ECC Flashcards
(211 cards)
1
Q
what does CPCR stand for?
A
cardiopulmonary cerebral rescusitation
2
Q
what is the aim of CPR?
A
perfusion of the heart, lungs and brain
ROSC
3
Q
why is it essential that the heart, lungs and brain are perfused?
A
require the most oxygen and glucose
4
Q
what is respiratory arrest?
A
apnoea
5
Q
what is caused by apnoea?
A
hypoxia
hypercapnia
6
Q
what can respiratory arrest lead to?
A
cardiac arrest
7
Q
what happens in the heart during cardiac arrest?
A
the patient has no cardiac output
8
Q
will patients in cardia arrest be breathing?
A
no
9
Q
what patients are at high risk of arrest?
A
trauma
systemically unwell
paediatric
geriatrics
iatrogenic causes like anaesthetic overdose
recently arrested
10
Q
when should CPR start?
A
as soon as we think a patient has arrested
11
Q
how long should you check for a heartbeat if a patient is not breathing?
A
no more than 2 seconds
12
Q
what will respiratory arrest rapidly lead to?
A
cardiac arrest
13
Q
who can help with CPR?
A
anyone
no schedule 3 procedures
ideally people with CPR training
14
Q
how can we best prepare for CPR?
A
regular training
have crash kit/box/trolley
crash alarm (or call for help)
15
Q
what is involved in BLS?
A
CPR cycle
oxygen therapy (including intubation)
16
Q
what is involved in ALS?
A
drug therapy
fluid therapy
cardioversion
17
Q
are BLS and ALS schedule 3 procedures?
A
BLS is not but ALS is and needs vet direction
18
Q
what position should patients be in ideally for CPR?
A
right lateral recumbancy
19
Q
where should the compressor be located in relation to the patient?
A
dorsal side
20
Q
what is the correct rate of cardiac compressions?
A
100-120 compressions per minute
21
Q
how deep should cardiac compressions be?
A
50% to 2/3 the width/depth of the thorax
22
Q
what should be felt for every cardiac compression?
A
femoral pulse
23
Q
what should be allowed for between each compression?
A
full elastic recoil of the chest
24
Q
what animals are cardiac pump compressions used on?
A
cats
small dogs
keel chest dogs
25
what animals are thoracic pump compressions used on?
medium to large breed dogs
26
if one cardiac compression technique isn't producing output what can be done?
change compression technique
27
what happens during cardiac pump compressions?
compression of the thorax directly over the heart
28
in what patient position are thoracic pump compressions performed?
lateral
dorsal
29
how are lateral thoracic pump compressions performed?
over the widest part of the thorax
30
how are dorsal thoracic pump compressions performed?
either over the caudal thorax or the xiphisternum
31
can nurses perform direct internal cardiac compressions?
yes but they cannot open the chest
32
in what animals may direct internal cardiac compressions be performed?
large breed dogs
33
when may direct internal cardiac compressions be performed?
if thoracotomy or laparotomy already being performed
if external compressions have not been effective
34
what rate should IPPV be given during CPR?
10-12 breaths per minute
35
how often should breaths be given during CPR?
every 6 seconds
36
when should ventilation start?
as soon as respiratory arrest suspected
37
how much should the chest be inflated by during CPR?
'normal amout"
guided by CO2 levels
38
what should patients be ventilated with?
ambubag on 100% O2
room air if no O2
39
what should be done if anything in the crash trolley is changed?
explain changes in training sessions
40
when should the crash trolley be checked and restocked?
checked monthly
restocked ASAP after every crash
41
what equipment is needed for airway access?
