Emergency Medicine Flashcards

(89 cards)

1
Q

Reversible causes of cardiac arrest

A

Hypoxia
Hypovolaemia
Hyperkalaemia/hypokalaemia, hypoglycaemia, hypocalcaemia
Hypothermia
Thrombosis
Tension pneumothorax
Tamponade
Toxins

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2
Q

Treatment of hypoxia in cardiac arrest

A

patient adequately ventilated with maximal inspired O2 (15L/min)

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3
Q

Treatment of hypovolaemia in cardiac arrest

A

o look for haemorrhage and restore volume with fluids and blood products
o Activate major haemorrhage protocol where required

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4
Q

Treatment of thrombosis in cardiac arrest

A

o Urgent angiography ± PCI
o Thrombolyse if PCI not available or PE

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5
Q

Treatment of tamponade in cardiac arrest

A

pericardiocentesis

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6
Q

Initial assessment in cardiac arrest

A
  • Assess for response and signs of life
  • Call for help and make bed flat
  • Open airway using head-tilt chin-lift
  • ABCDE
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7
Q

Chest compressions in cardiac arrest

A
  • Ratio chest compressions to ventilation 30:2
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8
Q

Ventilation in cardiac arrest

A
  • Bag-valve mask
  • Airway adjuncts used where required
  • Continuous with LMA, I-gel or endotracheal intubation
  • 10 breaths per minute
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9
Q

Defibrillation in cardiac arrest

A
  • Shockable rhythms: Ventricular fibrillation, pulseless ventricular tachycardia
  • Single shock followed by 2 mins of CPR
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10
Q

Adrenaline in cardiac arrest

A
  • Adrenaline 1mg ASAP for non-shockable rhythms (PEA, asystole)
  • Shockable rhythms give adrenaline once chest compressions have restarted after 3rd shock
  • Repeat adrenaline every 3-5 mins
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11
Q

Amiodarone in cardiac arrest

A
  • 300mg given to patients after 3 shocks
  • Further 150mg given after 5 shocks
  • Lidocaine used as alternative if amiodarone
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12
Q

Peri-arrest bradycardia adverse signs

A
  • Shock  hypotension, pallor, sweating, cold, clammy extremities, confusion, impaired consciousness
  • Syncope
  • MI
  • HF
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13
Q

Management of peri-arrest bradycardia

A
  • 1st line = 500mcg IV atropine
  • Continue atropine up to max 3mg
  • Transcutaneous pacing
  • Isopredaline/adrenaline infusion titrated to response
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14
Q

Risk factors for asystole

A
  • Complete heart block with broad complex QRS
  • Recent asystole
  • Mobitz type II AV block
  • Ventricular pause >3s
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15
Q

Management of peri-arrest tachycardia

A
  • If adverse signs present then synchronised DC shocks (up to 3)
  • Broad complex (regular)
    o Assume VT (unless previously confirmed SVT with BBB)
    o Loading dose of amiodarone followed by 24 hr infusion
    o Lidocaine  use with cuation in severe LV impairment
    o Procainamide
    o Electrophysiological study
    o Implantable cardioverter-defibrillator
  • Broad complex (irregular)
    o Seek expert help
    o AF with BBB
    o AF with ventricular pre-excitation
    o Torsade de pointes
  • Narrow complex (regular)
    o Vagal manoeuvres
    o IV adenosine
    o Atrial flutter  BB
  • Narrow complex (irregular)
    o AF
    o Onset <48 hr consider electrical or chemical cardioversion
    o BB
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16
Q

Causes of hypothermia

A
  • Primary hypothermia  environmental exposure with no underlying medical condition
  • Secondary hypothermia
    o Decreased heat production  hypothyroidism, hypoadrenalism, malnutrition
    o Increased heat loss  vasodilation, burns, erythroderma
    o Impaired thermoregulation  CNS trauma, stroke, sepsis, pancreatitis
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17
Q

