ENT Flashcards

1
Q

Features of a brachial cyst

A

An oval, mobile cystic mass that develops between the sternocleidomastoid muscle and the pharynx

  • mobile, soft and fluctuant
  • Swelling is intermittent

The cyst is filled with acellular fluid with cholesterol crystals and encapsulated by stratified squamous epithelium. Branchial cysts may have a fistula and are therefore prone to infection. They may enlarge following a respiratory tract infection.

Develop due to failure of obliteration of the second branchial cleft in embryonic development

Usually present in early adulthood

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2
Q

Thyroglossal cyst features

Location

Features

A

Persistence of the thyroid duct

More common in patients < 20 years old
Usually midline, between the isthmus of the thyroid and the hyoid bone (i.e. below the hyoid)

!Moves upwards with protrusion of the tongue!
Its connection with the foramen caecum means it will move on tongue protrusion.
May be painful if infected

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3
Q

Management of sore throat

A

CENTOR criteria: 3 out of following to give abc

1) Cervical lymphadenopathy
2) Exudate on tonsils
3) No cough
4) Temp >38

If abx given
- 7-10 day course of phenoxymethylpenicillin or erythromycin (if the patient is penicillin allergic)

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4
Q

Drugs that cause hearing loss

A
Aminoglycosides (e.g. Gentamicin)
Furosemide
Aspirin 
Quinine
and a number of cytotoxic agents
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5
Q

Indications for tonsillectomy

Complications of tonsillectomy + management

A

NICE: Meets all criteria
• Sore throats are due to acute tonsillitis
o 7 or more significant sore throats in the preceding 12 months OR
o 5 or more episodes in each of the preceding two years OR
o 3 or more in each of the preceding three years).
• Symptoms occurring for at least a year
• The episodes are disabling and prevent normal functioning

Other indications:

  • Recurrent febrile convulsions secondary to episodes of tonsillitis
  • Obstructive sleep apnoea, stridor or dysphagia secondary to enlarged tonsils
  • Peritonsillar abscess (quinsy) if unresponsive to standard treatment

Complications
Primary (< 24 hours): haemorrhage in 2-3% (most commonly due to inadequate haemostasis), pain
Secondary (24 hours to 10 days): haemorrhage (most commonly due to infection), pain

Haemorrhage is a feared complication following tonsillectomy. Primary, or reactionary haemorrhage most commonly occurs in the first 6-8 hours following surgery. It is managed by immediate return to theatre.

Secondary haemorrhage occurs between 5 and 10 days after surgery and is often associated with a wound infection. Treatment is usually with admission and antibiotics. Severe bleeding may require surgery. Secondary haemorrhage occurs in around 1-2% of all tonsillectomies.

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6
Q

Red flags for epistaxis

A

Unilateral - if recurrent unilateral epistaxis, think about cancer

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7
Q

Why does otitis media need urgent treatment

A

To prevent meningitis which is a complication of mastoiditis.

Other complications:
Ear drum perforation
- Spontaneously closes after 6/52
- Might see discharge => can cause secondary Otitis externa

Chronic supparative OM (chronic d/c through perf)

Chronic OM

Chronic OM with effusion (glue ear)

Mastoiditis

  • Spread of infection to mastoid cavity => loss of post-auricular sulcus, mastoid
  • Bogginess, pinna pushed forward and down
  • Urgent op needed due to risk of infection tracking into meninges and brain
Also to prevent
CN palsies
Hearing loss
Osteomyelitis
Carotid artery spasm
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8
Q

Malignant otitis externa

Who get’s it

Causative organism

Key features in history/presentation

Diagnosis

Management

A

Uncommon type of otitis externa that is found in immunocompromised individuals (90% cases found in diabetics)

most commonly caused by Pseudomonas aeruginosa

Infection commences in the soft tissues of the external auditory meatus, then progresses to involve the soft tissues and into the bony ear canal
Progresses to TEMPORAL BONE OSTEOLYTIS

