Eating and Feeding Disorders Flashcards

(58 cards)

1
Q

What is an Eating Disorder?

A
  • Consistently below or above a person’s caloric needs to maintain a healthy weight
  • Can be accompanied by anxiety and guilt (varies with each disorder)
  • Occurs without hunger or fails to produce satiety (comfortable fullness)
  • Results in physiologic imbalances or medical complications
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2
Q

Factors Contributing to Eating Disorders

A
  • Genetics
  • Onset of puberty
  • A vulnerable personality
  • Being female
  • History of obesity
  • Dieting that becomes uncontrolled
  • Major life changes or stressors
  • Family functioning style
  • Sociocultural emphasis on slimness
  • Perfectionism
  • Impulsivity
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3
Q

Physiological Factors causes of Eating Disorders

A
  • Low self-esteem
  • Feelings of inadequancy, lack of control in life
  • Depression, anxiety, stress, loneliness, trauma
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4
Q

Interpersonal Factors causes of Eating Disorders

A
  • Troubled relationships
  • Difficulty expressing emotions
  • Hx of being teased based on size/weight
  • Hx of physical or sexual abuse
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5
Q

Social Factors causes of Eating Disorders

A
  • Cultural pressues that glorify “thinness” or muscularity
  • Narrow definitions of beauty
  • Cultural norms that value people on the basis of physical apperance and not inner qualities/strength
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6
Q

Biological Factors

A
  • irregular hormone functions
  • Genetics
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7
Q

Neurobiological Etiology of Eating Disorders

A

Demonstrates that altered brain serotonin function contribute to the dysregulation of appetite, mood, and impulse control

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8
Q

Environmental Etiology of Eating Disorders

A
  • Childhood trauma and sexual abuse have been reported in 20%-50% of patients with eating disorders.
  • Those with abuse histories have a poorer prognosis.
  • Culture influences the development of self-concept and satisfaction with body size.
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9
Q

Treating Eating Disorders

A
  • Individuals with eating disorders rarely seek help
  • They typically are not motivated to change
  • They often leave treatment
  • Some recover spontaneously, whereas others have long-term problems
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10
Q

Anorexia Nervosa Characteristics

A
  • Intense fear of weight gain
  • A severely distorted body image
  • Restriction of calories relative to requirements with significantly low BMI
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11
Q

Restricting Type AN

A

Describes individuals that do not regularly engage in binge-eating or purging behavior (weight loss accomplished through dieting, fasting and/or excessive exercise)

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12
Q

Binge Eating and Purging Type

A

Refers to those who regularly engage in binge-eating or purging behavior (self-induced vomiting or misuse of laxatives, diuretics, or enemas)

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13
Q

Clinical Signs and Symptoms of AN

A
  • Low body weight (15% or more below what is expected for age, height, activity level)
  • Body mass index (BMI) is the gauge used to determine the severity of this disorder. (mild with a BMI of 17 or more, moderate with a BMI of 16 to 17, severe with a BMI of 15 to 16, and extreme when the BMI is less than 15)
  • Amenorrhea
  • Lanugo
  • Mottled, cool skin on extremities
  • Peripheral edema
  • Lack of energy, fatigue; muscular weakness
  • Constipation
  • Low blood pressure, pulse and temperature
  • Abnormal lab values
  • Impaired renal function
  • Decreased bone density
  • Anemic pancytopenia
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14
Q

Amenorrhea

A

the loss of menstrual periods in girls and women post-puberty

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15
Q

Lanugo

A

downy growth of body hair on the face and back

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16
Q

Epidemiology of AN

A
  • Lifetime prevalence is 0.5%
  • Female-to-male ratio 3:1
  • Commonly begins in adolescence or in young adults; uncommon before puberty or after age 40
  • Regardless of gender, disordered eating is more common among athletes who participate in sports that emphasize aesthetics or leanness for advantage in competition
  • The prevalence of eating disorders may also be higher among individuals who identify themselves as LGBTQ+
  • Anorexia nervosa is less common than bulimia nervosa
  • Co-morbid with bipolar disorder, anxiety disorders, OCD, depressive disorders, PTSD and trauma-related disorders and alcohol or substance use disorders
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17
Q

Biological Etiology of AN

A
  • Genetic and familial predispositions; 50-60%
  • Genetic correlations between AN and major depressive disorder, anxiety disorders, OCD and schizophrenia
  • Glucose and lipid metabolism ?
  • Neurobiological: Tryptophan and impact on serotonin synthesis
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18
Q