Presentation of hypothermia

A
  • Mild (32-35 degrees)
    o Shivering
    o Lethargy
    o Confusion
    o Tachycardia
    o Vasoconstriction
    o Loss of motor coordination
  • Moderate (28-32)
    o Dysrhythmias
    o Bradycardia
    o Hypotension
    o J waves (frosty Jacks)
    o Reduced reflexes
    o Reduced GCS
    o Dilated pupils
  • Severe (<28)
    o Agitation/delirium
    o Arrhythmias
    o Apnoea
    o Non-reactive pupils
    o Coagulopathy
    o Oliguria
    o Pulmonary oedema
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18
Q

Investigations of hypothermia

A
  • Rectal/ear temp
  • ECG  J waves or other arrhythmias
  • Bloods  U&Es, plasma glucose, amylase, TFTs, FBC, coagulation studies, blood cultures
  • ABG
  • CXR
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19
Q

Management of hypothermia

A
  • ABCDE
  • Aim for temp rise of 0.5 per hour using warm IV fluids, warm O2 and blankets
  • IV drugs avoided use to risk of drastic response
  • IF cardiac arrest, warming as quickly as possible
    o Warmed fluids infused into all orifices with access (IV, catheter, NG)
  • Patients not dead until warm and dead
  • If <30 and in arrest  no more than 3 shocks administered until rewarmed
  • Cardiac monitoring
  • Catheterise
  • Abx for prevention of pneumonia in patients <65 with temp <32
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20
Q

Management of haemorrhage

A
  • Major haemorrhage protocol
  • Call for help
  • Try to limit bleeding
  • Call blood bank
  • Bloods and 2x Group and Save
  • Two large bore cannulas
  • IV fluid bolus (no more than 1L)
  • 2 units of O- blood
  • Crossmatched blood
  • Additional blood products – platelets, FFP, cryoprecipitate
  • Definitive management
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21
Q

Causes of burns

A
  • Thermal  Solids, liquids, gases, smoke, fire
  • Chemical  Acid, alkali, toxins
  • Electrical
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22
Q

Severity classification of burns

A
  • Superficial epidermal (1st degree)
    o Red and painful, dry, no blisters
    o Blanching on pressure
    o Will heal without scarring
    o Don’t calculate total body surface area
    o E.g. sunburn
  • Partial thickness – superficial dermal (2nd degree)
    o Pale pink, painful, blistered, slow cap refill
    o Blanching
  • Partial thickness – deep dermal (2nd degree)
    o Typically white but may have patches of non-blanching erythema
    o Reduced sensation
    o Painful to deep pressure
  • Full thickness (3rd degree)
    o White/brown/ black in colour, leathery appearance
    o No blisters, non-blanching
    o Loss of sensation  no pain
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23
Q

Assessing extent of burn

A
  • Total Body Surface Area
    o Wallace’s rule of 9s
    o Lund and Browder chart (Paeds)
    o Palm rule  palm is 1% TBSA
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24
Q

