ENT Flashcards

(429 cards)

1
Q

Function of the nasal cavity

A

= most superior part of the respiratory tract

  1. Warms and humidifies inspired air.
  2. Removes and traps pathogens and particulate matter from the inspired air.
  3. Responsible for sense of smell.
  4. Drains and clears the paranasal sinuses and lacrimal ducts.
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2
Q

Respiratory vs Olfactory regions of nasal cavity

A

Respiratory region = lined by a ciliated pseudostratified epithelium, interspersed with mucus-secreting goblet cells.

Olfactory region = located at the apex of the nasal cavity. It is lined by olfactory cells with olfactory receptors.

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3
Q

What are the nasal conchae?

A

= curved shelves of bone projecting out of the lateral walls of the nasal cavity.

They project into the nasal cavity, creating four pathways for the air to flow

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4
Q

What are the four pathways of air flow created by the nasal conchae?

A
  1. Inferior meatus – between the inferior concha and floor of the nasal cavity.
  2. Middle meatus – between the inferior and middle concha.
  3. Superior meatus – between the middle and superior concha.
  4. Spheno-ethmoidal recess – superiorly and posteriorly to the superior concha.
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5
Q

What is the aim of the nasal conchae creating different paths of air flow?

A

to increase the surface area of the nasal cavity

to disrupt the flow of air to make it turbulent (so that it spends longer in the nasal cavity).

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6
Q

What structures drain into the nasal cavity?

A

Paranasal sinuses - frontal, maxillary, ethmoidal, sphenoid

Nasolacrimal Duct

Auditory (Eustachian) tube

Cribriform Plate

Sphenopalatine foramen

Incisive canal

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7
Q

What is the purpose of the Auditory (Eustachian) tube opening into the nasal cavity?

A

Connects the middle ear to the nasopharynx (opens onto the lateral wall of the nasal cavity)

It allows the middle ear to equalise with the atmospheric air pressure.

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8
Q

What is a problem that can occur due to the Auditory (Eustachian) tube opening into the nasal cavity?

A

Provides means for infection to spread from the upper respiratory tract to the ear.

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9
Q

Incisive Canal of nasal cavity

A

Pathway between the nasal cavity and incisive fossa of the oral cavity.

Transmits the nasopalatine nerve and greater palatine artery.

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10
Q

Sphenopalatine foramen of nasal cavity

A

Located at the level of the superior meatus

Sphenopalatine artery, nasopalatine and superior nasal nerves pass through

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11
Q

Cribriform plate of nasal cavity

A

part of the ethmoid bone

forms a portion of the roof of the nasal cavity

contains very small perforations, allowing fibres of the olfactory nerve to enter and exit.

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12
Q

Arterial Supply of nasal cavity

A

Internal carotid branches:

  • Anterior ethmoidal artery
  • Posterior ethmoidal artery

External carotid branches:

  • Sphenopalatine artery
  • Greater palatine artery
  • Superior labial artery
  • Lateral nasal arteries

These arteries form anastomoses with each other, especially in the anterior portion of the nose.

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13
Q

Little’s / Kiesselbach’s area

A

An area in the anterior portion of the nose, where there are lots of anastomoses of arteries

common site for nosebleeds (~90% from this area)

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14
Q

Venous drainage of the nose

A

The veins of the nose tend to follow the arteries.

They drain into the pterygoid plexus, facial vein or cavernous sinus.

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15
Q

How can the innervation of the nose be divided?

A

Special = the ability of the nose to smell

General = sensory innervation

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16
Q

Special sensory innervation of the nose

A

OLFACTORY NERVE

The olfactory bulb lies on the superior surface of the cribriform plate

Branches of the olfactory nerve run through the cribriform plate to provide special sensory innervation to the nose.

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17
Q

General sensory innervation of the nose

A

Innervation to the septum and lateral walls is delivered by the nasopalatine nerve (branch of maxillary nerve) and the nasociliary nerve (branch of the ophthalmic nerve).

Innervation to the external skin of the nose is supplied by the trigeminal nerve.

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18
Q

Parts of the external ear

A

Auricle
External Acoustic Meatus
Tympanic Membrane

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19
Q

Auricle

A

Functions to capture and direct sound waves towards the external acoustic meatus.

A mostly cartilaginous structure:
=> Helix, anti-helix, concha, tragus, antitragus

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20
Q

External acoustic meatus

A

A sigmoid-shaped tube
=> Initially superoanterior, then superoposterior, then inferoanterior.

Extends from the deep part of the concha to the tympanic membrane.

External 1/3 formed by cartilage
Internal 2/3 formed by the temporal bone.

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21
Q

Tympanic Membrane

A

Connective tissue structure; covered with skin on the outside and a mucous membrane on the inside

Connected to the surrounding temporal bone by a fibrocartilaginous ring

On the inner surface, the handle of malleus attaches to the tympanic membrane, at the umbo

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22
Q

Vasculature of external ear

A

Supplied by branches of the external carotid artery:

  • Posterior auricular artery
  • Superficial temporal artery
  • Occipital artery
  • Maxillary artery (deep auricular branch) – supplies the deep aspect of the external acoustic meatus and tympanic membrane only.

Venous drainage is via veins following the arteries listed above

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23
Q

Sensory innervation of external ear

A

Greater auricular nerve (branch of the cervical plexus) – innervates the skin of the auricle

Lesser occipital nerve (branch of the cervical plexus) – innervates the skin of the auricle

Auriculotemporal nerve (branch of the mandibular nerve) – innervates the skin of the auricle and external auditory meatus.

Branches of the facial and vagus nerves – innervates the deeper aspect of the auricle and external auditory meatus

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24
Q

Why can some individuals complain of an involuntary cough when cleaning their ears?

A

due to stimulation of the auricular branch of the vagus nerve (the vagus nerve is also responsible for the cough reflex).

