Emergency management of the pregnant woman Flashcards

1
Q

What anatomical and physiological changes occur in pregnancy?

A

CV system:
Blood volume increases 40-50%
Increasd HR by 10-15bpm
SV increases
Increased CO
Decreased BP (progesterone relaxes smooth muscle around blood vessels, placenta absorbs much of the circulating volume)
Supine Postural Hypotensive Syndrome = Enlarged uterus compresses the inferior vena cava reducing volume and preload

Respiratory system:
O2 consumption increases - therefore progesterone increases minute ventilation (Vt + RR)
Decreased secreton of bicarbonate due to decreased PaCO2 from increased MV - therefore ph can be slightly alkalitic
Diaphragm lifts but chest wallcircumference increases too to assist wth breathing.

Renal:
Vasodilation increases blood flow to kidneys, therefore increased GFR
Frequency due to increased GFR and pressure on bladder from uterus.
Pressure from uterus causes occlusion of ureters, causing build up of urine towards kidneys (increasing susceptibility to pylonephritis)

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2
Q

Discuss the medical history taken of the pregnant woman in the Emergency Department

A

History:

  • Expected DOB
  • Current pregnancy issues/concerns
  • Previous pregnancies and birth mode
    (Gravida - no of pregnancies, Para - no of babies over 20 weeks)
  • Maternal co existing medical conditions
  • Pregnancy related disorders
  • Incidental pathologies
  • Mental health illness in pregnancy
  • Domestic violence concerns
  • Drug and alcohol issues
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3
Q

Discuss the physical assessment of the pregnant woman in the Emergency Department

A
  • If estemated DOB alligns with uterus size
  • Vital signs - especially BP
  • Pain - where, how long, what sort and severity
  • Bleeding - how much, how long, where from
  • Other vaginal loss - odour, colour, amount, when noted
  • Abdominal palpation if gestation warrents and no contraindications of bleeding
  • Oedema
  • Fetal heart rate and wellbeing - growth, movements +/- CTG (cardiotocograph) - at appopriate gestation
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4
Q

Identify risk factors relevent to the pregnant or port partum woman presenting to ED

A

?

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5
Q

Define the parameters of HTN in a pregnant patient

A

SBP > 140

and/or

DBP > 90

On two occasions at least 4 hours apart

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6
Q

Define pre-eclampsia

A

Diagnosed after 20 weeks

A multi-system disorder characterised by HTN and associated involvement of one or more organ systems
(eg, heamatological, renal, hepatological, neurological, foetal/placental)

*affects 3-8% of pregnancies, can also develop or continue into the post natal period

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7
Q

Signs and symptoms of pre-eclampsia

A
  • Rapidly increasing facial oedema
  • Proteinuria
  • Vitual disturbance
  • Headache
  • RUQ pain
  • Hyper-reflexia/Clonus - involuntary muscle spasms

Late signs

  • Reduced fetal movements
  • LFT’s - skewed AST
  • Seizure/Eclampsia
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8
Q

Risk factors for pre-eclampsia

A
  • Previous personal or family history
  • Co existing medical conditions
    (diabetes, essential HTN, antiphospholipid syndrome, renal disease)
  • Multiple pregnancy
  • Nulliparity (hasn’t given birth before)
  • Obesity

*the risk of preeclampsia is increased when more than 1 risk factor is present

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9
Q

Maternal assessment/investigations for pre-eclampsia

A

Thourough history/general exam

  • Vitals: manual BP, ensure correct cuff size
  • Neuro exam: ?hyper reflexia
  • Abdo palpation (fetal lia, presentation, size)
  • UA
    +/- MSU and urine protein creatinine ratio (PCR) if FWT > 1+ proteinuria
  • Pre-eclampsia biochem screen: FBE, UEC, LFT, UA
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10
Q

Fetal assessment for pre-eclampsia

A
  • Fetal movement
  • CTG (Cardiotocography: to assess fetal HR)
  • U/S for AFI (Amniotic Fluid Index)
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11
Q

