Emergencies Flashcards

1
Q

How are pneumothoraces categorised?

A
Primary = YOUNG, Spont due to rupture of sub-pleural bleb
Secondary = >55YO SMOKERS, Lung pathology
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2
Q

What are the risk factors for a pneumothorax?

A
Asthma/COPD
Infection: TB, pneumonia, lung abscess
CT disorder: Marfan's, Ehlers Danlos
Trauma/iatrogenic
Lung Ca
Lung disease: CF, fibrosis, sarcoid
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3
Q

What are the common iatrogenic causes of a pneumothorax?

A
Pleural aspiration
Bronchial biopsy
Liver biopsy
\+ve pressure ventilation
Subclavian CVP line
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4
Q

How does a pneumothorax present?

A
Asymptomatic
Acute progressive dyspnoea
Pleuritic chest pain
Laboured breathing
Tracheal deviation
↓Ipsilateral: Expansion, tactile remits, breath sounds, air entry
↑Ipsilateral: Resonance 
Pleural rub
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5
Q

How is a pneumothorax investigated?

A

Determine 1o or 2o
CXR
Bloods: FBC, U&E, Clotting, ABG
ECG: Tachy

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6
Q

Was is classed as a small and a large pneumothorax?

A
Small = <2cm
Large = >2cm
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7
Q

How does a tension pneumothorax occur?

A
One way valve
Continually expanding pneumothorax into pleural space
No escape when expiring
Mediastinal shift to C/L side
Compresses great veins
Haemodynamic compromise
Cardio-respiratory arrest
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8
Q

How is a tension pneumothorax treated?

A

O2 15L/min NRB
Needle decompression: 2nd ICS midclav OR triangle of safety
Chest drain in triangle of safety

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9
Q

What are the borders of the triangle of safety?

A

Ant: Lat border of pec major
Post: Lat border of Lat Dorsi
Inf: 5th rib

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10
Q

How is a primary pneumothorax managed?

A

> 2cm/SOB: Aspirate 16-18G cannula- aspirate <2.5L. If successful consider discharge + review in 2-4w
NOT successful: Chest drain + admit
<2cm/No SOB: Consider discharge & review in 2-4w

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11
Q

How is a secondary pneumothorax managed?

A

> 2cm/SOB: Chest drain & admit
1-2cm: Aspirate 16-18G cannula- aspirate <2.5L. If successful (<1cm) = ADMIT (observe for 24hrs) If NOT successful Chest drain & admit
<1cm: ADMIT (observe for 24hrs)

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12
Q

What is the mechanism behind a PE?

A

Venous thrombi (DVT) dislodges (emboli)
Reaches pulmonary circulation
Unable to fit through & blocks blood flow to lungs

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13
Q

What are the risk factors for a DVT?

A

Pro-Coag states: COCP, malignancy, nephrotic syndrome
Venous stasis: Immobile, Rx surgery, Pelvic mass, Obesity, Pregnancy/childbirth, Long travel
Miscellaneous: Prev or Fhx of PE/DVT,

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14
Q

Apart from a DVT what other things can cause a PE?

A
RV thrombus: Post-MI
Septic emboli: Tricuspid endocarditis
Fat emboli: Long bone #
Air emboli: Venous lines, diving
Amniotic fluid emboli: Pregnancy
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15
Q

How does a PE present?

A
Pleuritic chest pain + Dyspnoea = SUDDEN onset
Haemoptysis
Tachycardia
Tachypnoea
↓O2 sats
↑JVP 
DVT
LARGE: Cyanosis, HypoT
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16
Q

How is a PE investigated?

A
CTPA = DIAGNOSTIC +ve = Heparinise
VQ scan
ABG: ↓PaO2, ↓PaCO2 = RESP ALKALOSIS 
ECG
CXR: Rarely signs
Bloods: APTT, Troponin, D-dimer
WELLS Score: >4 = likely
Pulm angio = last line
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17
Q

What signs may be seen on ECG to suggest PE?

A
Sinus Tachy
R axis deviation
RBBB
T wave inversion
S1 Q3 T3
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18
Q

What does the Wells Score tell you in ?PE ?

A

Whether a PE is likely or not:
>4 = Likely → CTPA
<4 = Unlikely → D-Dimer= rule out

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19
Q

What does a D-Dimer tell you in ?PE ?

A

+ve = may be PE → CTPA/VQ scan

-ve + Wells Score <4 = PE unlikely

20
Q

Which patients would get a VQ scan over a CTPA?

A

Allergic to contrast

CKD

21
Q

How is a haemodynamically stable pt with a PE treated?

