{ "@context": "https://schema.org", "@type": "Organization", "name": "Brainscape", "url": "https://www.brainscape.com/", "logo": "https://www.brainscape.com/pks/images/cms/public-views/shared/Brainscape-logo-c4e172b280b4616f7fda.svg", "sameAs": [ "https://www.facebook.com/Brainscape", "https://x.com/brainscape", "https://www.linkedin.com/company/brainscape", "https://www.instagram.com/brainscape/", "https://www.tiktok.com/@brainscapeu", "https://www.pinterest.com/brainscape/", "https://www.youtube.com/@BrainscapeNY" ], "contactPoint": { "@type": "ContactPoint", "telephone": "(929) 334-4005", "contactType": "customer service", "availableLanguage": ["English"] }, "founder": { "@type": "Person", "name": "Andrew Cohen" }, "description": "Brainscape’s spaced repetition system is proven to DOUBLE learning results! Find, make, and study flashcards online or in our mobile app. Serious learners only.", "address": { "@type": "PostalAddress", "streetAddress": "159 W 25th St, Ste 517", "addressLocality": "New York", "addressRegion": "NY", "postalCode": "10001", "addressCountry": "USA" } }

EMERGENCY (RESPI AND GI) Flashcards

(29 cards)

1
Q
  • the sudden and life-threatening deterioration of the gas exchange function of the lung and indicates failure of the lungs to provide adequate oxygenation or ventilation for the blood.
  • rapid onset respiratory impairment, which is severe enough to cause potential or actual morbidity or mortality if untreated.
A

acute respiratory failure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

decrease in arterial oxygen tension (PaO2) to less than 60mm Hg

A

hypoxemia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

increase in arterial carbon dioxide tension (PaCO2) to greater than 50 mm Hg

A

hypercapnia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

synonymous with perioperative respiratory failure and is related to atelectasis of the lung

A

Type III or “Perioperative” Respiratory Failure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

respiratory failure is related to an inability of (normal or relatively normal lungs) to keep up with increased ventilatory demands associated with systemic hypermetabolism (e.g., secondary to sepsis).

A

Type IV or “High –demand” Respiratory Failure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

most common form of respiratory failure and is defined by PaO2 < 60 mm Hg, with normal or decreased PaCO2

A

Type I or Classic “Hypoxemic” Respiratory Failure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

(PaCO2 > 45 mm Hg) represents the failure of the lungs to remove a sufficient amount of CO2 and is characterized by decreased alveolar minute ventilation

A

Type II or “Hypercapnic” Respiratory Failure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

beta 2 antagonist (relaxes smooth muscles) for ARF

A

albuterol, salbutamol

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

anti-cholinergic (blocks acetylcholine and prevents bronchodilation)

A

ipratropium bromide, xanthine derivatives (theophylline)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

nursing considerations for xanthine derivatives

A

assess tachycardia, xerostomia (the sensation of oral dryness, which can result from diminished saliva production)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Restlessness
Fatigue
Headache
Dyspnea
Air hunger
Tachycardia
Increased blood pressure

A

early signs associated with impaired oxygenation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Confusion
Lethargy
Tachypnea
Central cyanosis
Diaphoresis, and finally respiratory arrest

A

S&S hypoxemia progress for ARF

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

FOR ARF: Provide supplemental O2 to maintain Pao2 greater than ___ mmHg. The use of noninvasive methods for O2 administration (high flow nasal cannula or face masks)

A

60

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

characterized by non-cardiac pulmonary edema caused by increased alveolar-capillary membrane permeability and usually affects both lungs.

A

Acute Respiratory Distress Syndrome (ARDS)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

chronic inflammation of the BRONCHI (3 consecutive months and 2 consecutive years)

A

chronic bronchitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q
  • over dilatation of ALVEOLI
  • absence of recoil of ALVEOLI
  • hyperresonance due to overdistention, presence of air
  • terminal stage of COPD
  • INCREASED CO2 (above 45 mmHg)
A

pulmonary emphysema

17
Q
  • negative test for acid fast bacilli
  • no lesions in Xray
  • no hemoptysis (coughing up blood)
A

chronic bronchitis DIFFERENCE from TB

18
Q

another name for CHRONIC BRONCHITIS (hypoxic, RSCHF or enlargement of the heart)

19
Q

etiology of chronic bronchitis

A

exposure to pulmonary irritants and infection including RTI and influenza

20
Q

NI for chronic bronchitis

A
  • eliminate exposure to irritants
  • clear airway with chest physical therapy (percussion, vibration, position)
  • suctioning as ordered
  • mucolytics
  • deep breathing
  • teaching of adequate nutri
21
Q

etiology of pulmonary emphysema

A
  • age (usually geriatric)
  • hereditary (decreased Alpha T antitrypsin: below 75 mg/dL)
  • autoimmune deficiency
  • smoking
  • bronchitis
  • air pollution
  • chronic asthma (because it leads to OVERDISTENTION)
22
Q

ABG of pulmonary emphysema

A

respiratory alkalosis

23
Q

another name for pulmonary emphysema (pink skin color due to increased CO2)

24
Q

s&s of emphysema

A
  • easy fatigue
  • pursed lip breathing
  • barrel chest (due to air trapping)
  • wheezing on expiration
  • clubbing of nails
  • orthopnea
25
EMPHYSEMA: Impaired gas exchange r/t impaired ventilation: perfusion mismatching
- monitor ABG - sx of respiratory acidosis - sx hypoxia - monitor pulse ox (Spo2) - bronchodilator: "ol" and "ium" meds (check HR and BP may be increased) - good humidification - liquify secretions (3 to 5 L/day of water) - suction PRN - avoid narcotics: opium, codeine, heroin, demerol, darvon, morphine, methadone, vicodin, oxycotin (depresses RR) - low flow if COPD: 1 to 3 L/min (to avoid dependency and decreased o2 drive)
26
EMPHYSEMA: Ineffective airway clearance
- assess VS, cough, record sputum consistency, sx of hypoxia - respiratory therapy - antibiotics: "cillin", "mycin", or "in" meds or antihistamine: "ine" meds - administer steroids: "one" meds as ordered to decreased swelling of airway - avoid milk and cream products (can cause irritation = increase cough) - high fowler's
27
EMPHYSEMA: Impaired breathing pattern r/t airway obstruction
- position (high fowler) - pursed lip breathing - blow bottle exercises (10 mins per day to encourage muscle strength) - IPPB with with nebulization (mechanical ventilator, intermittent positive pressure breathing) - alternate activities with vent
28
EMPHYSEMA: High risk: complications
- ensure low flow o2 - monitor ABG - tracheostomy if necessary (if there is laryngospasm) - RISK FOR RSCHF: MEDS = diuretics due to edema, "ide" meds such as furosemide and "one" meds such as spironolactone and metolazone, FLUID = 1.5 - 1 L/day of o2 to not worsen edema, IV THERAPY = KVD 10 gtts/min to run for 24 hours
29