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25
Lymphatic drainage of external ear
The lymphatic drainage of the external ear is to the superficial parotid, mastoid, upper deep cervical and superficial cervical nodes.
26
Middle Ear
Lies within the temporal bone Extends from the tympanic membrane to the lateral wall of the inner ear. Main function = to transmit vibrations from the tympanic membrane to the inner ear via the auditory ossicles. Divided into two parts - Tympanic Cavity and Epitympanic Recess
27
Middle Ear - Tympanic Cavity
located medially to the tympanic membrane, contains the 3 auditory ossicles
28
Middle Ear - Epitympanic Recess
a space superior to the tympanic cavity, which lies next to the mastoid air cells
29
What are the bones of the middle ear?
1. Malleus 2. Incus 3. Stapes
30
Middle Ear - Malleus
Attaches to the tympanic membrane, via the handle of malleus. The head of the malleus lies in the epitympanic recess, where it articulates with the next auditory ossicle.
31
Middle Ear - Incus
Consists of a body and two limbs. => The body articulates with the malleus, => The short limb attaches to the posterior wall of the middle ear => The long limb articulates with the stapes
32
Middle Ear - Stapes
The smallest bone in the human body. It joins the incus to the oval window of the inner ear. It is stirrup-shaped, with a head, two limbs, and a base. => The head articulates with the incus, and the base joins the oval window.
33
Mastoid Air Cells
Located posterior to the epitympanic recess. A collection of air-filled spaces in the mastoid process of the temporal bone. The mastoid air cells act as a “buffer system” of air – releasing air into the tympanic cavity when the pressure is too low.
34
Middle Ear - Muscles
two muscles - serve a protective function => tensor tympani and stapedius They contract in response to loud noise, inhibiting the vibrations of the malleus, incus and stapes, and reducing the transmission of sound to the inner ear.
35
Acoustic reflex
The contraction of the tensor tympani and stapedius in response to loud noise, inhibiting vibrations of the bones and thereby reducing transmission of sound to the inner ear.
36
Why are middle ear infections more common in children?
The auditory tube is shorter and straighter in children, so there is an easier pathway for a URTI to spread to the middle ear.
37
Inner Ear - contents, location, connections to middle ear
Houses the vestibulocochlear organs Located within the petrous part of the temporal bone. Has two openings into the middle ear – the oval window and the round window
38
What are the main functions of the inner ear?
1. To convert mechanical signals from the middle ear into electrical signals, which can transfer information to the auditory pathway in the brain. 2. To maintain balance by detecting position and motion
39
Bony Labyrinth of inner ear
= a series of bony cavities within the petrous part of the temporal bone. It consists of three parts – the cochlea, vestibule and the three semi-circular canals.
40
Bony Labyrinth - Vestibule
Central part Separated from the middle ear by the oval window. Communicates anteriorly with the cochlea and posteriorly with the semi-circular canals.
41
Bony Labyrinth - Cochlea
Houses the cochlear duct of the membranous labyrinth It twists upon itself around a central portion of bone (the modiolus), producing a cone shape. Branches from the cochlear portion of the vestibulocochlear (CN VIII) nerve are found at the base of the modiolus. The presence of the cochlear duct creates two perilymph-filled chambers above and below: i. Scala vestibuli ii. Scala tympani
42
Bony Labyrinth - Semi-circular Canals
There are 3 canals – anterior, lateral and posterior. Contain the semi-circular ducts, which are responsible for balance (along with the utricle and saccule).
43
Inner Ear - Membranous Labyrinth
= a continuous system of ducts filled with endolymph. It lies within the bony labyrinth, surrounded by perilymph. It is composed of the cochlear duct, three semi-circular ducts, saccule and utricle.
44
Membranous Labyrinth - Cochlear Duct
Triangular shape Separated from the scala vestibuli by Reissner’s membrane Separated from the scala tympani by the basilar membrane The basilar membrane houses the epithelial cells of hearing – the Organ of Corti.
45
Membranous Labyrinth - Saccule and Utricle
= Two membranous sacs located in the vestibule. They are organs of balance – detect movement or acceleration of the head in the vertical and horizontal planes => The utricle receives the three semi-circular ducts. => The saccule receives the cochlear duct. Endolymph drains from the saccule and utricle into the endolymphatic duct.
46
Membranous Labyrinth - semi-circular ducts
Located within the semi-circular canals Upon movement of the head, the flow of endolymph within the ducts changes speed and/or direction. Sensory receptors detect this change, and send signals to the brain, allowing for the processing of balance.
47
Inner Ear - innervation
Innervated by the vestibulocochlear nerve (CN VIII). At the internal acoustic meatus it divides into the: - Vestibular nerve – supplies the utricle, saccule and three semi-circular ducts. - Cochlear nerve – supplies the receptors of the Organ of Corti
48
Bones of the neurocranium
Four singular bones centred on the midline (frontal, ethmoid, sphenoid, and occipital). Two sets of bones occurring as bilateral pairs (temporal and parietal).
49
Bones of the viscerocranium (facial skeleton)
Three singular bones lying in the midline (mandible, ethmoid, and vomer) Six paired bones occurring bilaterally (maxilla; inferior nasal concha [turbinate], zygomatic, palatine, nasal, and lacrimal bones).
50
Muscles of face
Occipitofrontalis Obicularis Oculi Obicularis Oris Buccinator Nose: 1. Nasalis – transverse and alar parts 2. Procerus 3. Depressor septi nasi Platysma
51
Sensory innervation of face
provided mainly by the trigeminal nerve (CN V) via the: 1. The ophthalmic nerve (CN V1) – only sensory 2. The maxillary nerve (CN V2) – only sensory 3. The mandibular nerve (CN V3) – sensory and motor
52
Motor innervation of face
1. Mandibular nerve (CN V3) – the muscles of mastication (masseter, temporal, medial and lateral pterygoids). 2. Facial nerve (CN VII) – the muscles of facial expression.
53
Where does the facial nerve emerge from the cranium?
stylomastoid foramen
54
Superficial lymph nodes of head and neck
Receive lymph from the scalp, face and neck * Submental * Submandibular * Superficial parotid/pre-auricular * Mastoid / retroauricular * Occipital
55
Deep (cervical) lymph nodes of head and neck
Receive all of the lymph from the head and neck – either directly or indirectly via the superficial lymph nodes. They are organised into a vertical chain, located within close proximity to the IJV within the carotid sheath.
56
Parotid gland - location
enclosed within a tough fascial capsule – the parotid sheath ``` apex = posterior to the angle of the mandible base = related to the zygomatic arch ```
57
Parotid duct
The parotid duct passes horizontally from the anterior edge of the parotid gland. At the anterior border of the masseter, it pierces the buccinator and enters the oral cavity through a small orifice opposite the second maxillary molar tooth.
58
Sensory innervation of the parotid sheath
greater auricular nerve (C2 and C3) provides sensory innervation to the parotid sheath and overlying skin.
59
Boundaries of anterior triangle of neck
* Superiorly – inferior border of the mandible (jawbone). * Laterally – anterior border of the sternocleidomastoid. * Medially – sagittal line down the midline of the neck.
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suprahyoid muscles
located superiorly to hyoid bone: Stylohyoid Digastric Mylohyoid Geniohyoid
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infrahyoid muscles
located inferiorly to hyoid bone: Omohyoid Sternohyoid Thyrohyoid Sternothyroid
62
Contents of carotid triangle
Common carotid artery (which bifurcates within the carotid triangle) Carotid sinus Internal jugular vein Hypoglossal and vagus nerves.
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Contents of submental triangle
the submental lymph nodes | => filter lymph draining from the floor of the mouth and parts of the tongue.
64
Contents of submandibular triangle
- The submandibular gland (salivary), and lymph nodes. | - The facial artery and vein also pass through this area.
65
Boundaries of posterior triangle of neck
* Anterior – posterior border of the sternocleidomastoid. * Posterior – anterior border of the trapezius muscle. * Inferior – middle 1/3 of the clavicle
66
Contents of the posterior triangle of neck
* many muscles (including a number of vertebral muscles which are covered by the pre-vertebral fascia) * the EJV * the accessory nerve (CN XI) * the cervical plexus * the trunks of the brachial plexus
67
Path of the external jugular vein
Formed by the retromandibular and posterior auricular veins Lies superficially, crosses SCM muscle to reach posterior triangle. Within the posterior triangle, the EJV pierces the investing layer of fascia and empties into the subclavian vein.
68
What are the 3 main distinct features of the cervical vertebrae?
1. Triangular vertebral foramen. 2. Bifid spinous process – this is where the spinous process splits into two distally. 3. Transverse foramina – holes in the transverse processes. => They give passage to the vertebral artery, vein and sympathetic nerves.
69
C-Spine - C1
= the atlas. * Has no vertebral body and no spinous process. * Has lateral masses which are connected by an anterior and posterior arch. * Each lateral mass contains a superior articular facet (for articulation with occipital condyles), and an inferior articular facet (for articulation with C2). * There is a transverse ligament of the atlas to secure the articulation with the dens of the axis.
70
C-spine - C2
= the axis. * Has the dens/odontoid process, which extends superiorly to articulate with the anterior arch of the atlas, in doing so creating the medial atlanto-axial joint. * The axis also contains superior articular facets, which articulate with the inferior articular facets of the atlas to form the two lateral atlanto-axial joints.
71
What and where is the pharynx?
a muscular tube that connects the oral and nasal cavity to the larynx and oesophagus. It begins at the base of the skull and ends at the inferior border of the cricoid cartilage (C6). Consists of: 1. Nasopharynx 2. Oropharynx 3. Laryngopharynx
72
Nasopharynx - location, contents, function
• Located between between the base of the skull and the soft palate • Performs a respiratory function by conditioning inspired air and propagating it to the larynx. => Lined with respiratory epithelium • Contains the adenoid tonsils.
73
Oropharynx - location, contents, function
* Located between the soft palate and the superior border of the epiglottis * Contains the posterior 1/3 of the tongue, the lingual tonsils, the palatine tonsils and superior pharyngeal constrictor muscle. * Involved in the involuntary and voluntary phases of swallowing.
74
Laryngopharynx - location, contents
* Located between the superior border of the epiglottis and inferior border of the cricoid cartilage (C6) * Continues inferiorly as the oesophagus * Contains the middle and inferior pharyngeal constrictors
75
Waldeyer’s ring
= the ring of lymphoid tissue in the naso- and oropharynx formed by the paired palatine tonsils, the adenoid tonsils and lingual tonsil.
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Motor/sensory innervation of the pharynx
MOTOR Most muscles are innervated by the vagus nerve (CN X). the only exception being the stylopharyngeus (glossopharyngeal nerve). SENSORY Receives sensory innervation from the glossopharyngeal nerve (CN IX)
77
