Dysrhythmias Flashcards

(61 cards)

1
Q

Sinus Pause/Arrest

A

May occur from increased vagal tone, myocarditis, MI and digitalis toxicity
Typically lasts 2 seconds - 2 minutes
Atropine may be given if hemodynamically unstable

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2
Q

What med should be given to a patient with symptomatic Sinus bradycardia?

A

Atropine 1mg IV

Temporary Pacemaker

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3
Q

Where does SVT originate?

A

Above the His bundle.

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4
Q

What might cause SVT?

A

digitalis, asthma meds, caffeine, ephedra, cocaine, meth Stimulants

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5
Q

What does SVT generally start with?

A

A PAC or PVC

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6
Q

S/S of SVT

A
Palpitations
Dizziness
SOB
Anxiety
CP/tightness
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7
Q

SVT Tx

A
Vagal maneuvers
Carotid Massage
Adenosine 6mg IV fast push.
or of unsuccessful...
Cardioversion, IV Beta blocker/CCB
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8
Q

SVT prevention

A

Beta Blockers
CCB’s
Digoxin
SVT ablation (permanent)

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9
Q

Wolff-Parkinson-White Syndrome (WPW)

A

Congenital defect - any age. Men > Women
Form of SVT where an accessory pathway bypasses the AV node.
AKA AV reciprocating arrhythmia
Bypassing AV node can cause re-entry tachycardia

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10
Q

WPW S/S

A
Palpitations
Tachycardia
DIzziness
Dyspnea
Anxiety
Syncope
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11
Q

WPW EKG symptoms

A
PR interval < .12ms
Delta wave (slurred QRS uptake)
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12
Q

WPW Long-term Tx

A

Depends on frequency and symptoms
Radiofrequency ablation
Beta blockers, CCB’s, Flecainide

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13
Q

WPW Acute Tx

A

Vagal maneuvers
IV adenosine (6-12 mg fast push)
OR
IV diltiazem or verapimil

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14
Q

Paroxysmal Atrial Tachycardia (PAT)

A

Atrial rate 150 - 250
P wave morphology varies
Usually requires no tx
Usually terminated with vagal maneuvers

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15
Q

Premature Atrial Contractions (PAC’s)

A

Discharge from non-sinus atrial pacemaker
P’ wave marks PAC
May be precursor to Afib

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16
Q

PAC Tx

A

Asymptomatic (avoid triggers (stimulants))

Symptomatic: Beta blockers

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17
Q

Wandering Atrial Pacemaker

A

Seen in normal hearts and diseased hearts
Variable rate
More than 3 different P wave morphologies
Usually no tx required
May lead to multifocal atrial tachycardia

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18
Q

Multifocal Atrial Tachycardia

A

Wandering atrial pacemaker with a rate over 100 bpm

Usually doesnt cause hemodynamic instability

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19
Q

Causes of multifocal atrial tachycardia

A
Lung dz, COPD
AMI
Sepsis
Hypokalemia
Low magnesium
Ma be a precursor to Afib
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20
Q

Multifocal atrial tachycardia tx

A

Tx underlying medical problem

Suppress rate with BB, or CCB

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21
Q

Atrial Fibrillation

A
Most common arrhythmia
Micro re-entry circuit
Atrial rate is 350 - 450
No distinguishable P waves
Irregularly irregular
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22
Q

Causes of Afib

A
Underlying cardiac dz
Valvular dz, Heart failure, IHD
Pericarditis
Thyrotoxicosis
PE
Pneumonia
Alcohol OD
Post-op thoracic surgery
Sleep Apnea
HTN
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23
Q

Afib may result in?

A

Decreased Cardiac Output

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24
Q

INR goals for Afib?

