ENT Flashcards

(78 cards)

1
Q

What do you look for in otoscopic exam?

A
  1. color
  2. other conditions
  3. mobility
  4. position
  5. Lighting
  6. entire surface
  7. translucency
  8. external auditory canal
  9. seal
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2
Q

Acute Otitis Media (without effusion)

A

Preschool children

Risks: pacifier use, smokers, daycare, not breast fed

S. pnemoniae, h. influenzae, m. catarrahalis, viruses

RETRACTED OR BULDGING TM, IMPARIED MOBILITY, FEVER, EAR PAIN, EAR PRESSURE, can be errythmateous, doens’t have to be, Triad: buldging, redness, and lack of motility, TYPICALLY COMES FROM URI THAT LEADS TO EUSTACHIAN TUBE DYSFUNCTION OR BLOCKAGE.

WATCHFUL WAITING WHEN WITHOUT SEVERE PAIN AND FEVER!! WAIT 3 DAYS WITH PAIN RELIEFAND SEE IF IT GETS BETTER UNLESS FEVER IS PRESENT THEN ANITBIOTIC, have them call back if symptoms get worse

COMPLICATIONS: MASTOIDITIS, Tm rupture

DOC: AMOXICILLIN, IN RESISTANCE USE AMOXICILLIN CLAVULANATE.

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3
Q

Acute Ottis Media (with effusion); what color is the membrane? is this an infection? what direction is the membrane going? what is the most important test you can do to confirm diagnosis?

A

LOOK FOR AIR FLUID LINE EFFUSION W/O SIGNS OF SYMPTOMS OF INFECTION

common in children, CAN EFFECT SPEECH

risk facors: AFTER AOM, eustachian tube dysfunction, smokers, alleries, pacifires

generally not as many signs of illness, hearing loss may be the only sympstom, CONDUCTIVE, STRANGE FORMULATION OF SPEECH, sleep disturbance, vertigo, may indicate tubes

TM: pearly gray, neutral or RETRACTED, AIR FLUID LEVELS, PNEUMATIC OTOSCOPY MOST IMPORTANT BECAUSE YOU WONT’ SEE THE MEMBRANE MOVE

NO antibtiocs!! watch and wait since not infected

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4
Q

What do these show?

A
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5
Q

Chronic Otitis Media; what is it associated with? what bacteria cause this? what do they need to avoid? what abx?

A
  • occurs from repeated episodes of acute otitis, trauma, or cholesteatoma. Associated with low socioeconomic status, smoking.
  • Different causitive organisms than AOM

p. aeruginosa, s. aureus, proteus

-perforated tympanic membrane for >2 weeks and chronic ear discharge (ottorhea), with or without pain, can cause conductive hearing loss

needs debriedment, avoidance of water exposure, topical antibiotic drops

***difference between AOM and COM is the time frame the TM is ruptured for, >2 weeks then chronic***

DOC: aural toilet irrigation with WARM water, prevents against vertigo. and vinegar rinse to dry it up. Topical antibiotic Cipro, oflaxacin

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6
Q

what do you not use aminoglycosides for topical ear infections?

aka Gentamycin and tobramycin

A

They are ototoxic!!

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7
Q

tympanometry

A

measuring the movement of the tympanic membrane

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8
Q

Cholesteatoma; what important things can this lead to? who is this common in? who is more likely to get it?

A

pocket of squamous epithelium that becomes hyperactive and makes tumors that go into middle and inner ear

-usually in children under 5 with cleft palate, Downs, facial abnormalties

ETreatment: excision

This is serious, don’t miss it, can lead to meningitis, cranial nerve palsies, hearing loss

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9
Q

Mastoiditis

A

can occur due to inadequate treatment of prolonged or chronic ottitis media when infection spread from middle ear in mastoid air cells

  • spiking fever and post auricular pain, fluctulant painful mass, swollen mastoid,
  • CT needed to diagnose
  • history is the biggest clue
  • DOC: IV cephalosporin, emergent EMT consult
  • can lead to meningitis, intercranial pressure
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10
Q

Perforated Tympanic Membrane; what are three things that can cause it? what should patients avoid? How does it heal? most important thing is?