What are the two main groups of pharyngeal muscles?
CIRCULAR - contract sequentially from superior to inferior to constrict the lumen and propel the bolus of food. LONGITUDINAL - act to shorten and widen the pharynx and elevate the larynx during swallowing
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Circular pharyngeal muscles
Superior pharyngeal constrictor (oropharynx) Middle pharyngeal constrictor (laryngopharynx) Inferior pharyngeal constrictor (laryngopharynx)
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Longitudinal pharyngeal muscles
Stylopharyngeus (styloid process to pharynx) Palatopharyngeus (hard palate to pharynx) Salpingopharyngeus (Eustachian tube to pharynx)
80
Vascular supply to the pharynx
ARTERIAL SUPPLY via branches of the external carotid artery: - Ascending pharyngeal artery - Branches of the facial artery - Branches of the lingual and maxillary arteries. VENOUS DRAINAGE Achieved by the pharyngeal venous plexus, which drains into the IJV.
81
Where is the Larynx?
- Suspended from hyoid bone - Spans between C3 and C7 - Covered anteriorly by the infrahyoid muscles - Covered laterally by the lobes of the thyroid gland.
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Function of the larynx
Several important functions: | => Phonation, cough reflex, protection of the lower respiratory tract.
83
Sections of the larynx
1. Supraglottis – from the inferior surface of the epiglottis to the vestibular folds (false vocal cords). 2. Glottis – contains vocal cords and 1cm below them. 3. Subglottis – from inferior border of the glottis to the inferior border of the cricoid cartilage.
84
What epithelium is there in the larynx?
Lined by pseudostratified ciliated columnar epithelium. An important exception to this is the true vocal cords, which are lined by a stratified squamous epithelium.
85
Vascular supply to the larynx
ARTERIAL SUPPLY - Superior laryngeal artery (from the external carotid) - Inferior laryngeal artery (from the thyrocervical trunk) VENOUS DRAINAGE - Superior laryngeal vein (=> IJV) - Inferior laryngeal vein (=> left brachiocephalic vein )
86
Motor/Sensory innervation of larynx
via branches of the vagus nerve: - Recurrent laryngeal nerve – provides sensory innervation to the infraglottis, and motor innervation to all the internal muscles of larynx (except the cricothyroid). - Superior laryngeal nerve – the internal branch provides sensory innervation to the supraglottis, and the external branch provides motor innervation to the cricothyroid muscle.
87
Cartilages of larynx
UNPAIRED: Thyroid Cricoid Epiglottis PAIRED Arytenoid Corniculate Cuneiform
88
What is Adam's apple?
= laryngeal prominence of thyroid cartilage | where the two sheets of cartilage joints anteriorly
89
Cricoid cartilage
= a complete ring of hyaline cartilage, completely encircling the airway Articulates with the paired arytenoid cartilages posteriorly and thyroid cartilage superiorly
90
Epiglottis
= a leaf-shaped plate of elastic cartilage Marks the entrance to the larynx During swallowing, the epiglottis flattens and moves posteriorly to close of the larynx and prevent aspiration.
91
What are the laryngeal folds?
- Vocal folds (= true vocal cords) | - Vestibular folds (= false vocal cords)
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Vocal folds / true vocal cords
Abducted, adducted, relaxed and tensed under control of the muscles of phonation to control the pitch of the sound created. Relatively avascular, appear white in colour Space between the vocal folds is called the rima glottidis.
93
Vestibular folds / false vocal cords
Pink in colour Act to provide protection to the larynx
94
Groups of muscles of the larynx
1. External muscles => Act to elevate or depress the larynx during swallowing 2. Internal muscles. => Act to move the individual components of the larynx => Play a vital role in breathing and phonation.
95
Innervation of the intrinsic muscles of the larynx
All innervated by the inferior laryngeal nerve, which is the terminal branch of the recurrent laryngeal nerve (except the cricothyroid – innervated by external branch of superior laryngeal nerve).
96
Thyroid gland - location and structure
Spans the C5-T1 vertebrae Two lobes (left and right); connected by a central isthmus anteriorly. Lobes are wrapped around the cricoid cartilage and superior rings of the trachea. Located within the visceral compartment of the neck (along with the trachea, oesophagus and pharynx) which is bounded by the pre-tracheal fascia.
97
Thyroid gland - arterial supply
= two main arteries (and one additional one) Superior thyroid artery - 1st branch of the external carotid a. Inferior thyroid artery - Arises from the thyrocervical trunk (branch of subclavian a.) (Thyroid ima artery) - Only ~10% of people have this additional artery - Arises from brachiocephalic trunk
98
Thyroid gland - venous drainage
Superior, middle, and inferior thyroid veins, which form a venous plexus around the thyroid gland. Superior and middle veins drain into the IJV Inferior vein drains into the brachiocephalic trunk.
99
Thyroid gland - innervation
Branches of sympathetic trunk
100
Thyroid gland - lymphatic drainage
To the paratracheal and deep cervical nodes.
101
Parathyroid Glands - location
Located on posterior aspect of thyroid gland (external to the thyroid but within the pretracheal fascia) Most people have 4 parathyroid glands (although variation from 2-6 is common) => Superior – located in the middle of posterior border of each thyroid lobe. => Inferior – usually found near the inferior poles of the thyroid gland, but location is inconsistent (can be found as far inferiorly as superior mediastinum)
102
Cervical Plexus
Located in the posterior triangle of the neck, within the prevertebral layer of cervical fascia Formed from the anterior rami of cervical spine nerves C1-C4 Muscular branches lie deep to sensory branches
103
Sensory branches of cervical plexus
Greater Auricular Nerve (C2 and C3) => external ear and skin over parotid gland Transverse Cervical Nerve (C2 and C3) => anterior neck Lesser occipital Nerve (C2) => posterosuperior scalp Supraclavicular Nerves (C3 and C4) => skin overlying supraclavicular fossa
104
Stertor
= noisy breathing due to partial obstruction ABOVE the larynx (tonsils, adenoids, tongue, angioedema)
105
Stridor
= noisy breathing due to partial obstruction BELOW the larynx NEEDS ENT REFERRAL.
106
Signs of severe airway obstruction
- Tracheal tug/recession - Tachycardia - Hypoxia - Use of Accessory mm. - Confusion
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Differentials of stridor
CONGENITAL Laryngomalacia VC web / VC Palsy Subglottic stenosis ``` ACQUIRED (acute) Laryngeal trauma Foreign body Croup Epiglottitis Allergic reaction ``` ``` ACQUIRED (chronic) VC Palsy VC polyp/cyst Tumour Thyroid mass Subglottic stenosis ```
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Laryngeal trauma - presentation and management
Stridor, neck bruising, surgical emphysema Mx = intubation +/- tracheostomy
109
Foreign body in airway - presentation, Ix, Mx
Feel something “stick” in throat, sharp pain, cannot eat/drink/swallow saliva Ix = lateral neck X-ray and CXR Mx = flexiscope and removal
110
Management of Stridor
1. Basic Hx and assess severity (cyanosis, RR, etc.) 2. A-E assessment 3. Secure Airway
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Methods of securing airway in stridor
a. Endotracheal Tube = 1st line b. Cricothyroidotomy c. Tracheostomy
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Cricothyroidotomy - method
Brown IV cannula through cricothyroid membrane & connect high flow O2 Incision in midline of cricothyroid membrane and insert ET tube with O2 bag
113
Tracheostomy - method and complications
Tube inserted between 2nd and 4th rings of cartilage Complications = tube blockage, wound infection, pneumothorax
114
Indications for tracheostomy
Stridor, Drain/prevent over-spilling of secretions, Respiratory failure.
115
What is tonsillitis?
= an acute bacterial infection of the tonsils Caused by: - strep. pyogenes, - staphylococci - m. catarrhalis
116
Tonsilitis - symptoms
- Sore throat + odynophagia - Pyrexia, malaise, etc. - Lymphadenopathy +/- pus on tonsils
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Tonsilitis - Centor Score
``` (1 point each): C – absence of [C]ough E – tonsillar [E]xudate N – tender cervical [N]odes T - >38oC [T]emperature ``` If Score >3 = high chance of strep A and need ABX
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Tonsilitis - complications
``` Peritonsillar abscess (quinsy) Deep neck space infection ```
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Tonsilitis - management
IF UNILATERAL Sx => ENT Referral Analgesia, fluids, soft food. ABX – PO penicillin V (or clarithromycin if pen allergic) Tonsillectomy if recurrent/complications
120
When is a tonsillectomy done?
Done if tonsilitis is recurrent/complications: 7x in 1 year 5x in each of 2 years 3x in each of 3 years 2 episodes of quinsy
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Tonsillectomy - complications
1-2 weeks of pain post-op Complications: a. 1o post-op haemorrhage – needs surgery b. 2o post-op haemorrhage – from infection => IV ABX
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What is peritonsillar abscess / Quinsy?
= pus between the tonsil capsule and lateral pharyngeal wall Caused by strep. pyogenes
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Presentation of peritonsillar abscess / Quinsy
SYMPTOMS - Sore throat, odynophagia, dysphagia - Trismus - “Hot potato voice” (muffled voice) - Referred otalgia SIGNS - Usually unilateral (DDx = tumour) - Unilateral swelling, LATERAL to tonsil. - Deviated tonsil and uvula to opposite side.
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What is trismus?
= restriction of the range of motion of the jaws
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Management of peritonsillar abscess / quinsy
ENT referral - Needle aspiration or incision & drainage - IV ABX +/- steroids for swelling - Analgesia, fluids, soft food.
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What is infectious mononucleosis?
"glandular fever" = EBV infection affecting LNs, tonsils and liver.
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Infectious mononucleosis - symptoms
- Prodromal illness: fever, malaise - Sore throat, dysphagia - Cervical lymphadenopathy - Abdo pain - Hepatosplenomegaly (50%)
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Infectious mononucleosis - investigations
FBC LFT Blood film Monospot test
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Infectious mononucleosis - management
Self-resolves in 2-4 weeks Supportive Tx – analgesia, fluids ABX ONLY if tonsilitis (but NOT amoxicillin) Monitor LFTs Advice - Avoid intimate contact - No contact sport => splenic rupture - No alcohol => liver damage
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What ABX should be avoided in a patient with glandular fever?
ABX are not routinely given as EBV is a virus => should only be given if there is evidence of bacterial tonsillitis Avoid ampicillin and amoxicillin => rash
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Pharyngitis - acute/chronic
1. Acute: Sudden-onset sore throat Usually viral (rhinovirus, coronavirus, influenza, HSV, VZV) May be bacterial (group A strep) 2. Chronic: Long-standing sore throat Specific (syphilis, TB, toxoplasmosis) Non-specific (GORD, tobacco, chronic sinusitis).
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Pharyngitis - management
fluids, analgesia, | gargle warm salty water.
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What causes epiglottis?
H. influenzae
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Epiglottitis - Sx
PHARYNX NORMAL O/E Very sore throat + high fever Dysphagia, drooling Stridor