Psychological and Cognitive Etiology of AN

A
  • Ego-syntonic disorder; know actions are harmful but believe benefits outweigh the harm
  • Struggle significantly with emotional identification, regulation, and processing
  • Exhibit low distress tolerance and deficits in behavioral control in response to distress.
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19
Q

Environmental Etiology of AN

A
  • Internalization of a thin body ideal
  • Associated with cultures that value thinness
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20
Q

Risk Factors AN

A
  • Being female
  • Family history of eating disorders
  • History of obesity
  • Dieting
  • Over exercising
  • Low self-esteem
  • Body dissatisfaction
  • Lack of assertiveness
  • Other eating disorders
  • History of abuse
  • Comorbid conditions
  • Distorted body image
  • Media
  • Fashion industry
  • Being an athlete
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21
Q

Warning Signs of AN

A
  • Development of food rituals (e.g., eating foods in certain orders, excessive chewing, rearranging food on a plate)
  • Consistent excuses to avoid mealtimes or situations involving food
  • Excessive, rigid exercise regimen; despite weather, fatigue, illness, or injury, the need to “burn off” calories taken in
  • Withdrawal from usual friends and activities
  • Behaviors and attitudes indicating that weight loss, dieting, and control of food are becoming primary concerns
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22
Q

Clinical Course of AN

A
  • Chronic condition with relapses characterized by significant weight loss; 1-year relapse rate is approx. 50%
  • Often continue to be preoccupied with food
  • 10% to 25% go on to develop bulimia nervosa
  • 1 in 5 anorexia nervosa deaths is by suicide
  • Poor outcome related to initial lower minimum weight, presence of purging, and earlier age of onset
    *** Difficult to treat, but recovery can occur in Anorexia Nervosa! **
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23
Q

Complications Due to Weight Loss/Starvation

A
  • Musculoskeletal: loss of muscle mass & fat, early onset osteoporosis
  • Metabolic: hypothyroidism (lack of energy, weakness, intolerance to cold & bradycardia), hypoglycemia, electrolyte abnormalities
  • Cardiac: bradycardia, hypotension, loss of cardiac muscle, small heart, cardiac arrhythmias, chest pain, sudden death
  • Gastrointestinal: delayed gastric emptying, bloating, constipation, abdominal pain, gas & diarrhea, GERD, hemorrhoids
  • Reproductive: amenorrhea, irregular periods, loss of libido, infertility
  • Dermatologic: dry, cracking skin & brittle nails due to dehydration, lanugo (fine baby-like hair over body), edema, acrocyanosis (bluish hands & feet), & hair thinning, yellowish discoloration of skin, poor wound healing
  • Hematologic: leukopenia, anemia, thrombocytopenia, hypercholesterolemia, hypercarotenemia
  • Neuropsychiatric: abnormal taste sensation(possibly due to zinc deficiency) apathetic depression, mild organic mental symptoms, sleep disturbances, & fatigue
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24
Q