A

trauma: foreign bodies, barotrauma, headtrauma

loud noises, AOM

-educate patient to avoid water in ear that will promote infection

  • don’t do peumatomotry because there is a hole, DUH.
  • Refer to audiology if hearing loss or vertigo

-usually will heal on its own, best treatment is PREVENTION, use antibiotic if infected

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11
Q

Otitis Externa; what is this known as? where do you see the pain? coloration? what happens in diabetics or immunocomprimised & where does it infect? what type of bacteria?

A

“swimmers ear” common exsposed to water, trauma where kids itch ears or cleaning, PSORIASIS OR ECZEMA

p. aerguginosa, s. aureus, fungal

  • patients complain of pain especially with movement of the auricle and tragus, black/cauliflower look in fungal infection
  • redness and swelling of the ear canal, PURLUENT EXUDATE

doc: antibiotic otic drops aminoglycosides (ok since membrane intact) or fluoroquinolones +- corticosteroids and avoid further moisture. CORTOSPORIN, use with a wick to get the drugs in!! may want to use benzocain with it to numb it up.

in diabetics and immunocompromised: malignant otis externa may develope; a necrotizing infection TEMPORAL BONE, requires HOSPITALIZATION AND PARENTRAL ANTIBIOTICS,

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12
Q

presbycusis; what goes first? how do you treat?

A

hearing loss with age, high pitches go first

Tx: hearing aids, cochlear implants, cerumin impactions

semisoneural hearing loss

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13
Q

cerumen impaction

A

increased risk with cotton swab use or hearing aids

only becomes an issue when impacted, secretions and epithelia cells

-fullness and hearing loss

irrigation, cerumlytics, manual removal, warm flushes, colace drops, use WARM water to prevent against vertigo when flushing

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14
Q

Tinnitus

Name the four types! BEES buzz

A

ringing in the ears

bruits: whoosh whoosh noise, work up

Endogenous, maskable tinnitus: feel better in noisy environment, ambient noise maker

exogenous tinnitus: feel better in a silent environment, hearing aids to filter

slow brainstem tinnitus: feel mentally unstable, non-focalized “noise”, dazed and disortiented

exo and endo are hard to treat but can use benzos in extreme cases or use tinitis retrain to respond differently to stimuli

NO GOLD STANDARD FOR TREATMENT

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15
Q

benign paroxysmal postional vertigo; what testing should you have them do? what can make it better? constant or comes and goes? what does that patient feel like?

A

vertigo when in certain positions, DIX HALLPIKE POSITION determines vertigo

“the room is spinning”, do orthostatics and EKG, look for nystagmus in vertigo patients, then do dix hallpike position to ellicit nystagmus in vertigo, dizziness comes and goes it is a vestibular issue rather than constant which is CNS. HEARING LOSS WITH PERIPHREAL TYPES.

-calcium in the labyrinth, TX by vestibular rehab, these are what get stuck on the hair cells and tell you that you’re moving when you’re not.

VERTIGO IS A SYMPTOM NOT A DIAGNOSIS

Tx: antihistamine (meclozine), but can make eldery confused, or the manuever

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16
Q

DIX hallpike manuever

A

used to determine vertigo BPPV

Have the patient tilt head 45 degrees and look for nystagmus, then have them lay down and look for nystagmus again for 30 seconds, once stopped, have then sit up and check again, repeat on the opposit side! BPPV confirmed and should fix the problem. Nystagmus will go away with position changes.

