ECG Flashcards

(20 cards)

1
Q

Intro

A

PID
ECG date + time
Calibration

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2
Q

Calibration
- speed
- voltage

A

speed : 25mm/s
voltage: 1mm/mV (i.e. 1mV deflection should be 2 large squares in height)

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3
Q

Rate

A

Calculate by dividing 300 by number of large squares between R peaks OR (if irregular) - total R waves on ECG multiplied by 6

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4
Q

Causes of bradycardia (HR<60bpm)

A

physical fitness, hypothermia, hypothyroidism, SA node disease, beta blockers/digoxin

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5
Q

Causes of tachycardia (HR>100bpm)

A

exercise, pain, anxiety, pregnancy, anaemia, PE, hyovolaemia, fever/sepsis, thyrotoxicosis

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6
Q

Rhythm

A
  1. Regularity: mark 4 R waves on piece of paper
    (if irregular - AF, ectopic, 2nd degree HB, sinus arrhythmia, atrial flutter with variable block)
  2. Sinus rhythm
    - look for AF or flutter (saw-tooth baseline)
    - narrow complex tachy with no p waves - SVT
    - broad “ “ = VF, VT or rarely SVT/AF with BBB
    - brady with no p wave - SA arrest with junctional escape rhythm
    - p waves present but inconstant PR - HB
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7
Q

Axis

A

Leads I and II
- QRS predominantly +ve in I and II

If (predominantly) +ve in I and -ve in II –> LAD (leaving each other = left axis)

If (predominantly) -ve in I and +ve in II –> RAD (Reaching towards each other = R axis)

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8
Q

Causes of LAD

A

More electricity to L due to
- LV hypertrophy
- LA hemiblock
- LBBB
- inferior MI
- WPW
- VT

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9
Q

Causes of RAD

A

More electricity to R
- tall and thin
- RV hypertrophy (e.g. PE, lung disease)
- left posterior hemiblock
- lateral MI
- WPW

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10
Q

P wave

A

Use rhythm strip
- Height: <2 squares (increased in atrial enlargement e.g. pulmonary hypertension
- Morphology
1. Bifid (look like m) -P mitrale (L atrial enlargement - mitral stenosis
2. Peaked = p pulmonale (R atrial enlargement in pulmonary disease)

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11
Q

PR interval

A

Use rhythm strip
- Length: 3-5 small squares (120-200ms)
- Decreased: accessory pathway conduction (look for delta wave in WPW)
- Increased: AV node block (‘heart block’)
1st degree - PR>200ms (5 squares) and regular
2nd degree -
* Mobitz 1 - PR extends until QRS skips (failure of conduction of atrial beat)
* Mobitz 2 - constant PR in conducted beats but some P waves not conducted
* 2nd degree with 2:1/3:1/4:1 block: alternate conducted and non-conducted atrial beats (P:QRS)
3rd degree / complete heart block - complete dissociation between P waves and QRS complexes

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12
Q

What happens in complete heart block

A

Normal atrial beats not conducted to ventricles, which results in ventricles self-depolarising at much slower rate (ventricular escape rhythm)

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13
Q

QRS complex: all leads

A

Check in all leads
- Q wave <1 square wide and 2 squares deep is normal in I, aVL and V6 due to SPETAL DEPOLARISATION
- pathological Q waves

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14
Q

QRS complex: chest leads

A

Chest leads
- R wave progression: from dominant S to dominant T through V1-V6 (from mostly -ve to mostly +ve): transition point at V3/4
* Clockwise rotation e.g. TP after V4 - RV dilatation 2ndary to lung disease
* Dominant R wave in V1/2 - RVH, posterior MI

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15
Q

QRS complex: rhythm strip

A

Rhythm strip
- Lenght <3 small squares (120ms). Increased = BBB
* RBBB - QRS in V1 has M (RSR) pattern and QRS in V6 has W - MaRRoW
* LBBB - QRS in V1 has W pattern and V6 has RSR M pattern - WiLLiaM

nb: W pattern usually not fully developed;
- RSR may be seen with normal QRS length - incomplete BBB and is of no clinical significance

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16
Q

Causes of LBBB

A

Aortic stenosis
Ischaemia
HTN
Anterior MI
Cardiomyopathy
Conduction system fibrosis
Hyperkalaemia

17
Q

Causes of RBBB

A

RVH
Cor pulmonale
PE
ASD
ischaemia
cardiomyopathy

18
Q

QRS complex: V1 and V5/6

A

Height - ventricular hypertrophy
- S wave depth in V1 + tallest R wave in V5/6 = > 7 big squares = LVH eg HTN, AS, AR, MR, coarctation of aorta, HOC
- R wave dominant in V1 + S wave dominant in V5/6 - RVH e.g. pulmonary HTN, MS, PE = other signs present too e.g T wave inversion in R chest leads (V1-3) and RAD

19
Q

ST segment: elevation/depression

A

Check in all leads - measure from J point (start of ST segment)
- Elevation 1 or more small squares - infarction (or pericarditis or tamponade if in EVERY LEAD)
- Depression 0.5 or more small squares - ischaemia (or reciprocal change in POSTERIOR MI)

20
Q

ST segment: morphology

A
  • Normally upwardly concave