Immediate management of burns

A
  • Stop the burn
    o Remove burning/source
    o Electrical burns  switch of power supply, cardiac monitoring
    o Chemical burns  Brush any powder off then irrigate with water (do not try to neutralise)
  • ABCDE
    o Airway issues get worse quickly with oedema
    o Oxygen and measure CO
    o 2x wide bore cannulae  fluid resus
    o Careful exposure
    o Monitor urine output
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25
General burns management
o Within 20 mins, irrigate burn with cool water for 10-30 mins o Cover burn using cling film, layered, rather than wrapped around limb o Symptomatic relief – analgesia, emollients o Tetanus o Cleanse wound o Leave blister intact o Non-adherent dressing o Avoid topical creams o Review in 24 hrs
26
Referral to secondary care for burns
- All deep dermal and full-thickness burns - Superficial dermal burns of >3% TBSA in adults or >2% in children - All circumferential burns - Unhealed burns >2/52 - Need for HDU/ITU - Pregnancy - Any inhalation injury - Suspicion of non-accidental injury - Discussion o Superficial dermal burns involving face, hands, feet, perineum, genitalia, or any flexure o Any electrical or chemical burn injury o Cold injury o Febrile/unwell children
27
Management of more severe burns
- Assess airway o Smoke inhalation can result in airway oedema o Look for singed nose hairs, facial burns, oropharyngeal burns, voice hoarseness, stridor o History of burns in enclosed space o Consider early intubation as oedema can make intubation more difficult later - Breathing o ABG  Carbon monoxide levels, lactate, oxygenation, cyanide poisoning o High flow O2 - IV fluids o Children with burns >10% total body surface area o Adults with burns >15% total body surface area o Parkland formula: total body surface area of burn % x weight (kg) x4 (adults) or x3 (children) o Half of fluid administered in first 8 hrs - Urinary catheter  monitor urine output - Transfer to burns unit o Complex burns o Burns involving hand perineum and face o Burns >10% in adults and 5% in children o Laser doppler imaging - Deroof blisters - Escharotomy o Divide burnt tissue in circumferential burns affect limb or severe torso burn impeding respiration o Relieve pressure to prevent compartment syndrome - Excision/debridement and skin grafting for complex burns o Remove necrotic tissue and aim to create viable tissue bed for healing o Autologous harvest of healthy skin o Meshed, fixed in place
28
Short term complications of burns
o Inhalation injury/poisoning o SIRS o Shock o DIC o Compartment syndrome o Multi-organ failure o Corneal ulceration
29
Medium term complication of burns
o Burn-associated infection o Paralytic ileus o Curling’s ulcer (acute gastric ulcer develop in response to severe physiological stress)
30
Long term complication of burns
o Contractures o Marjolin ulcer o Heterotopic ossification o PTSD, depression
31
Causes of airway obstruction
- Coma  loss of protective airway reflexes - Blood or vomit - Direct trauma - Haematoma - Oedema (following burns) - Choking
32
Airway maintenance
- Chin lift or jaw thrust manoeuvres - Airway control by holding mask onto face; inserting laryngeal mask airway; or intubation - Look in mouth and pharynx for foreign bodies, blood and vomit - Remove any obstruction with Magill’s forceps or Yankauer sucker
33
Management of choking
- Severe  patient cannot speak or breathe, attempts at coughing are silent - Mild  patient can speak, cough and breathe - Encourage to cough if mild - Cycle of 5 black blows (heel of hand between scapulae) and 5 abdominal thrusts (from behind, placing clenched hand under xiphisternum and pulling upwards and inwards - If lose consciousness, commence ALS protocol and try to retrieve object with foreceps/suction
34
Risk factors for sepsis
- Extremes of age - Immunosuppression  chemo, splenectomy, steroids, immunosuppressant meds, pregnancy - Recent trauma, Invasive procedure, Surgery in past 6 weeks - IVDU - Indwelling lines, drains or catheters
