Emergency medicine/surgery Flashcards

(39 cards)

1
Q

What do you hear on auscultation/percussion with effusion

A

Auscultation- absent breath sounds

Percussion- stony dullness

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2
Q

Acute severe asthma vs life threatening asthma

A
Acute severe: 
33-55% of best PEF 
Can't complete sentences 
Resp >25/min 
Pulse>110 beats/min
Life threatening asthma:
PEF <33% of best 
SpO2 <92% 
Silent chest, cyanosis, or feeble respiratory effort
Arrhythmia/hypotension
Exhaustion, altered consciousness
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3
Q

What is an important piece of info about an asthmatic in a&e

A

if they’ve ever been intubated… this means they were at the point of dying before so it is a big risk factor and you don’t want to send them home

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4
Q

Info to ask for asthma attack

A

Previous ITU
Other medical conditions
Allergies
Any infective symptoms

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5
Q

Pathophsiology of asthma

A

Reversible airway disease
Hyper reactivity
Airflow limitation

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6
Q

Clinical presentation of asthma

A

Wheeze?
-Other things which also cause wheeze. Not specific for asthma

Breathless?

Tachycardic?

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7
Q

Treatments for asthma

A
Nebulised salbutamol
IV salbutamol
Nebulised adrenaline
Nebulised magnesium
Oral steroids
IV steroids
 (IV magnesium if they are terrible)
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8
Q

What would you give if he is an acute asthmatic

A

Give them whatever is in the nebuliser IV (if they’re not breathing properly it probably won’t go in)

Call for specialist help

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9
Q

What is the difference between severe vs life threatening asthma

A

Decompensation

Unable to maintain adequate PO2 and PCO2

Drowsy due to rising PCO2, hypotension or exhaustion (note co2 should be low in asthma!)

Need intubation

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10
Q

What should CO2 be in an acute asthmatic

A

LOW (because they blow it off)

If co2 is normal you worry,

if it’s high then this is very bad

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11
Q

What is included in asthma review

A

How pathology creates the clinical signs
How physiology is explained- compensation vs decompensation
Treatment
Started to understand how history, examination and treatment might fit together in one package

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12
Q

What is special about meningitis rash

A

It is a vasculitic rash…. it is nonblanching

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13
Q

What will happen if you press on an uritcarial rash

A

It will go away (i.e. blanching)

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14
Q

Pathophsyiology of anaphylaxis

A

IgE mediated activation of mast cells leading to mediator release and airway airway obstruction

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15
Q

What is the treatment for anaphylaxis

A

Adrenaline
500mcg
0.5ml 1;1000 (IM not IV as it can predispose to arrythmia)

Lie flat and put legs in the air

Piriton

Hydrocortisone

Fluids

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16
Q

How does adrenaline help you in anaphylaxis

A

α1 – vasoconstriction and relaxation of GI tract

α2 – platelet aggregation and reduction in noradrenaline release from nerve terminals

β1 – inotropic and chronotropic cardiac effects and relaxation of GI tract

β2 – bronchodilatation, increase in noradrenaline release from nerve terminals, increase in intracellular cyclic adenosine monophosphate (cAMP) production in mast cells and basophils, reduction in the release of cellular mediators

17
Q

Priorities for MI

A
  1. Call for help
  2. Open his airway and start rescue breaths
  3. Get IV access
  4. Get a 12 lead ECG

You need to check for a pulse first (because an ECG could be normal despite no pulse and thus death)

18
Q

What pulse should you check

A

Carotid pulse (close to the heart)

19
Q

2 algoriths in CPR

A

Shockable or non-shockable rhythm

20
Q

Which rhythms are shockable

A

VF or pulseless VT (check this)

21
Q

What rhythms are non-shockable

A

PEA and asystole

22
Q

Reversible causes of cardiac arrest

A
Hs:
Hypoxia
Hypovolaemia
Hyper/hypokalamemia (&amp; other electrolyte disturbance)
Hypothermia
Ts:
Tension pneumothorax
Cardiac tamponade
Toxins
Thromboembolic
23
Q

Learn to recognise cardiac tamponade on echo!

24
Q

What do you do for a cardiac tamponade

A

An emergency pericardiocentesis (ultrasound guided)

25
What is the correct dose of adrenaline in anaphylaxis
0.5 1 in 1000
26
Primary survey for trauma
``` Catastrophic Haemorrhage? Airway with c spine control Breathing Circulation Disability Exposure ‘C-ABC’ ```
27
When do you get a dilated pupil What should you do
It means the brainstem is being pushed through the foramen magnum which compresses crainial nerves affecting pupils. Usually 1 eye first and then the second Before survery give mannitol
28
How to differentiate between subural and extradural
Subdural looks like banana | Extradural is convex
29
ED management of extradural haemorrhage
``` Optimise oxygenation -A, B, C Keep CO2 normal -A,B Maintain cerebral perfusion -(CPP= MAP-ICP) Make sure nothing more life threatening takes priority -Primary survey Neurosurgical input & theatre ```
30
Primary survey
Systematic approach C- ABC Treat life threatening problems as you find them Trauma team – lead by a senior doctor Simultaneous action But we teach it in order so you remember the priorities
31
What is the strongest predictor of injury
Mechanism of injury is the strongest predictor of injury Applies to all trauma- minor and major Create a mental picture of events in your head
32
What do you want to know about airway
Are they breathing? Is it normal or noisy? - Noisy = obstructed - Obstructed = do something! Do they need a ‘definitive airway’
33
Palpate for surgical emphysema
You have an airway leak going into the surrounding tissue
34
Assessment of circulation
Pulse, BP, capillary refill time | General appearance
35
Source of haemorrhage
'On the floor and 4 more': Abdomen (spleen and liver) Into leg (femur can bleed lots) Chest Pelvis
36
What fluid should you give to hypertensive patients
Transfusion | red cells+platelets+FFP
37
Circulation with haemorrhage control
``` Turn off the tap Fluids (blood) Warfarin Coagulopathy to correct TXA (within 1hr) ``` Damage control surgery
38
Assessment of disability
GCS- Glasgow Coma Score level of consciousness Pupils Blood sugar Limb movements
39
Potential injuries for burns
``` Direct Burns Inhalation Injury Smoke Inhalation Carbon Monoxide Poisoning Cyanide Poisoning Trauma ```