ECG Flashcards
(14 cards)
A nurse presents an ECG to you showing inferior ischaemia. What additional bedside investigation should you ask them for?
Right sided ECG
At minimum get a V4R lead
What % of new or possible new LBBB actually have ACS?
< 10%
STE in III >II suggest what?
right sided infarction
Look for STE in V1. Is specific to proximal right coronary artery occlusion
Posterior infarction may be isolated or in association with what other areas of infarction?
lateral or
inferior
STE in aVR and V1 suggest ischaemia in what coronar vessel?
LAD
What is the modified sgarbossa criteria?
- > 1mm concordant STE
- > 1mm concordant ST depression
- > 5mm or > 25% discordant STE
Used for those with LBBB or paced ECG
Excessive concordant STE is the weakest predictor of infarction
What are the two types of wellen’s t waves and where do you see them?
- biphasic
- deeply inverted
- anterior leads
Usualy V2, V3. May be seen in V4, occassionaly V5, V6
75% of pts with this finding have critical LAD stenosis
Why is it important to capture an ECG with a pt in pain and when pain resolves?
Show if ECG is dynamic
A pt has an inferior STEMI and also ST depression in V1 and V2. Is this reciprocal changes?
Get a posterior ECG to ensure STEMI not extending posteriorly
Are wellen T waves present during pain or when pain free
When pain free
Pain can actually makes them resolve
What is Wellen’s syndrome
- episode of angina
- typical T wave changes that come and go depending on pain state
- no pathological Q waves
- no loss of R waves
- none or minimal STE
- none or minimal troponin elevation
Without angina ECG may have Wellen’s sign but pt does not have Wellen’s syndrome
What are you looking for on a syncope ECG?
**The obvious **
- Ischaemia
- dysrythmia
- AV blocks
Intervalopathies
– Short PR (WPW)
– Long QTc
Genetic
–HCM
–Brugada
–ARVC (ACM)
What are the DDx of TWIs?
- CAD (ischaemia, Wellens, reperfusion)
- elevated ICP
- Pulmonary (PE, PTx, Pulm HTN, pneumonia, hyperventilation)
- ARVC (V1-V3), Brugada (V1-V2)
- wide QRS (BBB, PVC, paced, WPW)
- LVH, RVH with “strain”
- pericarditis, myocarditis
- hyperkalaemia, hypokalaemia
- mitraval valve prolapse
- normal in V1, aVR & IIII