massage inc. block

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14
Q

A Fib

A
irregularly irregular
undulating baseline
atrial rate 350-500 bpm
vent. rate variable
best seen in V1

Carotid massage may slow vent. rate

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15
Q

Multifocal Atrial Tachycardia (MAT)

A

at least 3 diff. P wave morphologies

100-200 bpm

WAT if <100 bpm

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16
Q

Paroxysmal Atrial Tachycardia (PAT)

A

100-200 bpm
warm-up period

Carotid massage - no effect or mild slowing

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17
Q

VT vs. PSVT

A

v - diseased heart, no effect from carotid massage, cannon A waves may be present, AV dissociation may be seen, slightly irregular, fusion beats may be seen, abnormal QRS

Everything is opposite in PSVT

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18
Q

1st degree AV block

A

PR interval >0.2 s

beats conducted through to ventricles

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19
Q

Wenckebach Type I 2nd degree AV block

A

Progressive prolongation of the PR interval until a QRS is dropped

Usually from block w/in AV node

20
Q

Mobitz Type II 2nd degree AV block

A

QRS complexes dropped w/o PR interval prolongation

Usually from block below AV node in His bundle

21
Q

3rd degree AV block

A

no beats are conducted through to the ventricles

complete heart block w/ AV dissociation

atria & vent. driven by independent pacemakers

22
Q

RBBB

A

wide QRS

RSR’ in V1&V2 w/ ST depression & T wave inversion
Bunny ears!!!

Reciprocal changes in V5, V6, I & aVL
(L lateral leads)

23
Q

LBBB

A

Wide QRS

broad or notched R wave, ST depression & T wave inversion
-bunny ears in L lateral leads

Reciprocal changes in V1&V2

L axis deviation may be present

24
Q

L anterior hemiblock

A

L axis dev >-30
w/ no other cause of dev.

more coomon

25
L posterior hemiblock
R axis dev. w/ no other cause of dev. only seen in diseased hearts
26
Bifascicular block
RBBB w/ L anterior or posterior hemiblock
27
Describe the effects from carotid massage
PSVT - slows/terminates A flutter - inc. block A fib - slow vent. rate
28
What is the most common cause of enlargment?
V overload
29
What is a normal P wave?
btwn 0 & 70 | 0.12s
30
Why do arrhythmias happen?
``` H - hypoxia I - ischemia & irritability S - sympathetic stimulation D - drugs E - electrolyte disturbances B - bradycardia S - stretch ```
31
Nonsinus pacemakers
Atrial 60-75 bpm Junctional 40-60 bpm Ventricular 30-45 bpm
32
What is the most common cause of paroxysmal supraventricular tachycardia?
PSVT | reentrant circuit w/in AV node
33
What is the most common cause for atrial flutter?
a reentrant circuit that runs around the annulus of the tricuspid valve sawtooth in II & III
34
What arrhythmia is irregularly irregular?
A fib
35
accelerated idioventricular rhythm
looks like Vtach but slower 50-100 bpm
36
Ashman phenomenon
wide aberrantly conducted supravent. beat after a QRS then long pause usually seen in Afib
37
Reciprocal changes w/ vent. hypertrophy
1. ST segment depression 2. T wave inversion Seen best in leads w/ tall R waves R - V1 & V2 L - I, aVL, V5 & V6
38
What is the most common nonsinus pacemaker?
junctional | 40-60 bpm
39
What is the common cause of PSVT?
reentrant circuit in AV node
40
What is Aflutter usually caused by?
reentrant circuit that runs around the annulus of the tricuspid
41
PVC
wide & abnormal QRS compensatory pause Bigeminy - 1 beat then 1 PVC Trigeminy - 2 beats then 1 PVC
42
What are the rules of malignancy w/ PVCs?
1. Frequent 2. Runs w/ 3 or + in a row 3. Multiform 4. PVC falling on T wave of previous beat = R on T phenomenon 5. PVC w/ acute MI
43
Uniform vs. polymorphic Vtach
U - often seen w/ healed infarcts P - common w/ ischemia, infarcts, electrolyte disturbances & conditions w/ prolonged QT intervals
44
Accelerated idioventricular rhythm
looks like PSVT but slower rate 50-100 bpm idioventricular rhythm - <50 bpm rarely req. Tx seen during acute MI or during reperfusion
45
Incomplete BBB
RBBB or LBBB w/ narrow QRS (0.10-0.12)
46
Who should get pacemakers?
1. 3rd degree AV block 2. AV block or bradycardia if symptomatic 3. Blocks w/ MI 4. Recurrent tachycardias
47
What is cardiac resynchonization therapy?
vent. pacemaker to correct timing only helps in Pts w/ wide QRS & LV dysfunction