A

2.0 - 3.0

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25
Rate goals for Afib?
between 60 and 110 Diltiazem Beta Blockers Digoxin
26
When is it safe to cardiovert without anticoagulation in Afib?
Less than 48 - 72 hours of Afib
27
Atrial Flutter
``` Macro re-entry circuit Atrial rate 250 - 350 Ventricular rate 150 2:1 3:1 4:1 pattern regularly irregular Classic sawtooth on EKG ```
28
A flutter is indicative of?
Diseased hearts (CHF) Precursor to Afib Tx depends on level of hemodynamic compromise
29
Aflutter Tx
Cardioversion Class 1A antiarrhythmics BB, CCB, Digoxin
30
Junctional Tachycardia
Junctional rhythm with a rate of 150 - 250 More common in women Usually initiated by PAC
31
Junctional Tach tx
Vagal maneuvers Adenosine* DOC Long-term: BB, CCB
32
1st degree AV block
PR interval > .20 Occurs in healthy and sick hearts Usually don't need tx
33
2nd Degree AV Block Type 1
Wenchebach Occurs in the AV node, above His bundle often transient Often asymptomatic - no tx
34
Wenchebach EKG
Variable rate | PR grows wider and wider until the QRS is blocked completely, then it resets.
35
2nd Degree AB block Type 2
Mobitz Usually occurs below His bundle May progress into higher degree AV block
36
Type 2 block EKG findings
Variable rate normal P wave Widened QRS associated w/ BBB PR may be normal until dropped QRS
37
Type 2 block Tx
More serious than Type 1 Artificial pacing Usually requires permanent pacemaker
38
3rd Degree Block
AV dissociation | Sinus impulses are completely blocked upstream of the ventricles
39
3rd Degree block causes
digitalis toxicity Acute infxn MI Degeneration of tissue
40
3rd Degree block EKG findings
Normal atrial rate Ventricular rate usually less than 70 QRS may be normal or widened
41
3rd degree block tx
External pacing and atropine for acute episodes. | Permanent pacing for chronic complete block
42
Premature Ventricular Contractions (PVC)
``` Increasing circulating catecholamines Coronary ischemia Hypokalemia Low magnesium Drug toxicities ```
43
PVC EKG findings
Variable rate P wave usually obscured by QRS Wide QRS May occur in singles, couplets, or triplets
44
Bigeminal PVC's
one normal beat and one PVC repeated
45
Trigeminal PVC's
two normal beats and one PVC
46
Ventricular Tachycardia causes
Triggered by ischemia, electrolyte abnormalities Hypokalemia is also an important trigger MI Cardiomyopathy
47
Pulse Vtach Tx
Cardioversion | Amiodarone
48
Pulseless Vtach
Defibrillation | Amiodarone
49
Torsades de Pointes
Twisting about the points | Upward and downward deflection of QRS
50
Torsades de Pointes causes
Long QT drugs electrolyte imbalances Myocardial ischemia
51
Torsades de Pointes Tx
Synchronized cardioversion IV magnesium, potassium Overdrive pacing
52
Ventricular Fibrillation
AKA sudden cardiac death ABsence of cardiac output Occurs with AMI
53
Vfib Tx
ACLS protocol Immediate defib Treat underlying cause Implantation of ICD
54
Idioventricular Rhythm
Widened WRS dying heart rhythm Pacemaker most likely needed Caused by myocardial ischemia, infarction
55
Asystole
In the presence of AMI is almost always fatal | Complete cessation of any electrical or mechanical activity
56
Asystole Tx
``` CPR, O2 IV, Intubate Transcutaneous pacing Epi 1mg IV push q3-5m Atropine ```
57
Pulseless Electrical Activity (PEA)
Electromechanical dissociation There is electrical activity, but no mechanical response EKG shows sinus, but NO pulse
58
PEA 6 H's
``` Hypoxia Hypovolemia Hypoglycemia Hydrogen Ions (Acidosis) Hypothermia Hypo/Hyperkalemia ```
59
PEA 6 T's
``` Toxins Tamponade Trauma Tension pneumothorax Thrombosis (cardiac) Thrombosis (pulmonary) ```
60
PEA Tx
Correct underlying cause Epi, Atropine CPR
61
Which pathway do electrical impulses take in WPW?
Through the kent bundles | You will see delta waves and short PR