ET tubes (cuffed, whole sizes)
laryngoscope
ET tube tie
cuff puff
guide wire
plain swabs
intubeaze
u cath
42
what equipment is needed for IV access?
various size IV catheters
IV/IO connectors - primed
tape
scissors
cut down kit
no 11 scalpel blade
43
what equipment is needed for ventilation?
paediatric ambubag with capnograph connector and flow regulator
adult ambubag with capnograph connector and flow regulator
in line capnograph
44
what is the benefit of an inline capnograph?
doesn't need to calibrate
instant reading
45
what are the main first line drugs in a crash trolley?
low dose adrenaline (0.1mg/ml)
high dose adrenaline (1mg/ml)
atropine
saline (pre drawn)
pre prepared needles and syringes
ECG pads
46
why is an ECG necessary for CPR?
ECG will dictate drugs and whether defibrillation used
47
what is the mg/ml of high dose adrenaline?
1mg/ml
48
what is the mg/ml of low dose adrenaline?
0.1 mg/ml
49
when is adrenaline given during CPR?
asystole
50
what environment is adrenaline less effective in?
acidic
51
why is it significant that adrenaline is less effective in acidic environments?
acidic environment seen in CPA
52
what does adrenaline do?
positive inotrope
positive chronotrope
potent vasopressor
profound vasoconstriction
increase SVR
increase MAP
53
what does a positive inotrope do?
increase cardiac contractility
54
what does a positive chronotrope do?
increase heart rate
55
what is the benefit of a vasopressor in CPA?
vessels have no tone so no SVR
56
when is low dose adrenaline given?
initial dose unless anaphylaxis
57
when is high dose adrenaline given?
second line if low dose non effective
anaphylaxis
58
how can adrenaline be administered?
IV
IO
intratracheal
59
how should adrenaline not be administered?
intra-cardiac
60
how should drugs be dosed intra-tracheally?
double dose
fill syringe with 100% more air
61
when is atropine given?
profound brady cardia
peri-arrest
PEA
62
what is the role of atropine?
positive chronotrope
63
how should atropine be given?
IV (ideally central line)
IO
intra-tracheal
64
via what route should atropine not be administered?
intra-cardiac
65
how often can atropine be given?
only once
66
what other drug types may be in the crash trolley?
antagonists
propofol
glucose
67
what can be used to antagonise opioids?
naloxone
68
what can be used to antagonise benzodiazepines?
flumazenil
69
what can be used to antagonise dexmedetomidine?
atipamezole
70
what is the role of amiodarone?
antidysrhythmic through Na channel blocking
71
when is amiodarone used?
second line for prolonged VT or VF
if unable to cardiovert
72
how should amiodarone be given?
IV (ideally central)
IO
73
how should amiodarone not be given?
intra-cardiac
74
what is a risk when giving amiodarone?
anaphylaxis
75
what bolus dose of dextrose can be given if patient is tanking?
0.5 ml/kg of 50% dextrose
76
what is a risk when giving neat dextrose?
phlebitis
77
what is propofol?
phenol as lipid IV anaesthetic agent
78
when is propofol given?
respiratory distress that may lead to arrest if airway not controlled
79
how can propofol be given?
IV
IO
(not intra cardiac)
80
what equipment is needed for thoracotomy during CPR?
long sleeved surgical gown
gloves (6.5 and 7.5)
drape (150x180cm)
small and large chloraprep
thoracoctomy kit
no 11 blade
small radioopaque swabs
lap swabs
small and large finochietto retractors
internal defibrillator paddles
100ml 0.9% NaCl
81
what additional equipment is necessary for CPR?
capnography
suction
crash chart
ECG
defib and gel
IO drill
82
what monitoring equipment is less crucial during CPR?
pulse ox
NIBP
invasive BP
83
what is capnography a representation of?
ventilation
84
why is capnography important for CPR?
show perfusion and gas exchange which then indicates if metabolism is occurring
85
what EtCO2 indicates that compressions are adequate?
12 mmHg
86
what EtCo2 indicates ROSC?