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Epiglottitis - Mx
Immediate admission Airway protection – intubation/tracheostomy IV ABX and steroids DO NOT TRY TO EXAMINE THROAT/MOUTH
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How do deep neck space infections occur?
Due to spread of throat infections (pus/abscess) via para or retro-pharyngeal space.
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Deep neck space infection - symptoms
``` Sore throat + odynophagia Dysphagia, drooling Fever Trismus Reduced neck movements “Hot potato voice” ```
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Deep neck space infection - signs
Poor Head Movement Neck mass Septic
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Deep neck space infection - investigations
CT – shows deep neck spaces USS – shows abscesses OPG – dental x-ray
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Deep neck space infection - management
Emergency A - E Assessment Airway protection IV ABX Surgical drainage
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Deep neck space infection - complications
- Airway compromise - Empyema - Pneumonia - Mediastinitis (50% mortality) - Carotid artery erosion
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Why can you get referred otalgia with a sore throat?
Ear has shared nerve supply with oro/laryngopharynx
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Globus Pharyngeus
Painless sensation of “sticking” / lump in throat even when not swallowing Causes – LP reflux, stress/anxiety, minor inflammation
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Globus Pharyngeus - Mx
Must exclude pathologies like cancer Mx – Treat any underlying cause, Avoid caffeine/smoking, Sip icy sparkling water
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Definition of sleep apnoea
30 or more episodes of cessation of breathing, each lasting at least 10 seconds, over a period of 7 hours of sleep
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Sleep Apnoea Index
measures the number of episodes to determine the severity.
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Causes of sleep apnoea
1. OBSTRUCTIVE – due to upper airway collapse (decreased O2 causes reflex of waking slightly and taking deep breath). 2. CENTRAL – fault with central respiratory drive (e.g. cerebral palsy, cognitive defect).
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Risk factors for sleep apnoea
``` Old age Male Down’s Syndrome Sedatives OBESITY Smoking/alcohol Craniofacial abnormalities Neuromuscular Disease ```
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Sleep apnoea - Sx
* Snoring/choking in sleep and witnessed apnoeas * Restless/non-refreshing sleep * Daytime sleepiness and decreased concentration * Irritability and decreased libido Kids – poor school performance Babies – poor feeding as blocked nasal breathing.
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Sleep apnoea - Ix
1. History 2. Examination – upper airway endoscopy 3. Sleep Studies - Measure pulse, ECG, O2 overnight - Audio/video recording of sleep - Polysomnography = gold-standard version
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Sleep apnoea - Mx
1. Lifestyle – weight loss, smoking/alcohol reduction 2. Conservative – nasal splints/tape & jaw advancers 3. Medical – CPAP via mask => Noisy and uncomfortable 4. Surgery – adenotonsillectomy, polypectomy, uvulopalatopharyngoplasty
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Management of snoring
1. Lifestyle – weight loss, smoking/alcohol reduction | 2. Conservative – nasal splints/tape & jaw advancers
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Major salivary glands
= 3 paired glands 1. Parotid – serous 2. Submandibular – mixed 3. Sublingual – mucous
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Why is the parotid gland painful if swollen?
it has a fibrous capsule which is painful if stretched
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What is xerostomia? What are some causes?
= dry mouth ``` Causes: • Depression/anxiety • Drugs – antimuscarinics/sympathomimetics • Radiotherapy to head/neck • Sjogren’s Syndrome ```
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Sjogren’s Syndrome
= an autoimmune disorder of decreased saliva/mucous.
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What is there increased risk of in people with Sjogren’s Syndrome?
increased risk of non-Hodgkin’s lymphoma.
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Sjogren’s Syndrome - symptoms
DRY MOUTH + EYES (+ vagina) Glossitis +/- Parotid gland enlargement
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Sjogren’s Syndrome - investigations
HLA, B8, DR2 Specific antigens – SSA, SSB Labial biopsy = diagnostic
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Sjogren’s Syndrome - management
Steroids Artificial saliva/tears (Parotidectomy if recurrent parotitis)
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What is Sialadenitis? What are the causes and risk factors?
= inflammation of salivary gland Causes – infection, stones, malignancy RFs – dehydration, poor oral hygiene, elderly.
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Sialadenitis - Sx
Swollen, tender gland +/- pus from duct +/- fever and systemic Sx
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Sialadenitis - Mx
Hydration & analgesia High dose ABX +/- pus drainage (Gland removal)
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Parotitis
= inflammation of parotid gland Causes: - Infection (measles, mumps, HIV, TB, candidiasis) - Sarcoid - Drugs
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Sialolithiasis
= Calculi in salivary glands
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scialectasis
Dilation of salivary ducts
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Sialolithiasis - Sx
Post-prandial swelling & pain | +/- palpable calculi in gland
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Sialolithiasis - Ix
CT/X-ray | Sialogram
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Sialolithiasis - Mx
Hydration & analgesia Duct massage Surgical stone removal (if necessary)
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Non-salivary causes of facial swelling
``` Masseter hypertrophy Lymphadenopathy Dental infection/abscess Mastoiditis Cysts ```
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Salivary causes of facial swelling
Sialadenitis Sialolithiasis Sjogren’s Syndrome Neoplasm – benign/malignant
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Process of normal voice production
= due to vocal cord vibration Oscillation of VCs causes sound wave that resonates within vocal tract Vowel production = vibration of OPEN VCs and mouth/tongue position Consonant production = force air through narrowed VCs
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Vocal cords - fundamental frequency (F0)
= PITCH (Hz) Determined by density of vocal fold Density altered by muscle contraction/relaxation Higher density = lower frequency (e.g. males, Reinke oedema)
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Vocal cords - Intensity/Pressure Level
= LOUDNESS (dB) Determined by subglottic pressure Pressure depends on degree of VC closure/length of closure Lower pressure = weaker voice (e.g. recurrent laryngeal nerve palsy).
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Dysphonia
= any voice impairment
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Dysarthria
= reduced voice muscle coordination
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Dysphasia
= receptive or comprehensive impairment
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Structural/neoplastic causes of voice disorders
Malignant = Laryngeal Carcinoma Benign – Polyp Benign – Reinke’s Oedema
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Laryngeal Carcinoma - causes
Causes = smoking, genetics, alcohol excess
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Laryngeal Carcinoma - Presentation
Sx = progressive hoarseness; +/- stridor, dysphagia, referred otalgia, cervical lymphadenopathy. Signs = irregular mass; leukoplakia/eythroplakia
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Laryngeal Carcinoma - Mx
Mx = radiotherapy; surgical excision.
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Vocal cord Polyp - cause and Sx
Causes = shouting Sx = Husky (deeper) voice Signs = smooth, grey swelling (usually UNILATERAL)
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Vocal cord Polyp - Mx
= surgical excision; +/- medical Tx; +/- voice therapy
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Reinke's Oedema
= a collection of fluid in Reinke's space Causes = smoking, voice overuse, LP reflux
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Reinke's Oedema - Presentation
Sx = deep, gravelly voice Signs = grey/red swelling (usually BILATERAL)
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Reinke's Oedema - Mx
Stop smoking/treat reflux; Surgical reduction; Voice therapy.
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Reinke's space
= a potential space between the vocal ligament and the overlying mucosa
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Inflammatory causes of voice disorders
Laryngitis | Laryngopharyngeal Reflux
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Laryngitis
= inflammation of the larynx Cause = Bacterial / fungal / HPV Sx: • Hoarse/croaky/voice loss; • Sore throat, odynophagia; • URTI symptoms Signs = erythematous, sloughy VCs
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Laryngitis - Mx
Self-limiting Voice rest, Supportive - analgesia, fluids; Steam inhalations
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Laryngopharyngeal Reflux - Sx
``` Sx • Strained voice + decreased pitch range • Dysphagia & globus sensation • Cough and constant throat clearing • May NOT have any heartburn ``` Signs • General erythema & oedema
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Laryngopharyngeal Reflux - Mx
* Gaviscon & PPI * Vocal hygiene * Dietary advice – avoid fatty/fried food & caffeine
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Neuromuscular causes of voice disorders
= recurrent laryngeal nerve palsy.
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Muscle tension imbalance causes of voice disorders
= excessive tension of laryngeal muscles.
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Recurrent laryngeal nerve palsy - causes
* Surgical trauma (e.g. thyroidectomy) * Malignancy * Idiopathic * Neurological disorders
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Recurrent laryngeal nerve palsy - Sx
* Weak, higher pitched voice * Tires with prolonged use * Choking on fluids * Weak “bovine” cough * Diplophonia (two tone voice)
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Recurrent laryngeal nerve palsy - Ix
1. Examination – listen to voice, head & neck exam, CNS exam 2. CXR – to exclude mediastinal mass/pancoast tumour 3. CT (skull base to mid-thorax) – check for lesions along nerve 4. Barium swallow – if oesophageal lesion suspected
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Recurrent laryngeal nerve palsy - Mx
Can just wait for spontaneous recovery Voice therapy VC medialisation – inject collagen/surgery
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Excessive tension of laryngeal muscles - causes
* Stress/anxiety * Following URTI * Long-term ineffective voice use * Compensation for underlying VC problem (e.g. cyst)
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Excessive tension of laryngeal muscles - Sx
* Husky voice – worse with use * Deeper or higher pitched than expected * Unstable voice * Sore throat
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Excessive tension of laryngeal muscles - Mx
* Vocal hygiene – steam inhalations * Lifestyle advice – avoid irritants (smoke, caffeine, spicy food) * Voice therapy