35
NICE criteria for sepsis
- 1 red criteria or 2 yellow criteria + symptoms, fever - Red = objective altered mental state, RR >25 or increased O2 requirement, HR >130, SBP <90 or >40 below normal, urine output <0.5ml/kg/hr or no output for >18h - Yellow = history of altered behaviour, RR >20, HR >100, SBP <100, urine output <1ml/kg/hr or no output for >12h, deterioration in function, rigors, immunosuppression, recent surgery
36
SOFA scoring in sepsis
PaO2, platelet count, bilirubin, MAP, GCS, creatinine, urine output
37
Assessment in sepsis
- A: normally patent but beware reduced conscious level - B: tachypnoeic and low SpO2 - C: hypotensive, establish IV access, take bloods, 500ml IV saline boluses, escalate to ICU - D: GCS decreased consciousness level - Erect CXR - Other Ix: abdo USS, CT, LP, echo, bronchoscopy,laparoscopy, sputum culture, skin/wound swabs, joint aspirate, ECG
38
Management of sepsis
(SEPSIS 6) = give 3, take 3/ BUFALO - Oxygen through non-rebreathe mask - IV fluid boluses to maintain SBP - IV broad spec Abx  co-amoxiclav or taxocin - ABG  lactate o Lactate >4 = high risk of death - Urine output  catheterise or commence hourly urine output monitoring - Blood cultures
39
Complications of sepsis
- Hypovolaemia/shock  SBP <90 or lactate >4 in absence of hypovolaemia - ARDS  increased O2 requirement to maintain sats - AKI  urine output <0.5ml/kg/hr, creatinine >75 - Hyperbilirubinaemia  >35 - DIC  platelets <100, INR >1.5 - Encephalopathy  new confusion/decreased GCS
40
Management of status epilepticus
- ABCDE - Insert cannula - IV lorazepam 4mg (repeat after 10 mins if seizure continues) - If seizure persists = IV phenobarbital or phenytoin
41
Causes of shock
* Hypovolaemic shock = low blood vol * Cardiogenic shock = heart not pumping * Distributive shock o Septic o Anaphylactic o Neurogenic * Anaemic shock = not enough O2 carrying capacity * Cytotoxic shock = cells poisoned
42
Presentation of shock
* Pulse is weak and rapid * Pulse pressure reduced * Reduced urine output * Confusion, weakness, collapse, coma * Skin is pale, cold, sweaty and vasoconstricted CRT >3s
43
Management of shock
ABCDE Give oxygen IV access and give IV fluids
44
Tension pneumothorax
- Laceration to lung parenchyma with flap - Pressure develops in thorax - Cause  mechanical ventilation in patient with pleural injury - Sx overlap with cardiac tamponade, hyper-resonant percussion note
45
Flail chest
- Chest wall disconnects from thoracic cage - Multiple rib fractures (at least 2 per rib in at least 2 ribs) - Associated with pulmonary contusion - Abnormal chest motion - Avoid over hydration and fluid overload
46
Haemothorax
- Commonly due to laceration of lung, intercostal vessel or internal mammary artery - Haemothoraces large enough to appear on CXR are treated with large bore chest drain - Surgical exploration is warranted if >1500ml blood drained immediately
47
Pulmonary contusion
- Most common potentially lethal chest injury - ABG and pulse oximetry important - Early intubation within hour if significant hypoxia
48
Blunt cardiac injury
- Usually occurs secondary to chest wall injury - ECG may show features of myocardial infarction - Sequelae: hypotension, arrhythmias, cardiac wall motion abnormalities
49
Diaphragm disruption
- Most due to motor vehicle accidents and blunt trauma causing large radial tears - More common on left side - Insert gastric tube which will pass into thoracic cavity
50
Mediastinal traversing wounds
- Entrance wound in one hemithorax and exit wound/foreign body in opposite hemithorax - Mediastinal haemtoma or pleural cap suggests great vessel injury - Mortality 20%
51
What is anaphylaxis
Life-threatening medical emergency caused by severe type 1 hypersensitivity reaction
52
Presentation of anaphylaxis
- Allergy symptoms o Urticaria o Itching o Angio-oedema with swelling around lips and eyes o Abdominal pain - Anaphylaxis symptoms o Shortness of breath o Wheeze o Swelling of larynx causing stridor o Tachycardia o Lightheadedness o Collapse
