Emergencies Flashcards

(38 cards)

1
Q

Which joint is usually implicated in septic arthritis?

A

Knee 75%

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2
Q

What are the causes of septic arthritis?

A
Staph Aureus: Single site, MOST COMMON
Strep: Multiple sites
HiB: Multiple sites
Gonorrhoea: Sexually active
Anaerobic organism: Penetrating trauma
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3
Q

What are the RFs for septic arthritis?

A
Steroids
RA
IVDU
Prosthesis
DM
ImmunoS
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4
Q

What can septic arthritis result from?

A

Adjacent osteomyelitis
Haematogenous spread
Puncture/direct trauma

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5
Q

How does septic arthritis present?

A
Single joint
Swollen
Hot 
Erythematous
Painful & tender 
↓RoM- Stiff
Held in slight flexion for comfort
Systemic: Fever, rigors, hypoT, Tachy, vomiting
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6
Q

How is a septic joint investigated?

A

Aspiration: MC&S, Gram stain, culture
Bloods: Cultures, FBC (↑WCC), ↑CRP
Xray: Widened joint space, ST swelling, effusion, erosion (LATE)
USS deep joint
Bone scan: If multiple sites

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7
Q

How is septic arthritis treated?

A

START IV Abx as soon as aspiration sent

1) Abx: Fluclox/Vancomycin, Cefotaxime (Gonococcal)
2) Splinting
2) Surgical debridement
4) Physio

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8
Q

What are the complications of septic arthritis?

A

Bone destruction <24hours

Chondrolysis

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9
Q

What is temporal arteritis?

A

Immune mediated granulomatous vasculitis of Medium & Large vessels

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10
Q

What are 50% of temporal arteritis’s associated with?

A

Polymyalgia rheumatica

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11
Q

Who is typically affected by temporal arteritis?

A

Female >55yo

if Female <55yo = Takayasu’s

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12
Q

What are the Sx of temporal arteritis?

A
Rapid onset <1m
Diffuse constant headache
Unilateral- 85%
Superficial pain over temporal region
Scalp tenderness
Jaw claudication
Distended, throbbing temporal artery
Acute transient visual loss- 10%
Other: N&amp;V, night sweats, ↓weight
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13
Q

How is temporal arteritis investigated?

A

Bloods:↑↑ESR > 40, ↑↑CRP, , ↓Hb, ↑WCC, ↑ALP

Temporal artery biopsy: Within 7d of starting steroids, if skip lesions = DEFINITIVE DIAGNOSIS

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14
Q

How is temporal arteritis treated?

A
Steroids: IMMEDIATE, Prednisolone 40mg for 2yrs
URGENT REFERAL to opthalmology- Same day
Consider:
PPI: Gastroprotection
Bisphosphonates
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15
Q

What criteria can be used to diagnose temporal arteritis?

A

GCA Classification 3/5:

  • > 50yo
  • New headache: New onset, new type, localised pain
  • Temporal artery abnormality: Tenderness on palpation, ↓ pulsation
  • ↑ESR >50
  • Abnormal artery biopsy: vasculitis, granulomatous inflammation, multinucleate giant cells
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16
Q

What are the complication of temporal arteritis?

A

Stroke/TIA

Visual loss- Temporal artery involvement

17
Q

How does spinal cord compression occur?

A

Direct spinal cord pressure or induction of vertebral collapse
Leads to SC or caudal equine compression
Neurological instability

18
Q

Where does SCC typically occur?

A

66% thoracic

33% lumbar

19
Q

What are the causes of SCC?

A
  • Malignancy: Extradural mets (Prostate, breast, lung, myeloma, lymphoma), extension of tumour from vertebral body
  • Trauma: crush #, transection or hemisection from penetration, prolapsed disc
  • Inflammatory: RA (high cervical spine compression)
20
Q

Where do prolapsed discs commonly happen?

21
Q

What are the Sx of SCC?

A

90% Back pain: Localised, nocturnal,
Motor: Weakness/pain, subtle onset 3m before Neuro Sx
Sensory: Paraesthesia, numbness, saddle anaesthesia
Bowel/bladder Sx

22
Q

How is SCC investigated?

A

?SCC = URGENT WHOLE SPINE MRI <24hours
Neuro: CNS & PNS ALL 4 limbs
Palpate spine for tenderness
Anal tone

23
Q

What happens to reflexes in SCC?

A

↑BELOW compression
ABSENT at level
NORMAL above level

24
Q

How is SCC treated?

A

1) Dexamethasone 16mg + PPI
2) Analgesia
3) Surgery/RT
Stabilise & immobilise spine
LMWH if admission
Catheterise if retention

25
What is the prognosis of SCC?
If motor function lost >48hours recovery is unlikely
26
What are the complications of SCC?
Paraplegia = Thoracic Quadraplegia = C3/4/5 Ileus
27
What are the Sx of caudal equine?
Saddle anaesthesia Loss of anal tone Bladder incontinence Localised pain
28
What are the Sx of Brown-Sequard syndrome?
CONTRALATERAL: Loss of pain & temperature IPSILATERAL: Paralysis, loss of proprioception & vibration
29
What is compartment syndrome?
Swelling of tissue in osseofascial compartment Leads to ↑compartment pressure Occludes vascular supply = hypoxia Final- acute ischaemia, oedema & necrosis
30
Where does compartment syndrome commonly happen?
Lower limb: Tibia most common (anterior & deep compartments) Upper limb (volar compartment) Gluteal region Abdomen
31
What are the causes of compartment syndrome?
``` Trauma: #, crushing Burns Infection Vascular: Haemorrhage, reperfusion Muscle hypertrophy: Athletes Orthopaedics ```
32
How does compartment syndrome present?
``` <48hours since injury ↑pain DISPROPORIONATE to injury Worse on passive stretching Muscle tenderness & swelling Sensory deficit Peripheral pulses present ```
33
What are the late signs of compartment syndrome?
Tissue ischaemia: Pallor, pulselessness, paralysis, cold, loss of capillary return Paralysis of muscle group
34
How is compartment syndrome investigated?
Intra-compartmental pressure monitoring: Wick catheter & needle manometry >40mmHg w/Sx = DIAGNOSTIC Difference of >30mmHg between dBP & compartment pressure = ↑risk Bloods: ↑CK, U&E Urine myoglobin
35
How is compartment syndrome treated?
Release any casts/dressings Elevate limb URGENT SURGICAL DECOMPRESSION <1hour- Fasciotomy ALL pt: Re-exploration at 48hours Amputation: >8hours WITH absence of muscle function
36
What are the complications of compartment syndrome?
Tissue necrosis: 6-12hours Mucsle necrosis: Fibrosis & ischaemic contracture (Volkmann's) Renal failure: Rhabdomyolysis from necrosis = ↑CK
37
What are the RFs for chronic compartment syndrome?
Athletes <40yo Sport: Football, cycling, tennis, gymnasts
38
How does chronic compartment syndrome present?
``` Severe pain & tenderness Worse on exercising & passive stretching Resolves on rest Muscle weakness Sensory change: numb, tingling Abnormal gait ```