DMS EKG I Flashcards
(47 cards)
What are the anterior leads?
V2
V3
V4
What are the inferior leads?
II
III
aVF
What are the left lateral leads?
I
aVL
V5
V6
What are the right leads?
aVR
V1
What happens when the wave of depolarization meets the myocardial cell?
releases Ca causing it to contract
Which leads should you look at for atrial enlargement?
II & V1
R atrial enlargement
look at II & V1
inc. height of 1st portion of P wave
possible R axis deviation of P wave
L atrial enlargement
look at II & V1
sometimes have inc. height of last portion of P wave
inc. P wave duration
aka P mitrale
R vent. hypertrophy
R axis dev
ratio of R:S height >1 in V1
<1 in V6
L vent. hypertrophy
- R wave in V5/V6 + S wave in V1/V2 >35
- R wave in V5 > 26
- R wave in V6 > 18
- R wave in V6 > V5
- R wave in aVL > 13
- R wave in aVF > 21
- R wave in I > 14
- R wave in I + S wave in III > 25
4 questions to ask about arrhythmias
- Are normal P waves present?
- Are the QRS complexes narrow or wide? (0.12s normal)
- What is the relationship btwn the P waves & the QRS complexes?
- Is the rhythm regular or irregular?
Paroxysmal Supraventricular Tachycardia (PSVT)
P waves retrograde if visible in II/III
150-250 bpm
Carotid massage - slows or terminates
A Flutter
Saw-toothed
2:1, 3:1, 4:1
Best seen in II & III
atrial rate: 250-350 bpm
vent. rate slower
Carotid massage inc. block
A Fib
irregularly irregular undulating baseline atrial rate 350-500 bpm vent. rate variable best seen in V1
Carotid massage may slow vent. rate
Multifocal Atrial Tachycardia (MAT)
at least 3 diff. P wave morphologies
100-200 bpm
WAT if <100 bpm
Paroxysmal Atrial Tachycardia (PAT)
100-200 bpm
warm-up period
Carotid massage - no effect or mild slowing
VT vs. PSVT
v - diseased heart, no effect from carotid massage, cannon A waves may be present, AV dissociation may be seen, slightly irregular, fusion beats may be seen, abnormal QRS
Everything is opposite in PSVT
1st degree AV block
PR interval >0.2 s
beats conducted through to ventricles
Wenckebach Type I 2nd degree AV block
Progressive prolongation of the PR interval until a QRS is dropped
Usually from block w/in AV node
Mobitz Type II 2nd degree AV block
QRS complexes dropped w/o PR interval prolongation
Usually from block below AV node in His bundle
3rd degree AV block
no beats are conducted through to the ventricles
complete heart block w/ AV dissociation
atria & vent. driven by independent pacemakers
RBBB
wide QRS
RSR’ in V1&V2 w/ ST depression & T wave inversion
Bunny ears!!!
Reciprocal changes in V5, V6, I & aVL
(L lateral leads)
LBBB
Wide QRS
broad or notched R wave, ST depression & T wave inversion
-bunny ears in L lateral leads
Reciprocal changes in V1&V2
L axis deviation may be present
L anterior hemiblock
L axis dev >-30
w/ no other cause of dev.
more coomon