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Aims of voice therapy
Aims to restore voice, eliminate benign nodules and avoid further vocal problems.
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Indications for voice therapy
LP reflux Nodules, cysts, polyps Muscle tension imbalance Psychological voice problems
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Components of voice therapy
Semi-occluded airflow exercises – reduce muscle straining Efficient respiration Voice resonance and projection Advice on vocal hygiene – steam inhalations, avoid irritants, etc.
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Types of head and neck malignancies
Aerodigestive tract (nasal/oral cavity, pharynx, larynx) = SCCs Glands Lymph nodes – lymphomas, secondary tumours Thyroid = papillary, follicular, medullary, anaplastic Skin = SCCs, BCCs, melanomas
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Risk factors for head and neck cancers
Tobacco/alcohol (including chewing tobacco) HPV 16 & 18 Occupation – woodwork, textiles, nickel Leukoplakia => 1/3 become cancerous Eythroplakia => ½ become cancerous
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Leukoplakia
Grey/White patches in the mouth Don’t come off when scraped => 1/3 become cancerous
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Erythroplakia
Red patches in the mouth Bleed easily if scraped => ½ become cancerous
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2WW referral for ?head & neck malignancy
- Odynophagia/Dysphagia >3 weeks - Hoarseness >3 weeks - Persistent, unexplained neck lump >3 weeks - Persistent mouth ulceration >3 weeks - Leukoplakia/Eythroplakia
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Signs of head & neck malignancy
- Persistent, unexplained neck lump >3 weeks * - Persistent mouth ulceration >3 weeks * - Leukoplakia/Eythroplakia * - Bleeding in mouth/throat or haemoptysis - General B symptoms – weight loss, night sweats, fever
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Symptoms of head & neck malignancy
OFTEN UNILATERAL - Odynophagia/Dysphagia >3 weeks * - Hoarseness >3 weeks * - Trismus - Referred otalgia - Dyspnoea/Stridor
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Head & neck malignancy - investigations
History and Head/neck exam + flexible nasal endoscopy Bloods – FBC, U&E, LFT, TFT, glucose, albumin Assess nutritional status Fine needle aspiration cytology (FNAC) BIOPSY = DIAGNOSTIC => But avoid if possible as need a GA CT/MRI of neck (for TNM staging) CXR/CT chest (for TNM staging)
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Glottic tumour
= most common head and neck cancer, and good prognosis Hoarseness first, then odynophagia/stridor Often no lymphadenopathy
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Supra/subglottic tumour
= late presentation and poor prognosis Odynophagia/stridor first, then hoarseness Referred otalgia
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Head & Neck cancer - management
Depends on TNM stage & age/health of patient. MDT involvement. 1. Surgery - Neck dissection to remove LNs +/- SCM, IJV, SAN - Laryngectomy 2. Radiotherapy/chemotherapy 3. Transoral Laser Resection
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What is a laryngectomy?
= “end tracheostomy” Remove the larynx and bring airway to surface Separated from the nose/mouth/oesophagus Unlike tracheostomy where still have airway above tube
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Laryngectomy - follow up
- Swallowing difficulties (SALT) | - Voice restoration (SALT)
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Thyroid cancer - Sx
Neck lump, moves when swallow/tongue out Hoarse voice / breathing difficulties
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Thyroid cancer - Mx
Thyroidectomy Neck dissection +/- radioactive iodine
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Causes of dysphagia
Structural changes – e.g. post-op Obstructive – e.g. malignancy, pharyngeal pouch Neurological – e.g. CVA/stroke Muscular Respiratory Gastro-oesophageal – e.g. LP reflux/GORD
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Signs/symptoms of dysphagia
Food/fluid pockets in mouth and/or “sticks” in throat Aspiration => coughing, wheezing, recurrent chest infections Dehydration Weight loss
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What should be done for a patient with unexplained dysphagia >3 weeks
2WW referral
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Dysphagia - Ix
Video-fluoroscopy Barium swallow Endoscopy
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Dysphagia - Mx
MDT management ``` Swallowing exercises Oral care – steam inhalations, artificial saliva Posture and positioning Adaptive equipment – cups/straws/ spoons Modified diet – pureed, thickeners ```
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What is the most common cause of a neck lump?
Reactive Lymphadenopathy (caused by infection)
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Head/Neck lump differentials - reactive lymphadenopathy
Enlarged LNs Infective cause Will have Hx of infective Sx
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Head/Neck lump differentials - lymphadenopathy due to neoplasm
Lymphoma Rubbery, painless lump B-cell Sx
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Head/Neck lump differentials - Thyroglossal cyst
Moves up with tongue protrusion Common in <20 yo
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Congenital neck lumps
Thyroglossal cyst Cystic hygroma Branchial cyst
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Head/Neck lump differentials - Branchial cyst
Smooth, mobile, oval Most commonly located along the anterior border and the upper 1/3 of SCM Present in early adulthood
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Head/Neck lump differentials - Cystic hygroma
Lymphatic lesion – soft, fluctuant, transilluminable 90% present <2 yo
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Head/Neck lump differentials - neoplasm of salivary glands
Features depend on type CN VII palsy if malignant
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Head/Neck lump differentials - infection of salivary glands
Swollen & painful Pain related to eating Xerostomia & dry eyes
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Head/Neck lump differentials - stone of salivary glands
Swollen & painful Pain related to eating Xerostomia & dry eyes
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Head/Neck lump differentials - carotid aneurysm
Pulsatile, lateral mass | +/- dysphagia, hoarseness
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Head/Neck lump differentials - Sebaceous cyst/lipoma
Soft, mobile | +/- pain
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Excess wax - management
Wax softening drops – e.g. sodium bicarbonate/olive oil Ear syringing
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Outer ear foreign bodies - management
Wax hook/forceps/suction to remove FB GA if uncooperative/deep in canal
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What are contraindications to ear syringing?
grommets, perforation, otitis externa
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Pinna haematoma
Blood collects between cartilage and perichondrium. Caused by Trauma Complications = Avascular necrosis & infection (= CAULIFLOWER EAR) Management = immediate drainage.
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What types of outer ear neoplasm are there?
Benign – papilloma or adenoma Malignant – BCC or SCC
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Otitis externa - signs and symptoms
= inflammation of the ear canal Symptoms: - Pain & swelling - Itching - Hearing loss - Discharge (from middle ear through perforated TM) Signs: - Tender pinna/tragus - Swollen/red canal - TM not visible
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Causes of otitis externa
Skin conditions (e.g. eczema, psoriasis) Generalised skin infections (e.g. impetigo) Localised skin infections (e.g. pseudomonas, S. aureus, candida) Trauma/foreign bodies Water exposure
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Otitis externa - Mx
Mild/simple (TM visible) • Analgesia and keep dry • Topical ABX +/- steroid ``` Severe/complex (TM not visible/Tx resistant): => ENT referral • Microsuction • Pope wick & drops • PO ABX if pinna cellulitis ```
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Differentials/complications of otitis externa
``` Necrotising OE Mastoiditis Pinna perichondritis Pinna cellulitis Middle ear infection (discharge but no canal swelling) ```
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What is Necrotising OE?
= complication of Acute otitis externa (AOE) where the infection spreads to skull base. Caused by pseudomonas aeruginosa.
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Necrotising OE - signs
Severe pain – worse at night and when chewing Nerve palsies – CN VII, IX, X, XI Canal granulations
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Necrotising OE - Mx
ENT referral High dose IV ABX CT/MRI
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Acute otitis media with effusion (OME)
= “Glue Ear” Symptoms: - Middle ear fluid with no Sx of Infection (painless) - Conductive hearing loss of 20-30 dB - Speech delay/school problems
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Acute otitis media with effusion - RFs
- Child - Smoking - Large adenoids - Nasal abnormalities
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Acute otitis media with effusion - cause
Cause = Eustachian tube dysfunction Due to: Nasal/sinus infection Allergic response Ciliary dysfunction
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Acute otitis media with effusion - Mx
50% spontaneous resolution If >3 months: - Grommets – ventilate middle ear (pop out in 18 months) - Hearing aids
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Acute Suppurative Otitis Media (ASOM)
= acute infection of the middle ear Causes – H. influenza (most common bacterial cause), S. pneumoniae, M. catarrhalis, RSV/rhinovirus
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Acute Suppurative Otitis Media - signs and symptoms
Symptoms: - PAIN! => crying/screaming child - Fever / systemic upset - Conductive hearing loss - Otorrhoea (pus +/- blood) => if TM perforated, this relieves pain Signs: - Bulging TM - TM perforation & pus/blood
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Acute Suppurative Otitis Media - management
by GP: 1. Analgesia & wait for resolution in 3-7 days ``` 2. PO amoxicillin +/- steroid ear drops ONLY if: • <6 months old • <2 years with bilateral Sx • Risk of complications • Systemically very unwell ```
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Acute Suppurative Otitis Media - complications
- Residual perforation/effusion (chronic SOM) - Ossicle necrosis - Tympanosclerosis - Intracranial sepsis/meningitis - Facial Palsy - Labyrinthitis - Mastoiditis
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Mastoiditis - Sx and Mx
Otalgia, hearing loss, malaise/pyrexia, post-auricular swelling, pinna down & forwards. Mx = ABX +/- surgery (ENT referral)
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What is Chronic Suppurative Otitis Media?
Recurrent ASOM (>6 weeks) leading to damage of the TM.
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Chronic Suppurative Otitis Media - Sx
Repeated ottorhoea CHL – 10-20 dB or more
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Chronic Suppurative Otitis Media - Mx
= ENT referral to assess possible complications: Regular aural toilet ABX + steroid ear drops
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What is cholesteatoma?
= accumulation of keratinising squamous epithelium attracting anaerobic bacteria (pseudomonas aeruginosa).