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17
Q

Vestibular neuritis AKA Labrynthitis

A

-gait imbalance, dizziness, hx of falling, tinnitus usually present, nausea

need to rule out cranial bleed/infarct, ACUTE SEVERE VERTIGO WITH HEARING LOSS AND VERTIGO FOR SEVERAL DAYS TO A WEEK

selflimiting, focus on treating the nausea, meclinazine/zofran

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18
Q

Menieres disease

A

most common 20-40 year olds

-endolymph disturbance- ion inbalance in inner ear

TO DIAGNOSE: NEED 2 SPONTANEOUS EPISODES OF VERTIGO FOR >20 MINS and audiometry to confirm sensioneural hearing loss, tinnitus, aural fullness

symptomatic, lifestyle changes decreasing salt intake, caffeine, alcohol, meclizine and benzo’s

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19
Q

Acoustic Neuroma; what are the two risk factors, what will be a sign on the eye? AGE?

A

A schwann cell derived tumor from vestibular nerve

found around age 50

risk factors: neurofibromatosis is a risk factor and so is radiation exsposure

EFFECT CN8

unilateral hearing loss, continous vertigo, MRI diagnosis, ipsilateral corneal reflex will be diminished

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20
Q

Types and characteristics of periphreal vertigo, what direction is the nystagmus?

A

labyrinthitis, benign paroxysmal postitional vertigo, Menieres, and vestibular neruitis

Periphreal is associated with suddent onset, nausea, vomiting, tinnitus, hearingloss and nystagmus (TYPICALLY HORSIZONTAL NYSTAGMUS)

if the dix-hallpike maneuver helps with benign parozymal positional vertigo, then it likely isn’t central vertigo, if this doens’t help then may be more serious

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21
Q

Central Vertigo; whats the onset like? what disease is associated with this that would be important?? nystagmus direction?

A

associated with gradual onset and vertical nystagmus NO AUDITORY SYMPTOMS

associated with brainstem vascular disease, arteriovenous malformations, tumors, MS

MRI suggested if CNS involvement is suspected

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22
Q

otosclerosis

A

abnormal bone growth at the oval window

causes: conductive hearing loss

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23
Q

What is the most common cause of conductive hearing loss?

A

cerumen impaction

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24
Q

Dental caries; what causes them? what prevents them in the mouth? whats the #1 prevention? Caution when a pt presents with this because?

A

bacteria form plaques on the teeth, erode the enamel and get into the tooth

Risk: not enough saliva since these contain enzymes, can be genetic or salivary glands

black and white plaques, degraded teeth, craters in the teeth

Tx: treat for pain, be careful of narcotic seekers, refer to a DENTIST and emphasize prevention. fluoride is the best prevention.

complications: can invade into pulp and gingiva, alveolar bone, and orofacial spaces and create absesses, invade the maxillary bone

