eating disorders - definitions Flashcards

(39 cards)

1
Q

define eating disorder

A

“a persistent disturbance of eating behaviour or behaviour intended to control weight, which significantly impairs physical health or psychosocial functioning”

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

issues with the definition of eating disorders (4)

A

in the eye of the beholder

do models and gymnasts and ballerinas fit this?
* maybe not cos their goal isn’t to become thin but to achieve a sporting goal

not just about weight

gender, age, and ethnicity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

younger cases of eating disorders (2)

A

feeding disorders
avoidant/restrictive food intake disorder

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

companies which determine what an eating disorder is (2)

A

DSM-5
ICD - international classification of disease - this follows DSM

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

incorrect stereotypes of eating disorders

A

only thin, white, young women
* only 15% with eating disorders are underweight - majority is normal weight

age, gender, and ethnicity doesn’t mean they don’t have an ED

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

BMI as a measure
* healthy rage
* in young people
* overweight categories

A

19-25 = healthy range (varies with ethnicity)

BMI is not meaningful in younger people - adjusted for age using expected weight for height:
<85% = underweight
< 70% = dangerously underweight

overweight = BMI > 25
obese = BMI > 30 (more levels within this e.g. morbidly obese > 40)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

issues of using BMI with treatments

A

can be used as a barrier to treatment - only when it is really bad
but many people with ED are healthy weight and so resources aren’t given to them

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

anorexia nervosa - diagnostic criteria (5)

A

persistent restriction of energy intake leading to significantly low body weight
* in context of age, sex, developmental trajectory, and physical health

either fear of gaining weight and becoming fat or persistent behaviour that interferes with weight gain

any of these:
* disturbance in way ones body weight or shape is experienced (body dysmorphia)
* undue influence of body shape and weight on self-evaluation
* persistent lack of recognition of seriousness of current low body weight

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

subtypes of anorexia nervosa (2)

A

restricting
binge-eating/purging

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

issues with anorexia nervosa diagnosis (6)

A

early stages might not be thin enough for intervention

athletes therefore defined as overweight due to muscle weight

ballerinas and gymnasts defined as underweight due to their sport

people differ in set point for weight - so BMI isn’t very useful

definitions related to BMI are arbitrary - they’ve changed over the years

self reported height and weight

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

difficulty spotting those with anorexia

A

better at spotting extreme examples
* especially seeing before/after states
* where BMI is lower than 17.5 it is easy to spot

however starvation could be due to other reasons - lack of resources

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

bulimia nervosa - diagnostic criteria (4)

A

recurrent episodes of binge eating (eating more than most people would in a discrete period of time under similar circumstances)
* with a lack of control over eating during the episode

recurrent inappropriate compensatory behaviour in order to prevent weight gain
* self-induced vomiting, laxative/diuretics misuse, fasting, excessive exercise

binges and compensatory behaviours occur on average at least once a week for 3 months

self-evaluation is unduly influenced by body shape/weight
* does not occur exclusively during episodes of anorexia nervosa

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

issues with bulimia nervosa diagnosis (3)

A

defining what a binge is
* subjective = loss of control
* objective = loss of control + excessive intake
* excessive intake = over 2000kcal in one go

defining compensatory behaviours
* vomiting isn’t always self-induced
* is exercise for health or to control weight

how often do these need to happen
* 1x week for 3 months - but this is ever changing too with new criteria

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

binge-eating disorder diagnostic criteria

A

recurrent episodes of binge eating
* eating in a set time period more than most people would under similar circumstances
* lack of control over eating in this episode

episodes have 3 or more of the following:
* more rapid eating
* eating till feeling uncomfortably full
* eating lots when not hungry
* eating alone from embarrassment
* feeling disgusted, depressed, or guilty

marked distress regarding binge eating
at least 1x week for 3 months
no purging/compensatory behaviours

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

issues with BED diagnosis

A

recent formal diagnosis - only 2013 with DSM-5 → so still debating levels for diagnosis

need to understand motivation for making this a category in DSM-5

distress and need for treatment

or for insurance money for clinicians as a way to treat lots of obese patients

BED patients can have trouble accessing services - prioritising anorexia over BED, viewed as higher risk - more immediate whilst BED causes premature deaths

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

OSFED diagnostic criteria

A

other specified feeding and eating disorders

atypical cases

present with many symptoms of other EDs but not full criteria for diagnosis

introduced due to:
* people with issues needing help
* insurance industry would only pay out on diagnosed cases

17
Q

OSFED - atypical disorders (5)

A

atypical anorexia nervosa
* weight loss but within or above normal range

atypical bulimia nervosa
* low frequency and limited duration

atypical binge-eating disorder
* low frequency and limited duration

purging disorder

night eating syndrome (eat at night whilst semi-conscious and don’t remember it the next day)

18
Q

ARFID diagnostics: who, what (4) and common treatment type

A

avoidant/restrictive food intake disorder
primarily in children and young people

  • disturbance in eating or feeding
  • substantial weight loss/lack of weight gain
  • nutritional deficiency - therefore dependence on supplements
  • absence of beliefs about food or fear of weight gain - more focus on feeding than eating consequences

treatments are primarily behavioural - focused on anxiety and exposure to food

19
Q

ARFID subtypes (3)