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Cholesteatoma - Sx and signs
FOUL SMELLING OTORRHOEA Attic retraction & squamous debris Conductive hearing loss
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Cholesteatoma - Ix
CT/MRI
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Cholesteatoma - Mx
ENT referral Surgical removal of sac Mastoidectomy if advanced disease
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Cholesteatoma - complications
= due to erosion of bone & nearby structures: - Facial nerve palsy - Vertigo - Intracranial sepsis - Conductive HL (FOUL OTORRHOEA + FN PALSY NEEDS ENT REFERRAL)
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Tympanic Membrane perforation - causes
``` AOM Foreign bodies Head injury – temporal bone fracture Barotrauma Sudden increase in air pressure – e.g. loud noise/slap ```
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Temporal bone fracture
Needs CT 20% transverse => sensorineural HL 80% longitudinal => conductive HL Complications – TM perforation, CSF leak, bleed, FN palsy
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Tympanic Membrane perforation - Sx
``` Conductive HL (10-20 dB) +/- pain, tinnitus, vertigo ```
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Tympanic Membrane perforation - Mx
Heals in 6 weeks => Keep dry & wait GP follow up in 6 weeks => If not healed, then ENT referral
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What is otosclerosis?
A familial condition where spongey bone forms around oval window, causing fusion with stapes.
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Otosclerosis - Sx
Progressive, bilateral conductive HL | +/- tinnitus
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Otosclerosis - Mx
Hearing aid | Stapedectomy
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Middle ear neoplasms
1. SCCs = malignant - Bloody otorrhoea & deep pain - May cause FN palsy 2. Glomus tumours (paraganglionic cells) = slow-growing and benign: - Pulsatile tinnitus & CHL - Pulsatile red mass behind eardrum - May cause FN palsy or CN IX/XII paralysis
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Process of hearing
Sound waves vibrate tympanic membrane => transmits to ossicles Ossicles amplify & transmit to oval window Pressure waves through perilymph vibrate tectorial membrane Hair cells are moved against organ of corti and stimulate cochlear nerve Signals carried to cortex
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What is tinnitus?
= the perception of noise with no external stimuli Due to incorrect information reaching the brain or incorrect processing in the brain
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Causes of subjective / intrinsic tinnitus
= only heard by the patient ``` Idiopathic Drugs Trauma Presbycusis Labyrinthitis Meniere’s Vestibular schwannoma Otosclerosis ```
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Causes of objective/extrinsic tinnitus
= heard by others as well Palatal myoclonus Insect in EAM Vascular
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Vascular tinnitus
= PULSATILE AVM / glomus jugular tumour Needs CT/MRI
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Tinnitus - Mx
NO CURE => control symptoms 1. Explain - Incorrect information reaching the brain OR - Incorrect processing in the brain 2. Masking: - Radio/TV in background - "Tinnitus maskers” – play noise into other ear 3. Counselling: - CBT, mindfulness - Tinnitus therapy – techniques to avoid stress response - Support groups 4. Heading aids – if associated with SNHL
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Unilateral SNHL - causes
``` Acoustic neuroma Trauma Vascular insult Post-labyrinthitis Otosclerosis Congenital ```
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Bilateral SNHL - causes
``` Presbycusis Noise induced Metabolic Otosclerosis Congenital ```
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Sudden onset SNHL - causes and Mx
Emergency => ENT Referral ``` Causes: • Meniere’s • Viral infection • Ototoxic drugs • Temporal bone fracture • Tumour (exclude acoustic neuroma with CT/MRI) ``` Management: • ENT Referral • PO Steroids ASAP! (prednisolone)
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When is the prognosis of SNHL worse?
Prognosis = worse if there is also severe vertigo
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Acoustic Neuroma - Sx
Tumour affects the IAM (containing CN VII and CN VIII) and if big then also the base of skull Sx: - Vertigo = Progressive and constant - Facial palsies - Headache - Ataxia - SNHL, Tinnitus If tumour is large – can get trigeminal paraesthesia
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Acoustic Neuroma - Ix
Must investigate for this with all unilateral SNHL * Pure tone audiometry * CT/MRI
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Acoustic Neuroma - Mx
5% regress and 80% won’t grow – “watch and wait” - Symptom management ~15% will grow - Radiotherapy to prevent further growth - Surgical excision (might end up with dead ear/no vestibular function/ facial nerve damage)
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Noise-induced hearing loss - Cause and features
Caused by chronic loud noise exposure (initially reversible, but eventually permanent) Features: • SYMMETRICAL SNHL and tinnitus • Dip at 4kHx on audiogram
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Noise-induced hearing loss - Management
Prevention is key Hearing aid
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What is non-organic hearing loss?
= feigned loss of hearing to get compensation.
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What is presbycusis?
= SNHL due to aging (>50 years), due to loss of outer hair cells of cochlea.
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Presbycusis - features
Bilateral high frequency SNHL +/- tinnitus Worse if background noise
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Presbycusis - Ix
Hx and otoscopy | PTA / tympanogram
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Presbycusis - Mx
Reassure – stress that low/mid frequency hearing is good, and decline is gradual Hearing aid Hearing tactics
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What are "hearing tactics"
Facing speaker, Decrease background noise, Be open; tell others you have difficulty hearing.
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Peripheral causes of vertigo
= ears, eyes, somatosensors ``` Labyrinthitis / Vestibular neuronitis Vestibular Migraine BPPV Meniere’s Ototoxic drugs ```
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Non-vertigo causes of dizziness
Postural hypotension / vasovagal Arrhythmias Presbystasis (age-related dysfunction of the vestibular system)
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What is labyrinthitis? | What are the symptoms?
Vertigo caused by inflammation of the VESTIBULOCOCHLEAR SYSTEM Caused by URTI/AOM Symptoms: - Persistent vertigo (sudden onset, lasts days to weeks) - N&V - Nystagmus - +/- SNHL
298
Labyrinthitis - Ix and Mx
Ix: - ENT exam - Pure Tone Audiometry Mx: = SUPPORTIVE => (Vestibular sedatives = SHORT TERM ONLY), Antiemetics, Bed rest => Vestibular rehabilitation – take away all vestibular sedatives so the system can recalibrate.
299
What is vestibular neuronitis?
Inflammation of VESTIBULAR NERVE, caused by viral infection. Symptoms: - Persistent vertigo (sudden onset, lasts days to weeks) - N&V - Nystagmus - No ear Sx
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Vestibular neuronitis - Ix and Mx
Investigations: - ENT exam - PTA Management = supportive: - (Vestibular sedatives), Antiemetics, Bed rest - Vestibular rehabilitation – take away all vestibular sedatives so the system can recalibrate.
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What is benign paroxysmal positional vertigo (BPPV)?
Vertigo caused by displaced semi-circular calculi Occur either spontaneously or caused by head injury. Vertigo is sudden and episodic (if head moved) Lasts seconds – minutes Symptoms: - Positional vertigo - Rotational Nystagmus - NO EAR Sx - +/- N&V
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BPPV - Ix and Mx
Investigations: - ENT Exam - PTA - Dix-Hallpike Manoeuvre Management: - Eply Manouvre - Exercises to do at home - Reassure – resolves in 12-18 months
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Dix-Hallpike Manoeuvre
* Performed by rapidly moving the patient from a sitting position to the supine position with the head turned 45° to the right. * After waiting approximately 20-30 seconds, the patient is returned to the sitting position. * The procedure is then repeated on the left side. * If the test is positive, the patient will complain of VERTIGO and you should be able to directly observe NYSTAGMUS
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What is Meniere’s Disease? What are the key symptoms?
Occurs due to excess endolymph, cause is unknown. Sudden onset and recurrent Lasts 30-40 mins ``` Triad of Symptoms (occurring at the same time as vertigo): 1. Vertigo 2. Tinnitus 3. Low frequency SNHL (+/- aural fullness, +/- N&V) ```
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Meniere’s Disease - Ix
Investigations: - ENT exam - PTA - Romberg Test +ve (during attacks) - + CT/MRI to r/o neuroma
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What is required for all facial palsies?
All facial palsies need a thorough ENT & neuro examination.
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Facial palsy with sparing of frontalis
= UMN problem
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Entire facial palsy
= LMN problem
309
Facial Palsy - Ix
Hx, ENT exam, neuro exam PTA Electroneuronography (= electrical stimulation of FN) MRI/CT – if suspicious case
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Facial Palsy Mx - eye care
Artificial tears, | Eye patch at night
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Bell's Palsy
Most common facial palsy. Caused by viral infection of FN Symptoms: - Sudden onset (hours) - Ipsilateral facial palsy (incl. frontalis) - +/- pain - No ear/CNS pathology
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What conditions cause an increased risk for Bell's palsy?
Increased risk in diabetes and pregnancy
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Bell's palsy - Mx
80% fully recover in 2 months Mx: - High dose PO steroids - Eye care + analgesia
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Ramsay Hunt Syndrome
Caused by HSV infection of facial nerve. Symptoms: - Ipsilateral facial palsy (incl. frontalis) - Ear pain - Vesicular rash in/on ear - +/- SNHL, vertigo, tinnitus
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Ramsay Hunt Syndrome - Mx
Palsy = irreversible PO Acyclovir +/- corticosteroids (unless systemically unwell, then IV acyclovir) Eye care + analgesia
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Causes of facial palsy in children
Congenital Forceps delivery Chickenpox Acute OM
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Red flags of facial palsy
Associated ear infection / foul otorrhoea => cholesteatoma Progressive palsy / parotid mass => neoplasm Associated neuro symptoms => CVA
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Nasal Polyps
= grey/white, soft & mobile pedunculated swelling in nose/sinuses Symptoms: - Nasal obstruction - Anosmia - Rhinorrhoea
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What should be considered with unilateral or bleeding nasal polyps?
RED FLAG => needs ENT referral
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Nasal Polyps - associations
Cystic fibrosis Infective sinusitis Samter’s triad – polyp + asthma + aspirin sensitivity
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Nasal Polyps - Ix