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25
Dental abscess
- bacteria from plaque gets in deeper - swelling in the gums, sometimes facial pain - fluctulant bulging gums and PAIN, don't have to see the buldge because it can be farther up and give **facial pain,** can *incision and drain* with 18 gage needle an, can numb with dental blocks with bruprivicane or **oil of clove** DOC: Penicillin or Clinda in allergies _ALWAYS PRESCRIBE SO INFECTION DOESN'T SPREAD_
26
Gingivitis; what bacteria causes it? what are interesting findings? what can it cause?
**\*most common periodontal disease in children\*** _Prevotella intermedia gram - anaerobes_ subgingival plaques are more common in pregnancy, drugs, and poor nuitrition **blue/purple discoloration of gums**, bleeding, bad breath, can cause **TRENCH MOUTH** DOC: penicillin or Clinda or augmentin, chorahexidine rinses
27
periodontits; what is it a progression of? what is diagnostic for it? what are two things you could protenially loose? how long does it take? who is it in?
- untreated gingivitis, common in children - **untreated gingivits, loss of alveolar bone and periodonal ligament, weeks to years progression, cauess loose or missing teeth** **_6mm depth is diagnostic_** early,= debrievment, surgery, and prevention is the biggest part
28
dental trauma; who is it common in? what do you want to check for (2)? important key thing to do when holding tooth?
common in children -fractures or **luxation: displaced but not falling out**, want to know if primary or secondary tooth, want to preserve peridontal lig, _hold by crown always_, often will loose teeth RF: those with imparied mobility **check maxillary fracture: bite down on TD and twist**
29
oral candidiasis
-**dentures, diabetes, immunocomprimised, children, corticosteroids, antibiotics** creamy white patches that can be scraped off to show erythematous mucosa, koh prep **pseudomembraneous (white) and atrophic (bright red in dentures)** DOC: nystatin or clotrimazole candies
30
angular chellitis; who is it found in? what two things cause it? what do you do to treat it?
- dentures/lip lickers - **candida albicans or staph impetigo**(school children) infection, need to determine if fungal or bacterial **red crusty lesions at the corner of the mouth** DOC **topical** FUNGAL: topical clotrimiazole, Bacterial: topical bactroban
31
Herpes Simplex mouth infection
**_vesicular lesion with erythematous base_** usually HSV1 or latten infection **prodromal: burning, numbness, tingling before lesions appear 24 hours prior to outbreak** **Tzank smear-**giant nucleated cells, Adults: acyclovir during prodromal phases! kids: magic mouth wash don't need to treat if only on the labia
32
apthous stomatitis; what are two interesting assocaited diseases?
chancre sore, children adolescent **round, grey lesions _WITHOUT ERYTHEMATOUS BASE_,** some genetic component and can be seen in patients with **Behcets disease: 3x a year, recurrent ulcers anywhere and vasculitis**, **IBS** difficulty clearing up on their own **herpes virus 6**
33
Coxsacki virus
**hand, foot, and mouth disease**, mostly kids Coxsackie virus painful blisters and ulcers on hands and feet, CONTAGIOUS, keep them out of school and isolate them
34
oral/throat cancer; who is it common in? which virus causes it? where should you always look?
- Males - **HPV16, 50 fold increase** RF: tobacco, alcohol uclers and lesions that just won't heal, dentures stop fitting, trouble swallowing, *localized papules in the back of the throat*, important to examine under the tongue **biopsy, CT, worry about metastisies**
35
Leukoplakia
**hyperplasia of squamous epithelium** benign but can turn cancerous, *trauma or _repetitive lip biting_*, tabacco, *stays on when you rub it unlike thrush* biopsy them, constant monitoring to make sure not cancerous
36
viral pharyngitis; what can cause it and how do you differentiate? who is it commonly found in?
children/adolescents **rhinovirus, influenza, parainfluenza, EBV** flu swab, monospot/post cervical lymph nodes, and splenomegaly for EBV need to differentiate what is causing it, tamiflu or symptomatic
37
bacterial pharyngitis; who is it in? what 3 interesting things can cause it? DOC?
kids mostly with daycare **strep, nisseria, chlamydia** **Centor swabbing criteria** strep: DOC is penicillin Strep Kids: DOC is amoxicillin or cephalosporins, if allergic them azithromax
38
what is the centors criteria for swabbing for strep and how does this influence treatment?