Key features in history
Diabetes (90%) or immunosuppression (illness or treatment-related)
Severe, unrelenting, deep-seated otalgia
Temporal headaches
Purulent otorrhea
Possibly dysphagia, hoarseness, and/or facial nerve dysfunction

Diagnosis
A CT scan is IMPORTANT

Treatment
non-resolving otitis externa with worsening pain should be referred urgently to ENT
Intravenous antibiotics that cover pseudomonal infections - ciprofloxacin

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9
Q

What is osteoclerosis

Cause

Features

Management

A

Otosclerosis describes the replacement of normal bone by vascular spongy bone. It causes a progressive conductive deafness due to fixation of the stapes at the oval window. Otosclerosis is autosomal dominant and typically affects young adults

Onset is usually at 20-40 years - features include:

  • Conductive deafness
  • Tinnitus
  • POSITIVE FH

Management
hearing aid
stapedectomy

*10% of patients may have a ‘flamingo tinge’, caused by hyperaemia

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10
Q

Management of nasal polyps

Associated conditions

A

When bilateral, nasal polyps should be referred to ENT non-urgently for assessment. Topical corticosteroid therapy is first-line management, which shrinks polyps in 80% of cases.

Unilateral nasal polyps are a red-flag sign for nasopharyngeal cancer and therefore warrant an urgent referral to ENT

Associations
Asthma* (particularly late-onset asthma)
Aspirin sensitivity*
Infective sinusitis
Cystic fibrosis
Kartagener's syndrome
Churg-Strauss syndrome

*the association of asthma, aspirin sensitivity and nasal polyposis is known as Samter’s triad

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11
Q

Features of a cytsic hygroma

A

A congenital lymphatic lesion (lymphangioma) typically found in the neck, classically on the left side
Most are evident at birth, around 90% present before 2 years of age

Swelling in the subcutaneous tissue of the posterior triangle which transilluminates.

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12
Q

What is black hairy tongue

Predisposing factors

Investigation

Management

A

Black hairy tongue is relatively common condition which results from defective desquamation of the filiform papillae. Despite the name the tongue may be brown, green, pink or another colour.

Predisposing factors
poor oral hygiene
antibiotics
head and neck radiation
HIV
intravenous drug use

The tongue should be swabbed to exclude Candida

Management
Tongue scraping
Topical antifungals if Candida

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13
Q

Causes of vertigo

A

Viral labyrinthitis

  • Recent viral infection
  • Sudden onset
  • Nausea and vomiting
  • Hearing may be affected

Vestibular neuronitis

  • Recent viral infection
  • Recurrent vertigo attacks lasting hours or days
  • No hearing loss

Benign paroxysmal positional vertigo

  • Gradual onset
  • Triggered by change in head position
  • Each episode lasts 10-20 seconds

Meniere’s disease
- Associated with hearing loss, tinnitus and sensation of fullness or pressure in one or both ears

Vertebrobasilar ischaemia

  • Elderly patient
  • Dizziness on extension of neck

Acoustic neuroma

  • Hearing loss, vertigo, tinnitus
  • Absent corneal reflex is important sign
  • Associated with neurofibromatosis type 2

POSTERIOR CIRCULATION STROKE

Trauma

Multiple sclerosis

Ototoxicity e.g. gentamicin

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14
Q

What is gingivitis

Spectrum of disease

Management

A

Gingivitis is usually secondary to poor dental hygiene. Clinical presentation may range from simple gingivitis (painless, red swelling of the gum margin which bleeds on contact) to acute necrotizing ulcerative gingivitis (painful bleeding gums with halitosis and punched-out ulcers on the gums).