>24 mmHg
87
what is the benefit of having suction during a crash?
removal of airway secretions
improve visualisation of the larynx
reduce risk of aspiration
88
what is the purpose of a crash chart?
clear record of what happened
timings and drugs
used for clinical governance
89
if a crash record cannot be completed at the time what should be done?
asap afterwards
its not always possible to record at the time
90
what is ECG used for?
shows if there is electrical impulse or conduction in the heart
shows whether cardioversion or drugs are necessary
91
does ECG show perfusion?
no
92
is defibrillation a schedule 3 procedure?
no - anyone can perform
93
when is defibrillation used?
non-perfusing rhythms
VF
pulseless ventricular tachycardia
94
what method other than defibrillation can be used for VF?
precordial thump
95
what is VF?
disorganised and random depolarisation all over the heart
96
what is VT in CPA?
Hr >180 with ventricular complexes
97
how much should the voltage be increased by each round of defibrillation?
50% each time
98
who is responsible for ensuring staff safety during defibrillation?
user
99
when should defibrillation not be used?
if the patient is very wet
if lots of spirit used
100
what is the go to for drug access?
IO
101
would IVFT be used during a crash?
unlikely - makes CO harder to acchieve
102
what must you be aware of following a crash?
patient may rearrest
103
what is involved in a CPR debrief?
what happened (facts)
what went well
what went less well
suggested improvements
no blame culture
be kind
104
what is type 1 decision making?
These are irreversible decisions that cannot be changed once executed. Therefore, they require careful thought
105
what are type 2 decisions?
These are reversible decisions. Even after executing them, you can change them if you like. Therefore, you must act on such decisions quickly
106
what is a risk with seizures?
hyperthermia
107
what is ARDS?
distributive shock within the lungs
108
what is involved in tracheostomy management?
minimal handling
keep clean
no water in kennel
nebulisation - 20mins QID
change SID or if causing an issue
109
what is the benefit of high flow oxygen?
comfort for patient as warmed / humidified
lung recruitment increased due to high pressure
less aggressive than ventilation
110
what is CPAP?
provision of positive pressure (PEEP) to airway while patient breathes to keep airway open (prevents collapse)
111
what areas of the body can be used for IO access?
greater tubercle of the humerus
trochanteric fossa
flat medial surface of te proximal tibia
tibial tuberosity
wing of ilium
112
what are the preferred IO access points?
greater tubercle of the humerus
flat medial surface of the proximal tibia
wing of ilium
113
via what route should glucose be given?
IV ideally central catheter
114
how can a 5% dextrose solution be made up?
0.5ml neat 50% dextrose to 9.5ml 0.9NaCl
gives 10ml of 5% solution
115
what is the rescue dose of dextrose?
0.5ml/kg of 5% dextrose
116
how is invasive blood pressure measured?
through arterial catheter and transducer to a monitor
117
how is central venous pressure measured?
via central line
118
what patients is CVP used in?
those at risk of volume overload
119
what lactate level is considered normal?
<2.4 mmols
120
what needs to be carried out before a blood transfusion?
cross matching
121
what is pH a measure of?
hydrogen ions present
122
what is base excess?
H ions needed to return the pH back to normal
123
what is the anion gap?
unmeasured ions in the blood (e.g. ketones, uric acid) that may be contributing to acidaemia
124
when is an elevated anion gap seen?
end stage renal failure
125
what is normal pH?
7.35-7.45
126
what is normal PaO2?
80-100 mmHg
127
what is normal PaCO2?
35-45 mmHg
128
what is normal bicarbonate?
21-24
129
what is normal base excess?
+2 - -2 mEq
130
what is the normal anion gap in cats?
12-24 mEq/L
131
what is the normal anion gap in cats?