A

O2: 15L/min NRB
LMWH: Tinz 175u/kg OR Fondaparinux OR UFH for 5d
Cardiac monitoring
NSAIDS

22
Q

What should all people with an ‘unprovoked’ PE get?

A

Cancer screen!

CT

23
Q

How is a haemodynamically unstable pt with a PE treated?

A

500ml NaCl if hypoT

1) Thrombolysis: Alteplase OR Streptokinase
2) Embolectomy if thrombolysis CI
3) IVC filter

24
Q

How is a patient with a PE anticoagulated?

A
Confirmed PE = Warfarin OR NOAC
Overlap LMWH w/Warfarin or NOAC until INR 2-4
Stop LMWH
Provoked = Warfarin for 3m
Unprovoked = Warfarin for 6m
Continuing RFs (malignancy) = Lifelong
25
How does a DVT usually present?
``` Leg discomfort Erythema Swelling Dilated superficial veins Homan's sign: Calf pain of dorsiflexion ```
26
How is a DVT investigated?
Wells Score for DVT: >2 = DVT likely → USS WITHIN 4HRS <2 = DVT unlikely → D-dimer USS +ve → refer to DVT team + Anti-coag + Heparinise USS -ve → D-dimer → +ve = heparinise + redo USS in 1w
27
How is a DVT treated?
1) LWMH: Tinz sc OR Fondaparinux OR UFH for 5d | 2) Warfarin: ALL w/confirmed DVT within 24hrs OR NOAC
28
Who gets thrombolysis in DVT?
``` I/L w/Sx ilio-femoral DVT with: -Sx >14d -Good functional status -Low bleed risk -Survival rate >1yr USE: Catheter directed streptokinase ```
29
How does a foreign body in the airway cause problems?
Inhalation of foreign body → acute airway obstruction = ↓GCS, Asphyxia → arrest
30
Who is most likely to have a foreign body obstruction?
Toddlers- small toys, peanuts/food Elderly Alcoholics Recent sedation
31
How does a mild obstruction present?
Still able to breathe & talk Effective, loud cough/cry Fully responsive
32
How does a severe obstruction present?
``` Unable to breathe/talk Ineffective cough Stridor Wheezing Cyanosis ↓GCS or unconscious = T2RF ```
33
How is choking in a child managed in those with an ineffective cough?
CONSCIOUS: 5back blows → 5 abdo thrusts → assess + repeat UNCONSCIOUS: Open airway → 5 rescue breaths → CPR 15:2
34
How does carbon monoxide cause tissue hypoxaemia?
CO has higher affinity for Hb binding than O2 COHb (carboxy-haemoglobin) shifts O2 saturation curve LEFT and DOWN = Tissue hypoxaemia
35
What are the signs of CO poisoning?
EARLY: N&V, headache (90%), poor conc/memory LATE: Dizzy + Vertigo (50%), Cherry red skin, irritable confusion, disorientated, seizure/coma
36
In CO poisoning typically when does encephalopathy occur?
2-6w after
37
What are signs of inhalation injury that could lead to CO poisoning?
``` EARLY INTUBATION Burns to face Soot in nostrils/mouth Stridor/hoarseness/drooling ↓GCS ```
38
How is CO poisoning investigated?
``` Spectrometer = DIAGNOSTIC <10% = common in smokers 10-30% = Symptomatic >30% = Severe ABG: BE AWARE machine reads HbCO as HbO so sats may look normal when not ECG: Myocardial ischaemia (↓ST, VF, VT) ```
39
How is CO poisoning treated?
100% O2 | Hyperbaric O2
40
In CO poisoning what are indications of hyperbaric O2 therapy?
LOC Neuro signs Pregnancy MI/arrhythmia
41
What is the mechanism of ARDS?
Lung damage leads to release of inflammatory mediators ↑permeability of capillaries or loss of oncotic pressure Leads to ↑Alveolar fluid accumulation
42
What are the causes of ARDS?
Pulmonary: Pneumonia, gastric aspiration, inhalation, injury, vasculitis, contusion Other: Post-op, shock, sepsis, DIC, haemorrhage, acute LF, burns, pancreatitis
43
How does ARDS present?
``` ACUTE ONSET- within 1w of insult Cyanosis Hypoxia Tachycardia & tachypnoea Peripheral vasoD B/L fine crackles ```
44
How is ARDS investigated?
CXR ABG Bloods: FBC, LFT, Amylase
45
How is ARDS managed?
Early intubation = burns or inhalation injury ICU!! Oxygenation & ventilation (CPAP) Vasopressors may be needed
46
What is the diagnostic criteria for ARDS?
1. Acute onset within 1w 2. CXR w/ B/L pulmonary infiltrates 3. PCWP <19 4. Refractory hypoxaemia w/ PaO2:FiO2 <200