If the patient has simple gingivitis
should be advised to seek routine regular review by a dentist. Antibiotics are not usually necessary

If a patient presents with acute necrotizing ulcerative gingivitis CKS recommend the following management:
refer the patient to a dentist, meanwhile the following is recommended:
Oral metronidazole* for 3 days
Chlorhexidine (0.12% or 0.2%) or hydrogen peroxide 6% mouth wash
Simple analgesia

*the BNF also suggest that amoxicillin may be used

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15
Q

Causes of gingival hyperplasia

A

Drug causes of gingival hyperplasia
Phenytoin
Ciclosporin
Calcium channel blockers (especially nifedipine)

Other causes of gingival hyperplasia include
acute myeloid leukaemia (myelomonocytic and monocytic types)

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16
Q

Submandibular triangle borders

Masses cause

A

Borders

  • Mandible
  • Anterior digastric
  • Posterior digastric

Causes:

1) Sialolithiasis = stones
80% of all salivary gland calculi occur in the submandibular gland. 70% of these calculi are radio-opaque
- Stones are usually composed of calcium phosphate or calcium carbonate
- COLICKY pain and post prandial swelling of the gland
Investigation involves sialography to demonstrate the site of obstruction and associated other stones
- Stones impacted in the distal aspect of Wharton’s duct may be removed orally, other stones and chronic inflammation will usually require gland excision

2) Sialadenitis
Usually occurs as a result of Staphylococcus aureus infection
Pus may be seen leaking from the duct, erythema may also be noted
Development of a sub mandibular abscess is a serious complication as it may spread through the other deep fascial spaces and occlude the airway
Foul taste in mouth

3) Submandibular tumours
Only 8% of salivary gland tumours affect the sub mandibular gland
Of these 50% are malignant (usually adenoid cystic carcinoma)
Diagnosis usually involves fine needle aspiration cytology
Imaging is with CT and MRI
In view of the high prevalence of malignancy, all masses of the submandibular glands should generally be excised.

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17
Q

Sudden sensorineural hearing loss

Cause

Test

Management

A

In the vast majority of cases is idiopathic.

PTA
- Both bone and air conduction reduced

There is some evidence that high dose steroids (60mg/day) for seven days improves prognosis, so all patients should start treatment as soon as possible.
ENT assessment should be arranged as soon as possible to allow pure tone audiometry testing and to arrange an MRI to exclude an acoustic neuroma.
Intra-tympanic steroids can also be given if there is no response to oral steroids.

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18
Q

Otitis externa

Causes

Features

Investigations

Management

2nd line options

A
Causes of otitis externa include:
Infection: bacterial (Staphylococcus aureus, Pseudomonas aeruginosa) or fungal
Seborrhoeic dermatitis
contact dermatitis (allergic and irritant)

Features
Ear pain, itch, discharge, commonly 2ndary to eczema/FB
O/E: palpate pinna and tragus - pain = +ve sign
Otoscopy: red, swollen, or eczematous canal, visible discharge (swab)

Investigations

  • Swabs if discharge
  • Bloods

The recommended initial management of otitis externa is:
Topical antibiotic or a combined topical antibiotic with a steroid e.g. gentamycin + hydrocortisone drops
[If the tympanic membrane is perforated aminoglycosides are traditionally not used*]
If there is canal debris then consider removal (micro suction)
If the canal is extensively swollen then an ear wick is sometimes inserted

Second-line options include
Consider contact dermatitis secondary to neomycin
Oral antibiotics (flucloxacillin) if the infection is spreading
Taking a swab inside the ear canal
Empirical use of an antifungal agent

If a patient fails to respond to topical antibiotics then the patient should be referred to ENT.

Malignant otitis externa is more common in elderly diabetics. In this condition, there is extension of infection into the bony ear canal and the soft tissues deep to the bony canal. Intravenous antibiotics may be required.

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19
Q

Nasopharyngeal carcinoma

Features

Imaging

Treatment

A

SQUAMOUS CELL of the nasopharynx
Rare in most parts of the world, apart from individuals from Southern China
Associated with Epstein Barr virus infection

Features
Systemic: Cervical lymphadenopathy - painless
Local: Otalgia; Unilateral serous otitis media; Nasal obstruction, discharge and/ or epistaxis; Cranial nerve palsies e.g. III-VI

Imaging
Combined CT and MRI.

Treatment
Radiotherapy is first line therapy.