13-27 mEq/L
132
what is the effect of respiratory acidosis on RR?
hyperventilation to blow off CO2
133
what is the effect of respiratory alkalalosis on RR?
hypoventilation to retain CO2
134
what are the effects of compensatory mechanisms in metabolic acidosis?
pH WNL
HCO3 very low
PaCO2 low
135
what are the effects of compensatory mechanisms in metabolic alkalosis?
pH WNL
HCO3 very high
PaCO2 high
136
what effect can lactate have on acid base?
cause metabolic acidosis
137
what can cause metabolic acidosis?
diarrhoea
DKA
renal failure
Addisons
lactic acidosis (sepsis)
138
what can cause metabolic alkalosis?
vomiting (loss of H+)
hypoalbuminaemia
upper GI obstruction
139
why does albumin affect acid base?
weak acid
140
what is Kirbys rule of 20?
lists critical parameters to be checked in critical care patients
141
what determines stroke volume?
preload
afterload
contractility
142
what drugs may be used to increase BP?
vasopressors
inotropes
143
what determines cardiac output?
HR
SV
144
what are the sections within Kirbys rule of 20?
fluid balance
oncotic pull (albumin)
glucose
acid base and electrolytes
oxygenation and ventilation
demenour/mentation
BP
HR and rhythm
temp
coagulation
RBC and Hb
renal function
immune status
GI motility
drug metabolism
nutrition
Pain
mobilisation
wounds
TLC
145
what reflex may be stimulated in head trauma patients?
cuchings (hypertension, reflex bradycardia)
146
what electrolyte derangement may be seen with head trauma?
hyperglycaemia (mechanism unknown)
147
what basic function may be affected by head trauma?
ventilation
148
why is ventilation affected by head trauma?
altered drive
149
why is it important to elevate raised ICP patients 30 degrees?
reduce CSF flow in brain
regurge risk
150
will mannitol be given to all head trauma patients?
potential to worsen haemorrhage
hypertonic saline instead
151
what assisted feeding tube could be used for head trauma patients?
O or PEG
NO is contraindicated due to ICP risk
152
what may be given to manage hyperfibrinolysis?
TXA
153
why may TXA be contraindicated in head trauma patients?
induces emesis - raising ICP
(dilute and give slow)
154
how can oral care reduce aspiration pneumonia risk?
removal of bacteria / saliva and secretions which can reduce incidence and severity of AP
155
what are the main considerations for patients on high flow O2?
lots of ongoign losses even though air is warmed and humidified
eye care crucial
156
what effect can reduction of pyrexia have on AP?
may make AP worse
157
why may TPN be needed in AP cases?
to prevent further regurge and allow lungs to rest
158
what is a preferable option to ventilation?
HFO2
159
what is needed before moving tetanus patients?
propofol / muscle relaxant bolus to facilitate movement
160
what is involved in the nursing care of the ventilator patient?
eye care
oral care
airway management (suction)
humidifcation
cleaning of tubes and lines
physio
positioning
U+/F+ managed
drugs needed
treat underlying disease
communication with team and owners
records
acid base/BG/electrolytes
161
what is the intervention value for GCPS?
6/24
5/20
162
what drop in MGCS should clinicians be notified about?
2 or more
163
what is most body heat produced by?
muscular activity (seizures / exercise)
164
where are thermoreceptors located?
peripherally
centrally
165
where is the thermoregulation centre located in the body?
anterior hypothalamus
166
what is indicated by thermoreceptors?
when temperature is above or below the set point
167
what does temperature above the set point trigger?
heat dissipation
168
what does temperature below the set point trigger?
heat conservation and production
169
define hyperthermia
increased body temperature (>39.2)
170
what are the causes of hyperthermia?
pyrexia
increased heat production due to increased muscular activity (seizures/exercise)
heat stroke
171
what are the 2 types of heat stroke?
classic
exertional
172
what is classic heat stroke caused by?
reduced heat loss
173
what is exertional heat stroke caused by?
overheating due to exercise in high temperatures
174
what is heat stroke caused by?
failure of heat dissipation
175
what factors may cause failure of heat dissipation?
Upper respiratory obstruction
Increased environmental temperature/humidity
Poor environmental ventilation
Circulatory compromise
Obesity
Breed predisposition
176
at what temperature is the patient at risk of permanent organ damage or failure?