Anterior rhinoscopy – biopsy if suspicious
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Nasal Polyps - Mx
Medical – antihistamines, steroid drops/spray, decongestants Surgical – polypectomy (if significant blockage/red flag features)
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Nasopharyngeal Carcinoma - RFs and Sx
= SCC RFs – southern Chinese origin, EBV Symptoms: - Cervical lymphadenopathy - Unilateral otalgia - Unilateral OME - Nasal obstruction - CN palsies
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Nasopharyngeal Carcinoma - Mx
CT & MRI Radiotherapy Surgery
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What can tenderness on palpation of tragus / pinna indicate?
often tender in otitis externa
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Things to cover in an ear history of presenting complaint
Hearing loss – when, progression, side Otalgia / Pain – side, nature of pain Otorrhoea / Discharge – nature (e.g. foul smelling, blood, CSF), side, duration Tinnitus – pulsatile or not, severity, sleep, side Vertigo / Dizziness – what they mean, duration, associated Sx Facial Nerve Sx
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Potential presenting nose complaints What are red flags?
- Blockage - Discharge - Change in smell - Facial Pain - Bleeding Red flags – numb face, neck lump, unilateral Sx, proptosis, diplopia, eye displacement.
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How is an examination of the nose performed?
Inspection of outer nose => Front, side, top, bottom Palpation Anterior rhinoscopy => Use finger to lift up tip of nose Nasal airway patency => Use a metal speculum to occlude one nostril gently from underneath.
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How is an examination of the oral cavity performed?
``` General Inspection => Swelling on face   Inspection of mouth - Lips - Gums and Teeth - Tongue – dorsum, sides, underneath - Buccal mucosa - Parotid duct ``` Inspect palate and uvula - Use tongue depressor to gently depress the tongue - Candidiasis/papilloma/ulceration - Deviation of uvula Inspect tonsils, pharyngeal arches - Use tongue depressor to gently depress the tongue Inspect floor of mouth: - Assess for abnormalities of submandibular gland duct - Assess for ulceration Palpation: - Palpate any identified lumps - Palpate lateral walls for parotid gland and duct - Palpate floor for submandibular gland and duct
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How is an examination of the neck performed?
General inspection Inspection: - Lumps, Asymmetry, Scars, Skin changes - Distended neck veins Palpate: - Lumps - Trachea - Carotid pulse (one side at a time) Palpate Lymph Nodes systematically (anterior and posterior triangle)
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If a neck lump is found, what should be done.
Inspection - - ask the patient to swallow (+/- sip of water). - Any lump attached to the pre-tracheal fascia will move upwards on swallowing – i.e. a thyroid lump or thyroglossal cyst Palpation => Ix = USS
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What are some ototoxic drugs?
Aminoglycosides (e.g. Gentamicin), Furosemide, Aspirin A number of cytotoxic agents
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Causes of epistaxis
Idiopathic or Nose-picking = most common Trauma Infection Tumours
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Features of posterior epistaxis
- Profuse - Bilateral - Failed anterior packing
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Epistaxis - predisposing factors
- HTN - Anticoagulants, NSAIDs, aspirin - Coagulopathies - Hereditary Haemorrhagic Telangiectasia
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What should be considered with unilateral Epistaxis in adolescent boys?
consider juvenile angiofibroma (a nasopharyngeal tumour) => Needs CT and excision
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Epistaxis - Mx
1. First aid 2. Resuscitation (if severe) 3. Cauterisation (if anterior bleed) => Using silver nitrate or bipolar diathermy 4. Packing (anterior = 1st line, posterior if anterior fails) 5. Sphenopalatine artery ligation 6. Surgery – if cannot stop bleed
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Epistaxis first aid
* Lean forward, pinch fleshy part – for 10 mins * Apply ice to bridge of nose * Avoid swallowing blood
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Epistaxis - resuscitation
* Estimate blood loss, measure pulse/BP * FBC, coag screen, G&S * IV fluids if needed
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Epistaxis - packing
Done if cannot visualise or cauterise bleed NEED PROPHYLACTIC ABX * First line – anterior packing * Second line – posterior packing
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Management of nose fracture
1. Manage Epistaxis / acute problems 2. MUST Rule out serious complications 3. Clinic 5-7 days later => Assess bony nose injury once swelling has subsided 4. Manipulation of bony deformity (within 14 days of injury)
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Nose fracture - serious complications to rule out
* Zygomatic/facial fracture – diplopia, facial numbness, trismus * Head injury – LOC, N&V, amnesia, pupils * CSF leak – unilateral, clear nasal discharge * Obstructed airways * Chest/abdo injuries * Septal haematoma
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what is the timeframe for manipulating the bony deformity of a nose fracture?
Must be done within 14 days of injury
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Septal haematoma
= a bleed between the septum and perichondrium Appears as bilateral red/purple bulge
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Septal haematoma - complications
* Blocks nose and gets infected | * Necrosis and septal perforation
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Septal haematoma - management
Immediate ENT referral Surgical drainage & IV ABX
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Septal perforation - causes
Usually Trauma/surgery Also: • Avascular necrosis (septal haematoma/cocaine use) • Granulomatous infection (syphilis, TB, Wegener’s)
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Septal perforation - symptoms
* Sense of nasal obstruction * Whistling * Crusting / bleeding
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Septal perforation - management
* Douching and Vaseline | * Surgery
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When should a FB in the nose be considered? How is this managed?
Suspect in kids if unilateral offensive discharge (+/- epistaxis) Management = removal with forceps/Johnson probe / suction
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Acute rhinosinusitis
< 4 weeks
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Subacute rhinosinusitis
4 - 12 weeks
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Chronic rhinosinusitis
> 12 weeks
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What is rhinosinusitis?
= inflammation of the nasal and sinus mucosa, causing URTI Sx for >10 days.
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Risk factors for rhinosinusitis
* Polyps * Deviated septum * Dental infection * Smoking
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Rhinosinusitis - pathophysiology
Viral URTI causes hyperaemia & oedema of mucosa and increased secretions Stagnant secretions become infected by bacteria => H. influenzae, Strep. pneumoniae
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Rhinosinusitis - Sx
Mucopurulent rhinorrhoea Nasal obstruction/congestion Reduced smell/taste Facial pain – over infected sinus, worse when bending forwards Malaise/pyrexia
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Rhinosinusitis - Ix
Anterior rhinoscopy – inflamed mucosa | Flexible Nasal Endoscopy – mucous in oropharynx
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Rhinosinusitis - Mx
Conservative: => Simple analgesia, Steam inhalations, Nasal decongestants Medical: • Steroid nasal spray (e.g. beconase) • Amoxicillin - only if severe pain/high fever/persistent Sx Surgical (ENT referral): • Maxillary sinus washout - only if progressive pain / complications
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Rhinosinusitis - complications
Chronic sinusitis Mucocele Osteomyelitis Intracranial problems (need CT) – meningitis, brain abscess Facial cellulitis Periorbital cellulitis
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Facial Cellulitis - Sx, sources, Mx
Infection spreads to the skin of the face Symptoms => Red, warm, painful skin Sources: - Orbital cellulitis - Sinusitis - Osteomyelitis Management = high dose ABX + sinus drainage
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What is mucocele?
A collection of sterile mucous in an obstructed sinus Over years, increasing pressure causes sinus expansion
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Mucocele - Sx and Mx
Symptoms: - Eye displacement - Visual problems - Facial swelling Management: - Surgical sinus drainage
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Periorbital cellulitis following sinus infection
Infection spreads into orbit (usually ethmoid sinus through ethmoid bone). Symptoms: - Unilateral eyelid swelling, pain, redness - Blurred vision - Fever, headaches, meningism, sepsis
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Periorbital cellulitis - Mx
Urgent ENT referral and CT High dose IV ABX Nasal decongestant Careful eye obs (for signs of abscess pressing on optic n.)
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Chronic rhinosinusitis - pathophysiology
INFECTION – viral / bacterial (anaerobes, staph. aureus, gram -ve) ALLERGENS – dust mites, pollen, animal hair - Specifically known as allergic rhinitis - Sneezing, itchy eyes, rhinorrhoea
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Chronic rhinosinusitis - Sx
``` Nasal obstruction / congestion POST-NASAL DRIP – worse at night, morning cough to clear Reduced smell/taste or unpleasant smell Intermittent facial pain Crusting / bleeding ```
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Chronic rhinosinusitis - Ix
Diagnosis based on Hx Anterior rhinoscopy AND FNE / endoscopy
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Chronic rhinosinusitis - Mx
Infection: - Broad spectrum PO ABX (3+ weeks) - Topical nasal steroids (2 months) - Steroid nasal spray (after finishing drops) - Nasal douching Allergic: - Avoid allergens - Antihistamines - PO steroids If no improvement in 8 weeks - ENT referral - Confirm Dx and CT & surgery to clear drainage pathway
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How is the best way to apply nasal steroid drops (e.g. betamethasone drops)?
applied with the head upside down over the edge of a bed.
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Nasal douching
½ tsp salt, ½ tsp sugar, ½ tsp bicarb dissolved in boiling water Draw up some with a syringe Block one nostril with finger and sniff up mix with other nostril Let it run out after Important to do this BEFORE any nasal spray/drops (not after)
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What is vertigo?
= abnormal sensation of movement with ROTATIONAL component / “room spinning” Usually with nausea and vomiting. Can be persistent or episodic
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Central causes of vertigo
Involve brainstem ``` Space-occupying lesion Head injury Alcohol/drugs Degenerative disease (e.g. MS) Vascular ischaemia ```
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what is an unlikely cause of vertigo with LOC / collapse ?
Unlikely to be a peripheral cause of vertigo (more likely to be central)
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What are vestibular sedatives?