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20
Q

Cholesteatoma define

Main features

Ottoscopy findings

Management

Complications

A

Cholesteatoma is a non-cancerous growth of squamous epithelium that is ‘trapped’ within the skull base causing local destruction. It is most common in patients aged 10-20 years. Being born with a cleft palate increases the risk of cholesteatoma around 100 fold.

Main features
foul-smelling, non-resolving discharge
hearing loss

Other features are determined by local invasion:
vertigo
facial nerve palsy
cerebellopontine angle syndrome

Otoscopy
‘attic crust’ - seen in the uppermost part of the ear drum

Management
patients are referred to ENT for consideration of surgical removal (MASTOIDECTOMY)

Complications

  • Cerebrall abscess
  • Meningitis
  • Deafness
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21
Q

Otitis media

Features

Management

A

Can be 2ndary to URTI –> Eustachian tube

Features
Most common in children and rare in adults
May present with symptoms elsewhere (e.g. vomiting) in children
Severe pain and sometimes fever
May present with discharge is tympanic rupture occurs

Management
Antibiotics (usually amoxycillin 500mg QDS for 7 days) or erythromycin

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22
Q

When to 2 week wait to oral surgery

A

2 week wait referrals to oral surgery should be done in all of the following cases:

1) Unexplained oral ulceration or mass persisting for greater than 3 weeks
2) Unexplained red, or red and white patches that are painful, swollen or bleeding
3) Unexplained one-sided pain in the head and neck area for greater than 4 weeks, which is associated with ear ache, but does not result in any abnormal findings on otoscopy
4) Unexplained recent neck lump, or a previously undiagnosed lump that has changed over a period of 3 to 6 weeks
5) Unexplained persistent sore or painful throat
6) Signs and symptoms in the oral cavity persisting for more than 6 weeks, that cannot be definitively diagnosed as a benign lesion

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23
Q

Causes of hoarseness

Investigations

When to 2 week wait

A
Causes of hoarseness include:
voice overuse
smoking
viral illness
hypothyroidism
gastro-oesophageal reflux
laryngeal cancer
lung cancer

When investigating patients with hoarseness a chest x-ray should be considered to exclude apical lung lesions.

Suspected laryngeal cancer: referral guidelines-

A suspected cancer pathway referral to an ENT specialist should be considered for people aged 45 AND OVER with:

  • Persistent unexplained hoarseness or
  • An unexplained lump in the neck.
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24
Q

Thyroid surgery complications

A

Anatomical such as recurrent laryngeal nerve damage.

Bleeding. Owing to the confined space haematoma’s may rapidly lead to respiratory compromise owing to laryngeal oedema.
-If a bleed occurs, the pressure behind the suture line increases and the trachea becomes compressed resulting in stridor. Thereforere remove sutures + urges Senior assistance will be required as this patient will require further surgery for haemostasis.

Damage to the parathyroid glands resulting in LOW CALCIUM.

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25
Q

Perforated tympanic membrane

Causes

Management

A

Causes
Infection.
Other causes include barotrauma or direct trauma.

Management
No treatment is needed in the majority of cases as the tympanic membrane will usually heal after 6-8 weeks. It is advisable to avoid getting water in the ear during this time
it is common practice to prescribe antibiotics to perforations which occur following an episode of acute otitis media. NICE support this approach in the 2008 Respiratory tract infection guidelines
myringoplasty may be performed if the tympanic membrane does not heal by itself

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26
Q

When to 2 week wait for oral cancer

A

Unexplained ulceration in the oral cavity lasting for more than 3 weeks or
a persistent and unexplained lump in the neck.

Consider an urgent referral (for an appointment within 2 weeks) for assessment for possible oral cancer by a dentist in people who have either:
A lump on the lip or in the oral cavity or
A red or red and white patch in the oral cavity consistent with erythroplakia or erythroleukoplakia.

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27
Q

What is Ramsay hunt syndrome

Features

Management

A

Ramsay Hunt syndrome (herpes zoster oticus) is caused by the reactivation of the varicella zoster virus in the geniculate ganglion of the seventh cranial nerve.

