EKG Flashcards

(46 cards)

1
Q

P wave

A

atrial depolarization

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2
Q

QRS complex

A

ventricular depolarization and atrial repolarization

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3
Q

T wave

A

ventricular repolarization

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4
Q

PR interval

A

movement of electrical activity from atria to ventricles

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5
Q

normal PR interval

A

0.12 - 0.20

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6
Q

what is happening in the PR interval

A

time impulse travels from SA node through internodal pathways in atria toward ventricles

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7
Q

long PR interval

A

greater then 0.20 seconds
indicates the impulse was delayed as it passed through atria, AV node, or AV bundle

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8
Q

short PR interval

A

less than 0.12 seconds
may be seen when the impulse originates in the atria close to the AV node or in the AV bundle

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9
Q

QRS complex represents

A

conduction of impulse form Bundle of HIS through ventricular msucle

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10
Q

normal QRS complex

A

0.12 seconds

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11
Q

0.12 seconds = ___ small boxes

A

3

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12
Q

R-R interval

A

ventricular rate and regularity

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13
Q

QT interval

A

time take from ventricles to depolarize, contract, and repolarize; represents total ventricular activity

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14
Q

ST segment

A

time between ventricles depolarized and repolarization of ventricles begins (ventricular contraction)

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15
Q

T waves

A

“resting phase” of cardiac cycle

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16
Q

Most important assessment other or while doing ECG

A

looking at your patient and there presentation

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17
Q

8 step ECG interpretation

A

(1) measure HR
(2) examine R-R interval
(3) examine P wave
(4) measure PR interval
(5) determine if each P wave is followed by QRS
(6) examine and measure QRS
(7) examine and measure QT
(8) diagnose the rhythm

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18
Q

six-second method

A

count the number of complete QRS complexes within a period of 6 seconds and multiply it by 10 to determine the number of QRS complexes in 1 min

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19
Q

Sinus Bradycardia

A

HR less than 60
originates from SA node

20
Q

treatment for sinus bradycardia

A

atropine / pacemaker

21
Q

sinus tachycardia

A

rate greater than 100bpm and less than 150bpm

22
Q

what causes sinus tachycardia

A

sympathetic stimulation, fever, hypovolemia, pain

23
Q

treatment for sinus tachycardia

A

beta blockers, calcium channel blockers, vagal nerve stimulus, antipyretics, pain management, antianxiety measures, carotid artery massage

24
Q

sinus node dysfunction

A

impulses originate elsewhere in atria
- premature atrial contractions
- SVT
- may decrease cardiac output secondary to tachycardia

25
Atrial dysrhythmias
characterized by rapid atrial rate - rapid ventricular response to symptoms
26
PAC's
often benign but may be warning sign for something else - enhanced automaticity of cardiac cells
27
sinus arrhythmia
rate 63-81 bpm
28
who is sinus arrhythmia common in
athletes and children
29
atrial fibrillation
atria contracting very rapidly, unable to empty, discharging greater than 400bpm - absent P waves
30
what happens in afib
- atrial chambers are unable to refill before contraction - inadequate ventricular filling - decreased stroke volume by 25% - blood in atria prone to form clots - increase risk for thrombotic stroke
31
supraventricular tachycardia (SVT)
- rate between 150 and 250 bpm - regular rhythm - undistinguishable P wave - QRS complex normal - R-R irregular
32
treatment for SVT
valsalvas maneuver, calcium channel blockers, digoxin, beta blockers, adenosine, cardioversion
33
atrial flutter
- rate greater than 250 (up to 350) - ventricular rate regular or irregular - atrial oscillations appear as saw tooth or flutter waves -ventricular rate ~80, atrial rate ~375 - QRS complex 0.10
34
treatment for atrial flutter
cardioversion, calcium channel blockers to regulate HR, digoxin, beta blockers
35
heart blocks are like __________
relationships
36
first degree AV block
- PR interval > 0.20 seconds - delayed conduction through AV node - pt usually asymptomatic; no treatment necessary - related to acute MI, CAD
37
second degree AV block
SA node impulse conduction is delayed or completely blocked - occurs in AV nodal area - P wave is present - PR interval is irregular or not measurable
38
difference between second degree AV block type 1 and type 2
Type 1 - P and QRS are far apart and then close together again Type 2 - P and QRS are normal, but P sometimes moves father or closer
39
junctional dysrhythmias
- SA node fails to fire - intrinsic rate 40-60 bpm - AV node become pacemaker - inverted or absent P wave
40
ventricular dysrhythmias
- life threatening - inadequate ventricular ejection - insufficient stroke volume - decrease cardiac output - decrease tissue perfusion
41
ventricular bigeminy
- beats originate in the ventricles - regular pattern - may lead to v tach or v fib
42
treatment for ventricular bigeminy
O2, antiarythmatics
43
ventricular tachycardia
- 3 or more PVC's occurring at a rapid rate, usually greater than 100bpm - may deteriorate into ventricular fibrillation
44
ventricular fibrillation
- most common cause of sudden cardiac arrest
45
treatment for ventricular fibrillation
defibrillation is treatment of choice and epinephrine
46
asystole
- represents complete cessation of electrical impulses - terminal rhythm