53
Management of anaphylaxis
- IM adrenaline (repeat after 5 mins if required) o <6m give 100-150 micrograms 1 in 1000 o 6m-6y give 150 micrograms 1 in 1000 o 6-12y give 300 micrograms 1 in 1000 o Adult and child >12y give 500 micrograms - Refractory anaphylaxis  symptoms persist despite 2 doses of adrenaline o IV fluids for shock o Expert help and consider IV adrenaline
54
Post-event management of anaphylaxis
- Period of assessment and observation for biphasic reactions - Admit to paediatric unit for observation - Measure serum mast cell tryptase within 6 hours - Antihistamines = oral chlorphenamine or certirizine - Education and follow-up of family and child - Train parents in BLS - Specialist referral for diagnosis in allergy clinic, education, follow up and training to use adrenalin auto-injector
55
Presentation of moderate acute asthma attack
Peak flow >50-75% predicted Normal speech No features listed
56
Presentation of severe asthma attack
Peak flow 33-50% Sats <92% Unable to complete sentences in 1 breath Signs of resp distress Accessory muscles use Inability to feed RR >40 in 1-5yrs or >30 in over 5s or >25 in over 12s HR >140 in 1-5 yrs or >125 in over 5s or >110 in over 12s
57
Presentation of life-threatening asthma attack
Peak flow <33% predicted Sats <92% Exhaustion and poor resp effort Hypotension Silent chest Cyanosis Altered consciousness/confusion Cardiac arrhythmia
58
Management of acute asthma attack
- Admit all with life-threatening asthma and severe asthma that persists after initial bronchodilator Tx - Supplementary O2 if required - Abx if bacterial cause - Moderate to severe cases in stepwise approach 1. Salbutamol inhalers via spacer device (10 puffs every 2 hrs) 2. Nebulisers with salbutamol/ipratropium bromide 3. Oral prednisolone 4. IV hydrocortisone 5. IV magnesium sulfate 6. IV salbutamol 7. IV aminophylline - Then step down ladder as control is reached
59
Causes of DKA
- Interruption of insulin therapy - Stress of intercurrent illness (surgery/infection) - Patient reduces/omits insulin because unable to eat (nausea/vomiting) - Undiagnosed diabetes
60
Presentation of DKA
- Gradual drowsiness - Nausea and vomiting - Abdominal pain - Deep breathing - Polyuria and polydipsia - Breath smells of pear drops - Dehydration and hypotension - Acidotic - Coma
61
Investigations of DKA
- Hyperglycaemia = blood glucose > 11mmol/L - Raised plasma ketones = >3mmol/L - ABG/VBG = low pH, K+ low, O2 low, bicarbonate <15mmol/l - FBC, U&E, LFT, clotting, CRP - CXR - ECG - Urine C+S
62
Management of DKA
FIGPICK - Fluid resuscitation (Saline bolus) - IV Insulin continuous infusion rapid acting 0.1 unit/kg/hr - Glucose  once blood glucose <14mmol/l 10% dextrose infusion added - Potassium = monitor and correct - Infection = treat underlying triggers - Chart fluid balance - Ketones = monitor blood ketones or bicarbonate - Anticoagulation
63
Complications of DKA
- AKI - Gastric stasis - Thromboembolism (DVT) - Acute respiratory distress syndrome - Cerebral + pulmonary oedema
64
Causes of hyperosmolar hyperglycaemic state
- Infection = most common precipitating cause (pneumonia) - Consumption of glucose rich fluids - Concurrent mediation = thiazide diuretics or steroids
65
Presentation of hyperosmolar hyperglycaemic state
- Longer history = 1 week - Decreased level of consciousness = due to elevated plasma osmolality - Clinical signs of dehydration - Polyuria and polydipsia - Lethargy - N+V - Focal neurological deficits - Stupor or coma
66
Investigations of HHS
- Marked hyperglycaemia (>30mmol/L) - Urine stick test = heavy glycosuria - Mild or no ketosis = No ketones in blood or urine - Bicarbonate not lowered - Plasma osmolality = extremely high - Total body K+ is low = Due to osmotic diuresis o Serum K+ is raised  absence of insulin  K+ shift out of cells
67
Management of HHS