Features
Auricular pain is often the first feature
Facial nerve palsy
Vesicular rash around the ear
- may also be seen on the anterior 2/3rds of the tongue and the soft palate.
Other features include vertigo and tinnitus

Management
oral aciclovir and corticosteroids are usually given

28
Q

What is meniere’s disease

Features

Natural history

Management

A

Disorder of the inner ear of unknown cause. It is characterised by excessive pressure and progressive dilation of the endolymphatic system. It is more common in middle-aged adults but may be seen at any age. Meniere’s disease has a similar prevalence in both men and women.

Features
Recurrent episodes of vertigo, tinnitus and hearing loss (SENSORINEURAL).
- Vertigo is usually the prominent symptom
- A sensation of aural fullness or pressure is now recognised as being common
- other features include nystagmus and a positive Romberg test
- episodes last minutes to hours
- typically symptoms are unilateral but bilateral symptoms may develop after a number of years

Natural history
Resolve in the majority of patients after 5-10 years
Majority will be left with a degree of hearing loss
Psychological distress is common

Management
ENT assessment is required to confirm the diagnosis
Patients should inform the DVLA. The current advice is to cease driving until satisfactory control of symptoms is achieved

Acute attacks:
- buccal or intramuscular Prochlorperazine. Admission is sometimes required

PREVENTION:
- BETAHISTAMINE and vestibular rehabilitation exercises may be of benefit

29
Q

Quincy

Define

Features

Management

A

A peritonsillar abscess typically develops as a complication of bacterial tonsillitis.

Features include:
• Severe throat pain, which lateralises to one side
deviation of the uvula to the unaffected side
• Trismus (difficulty opening the mouth)
• Reduced neck mobility
• Hot potato voice
• Anorexia - can’t eat

Patients need urgent review by an ENT specialist.

Management
• Needle aspiration or incision & drainage + intravenous antibiotics - LA through moth
• Tonsillectomy should be considered to prevent recurrence

30
Q

Vestibular neuronitis

Features

Differential diagnosis

A

Vestibular neuronitis is a cause of vertigo that often develops following a viral infection.

Features
recurrent vertigo attacks lasting hours or days
nausea and vomiting may be present
horizontal nystagmus is usually present
no hearing loss or tinnitus

Differential diagnosis
viral labyrinthitis
posterior circulation stroke: the HiNTs exam can be used to distinguish vestibular neuronitis from posterior circulation stroke

Management
vestibular rehabilitation exercises are the preferred treatment for patients who experience chronic symptoms
Buccal or intramuscular prochlorperazine is often used to provide rapid relief for severe cases
a short oral course of prochlorperazine, or an antihistamine (cinnarizine, cyclizine, or promethazine) may be used to alleviate less severe case

31
Q

Sinusitis

Investigations

management

Complications

Name the sinuses

A

Investigations
• Usually CT done
• Nasal swabs? (commonly strep pneumonia, haemophillus, moraxella catarrhalis)

Management of acute sinusitis

  • Analgesia
  • Intranasal decongestants or nasal saline may be considered but the evidence supporting these is limited

NICE CKS recommend that intranasal corticosteroids may be considered if the symptoms have been present for MORE THAN 10 DAYS

Oral antibiotics are not normally required but may be given for severe presentations. The BNF recommends phenoxymethylpenicillin first-line, co-amoxiclav if ‘systemically very unwell, signs and symptoms of a more serious illness, or at high-risk of complications’

Complications
• Orbital cellulitis
• Frontal abscess/subdural empyema/cerebral abscess

Sinuses
- Frontal 
- Maxillary
- Ethmoid 
- Sphenoid 
They drain into the nose through tiny openings - mucus should go down to throat
32
Q

What is allergic rhinitis

Types

Features

Management

A

Allergic rhinitis is an inflammatory disorder of the nose where the nose become sensitized to allergens such as house dust mites and grass, tree and weed pollens. It may be classified as follows, although the clinical usefulness of such classifications remains doubtful:

1) Seasonal: symptoms occur around the same time every year. Seasonal rhinitis which occurs secondary to pollens is known as hay fever
2) Perennial: symptoms occur throughout the year
3) Occupational: symptoms follow exposure to particular allergens within the work place

Features
sneezing
bilateral nasal obstruction
clear nasal discharge
post-nasal drip
nasal pruritus

Management of allergic rhinitis
allergen avoidance
if the person has mild-to-moderate intermittent, or mild persistent symptoms:
- oral or intranasal antihistamines
if the person has moderate-to-severe persistent symptoms, or initial drug treatment is ineffective
- intranasal corticosteroids = 1st line e..g. fixonase
- a short course of oral corticosteroids are occasionally needed to cover important life events

there may be a role for short courses of topical nasal decongestants (e.g. oxymetazoline). They should not be used for prolonged periods as increasing doses are required to achieve the same effect (tachyphylaxis) and rebound hypertrophy of the nasal mucosa (rhinitis medicamentosa) may occur upon withdrawal

33
Q

Causes of sensorineural hearing loss

A

Disorders of the cochlear apparatus
• Congenital disorders: from rubella or syphilis in the pregnant mother
• Infection: basal meningitis, spread of infection from the middle to the inner ear, mumps or measles
• Medication: aminoglycosides, diuretics, salicylates, quinine (deafness is transient with the last two but can be permanent with aminoglycosides)
• Presbycusis: neuronal degeneration in the elderly causing high-frequency hearing loss
• Noise-induced disorders: high-frequency hearing loss from, e.g., gun blasts or industrial machinery
• Ménière disease: vertigo, fluctuating tinnitus and deafness
• Head injury: fractures through the base of the skull and petrous temporal bone can lead to damage to the cochlea and eighth nerve

Disorders of the cochlear nerve
• Lesions of the cerebellopontine angle (CPA), e.g., vestibular schwannoma (NF2?), other tumours, granulomatous disease, arteriovenous malformation, stroke (anterior inferior cerebellar artery)
• Lesions of the base of the skull e.g. infective (meningitis), inflammatory (sarcoidosis), carcinomatous meningeal seeding such as nasopharyngeal carcinoma

Disorders of the brainstem
• Multiple sclerosis: plaques of demyelination involving the cochlear nuclei
• Infarction
• Neoplastic infiltration

Disorders of the supranuclear connections
Unilateral lesions of the supranuclear pathways will not cause deafness as the cochlear nuclei on each side have bilateral connections projecting to the temporal cortices.

34
Q

Causes of conductive hearing loss

Define

A
Wax
Osteosclerosis
Trauma
FB
Ottitis externa
Otitis media with TM perforation

Sound not getting through to the cochlea for some reason

35
Q

What is glue ear

Management

A

OM with effusion

More common in children than adults (important to monitor effect on hearing, development, schooling)

Build up of fluid in middle ear - blocked Eustachian tube

Red flag in adults as could be cancer impairing drainage through Eustachian tube

Mx

Conservative – Valsalva maneuvers, auto-inflation in younger children,
- xhearing aids

Surgical – myringotomy +/- Grommet (ventilation tubes) insertion

  • Grommets fall out spontaneously in a year or so
  • Common for pts to need re-insertion

Parents should stop smoking

36
Q

Define sensorineural hearing loss

A

Something happing in the cochlea, the nerve of hearing or the brain which is stopping the brain picking up that sound

37
Q

What is labrythitis

Presentation

Treatment

A

Most common cause of acute vertigo – usually due to viral infection getting into ear

Presentation
• Acute vertigo, nausea, imbalance
• Nystagmus
• No neurological deficit or headache. If they do  urgent investigation

Treatment
• Vestibular sedatives: Stemetil (asap)
• Vestibular rehab after acute phase
• Admit if lives alone, unable to cope, severe vomiting

38
Q

What is BPPV

Test

Management

A

Severe brief episodic vertigo lasting 30s on head turning (always positioning)
+ Nystagmus

Cause
- Displacement of otoliths in semicircular canals - common after head injury

