ECG Flashcards

(60 cards)

1
Q

What is an ECG concerned with?

A

collection of cells

rapid conduction between cells via intercalated discs

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2
Q

In a unipolar electrode, what deflection would depolarisation moving towards a positive electrode give?

A

Upward deflection

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3
Q

In a unipolar electrode, what deflection would depolarisation moving away from a positive electrode give?

A

Downward deflection

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4
Q

When is amplitude maximal?

A

When positive electrode is on the vector

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5
Q

When is amplitude minimal/biphasic?

A

When positive electrode is perpendicular

No charge

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6
Q

In a unipolar electrode, what deflection would depolarisation moving towards a negative electrode give?

A

Downward deflection

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7
Q

In a unipolar electrode, what deflection would depolarisation moving away from a negative electrode give?

A

Upward deflection

Ie. Focally positioned negative electrode records same thing as diametrically opposite positive electrode

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8
Q

Describe a model for unipolar electrode

A

Strip of cells in bath with conducting fluid
Focally positioned positive electrode
Corners of bath connected to negative electrode

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9
Q

How is the strip affected in a unipolar electrode when it depolarises?

A

Inside positive, outside negative
Cells closest to electrode most influenced
Indifferent electrode sees average potential of strip

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10
Q

What does the potential difference reflect?

A

Movement of current in relation to positive electrode

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11
Q

Describe bipolar electrode

A

2 recording electrodes
Strip of cells in bath with conducting fluid
Focally positioned positive and negative electrode

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12
Q

How is the strip affected in a bipolar electrode when it depolarises?

A

Inside positive, outside negative
Wave of depolarisation moving towards positive - upward
Also moving away from negative - upward
Combined means bipolar deflection is larger than unipolar potential difference

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13
Q

How is the strip affected in a unipolar electrode when it repolarises?

A

Potential change is opposite to depolarisation - inside negative, outside positive
Towards positive - downward deflection
Away from positive - upward deflection

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14
Q

How does electrical current flow in the heart?

What does this allow?

A

Flows as if between 2 termInals in a volume conductor
Intra-thoracic contents also act as volume conductors

This allows surface recordings of potential differences provide 3D picture of electrical events in the heart

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15
Q

What is the recorded potential difference at any instant?

A

Vectorial resultant (mean vector) of several differently directed wavefronts

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16
Q

What would be recorded on ECG I if the positive electrode is at apex at atrial depolarisation?

A

Upward deflection - toward positive electrode

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17
Q

What would be recorded on ECG I if the positive electrode is at apex afteratrial depolarisation?

A

No deflection, due to delay as current passes through AV node

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18
Q

What would be recorded on ECG I if the positive electrode is at apex at ventricular depolarisation?

A
  1. Septum (LV to RV) - small negative, away from electrode (small number of cells)
  2. Main ventricular depolarisation - large upward, towards electrode (left bias, more cells, endo to epi)
    Repolarisation
  3. Base of ventricles - small negative, away from electrode
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19
Q

What would be recorded on ECG I if the positive electrode is at apex at ventricular repolarisation?

A

Away from electrode, therefore positive deflection

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20
Q

Which ventricular cells are first to repolarise?

A

Those which are last to depolarise (epicardial cells) - shorter action potentials

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21
Q

Limb leads of unipolar

A

One focally positioned positive electrode, others are indifferent (RL earth)

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22
Q

aVR

A

Right shoulder

Positive RA

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23
Q

aVL

A

Left shoulder

Positive LA

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24
Q

aVF

A

Feet (groin direction)

Positive left leg

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25
Chest leads
One focally positioned positive electrode in 6 specific positions on the chest V1-V6 4th intercostal space at right margin of sternum to 5th intercostal space at mid-axillary line
26
Bipolar limb leads
Two focally positioned electrodes (positive and negative) | Records PD between them
27
Limb lead I bipolar
Positive - LA (-30) negative - RA (-150) Lead - 0 degrees
28
Limb lead II bipolar
Positive - LL (groin) Negative - RA (-150) Lead - 60 degrees
29
Limb lead III bipolar
Positive - LL (90) Negative - LA (-30) Lead - 120 degrees
30
What is the p wave?
First wave, irrespective of polarity
31
What is the T wave?
Final wave | Unless U waves (rare) are present
32
What is the R wave?
First positive wave after P wave
33
What is a Q wave?
Any negative wave after P wave but before R wave
34
What is an S wave?
Any negative wave after R wave
35
What is a R’ wave?
Any positive wave after S wave
36
What does a P wave show?
Atrial depolarisation
37
What does QRS show?
Ventricular depolarisation
38
What does T show?
Ventricular repolarisation
39
P duration
Around 0.08s
40
P-R interval
<0.2s | Wave of excitation through AV node
41
QRS duration
Around 0.1s
42
What occurs during S-T?
Ejection of blood
43
What would be diagnosed if S-T duration is above baseline?
Recent heart attack
44
What occurs in T-P interval?
Ventricles filling with blood
45
In a normal ECG where would the largest QRS wave be detected?
LL II
46
Is there is a left axis deviation, where would this be detected?
LL I
47
Cause of left axis deviation
Hypertension Valvular heart disease LV hypertrophy
48
Where would right axis deviation by detected?
LL III
49
Cause of right axis deviation
COPD | Pulmonary hypertension
50
What would 1st degree heart block look like?
Long P-R
51
What would 2nd degree heart block look like?
Some P with no QRS
52
What would 3rd degree heart block look like?
Complete block | No AV conduction
53
Examples of ectopics
Ventricular extrasystole Tachycardia Atrial fibrillation Ventricular
54
Ventricular extrasystole
No preceding P wave | Next p missed, therefore gap before next complex
55
Tachycardia
Atrial/ventricular | Lots of P, not as many QRS
56
Atrial fibrillation
Rhythm not set by SA node, uncoordinated QRS complex irregular, gaps between No defined P wave
57
What else can influence electrical activity ?
Anti-arrythmic drugs
58
How are dysrythmias classified?
Site of origin of abnormality (atrial, junctional, super ventricular) Rate of change (tachycardia, bradycardia)
59
4 basic phenomena that cause changes
1. Abnormal pacemaker activity 2. Heartblock - unstable/inappropriate conduction via AV node 3. Delayed after depolarisation - XS Ca2+ -> Na+ influx 4. Re-entry - no extinction collision
60
Classes of drugs
1. Block Na+ channels eg. Lidocaine , reduce max rate of depolarisation 2. Beta-adrenoceptor antagonists eg. Atenolol 3. Block K+ channels eg. Amiodarone, slow repolarisation, prolong AP, increase refractory period 4. Ca2+ channel antagonists eg. Verapamil, slow conduction in nodes