Initial goals of AN treatment

A
  • Depends on acuity of the patient
  • Initiating nutritional rehabilitation; health teaching and promotion
25
Later goals of AN treatment
* Resolving conflicts around body image disturbance * Increasing effective coping * Addressing underlying conflicts relating to maturity fears and role conflict * Assisting family with healthy functioning and communication
26
Treatment modalities AN
* Hospitalization usually necessary * Intensive therapies * Outpatient partial hospitalization (for patients who have been stabilized)
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Criteria for Hospitalization for AN
* Extreme electrolyte imbalance or weight below 75% of ideal body weight * Less than 10% body fat * Daytime heart rate less than 50 beats/minute * A systolic blood pressure of less than 90 * Temperature less than 96 degrees F * Arrhythmias
28
Once medically stable tx for AN
* A weight restoration program begins that allows for incremental weight gain * Treatment goal is set at 90% of ideal body weight * Precise mealtimes, adherence to selected menu, observation during and after meals and regularly scheduled weighing * Constant monitoring during bathroom trips * A potential complication is refeeding syndrome or a serious and potentially fatal condition that can occur during refeeding. It’s caused by sudden shifts in the electrolytes that help your body metabolize food * During refeeding the individual participates in milieu therapy focusing on eating behavior and underlying feelings of anxiety, dysphoria, low self-esteem, and lack of control
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Pharmacotherapy AN
* No FDA approved medications and research doesn't support pharmacological agents * Fluoxetine has proven helpful for obsessive-compulsive behavior (after the client has reached a maintenance weight)
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Integrative Therapy AN
* Yoga * massage * acupuncture * bright light therapy
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Psychological Therapies AN
No empirical evidence to support any specific psychotherapy model in adults with anorexia nervosa. However, in adolescent patients with anorexia, there is evidence to support its use (insight-oriented therapy, family-based treatment, adolescent focused therapy and CBT)
32
Bulimia Nervosa (BN)
An eating disorder in which the individual engages in recurrent episodes of uncontrollable binge eating and compensatory behavior to **avoid weight gain** through purging methods such as self-induced vomiting, use of laxatives, diuretics or excessive exercise
33
Bulimia Nervosa vs. Anorexia Nervosa
* More prevalent * Older at onset (mean onset 18) * Typically, of normal weight * Generally, not life-threatening * Treatment is usually outpatient * Outcomes better * Mortality rates lower * Medications are effective
34
Biological Etiology of BN
* Neuropathological; changes in the brain may be due to eating dysregulation * Genetic and familial predispositions; 60% * Gene connection * Biochemical: Lower brain serotonin
35
Psychological and Cognitive Etiology BN
* Anxiety disorders or low self-esteem * Impulsivity and compulsivity * Chaotic, non-nurturing family relationships * Difficult interpersonal relationships * Triggers: stress, poor body self-image, food, restrictive dieting, or boredom
36
Environmental Etiology of BN
* Internalization of a thin body ideal; weight-based teasing or bullying * Childhood sexual or physical abuse, traumatic events and environmental stress
37
Clinical Course BN
* Few outward signs; initially do not appear physically ill * Often at or close to normal weight * Binge and purge in secret/doesn’t come to the attention of others * Treatment often delayed for years * Treatment initiated when control of eating is lost * Once treatment is complete typically there is complete recovery, except if depression &/or personality disorders are present * Individuals present as overwhelmed, overly committed, “social butterflies”, who have difficulty setting limits & establishing boundaries * They have enormous rules about food & food restriction * They **feel shame, guilt & disgust** regarding their binge eating & purging * They may also be compulsive and impulsive in other areas of their life
38
Risk Factors for BN
* Binge-eating behaviors * History of AN (1/4 - 1/3 of individuals) * Depressive signs and symptoms * Problems with interpersonal relationships * Impulsive behaviors * Increased levels of anxiety and compulsivity * Possible substance use disorders
39
Warning Signs of Bulimia Nervosa
* Evidence of **binge eating**, including disappearance of large amounts of food in short periods of time or finding wrappers and containers indicating the consumption of large amounts of food * Evidence of **purging** behaviors, including frequent trips to the bathroom after meals, signs and/or smells of vomiting, presence of wrappers or packages of laxatives or diuretics * Excessive, rigid exercise regimen; despite weather, fatigue, illness, or injury, the compulsive need to “burn off” calories taken in * Creation of lifestyle schedules or rituals to make time for binge-and-purge sessions * Withdrawal from usual friends and activities * In general, behaviors and attitudes indicating that weight loss, dieting, and control of food are becoming primary concerns * **Parotid swelling** or unusual swelling of the cheeks or jaw area * Calluses and/or scars on the back of the hands and knuckles from self-induced vomiting,** Russell’s Sign** *** Dental caries, tooth erosion** and discoloration or staining of the teeth
40
Complications of Eating Disorders due to **Purging**
* **Dental**: erosion of dental enamel * **Neuropsychiatric**: seizures, fatigue, weakness, mild organic mental symptoms * **Cardiac**: Ipecac cardiomyopathy, arrhythmias * **Dermatologic**: Russell’s Sign
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Recognize Cues of BN
* Determine the patient’s perception of the problem * Complete physical assessment, including labs * Psychosocial history including screening for suicide or self-harm behaviors, interpersonal and social functioning * Eating habits and fluid intake; history of dieting * Daily activities, including exercise * Value attached to a specific shape and weight
