Dysrhythmias Below The AV Node Flashcards

(35 cards)

1
Q

Ectopic focus from ventricle (cell decides to contract early/out of order)
Ventricular depolarization occurs BEFORE SA node fires
WIDE QRS bc stimulation occurs BELOW AV node
NO P wave
COMPENSATORY PAUSE to reorganize SA and AV nodes for impulse conduction

A

PVC

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

PVC

A

Ectopic focus from a ventricle (cell decides to contract early/out of order)
Ventricular depolarization occurs BEFORE SA node fires
WIDE QRS bc stimulation occurs BELOW AV node
NO P wave
COMPENSATORY PAUSE after the PVC to reorganize SA and AV for impulse conduction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Are PVCs abnormal?

A

No

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

PVC telemetry characteristics…

A
Compensatory pause ==> beat after PVC feels like a "thump in the chest" bc the ventricles have a lot of time to fill c blood bc of the compensatory pause 
Bigeminy 
Trigeminy 
Couplet 
Unifocal 
Multifocal
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Bigeminy

A

Every OTHER beat is PVC

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Trigeminy

A

Every THIRD beat PVC

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Couplet

A

TWO PVCs paired together

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Unifocal

A

PVCs all the same –> same irritated area in Vs causing the early contraction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Multifocal

A

PVCs appear different –> more than one area irritated and they all contribute to early contraction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

PVC potential causes…

A
Caffeine/energy drinks
Nicotine
Stress
Overexertion
Hypo- or hyperkalemia
Ischemia
Cardiomyopathy
Cardiac irritation
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

PVC characteristics

A

Increased frequency ==> decreased filling time ==> DECREASED PRELOAD ==> decreased SV ==> decreased CO

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Decreased CO is dangerous c PVCs when…

A
Couplet
Multifocal
> 6/min.
Triplets 
*termination points for activity ==> can degrade to life-threatening rhythm*

May progress to V-tach or V-fib
Anti-arrhythmia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

It is important to note if PVCs…

A

Increase c exercise
Decrease c exercise
Stay the same c exercise

monitor BP and sx for termination points

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Should premature beats be taken into consideration when calculating HR?

A

No - HR should be calculated on a part of the strip that does not contain the PVCs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

3 or more consecutive PVCs
NO P wave
WIDE QRS (bc impulse BELOW AV node)
V rate 100-250bpm

A

V-tach

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

When is the QRS wide?

A

When the impulse comes from BELOW the AV node

17
Q

V-tach

A

3 or more consecutive PVCs
NO P wave
WIDE QRS (bc impulse comes from BELOW AV node)
V rate 100-250bpm

18
Q

V-tach V rate…

19
Q

V-TACH =

20
Q

V-tach possible causes…

A
Ischemia/infarction
CAD
HTN
Digoxin
Electrolyte imbalance
21
Q

V-tach management

A

Cardioversion (time c QRS)

Defibrillation (increased Joules of energy to reset the rhythm)

22
Q

V-tach characteristics

A
CO greatly affected ==> decreased HR, decreased filling time, NO atrial contraction
Lightheadedness
Syncope
Angina
Weak, thready pulse
Disorientation
23
Q

V-tach telemetry characteristics…

A

NO P wave

WIDE QRS

24
Q

“Twisting of the points”

A

Torsades de Pointes

25
Torsades is a characteristic of...
V-tach
26
Torsades occurs when...
V-tach changes axis ==> bad prognostic factor that makes the V-tach harder to defibrillate
27
Characteristics of torsades
Twisting around the isoelectric line Occurs during V-tach Significant drop in CO Cardioversion
28
Quivering of ventricles, so no real contraction, and the atria do not really contract much NO CO Multiple ectopic foci --> contractions not synchronous Zig-zag pattern Progression of V-tach
V-fib
29
V-fib
Quivering of ventricles, so no real contraction, and the atria don't really contract much NO CO Multiple ectopic foci c no synchronous contraction Zig-zag pattern Progression of V-tach
30
V-FIB =
EMERGENCY
31
V-fib possible causes...
Progression of V-tach Infarction/ischemia HTN Digoxin toxicity
32
V-fib management
Defibrillation CPR O2 Meds
33
Altered electrical conduction during angina T wave inversion T wave flattened or peaked Changes in ST segment at least 1mm (elevation is STEMI; depression if NSTEMI)
MI
34
The zone of ischemia in an MI looks like what on a telemetry strip?
T wave inversion or flattening
35
The zone of infarction in an MI looks like what on a telemetry strip?
Q wave/STEMI (elevated) (transmural MI) --> > 0.04 sec; at least 1/4 the heigh of R wave in same QRS Non Q-wave/NSTEMI (depressed) (sub-endocardial MI) --> area of myocardium can re-infarct if not managed Bundle branch blocks