Causes

  • head injury
  • Idiopathic
  • Otosclerosis
  • Post viral

Test = Hallpike = upbeat torsional nystagmus + dizziness

Management

  • Epley manœuvre
  • Betahistine - often given but limited use
39
Q

What is ossiculoplasty

A

For conductive hearing loss with damaged or fixed ossicles
• Synthetic implant or
• Shaped pieces of cartilage

Between the ear drum and the stapes foot plate where the sound is conducting into the cochlea

40
Q

What is an acoustic neuroma

Fetaures

Associated conditions

Investigations

Management

A

AKA Vestibular schwannomas account for approximately 5% of intracranial tumours and 90% of cerebellopontine angle tumours.

The classical history of vestibular schwannoma includes a combination of vertigo, hearing loss, tinnitus and an absent corneal reflex. Features can be predicted by the affected cranial nerves:

  • CN VIII: vertigo, unilateral sensorineural hearing loss, unilateral tinnitus
  • CN V: absent corneal reflex
  • CN VII: facial palsy

Bilateral vestibular schwannomas are seen in neurofibromatosis type 2.

Patients with a suspected vestibular schwannoma should be referred urgently to ENT. It should be noted though that the tumours are often slow growing, benign and often observed initially.

MRI of the cerebellopontine angle is the investigation of choice. Audiometry is also important as only 5% of patients will have a normal audiogram.

Management is with either
- surgery
- radiotherapy
or observation.

41
Q

Tonsilitis

Symptoms

Causes

Criteria

Management

A

• 60-90% Viral
Sore throat, fever, malaise

Signs
• Lymphadenopathy
• Inflamed tonsils and oropharynx
• Exudates

Organisms
• Precedes bacterial infection
• Usually Streptococcal (Gp A Strep pyogenes)
• Strep pneumoniae, staph, Haemophilus,
• Rarely Diptheriae, Gonococcus,
• EBV (Glandular Fever) – Don’t give amoxicillin else rash

Criteria - CENTOR
Cervical lymphadenopathy
Exudate
No cough
Temperature

Management
• Throat Swab
• Penicillin V 500mg QDS 10 days (Clarithromycin if allergy to pen) - if CENTOR 3 or more
• Hydration, rest
• Strong analgesia
• If persists Throat swab, add metronidazole, Monospot

42
Q

Adenoidectomy Indications

A

Indications
• OSA in children (+/- adults)
• Glue ear with failed grommets x 1-2
• For biopsy

43
Q

Tracheostomy vs Laryngotomy

A

Tracheostomy:
• Tube coming from neck into airway but still have a normal airway (voice box and airway above there so can breathe both ways)

If cuffed tracheostomy:
• Can only breathe through the tube

Laryngectomy:
• Trachea is brought out and sutured to the skin – no voice box/larynx

44
Q

Anterior neck triangle borders

A

1) Anterior Sternocleidomastoid (SCM)
2) Mandible
3) Midline

Broken further down by other muscles
• Digastric
o Submandibular triangle
o Carotid triangle

45
Q

Posterior neck triangle borders

A

1) Posterior border of SCM
2) Clavicle
3) Trapezius

Further divided by the omohyoid muscle: Into upper and lower part

46
Q

Causes of anterior neck triangle lumps

Investigations

A
Divide by pulsatility 
1) Pulsatile
•	Carotid artery aneurysm
•	Tortuous carotid a.
•	Carotid body tumour
2) Non-Pulsatile
•	Branchial cyst
•	Laryngocele
•	Goitre
•	Parotid tumour (lump in postero-superior area)
47
Q

Causes of posterior Triangle neck lumps

A
LNs
Cervical ribs
Pharyngeal pouch
Cystic hygromas
Pancoast's tumour
48
Q

Causes of midline lumps

A

<20yo
• Thyroglossal cyst
• Dermoid cyst

> 20y
• Thyroid isthmus mass
• Ectopic thyroid tissue

49
Q

Brachial cyst

What is it

Presentation

Management

A

Embryological remnant 2nd brachial cleft

Presentation

  • Age <20
  • Anterior margin of SCM at junction of upper and middle 3rd
  • May be infected –> abscess
  • May be associated with branchial fistula