Management of pre-eclampsia

A
  • IVC, path
  • Control HTN
  • Seizure prophylaxis
  • *Mg sulphate** (diaz not appropriate)
    • slows neuromuscular conduction and decreases CNS irritability
  • fetal neuroprotective effect if anticipating pre-term birth*
  • Fetal maturation
    • maternal corticosteroid administration for 24 - 34+6 weeks gestation for those at risk of preterm birth within 7 days*
    • Strict FBC*
    • Continuous fetal monitoring (CTG)*
    • Birth plan*
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12
Q

Define eclampsia

A

The development of seizures usually with pre-existing pre-eclampsia

  • Life threatening
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13
Q

Management of eclampsia

A
  • Control seizures with Magnesium sulphate (not diazepam)
  • Control HTN with labetolol, nifedipine or hydrazazine
  • Stabilising woman is primary goal over fetus
  • Evaluate for stabalisation and immediate delivery
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14
Q

What is HELLP syndrome?

A

A subset of women with severe pre-eclampsia characterised bu

Haemolysis

Elevated Liver enzymes

Low Platelets

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15
Q

What are the 4 main hypertensive disorders in pregnancy

A
  • *Chronic Hypertension**
  • hypertension (>140/80mmHg) that predates the pregnancy or is diagnosed prior to 20 weeks gestation
  • *Gestational Hypertension**
  • New onset of hypertension after 20 weeks gestation.
  • No assoiated signs of pre-eclampsia
  • Should resolve within 6 months of birth
  • *Pre-eclampsia/Eclampsia**
  • >20 weeks gestation, proteinuria and evidence of end organ compromise.
  • End organ compromise may manifest as CNS symptoms, hepatic dysfunction, renal insufficiency, pulmonary oedema.
  • Eclampsia = new onset of seizures in the setting of pre-eclampsia
  • *Pre-eclampsia superimposed in chronic hypertension**
  • Pre-eclampsia in women with chronic hypertension.
  • Higher risk of adverse outcomes
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16
Q

What are the 5 main causes of bleeding in pregnancy?

A

Early pregnancy

  • *Ectopic pregnancy**
  • *-** implantation occurs outside the uterus
  • *Miscarriage**
  • pregnancy loss <20weeks
  • threatened (bleeding, cervix closed), inevitable (cervix open) or complete (all products expelled)

Post first trimester

  • *Placenta previa**
  • placenta has implanted at lower uterine segment, over the cervix or cose by
  • Presents as painless bright red blood
  • *Placental abruption**
  • placenta prematurely becomes detached from the wall of the uterus (partially or totally)
  • *Vasa previa
  • ** fetal umbillical cord blood vessels run throught eh amniotic membrane near the cervix
17
Q

Causes of bleeding in early pregnancy

A
  • Implantation bleed
  • Cervical lesions
  • Miscarriage (spont abortion)
  • Ectopic pregnancy
  • Incompetent cervix
  • (does not remain closed during the pregnancy)*
  • Hydatiform mole
18
Q

Management of miscarriage

A
  • *Expectant Mx**
  • waiting and seeing
  • *Medication**
  • misoprostil helps products to pass
  • used in incomplete miscarriage
  • not indicated with heavy bleeding/infection/>9 weeks
  • *Surgical**
  • Suction, dialation and curette (D&C)
  • indicated for heavy bleeding/pain
19
Q

S&S of ectopic pregnancy

A

S&S

  • mild lower abdo pain
  • ocasionally sharp, stabbing, increased intensity over a few days especially with bowels*
  • -* Nausea
  • +/- bleeding or slight brown discharge
  • Pathology: decreased progesterone and bHCG
  • U/S: uterus empty

Ruptured

  • Sudden, severe abdo pain
  • profound shock - collapse
  • referred shoulder tip pain
  • internal bleeding
20
Q

Management of ectopic pregnancy

A

Dependent on presentation

Medical

  • If dx early or embryo diameter <3.5cm and no severe bleeding
  • IM Methotrexate stops rapid growth
  • needs serial bHCG to show that it is reducing in size
  • avoid pregnancy for 3 moths

Surgical
- laproscopic, may involve removing part or all of the fallopian tube to control bleeding

21
Q

Define an Antipartum Heamorrhage (APH)

A

Bleeding from the birth canel of 10-15ml >20wks gestation

Mostly unknown cause but can be due to placenta previa, placantal abruption, vasa previa