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Hospitalization in Bulimia Nervosa
Only if experincing life-threatening complications and suicide risk
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Treatment or Take Action for BN
* Treatment is usually on an outpatient bases unless the individual is suicidal or past outpatient treatments have failed * Stabilizing and normalizing eating; interrupt the binge-purge cycle * Restructuring dysfunctional thoughts and attitudes about eating, weight, & shape * Teaching healthy boundary setting * Nutrition counseling (stabilize & normalize eating) * Behavioral techniques such as using a diary to record binges and purges and precipitating emotions and environmental cues
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Pharmacotherapy BN
SSRIs (effective in treating binge eating and purging); fluoxetine is the only FDA approved medication for the treatment of BN in adults; most effective in conjunction with CBT
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Psychological Therapies BN
* Cognitive behavioral therapy (CBT)-**first line treatment for BN** * Dialectical behavioral therapy (DBT) * Group psychotherapy & support groups * Family therapy not usually used due to age of individual
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Binge Eating Disorder (BED)
An eating disorder characterized by recurrent episodes of binge eating, with accompanying marked distress and impaired control over such behavior
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Epidemiology & Comorbidity of BED
* Most common eating disorder * Lifetime incidence is 3.6 for women, 2.1% for men * Prevalent among females * All racial and ethnic groups seem to be represented fairly equally * BED occurs in normal-weight/overweight and obese individuals * Approximately half of the risk for BED is genetic * 79% of those with BED have another psychiatric disorder * The most prevalent psychiatric disorders associated with BED are specific phobia, social phobia, PTSD and alcohol abuse or dependence * Impulsivity and reward sensitivity, Low self-esteem, body dissatisfaction, difficulty coping with feelings * History of trauma and adverse childhood events * History of food insecurity
48
Signs & Symptoms of Binge Eating Disorder
* Frequent episodes of eating large quantities of food in short periods of time * Feeling out of control overeating behavior during the episode * Feeling **depressed, guilty, or disgusted** by the behavior * There are also several behavioral indicators of BED including eating when not hungry, eating alone because of embarrassment over quantities consumed, eating until uncomfortably full * Often have upper and lower GI problems that bring them to the attention of healthcare professionals
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Recognize Cues or Assessment BED
* Determine the patient’s perception of the problem * Complete physical assessment, including labs * Psychosocial history * Assess nutritional pattern * Assess the history of weight cycling (i.e. gains and losses) * Careful history of binge-eating triggers, foods and frequency
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Health Consequences of Binge Eating Disorder
* Hypertension * High cholesterol levels * Heart disease * Diabetes mellitus * Gastrointestinal diseases * Gallbladder disease * Musculoskeletal problems
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Treatment for BED
* Hospitalization **not usually required**; outpatient treatment * Health teaching and health promotion; healthy eating and excercise
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Pharmacotherapy for BED
SSRI’s Lisdexamfetamine dimesylate
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Psychological for BED
* Individual or group CBT * DBT * Support Groups
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PICA
* **Ingestion of substances that have no nutritional value, such as dirt or paint** * The prevalence of pica is unknown but among institutionalized children, the prevalence of this disorder may be as high as 26% * Usually begins in early childhood and lasts for a few months; condition can also appear in adolescence or adulthood. * Males and females are affected equally. * Monitoring of the individual’s eating behavior is obviously an essential aspect of treating this problem. * Behavioral interventions such as rewarding appropriate eating can be helpful. *
55
Feeding Disorder: Rumination Disorder
* Characterized by **undigested food being returned to the mouth. It is then rechewed, reswallowed, or spit out** * May be diagnosed after 1 month of symptoms; Can begin at any age. In infants, the onset is usually between 3 and 12 months * Ruminating behaviors occur more frequently among people with intellectual disabilities. * Childhood neglect is a predisposing factor to the development of this disorder
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Interventions for Rumination Disorder
* Repositioning infants and small children during feeding * Improving the interaction between caregiver and child and making mealtimes a pleasant experience often reduces rumination * Distracting the child when the behavior starts can also be helpful * Family therapy may be required
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Feeding Disorder: Avoidant/Restrictive Food Intake Disorder
* Can result in significant weight loss, nutritional deficiency, dependence on supplements or enteral feeding, and marked interference with functioning. * For some, **food avoidance **may be related to **strong dislikes** related to sensory qualities of food. Appearance, color, smell, texture, temperature, and taste are implicated in food refusal. * Males and females are equally affected by this eating disorder of infancy and early childhood. * Personal anxiety and family anxiety seem to be risk factors
58
Treatments for Avoidant/Restrictive Food Intake Disorder
* The primary treatment modality is some form of behavioral modification to increase regular food consumption. * Families caring for a child with a feeding disorder often need support and education in specific behavioral techniques, but family therapy is not usually necessary. * The treatment of anxiety and depressive symptoms may be helpful in some cases.