Centor Criteria to swab for strep: 1. tonsular exudate 2. tender cervical lymphnodes 3. fever by history 4. absence of cough **if 2-3 then just go ahead and swab, if 3-4 then just go ahead and treat without swabbing**
39
why is it so important to treat strep when there is a bacteria pharyngitis?
Rheumatic Fever(heart and joints)-strawberry tongue Strep TSS
40
xerostomia (dry mouth); what do you suspect? what do you treat with?
older adults dry mouth, cobblestone tongue, cracked lips -radiation, tobacco, chemo, chronic viral infection **sailometry: how much saliva, biopsy saliva for Sjogren's syndrome (decreased mucous production throughout)** TX: **pylocarpine (increases secretions)**
41
sialadenitis (parotitis); what can this help predict?
eldery **causes: mumps, staph, anaerobes** viral or bacterial infection that happens because of **salivary stasis, swelling, or obstruction of salivary gland, ID within 48 hours if not better!!** can be used as a indicator for _mumps prodromal phase**,** with involvement of the poratid gland_ (see bilaterally, and with trismus (lockjaw), if bacterial then it will be purlulent DX: **serum amylase, PCR MUMPS** can lead to meningitis, encephalitis, pancreatitis (amylase)
42
sialadentitis (sialthiasis)
common 30-60 y/o males **_stones_ in submandibular and parotid** pain especially with thinking about food because increases pressure **milk duct, _heat, lemon drops_, hopefully they can pass the STONE on their own** DOC if infection: naphacillin + clindamycin
43
peritonsilar abcess
assymetric pharynx, voice muffled. uvular displaced to uneffected side **trismus (lockjaw), _deviation of the uvula and asymmetric rise_, "hot potato" voice** aspiration and incision DOC: amoxicillin or clinda
44
breathing allows for what four things to happen?
1. intake- 80/20 relationship 2. warms air to 75-80% humiditiy and 36\* C, 1 degree less than body temp 3. cleanses inspired air 4. antiseptic-IgA prevent viruses from getting into epithelia cells
45
how much mucous does the nose produce a day?
1 liter, sweeps it away from the lungs and to the ouside world
46
how does the nose cleanse the air?
1. vibrassae (nasal hairs), filters 2. mucous membrane- cillia sweep to the nasopharynx in **10 minute cycles**, 50-200 cilia per cell
47
all five arteries in the nose anastamose on....
**kiesselback plexus**
48
where are two places in the nose that can cause injury? what are the negative effects of each?
Trauma to the septum: 1. physical obstruction of flow 2. loss of nasal reparative function Trauma to the turbinates: 1. loss of **flue** effect 2. loss of reparative effect
49
what happens in dryness? what happens if too many particulates? what happens if too many allergens? what happens if too many toxins or noxious material?
dryness: IgA can't function too many particulates: overwhelm cilia allergens: activate mast cells, realse histamine and cause disruptions toxins: inactivate cilia
50
nasal septum deviation can....
disturb air flow and mucosal function, moves faster through horizontally than vertically.
51
how much can a blockage of the nose decrease the oxygen level in blood?
by 10%, this is a big deal in elderly, not so much in younger people
52
Epitaxis (nose bleeds);where do the majority take place? the minority? what are 5 risk factors for epitaxis?
**90% epitaxis occur in middle or anterior nares** Kiesselbach Plexus RF: nasal trauma ie picking, dry nasal mucosa, hypertension, nasal cocaine, alcohol
53
arteriole vs venous epitaxis; where do you have to go for each?
arteriole **(may not work with pressure)** and venous: probs will stop bleeding **posterior go to ED**
54
what can cause a nose bleed anteriorly at the kiesselbach plexus? what can happen if this happens frequently? what do you do about it? what do you need to be really careul of in males with anterior epitaxis?
caused by **trauma or in elderly when the mucosa _thins_ out** if recurrent causes nasal septal perforation **hold pressure 10 mins** (trauma, hypertensions, extreme dryness, foreign body), review meds like warfarin **CAUTION: IN MALES BLEEDING WITH COEXISTING NASAL OBSTRUCTION=JUVENILE NASOPHARYNGEAL ANGIOFIBROMA-**mass of small blood vessels if you put someting in you could make it a lot worse
55
posterior epitaxsis happens at which arter and in how many people?
10% of people and the sphenopalatine artery, balance of the nose ## Footnote **5% associated with hypertension and athlerosclerosis**
56
57
what is an interesting illicit substance you can use to stop the bleeding in epitaxis? what else can you use thats more boring?