22
Q

Presentation and management of placenta previa

A

Presentation

  • painless recurrent bleed of various amounts
  • premature labout
  • shock

Management
Depends on grade

  • Grade 1&2 (cervix not covered)*
  • repeat U/S at 32 weeks - placenta may have moved
  • Grade 3&4 (cervix covered)*
  • cannot have vaginal birth, must be cesarian
  • will likely require hospital admit when they start bleeding
  • close watch and wait to maximise fetal growth and minimise blood loss for mother
23
Q

Placental abruption causes

A

Cause

  • pre-eclampsia
  • HTN
  • Trauma
  • Unknown
24
Q

Placental abruption types

A

Concealed
Bleeding retained behind the placenta
- forced back into uterine muscle fibres
- extravasation causes damage to uterus
- can have hypovolaemic shock but no bleeding

  • *Revealed**
  • blood escapes from placental site
  • seperates membrane from uterine wall
  • draines out the vagina.
25
Q

Presentation of placental abruption

A
  • +/- vaginal bleeding
  • tachycardia, hypotension: hypovolaemic shock
  • decreased/no fetal moveemnts, loss of fetal HR
  • anxious
  • hard and rigid abdomen, extreme pain
26
Q

Complications from antipartum heamorrhage

A
  • DIC (Disseminated Intravasculat Coagulation)
  • Post Partum Haemorrhage: may result in hysterectomy
  • Renal failure
  • Pituitary necrosis
  • Distressed baby/death in utero
27
Q

Presentation of vasa previa

A
  • Painless vaginal bleeding (fresh blood)
  • Limited bleeding, usually when membrane ruptures
  • Fetal distress (post membrane rupture)
  • Usually late 3rd trimester associated with labour
  • Can be detected on U/S
28
Q

Management of vaso previa

A
  • Monitor FHR
  • 1st stage labour - emergency csection
  • 2nd stage labout - expidite vaginal birth, otherwise cesarian
  • High fetal mortality as blood loss is from fetal blood supply
29
Q

Management of PPH

A
  • Immediate and extensive resus if required
  • Vitals, birth history
  • Assess loss: volume, colour, odour, clots
  • Swab for MCS
  • Speculum exam
  • Possible D&C
  • ABx
30
Q

Management of unexpected birth in the ED

A
  • Woman adopts most comfortable birthing position
  • Wipe perineal area if contaminated with faeces
  • Head will advance with contractions
  • Apply gentle pressure to advancing head, following birth of head, next contraction should be shoulders
  • Place hands on either side of head, exert gentle traction, birth anterior shoulder under symphysis pubis, gentle upwards pressure to birth anterir shoulder
  • grasp under armpits to bring baby up to mum
  • vigerously dry baby, skin to skin contact
  • allow spontaneous birth of placenta and membrane, unless oxytocic available (syntocinon, syntometrine, ergometrine)
  • doccument, vitals
31
Q

Definition and Management of Shoulder Dystocia

A

Impactation of the anterior shoulder against the symphysis pubis following delivery of the head

Suspect if:

  • Difficulty with the birth of the face and chin
  • Chin retracts into the perineum (turtle sign)
  • Anterior shoulder does not birth with normal downward traction

Management

  • Note the time of birth of the head
  • Ask woman to stop pushing
  • Knees to nipples, apply suprapubic pressure behing baby’s anterior shoulder, attempt birth
  • If unsuccesful, try on all fours
  • Expecta compromised baby, prep for neonatal resus
32
Q

Recognition and Management of Breech Birth

A
  • Passing fresh meconium from vagina
  • Pushing with breech presentation
  • Foot hanging through the vagina

Management

  • Escalate to maternity
  • Gather equiptment for neonatal resus
  • Vaginal birth only if frank or complete breech (not footling) and no absolute contraindication to vaginal birth (eg. placenta praevia)
33
Q

Common Antenatal and Postnatal presentations to Emergency

A

Antenatal

  • PV bleed
  • Placenta Previa
  • Placental Abruption
  • Decreased Fetal movements
  • Pre-term rupture of membranes

Postnatal

  • Wound infection
    -Secondary PPH
  • Mastitis
  • Postnatal depression
    Sepsis