A

sensory-based avoidance
* refuse intake based on smell, texture, colour, brand, presentation

lack of interest
* in consuming or tolerating food

food associated with fear-evoking stimuli
* developed from learned history

20
Q

issues with diagnoses of EDs (5)

A

diagnosis of EDs doesn’t do what it should do
* 40-50% don’t fit into main diagnoses
* OFSED is largest group - all atypical
* many fail to stay in one diagnosis
* diagnosis doesn’t indicate best treatment

therefore shift away from rigid diagnosis → move towards transdiagnostic model

21
Q

comorbidity of EDs with other psychological problems (4)

A

anxiety - OCD, social anxiety

depressed

personality disorder - anxiety and impulsivity based

alcohol and substance use - alcohol as higher risk

22
Q

reasons for high mortality with EDs (6)

A

cardiac complications

muscular weakness (including cardiac failure)

osteoporosis

liver damage

oesophageal tearing

fainting

23
Q

epidemiology - incidence and prevalence - which is used with EDs most

A

incidence = number of new cases in set window of time

prevalence = number of current cases or people who have had it in the last year

slow onset + secrecy + slow diagnosis = hard to calculate incidence

so focus on prevalence

24
Q

prevalence of different disorders (4)

A

lifetime prevalence rates:

anorexia nervosa = 4% women, 0.3% men

bulimia nervosa = 3% women, 1% men

binge eating disorder = 2.8% women, 1% men

indication that OFSED has highest lifetime prevalence

western cultures = binge eating disorder up to 6.1% women and 0.7% men

25
issues using prevalence
can’t rely on medical records * make assumptions about who would have eating disorders → GPs do this too * only can measure those with diagnosis not how many which aren’t caught * most focus on young women (14-30 years) increase in numbers doesn’t mean actual increase in cases but just increase in diagnosis rates → more awareness
26
impact of westernisation on EDs
westernisation = increasing identification and prevalence of EDs more cases amongst non-whites in recent years study in Fiji → linked introduction of western media and EDs why - body image and control issues?
27
proposed causal factors of EDs (2)
sociocultural factors neurobiological factors
28
sociocultural factors of EDs (5) - strength of evidence (4)
early parenting, abuse, bullying, emotional invalidation, childhood obesity etc. weak causal evidence: * lack longitudinal data * selective sampling * risk of selective memory * misinterpretation of associations - does chaotic lifestyle cause ED vice versa
29
neurobiological factors of EDs (3) + issue with these
hypotheses are more common than actual evidence **genetics** * some evidence from twin studies * unknown which genes are responsible → directly or do they increase risk e.g. perfectionism or serotonin mechanisms that predispose impulsivity or compulsivity **hypothalamic damage** - prevents hunger * but anorexia patients report being hungry starvation effects go away when the person eats * mood and cognitive deficits, social isolation, behavioural inactivation issue of causality * does biology cause ED or ED cause changed biology
30
maintenance of EDs - why look at this
related to treatments → idea of interrupting maintenance mechanisms to stop disorder can start in many ways - limited ways of maintaining it if we can’t identify causes can look at maintenance mechanisms instead
31
cognitive patterns of ED maintenance (3)
low self-esteem negative self-attribution perfectionism each have a self maintaining cycle = low self-esteem → don’t look for good parts of yourself → don’t see the positives → feeds into low self-esteem
32
cognitive central belief systems (2)
broken cognitive link between eating and weight * drives restriction, then binging, then weight gain in a cycle * strong cognitive dissonance overvaluation of appearance and weight * used to define self as being acceptable person
33
cognitive maintenance of EDs - safety behaviours
behaviours which calm temporarily when anxious * binge-eating, restricting, body avoidance/checking, exercise, purging * can make you feel more in control short term have long-term consequences * feel worse so start these behaviours again * forms a cycle
34
emotional factors of ED maintenance
anxiety - triggers and maintains emotion for EDs * related to safety behaviours impacts other emotions * anger, loneliness, boredom * causality goes both ways with these depression as a consequence not cause → serotonin deficits following lack of eating
35
perceptual factors of ED maintenance
those with ED overestimate body size by 25-30% misperceive our weight - thought-shape fusion when we see food * idea thinking about eating is as bad as eating
36
social factors of ED maintenance
social pressure to be thin or muscular worsening body image as a result of being exposed to images on social media images based on criticism and self-comparison to “thinspiration”
37
formulating behaviours - binges triggers and setting conditions
done with individual to normalise what they eat when they binge-eat * identifies risks triggers of binging * emotional distress * availability of food/cue exposure setting conditions * teach better ways to deal with emotions and cope around food * starvation * disinhibition/permissive cognitions
38
formulating cases with EDs
way of understanding functions of ED - not diagnostic process allows for individual differences in history and potential causal/maintaining factors assumes core functions and processes that underpin most cases
39
models of formulating ED cases (2)
ABC model * antecedent → behaviour → consequence * focus on feedback loops that maintain the problem models have different levels of evidence and complexity → too complex means it isn’t easily implemented Slade (1982) model * central role of control * stress is one of the maintenance elements * food becomes more critical later in model but other cognitions and behaviours cause it earlier