- Fluid replacement with IV 0.9% saline slowly o 0.5-1L per hour o Aim for +ve fluid balance of 3-6L by 12 hrs o Encourage food and drink ASAP - Patients more sensitive to insulin = lower rate of infusion o Only use if glucose not falling or ketonemia - VTE prophylaxis  Low molecular weight heparin - Monitor K+ and give when urine starts to flow
68
Complications of HHS
- Risk of cerebral and pulmonary oedema o Rapid lowering of blood glucose and osmolality of blood - Severe dehydration (hyperviscosity)  DVT, stroke, MI, arterial insufficiency of lower limbs - Foot ulcers
69
Causes of hypoglycaemia
- Insulinoma - Self-administration of insulin/sulphonylureas - Liver failure - Addisons’s - Alcohol - Nesidioblastosis
70
Presentation of hypoglycaemia
- Autonomic symptoms o Sweating o Shaking o Hunger o Anxiety o Nausea - Neuroglycopenic symptoms o Weakness o Vision changes o Confusion o Dizziness - Severe  convulsion, coma
71
Management of hypoglycaemia
- Oral glucose 10-20g in liquid, gel or tablet form - Quick-acting carbohydrate  GlucoGel or Dextrogel - SC or IM glucagon - IV 100ml 20% glucose
72
Risk factors for paracetamol overdose
rifampicin, phenytoin, carbamazepine, chronic alcohol excess, St John’s Wort, anorexia nervosa, malnourished, HIV, P450 inducers
73
Management of paracetamol overdose
o Activated charcoal within 1 hr o Acetylcysteine if  conc over line after 4 hrs, or  staggered overdose or  Present 8-24 hrs of ingestion >150mg/kg  Present >24 hrs if jaundiced or hepatic tenderness or ALT above upper limit o Liver transplant  Arterial pH <7.3, 24 hrs after ingestion  PT >100s and creat >300 and grade III or IV encephalopathy
74
Features of salicylate overdose
hyperventilation, tinnitus, lethargy, sweating, pyrexia, N+V, hyper/hypoglycaemia, seizures, coma (mixed respiratory alkalosis and metabolic acidosis)
75
Management of salicylate overdose
o Mx: charcoal, IV sodium bicarbonate, o Indications for haemodialysis  Serum conc >700  Metabolic acidosis resistant to Tx  Acute renal failure  Pulmonary oedema  Seizures  Coma
76
Management of opioid overdose
IV naloxone give in smaller increments. May need an infusion. Careful in palliative care setting.
77
Features of TCA overdose
arrhythmias (wide QRS and prolonged QT), seizures, metabolic acidosis, coma
78
Management of TCA overdose
IV bicarbonate
79
Features of lithium overdose
coarse tremor, hyperreflexia, confusion, polyuria, seizure, coma
80
Management of lithium overdose
o Mild – mod  normal saline o Severe  Haemodialysis
81
Management of BB overdose
atropine (if resistant glucagon)
82
Management of iron overdose
o <40mg/kg + asymptomatic  observed at home o >40mg/kg or symptomatic  measure iron levels 2-4 hrs post ingestion and AXR o Whole bowel irrigation for all patient within 4 hrs of ingesting >60mg/kg o Desferrioxamine  Iron level >90  Iron level 60-90 + symptomatic or persistent iron on AXR despite whole bowel irrigation  Shock, coma, metabolic acidosis
83
Over fluids/fasting before surgery
- Clear fluids until 2 hrs before surgery o Helps reduce headaches, nausea and vomiting o E.g. water, fruit juice, coffee/ tea without milk, ice lollies - Fast for 6 hrs before surgery
84
Complications of diabetes and surgery
o Undetected hypoglycaemia o Increased risk of wound and resp infections o Increased risk of post-op AKI o Increased length of hospital stay
85
Insulin and surgery
o Good glycaemic control (HbA1c <69) and minor procedure managed by adjustment of usual insulin regimen o Variable rate IV insulin infusion if long fasting period and poorly controlled diabetes o Reduce once daily insulin by 20% o Half morning dose of biphasic or ultra-long acting insulins
86
Causes of malignant hyperthermia
Anaesthetic agents  halothane, suxamethonium, antipsychotics
87
Management of malignant hyperthermia
Dantrolene
88
Presentation of postoperative ileus
- Abdo distention/bloating - Abdo pain - N+V - Inability to pass flatus - Inability to tolerate oral diet
89
Management of postoperative ileus
- NBM - NG tube if vomiting - IV fluids + correct electrolytes - TPN for severe/prolonged cases