topical cocaine, anestetic and vasoconstrictor, oxymetazalone, can cauderize if you find the area it is coming from
58
allergic rhinitis
inflammation of the nasal mucosa **IgE** mediated reactivity to airborne antigens (pollen mold etc) common in those with atopic diease like asthma eczema, or family history **allergic shiners**, **bluish coloration below the eyes increased pigmentation, AVASCULAR POLUPS IN THE SINSES TO ETHMOID (PEELED GRAPES), BILATERAL OR CONCERNTED WITH CANCER**, rhinnorhea itchy watery eyes, sneezing nasal congestion, allergic salute or a horizontal line on the nose from pushing up **clear and watery discharge** DOC: antihistamines including cromolyn sodium (mast cell inhibitor nasal spray), systemic corticosteroids, DONT USE DECONGESTANTS BECAUSE OF ADDICTION
59
what drugs can cause rhinitis?
1. cocaine 2. anticholenergic 3. oxymatazalone 4. beta blockers 5. estrogens/viagra
60
what does rhinitis occur in pregancy? what do you do about it?
response to increase in estrogen, irrigations
61
vasomotor rhinitis; what is increased here and why? what makes this worse and you shouldn't give to an elderly patient? what can you try to make it better?
imbalance of autonomic nervous system activity in elderly _ironic_: chronic dry nasal mucosa but increased rhinorreah _this rhinnoreah doesn't have the same concentration as nasal secretions so it tries to make more_ **"bogginess of nasal mucosa associated with stuffiness, and rhinorreah"** Tx: nothing that works really well, saline nasal spray, corticosteroid NS ANTIHISTAMINES MAKE IT WORSE!!
62
what are the two types of rhinitis that are commonly confused? which isn't typically found in elderly and what happens if you put them on antihistamines?
allergic rhinitis and vasomotor rhinitis ## Footnote **typically no allergic rhinitis in elderly, if given antihistamines they get worse**
63
what is rhinitis medicamentosa?
overuse of decongestants drops or sprays with oxymatazalone causing rebound congestion, which causes the pt to use it more pts get increased pain and congestion DISCONTINUE USE--may use corticonasal spray in the transition
64
bacterial rhinitis; what bacteria cause this and where do you find it?
ethmoid **same bacteria that cause ottis media** **_s.peumoniae, h. influenzae, m. catarrhalis_** DOC: amoxicillin, doxy
65
atrophic rhinitis; what do you treat with?
usually in elderly, dry nose **anatomical deformity in the nasal septum, turbinates exacerbate** DOC: premarin cream, corticosteroid spray
66
paranasal sinuses are lined with...
respiratory epithelia, cilia
67
superior turbinates drain the...(2)
posterior ethmoids, sphenoids
68
middle turbinates drain...(3)
maxillary, frontal, anterior ethmoids
69
inferior turbinate drain the...(1)
nasolacriminal duct
70
the ethmoid sinuses
present at birth but enlarge until puberty
71
maxillary sinuses; what may extend into the floor? drains into what? depends on what for clearance?
- dental roots may extend into the floor - depends on cillia for clearance - drains into middle turbinate
72
frontal sinuses; often\_\_\_in shape;at birth?
- asymmetric - not present at birth and may not ever develope
73
the cilia in the sinuses are very\_\_\_\_\_\_ to \_\_\_\_\_\_. the sinuses are painful if they arent \_\_\_\_\_. ;when can this happen? sinuses are lined with?
the cilia in the sinuses are very sensitive to toxins the sinuses are painful if they arent AERATED ;when can this happen; EDEMA sinuses are lined with, RESPIRATORY EPITHELIUM
74
acute sinusitis; how long do you have it? what distinguishes it from rhinitis? what happens with the symptoms?
mostly viral, very few bacteria **inflammation near the osteomeatal complex distinguishes from rhinitis** **often follows URI** **same bacteriology as ottis media, _strep pneumonia, m. catarralis, h. influenzae_** **10 days-4 weeks** **initial improvement but then worsening of symptoms** sinus tendernous, decreased transillumination of sinuses
75
chronic sinusitis; how long do they have it; who is it common in; how does it present. ONE INTERESTING THING THAT IT PRESENTS WITH?
**present 12 weeks durations** **chronic mucopurlulent discharge, pain/pressure** mucosa damaged, thickened, and **ciliary spotty** **_mucoeles: chronic thickened mucosa_** **tabacco smokers**
76
what test do you do to evaluate sinusitis?
CT
77
barosinusitis
**negative air pressure in the sinuses** acute forms with rapid change in ambient pressure chronic: obstruction of maxillary sinus ostia
78
osteomeatal complex
hole/drainage in the wall of the sinuses