>41.6
177
why are organs at risk at temperatures >41.6?
cell death and increased oxygen demand leads to DIC
178
what are the main clinical signs of heat stroke?
Stress
Hyperthermia
Tachycardia
Hypovolaemia
Hyperdynamic pulses
Peripheral vasodilation
Collapse
Hyperaemic MM (bright red) with rapid CRT
179
what temperature may patients be if heat stroke is advanced?
normothermic
180
why may patients with advanced heat stroke be normothermic?
due to impaired peripheral perfusion leading to colder extremities but hyperthermic core
181
what may be seen in advanced heat stroke?
Hypovolaemia
GI losses
Vasodilation
Increased intestinal mucosal permeability and impaired GI perfusion
Tachypnoea
Secondary respiratory complications
182
what can cause hypovolaemia in heatstroke patients?
GI losses
vasodilation and so relative hypovolaemia due to systemic compromise
183
what can increased GI mucosal permeability ad reduced perfusion lead to?
endotoxin translocation = sepsis
184
what may patients with severe GI compromise require?
mucosal protectants (omeprazole)
IV antibiotics
185
what secondary respiratory complications may be seen with heatstroke?
AP
pulmonary oedema
pulmonary haemorrhage (DIC)
obstruction risk
186
why do secondary respiratory complications occur?
panting
DIC
187
what are secondary complications of heat stroke?
impaired renal perfusion leading to AKI
CNS compromise
coagulopathies - DIC
188
why is impaired renal perfusion seen?
distributive shock
vasodilation to loose heat
hypovolaemia due to losses
189
how can heatstroke lead to CNS compromise?
direct thermal damage
secondary effects e.g. hypoglycaemia
190
what are the signs seen on blood work that indicate heatstroke?
Hypoglycaemia
Hyperbilirubinemia
Epithelial desquamation
Thrombosis
Myopathy
Electrolyte derangements
191
where is most heat lost when body temperature increases?
body surface
then panting
192
what does heat loss through the body surface lead to?
increase in peripheral circulation through vasodilation and so increase in cardiac output
results in overall decreased perfusion
193
at what temperature should patients be actively cooled?
>41 degrees C
194
when should active cooling end?
39.4 degrees C
195
why should active cooling end at 39.4 degrees C?
prevent hypothermia
196
how often should patients temperature be taken if they have heat stroke?
every 5 mins minimum
197
what is involved in emergency management of heatstroke patients?
active cooling
O2
maintain patent airway
IVFT
bloods
consider raised ICP
198
how can patients be actively cooled?
lukewarm water
run over the body
wipe over with cloths
DO NOT USE TOWELS LAID OVER
199
why is oxygen supplementation needed?
ventilation may be inadequate due to panting
200
what bloods may be required for heatstroke patients?
PCV
TP
glucose
electrolytes
coags
ABG
201
how may coagulation be affected by hyperthermia?
increase clotting time
202
why can heat stroke case increased ICP?
cerebral vasodilation due to peripheral vasodilation leading to raised ICP
203
should pyrexic patients be actively cooled?
no - it is beneficial
204
why should pyrexic patients not be actively cooled?
slows replication of pathogens
increases function of WBC
205
what are examples of antipyretic drugs?
paracetamol
NSAIDs
206
when may antipyretic drugs be given to patients?
patients with
cancer and immune compromise
207
should antipyretic drugs be given to all pyrexic patients?
no - pyrexia is of benefit to the patient
208
what can cause pyrexia?
Inflammatory disease
Neoplastic disease
Other causes
209
what types of inflammatory disease may cause pyrexia?
infectious
immune mediated
210
what are some causes of pyrexia other than inflammatory or neoplastic disease?
opioids
hepatic encephalopathy
transfusion reaction
211
what type of transfusion is particularly likely to lead to pyrexia?
xenotransfusion