"Anti-dizziness" medications prochlorperazine, cinnarizine, cyclizine, or promethazine Given as a SHORT course - prolonged use may delay recovery
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What is the difference between labyrinthitis and vestibular neuritis?
Both follow viral infection, BUT: Vestibular neuronitis = inflammation of vestibular nerve (vertigo with no loss of hearing) Labyrinthitis = inflammation of entire inner ear (vertigo with hearing often affected)
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Meniere's Disease - Mx
ENT Referral Prevention = DIET – low salt/caffeine Symptomatic Tx = Vestibular sedatives “anti-dizzy”, Antiemetics MEDICAL - Betahistine (1st line) - Chemical labrinthectomy – intratympanic steroids, intratympanic gentamycin (Surgical – surgical labyrinthectomy)
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Reassurance and advice for Meniere's disease
Advise that an acute attack of vertigo will normally settle within 24 hours in most people Advise the person not to drive when they are feeling dizzy Discuss reliable sources of information
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Vestibular migraine - Sx
- Vertigo (tends to last minutes to hours) - Possibly tinnitus - Photophobia / Phonophobia / aura associated with vertigo is almost diagnostic. - Sympathetic Sx – sweating, flushing
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Vestibular migraine - Mx
``` Diet = 1st line – avoid 5C’s • Caffeine • Cheese • Chocolate • Claret (wine / alcohol) • Chinese food ``` Lifestyle – avoid too much/too little sleep and too much/too little fluids (Medical – low dose antidepressants)
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What is an acoustic neuroma?
= vestibular schwannoma Compression of vestibular nerve due to benign tumour of the schwann cells of vestibular nerve.
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Assessment of hearing
Hx Otoscopy Audiometric tests
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Conductive hearing loss
= problem with the outer/middle ear Bone conduction normal, reduced air conduction
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Sensorineural hearing loss
= problem with the inner ear/ auditory nerve/ brain Reduced air AND bone conduction
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Mixed hearing loss
= CHL and SNHL Decreased air conduction will be greater than decreased bone conduction
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Limits with tuning fork tests
Just used for SCREENING Cannot be performed on patient’s whose loss is too severe to be able to hear the tuning forks (512-Hz or 1024Hz )
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Weber's Test
1. Strike the tuning fork and place it on the middle of the forehead. 2. Note where the sound is best heard – the left ear, the right ear, or both equally.
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Rinne's Test
1. Strike a tuning fork and place it 25mm from entrance to ear canal for 2-3 seconds 2. Without delay, press the base of the tuning fork against the mastoid process for 2-3 seconds. 3. Ask the patient which they heard louder (2-3 second timing is important in order for the sound to not disappear and alter the results of the test.)
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Normal Weber's and Rinne's
Weber’s - Central / no lateralisation Rinne’s - Positive – AC > BC
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Rinne's test Positive
when AC > BC Normal hearing or SNHL
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SNHL - Weber's and Rinne's
Webers = Lateralises to the side with the better cochlea (i.e. opposite side to loss) Rinnes = Positive – AC > BC
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Why does weber's test lateralise to the opposite side of SNHL?
Cochlear damage = no sound detection on that side therefore sound lateralises to the better cochlea
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CHL - Weber's and Rinne's
Weber's = lateralises to the side with the greater conductive loss Rinne's = Negative – BC > AC
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Rinne's test Negative
when BC > AC Conductive hearing loss
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Why does weber's test lateralise to the same side of CHL?
Distracting external sounds not heard, so sound from fork seems louder on that side
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What is cross hearing?
When sound is applied to one ear, the opposite cochlea can be stimulated to varying degrees. This occurs either by escaping sound travelling through air to the opposite ear, or via vibrations through the bone of the skull. => CAN GIVE A FALSE RINNE NEGATIVE => CAN AFFECT PTA RESULT e.g. if the patient has a normal ear and a dead ear, audiological tests would result in a better threshold in the dead ear due to cross hearing.
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FALSE rinne Negative
the tone may appear louder by bone due to cross hearing from the better ear
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What is masking in audiology?
The process of artificially raising the hearing threshold of the non-test (better) ear, to get a more accurate result of the test ear's hearing.
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Pure Tone Audiometry
Use electrical equipment to control frequency and intensity of sound to quantify hearing loss. Tests the range of speech frequency Used for: 1. Diagnosis 2. Rehabilitation 3. Monitoring hearing
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When is monitoring of hearing required?
a. Patients working in high noise environments (annual screening) b. Patients on ototoxic drugs c. Pre- and post-surgery (e.g. grommets)
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What ranges are tested in PTA?
Hearing is tested over the range of speech frequency – 250 Hz to 8000 Hz. Bone conduction is tested over 500 to 4000Hz.
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decibels normal hearing level
Normal hearing = 0 dB nHL This means that the patient is able to hear the sound at an intensity that is 0 dB louder than a normal hearing person would be able to hear – i.e. it is the same as a normal hearing person. 35 dB nHL means that the patient is able to hear the sound at an intensity that is 35 dB LOUDER than a normal hearing person would be able to hear
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Audiogram symbols - Air conduction
Right ear = O (red) Left ear = X (blue)
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Audiogram symbols - Bone conduction (not masked)
Right ear = Triangle (red) Left ear = Triangle (blue)
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Audiogram symbols - Bone conduction (masked)
Right ear = [ open bracket (red) Left ear = ] close bracket (blue)
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Audiogram symbols - Masking applied to air conduction, but no change in normal threshold
Right ear = half-coloured circle (red) Left ear = half-coloured X (blue)
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Audiogram symbols - Shadow response to masking
Right ear = coloured circle (red) Left ear = coloured X (blue)
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WHEN is masking applied to audiogram?
Used to prevent cross-hearing Applied when: 1. AC – 40dB or greater air to air difference (right vs. left) 2. BC – 10dB or greater air to bone difference in the same ear (AC vs. BC)
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Audiogram - normal hearing
Hearing level within normal threshold (-10 to 20)
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Audiogram - SNHL
AC decreased and BC decreased | Bone/air gap <5-10 dB
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Audiogram - CHL
AC decreased, BC within normal range (Bone/air gap >15 dB)
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Audiogram - mixed HL
AC and BC both lower than normal range, but decrease is greater for AC (Bone/air Gap >15 dB)
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Tympanometry
= a test of middle ear function. Measures sound compliance into the middle ear Measured over a range of pressures (from negative to positive) Information is plotted on a tympanogram.
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Tympanometry - Normal range for Ear Canal Volume
0.6 – 2.5 mL
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Tympanometry - Normal range for Middle Ear Pressure
+50 to -100 daPa
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Tympanometry - Normal range for Compliance
0.3 – 1.6 mL
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Tympanometry - TYPE A
Peak at atmospheric pressure (0daPa) | Normal compliance
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Tympanometry - TYPE Ad
= Like Type A with taller peak Increased Compliance: - Healed TM perforation - Retraction pocket - Ossicle disarticulation
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Tympanometry - TYPE As
= Like Type A with smaller peak Decreased Compliance: - TM scarring - Fluid in middle ear
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Tympanometry - TYPE B
= Flat line, no peak No Peak Compliance - Middle ear effusion / tumour - TM perforation - Grommet
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Tympanometry - TYPE C
Type C Peak in negative pressures - Peak compliance at low frequency - Eustachian tube dysfunction
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Paediatric audiometry - timings
Newborn Hearing Screening Programme (NHSP) => within 5 weeks of birth (ideally before discharge) Behavioural Observation Audiometry (BOA) => 0 – 6 months Visual Reinforcement Audiometry (VRA) => 6 months – 3 years Performance Test / Conditioned Play Audiometry (CPA) => 30+ months Conventional Pure-tone Audiometry => 5+ years
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Newborn Hearing Screening Programme
Offered to all babies within 5 weeks of birth 2 tests: 1. Automated Otoacoustic emission (AOAE): - Tests function of outer hair cells. - If child fails 2 of these, then AABR is performed. 2. Automated Auditory Brainstem Response (AABR) - Uses electrodes to monitor brain activity response to sound stimulus. - Can be done at any age, but will often need to be sedated after 6 months.
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Behavioural Observation Audiometry
Age 0 – 6 months Observe the child in a normal (quiet) state and also during presentation of loud sound to see if there is any change in behaviour (e.g. startle, eye movement/widening, head turn, etc.) Does not assess laterality.
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Visual Reinforcement Audiometry
Age 6 months – 3 years. Observation of conditioned response to sound stimulus (sound field or insert) Child is conditioned to turn when sound is heard, during conditioning the child is provided with a visual reward (toy).
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Performance Test / Conditioned Play Audiometry
From age 30+ months. Performance test – child presented stimulus in sound field, plays a game with dropping men in a boat. CPA = continuation from performance test, but with headphones to obtain more specific results for each ear.
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What are the branches of the facial nerve?
" two zoologists butchered my cat" ``` Temporal Zygomatic Buccal Marginal Mandibular Cervical ```
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What can cause gingival hyperplasia?
Drugs: - - phenytoin - - ciclosporin - - calcium channel blockers (especially nifedipine) Other causes: -- Acute myeloid leukaemia
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What is the most common bacterial cause of otitis media?
H. influenzae