Management

  • Med: Abx for infection, Sclerotherapy
  • Surgery: Definitive Rx
50
Q

What is chronic rhinosinutisis

Symtoms

Investigations

Management

A

Not always related to allergy
• +/- Nasal polyps
• Samters triad – asthma, nasal polyps and aspirin allergy
• Difficult to treat

Investigation
• Allergy testing

Management
•	Long term steroid nasal sprays/drops
o	(fluticasone / mometasone)
•	Oral steroids occasionally
•	Antibiotics (azithromycin)
•	Endoscopic sinus surgery
51
Q

Epistaxis managemnet

A
  • Sit up and forwards
  • Reassure
  • Mouth open (cork)
  • Pinch firmly cartilaginous nose
  • Ice over bridge of nose
If that doesn’t work
•	Calm. Reassure, fluids, bloods
•	Protection – apron, mask, goggles
•	Assistant
•	LA spray – cophenylcaine and wait 3-4mins

Consider cautery or packing. Cautery should be used if the source of the bleed is visible and cautery is tolerated- it is not so well tolerated in younger children! Packing may be used if cautery is not viable or the bleeding point cannot be visualised. If the nose is packed in primary care, the patient should be admitted to hospital for review.

Cautery:
Ask the patient to blow their nose in order to remove any clots. Be wary that bleeding may resume.
Use a topical local anaesthetic spray (e.g. Co-phenylcaine) and wait 3-4 minutes for it to take effect
Identify the bleeding point and apply the silver nitrate stick for 3-10 seconds until it becomes grey-white. Avoid touching areas which do not require treatment, and only cauterise one side of the septum as there is a risk of perforation.
Dab the area clean with a cotton bud and apply Naseptin or Muciprocin

Packing:
Anaesthetise with topical local anaesthetic spray (e.g. Co-phenylcaine) and wait for 3-4 minutes
Pack the patient’s nose while they are sitting with their head forward, following the manufacturer’s instructions
Pressure on the cartilage around the nostril can cause cosmetic changes and this should be reviewed after inserting the pack.
Examine the patient’s mouth and throat for any continuing bleeding, and consider packing the other nostril as this increases pressure on the septum and offending vessel.
Patients should be admitted to hospital for observation and review, and to ENT if available

  • Foley, Brighton Balloon – insert into postnasal space then inflate the balloon, pull it forward – puts pressure on sphenopalatine artery at back of nose
  • Often a combination of packing and balloon catheter

Self-care advice involves reducing the risk of re-bleeding. Patients should be informed that blowing or picking the nose, heavy lifting, exercise, lying flat, drinking alcohol or hot drinks should be avoided. The same applies for patients who have just been cauterised, as any strain on the nostril may induce a re-bleed.

52
Q

Instrumenst used to examine the nose

A

Headlight
Thudicum’s speculum
Otoscope
Nasal Endoscope (rigid or flexible)

53
Q

When to x-ray for nasal bone injury

Management

A
Only x-ray acutely depending on:
•	Mechanism
•	Diplopia
•	Abnormal eye movements
•	Depressed zygoma
•	Malocclusion
•	LOC
•	Loss of facial sensation

Management
• If seen v early release immediately pre swelling
• Otherwise see in 7d for reassessment
• If compound: Clean thoroughly and close (broad spectrum abx)
• Septal haematoma –> refer for drainage

54
Q

Septal haematoma

Cause

Management

A

Blood collection between skin and lining of the nose and the nasal septum

Cause
• Nasal bone fractures

Management
• DRAIN - as blood supply from cartilage comes from perichondrium and if elevated off - they lose blood supply - Can cause necrosis and leave hole in nasal septum

55
Q

Septal deviation

Causes

Presentation

Management

A

Causes
• Trauma
• Congenital (tend to be more severe)

Presentation
• Nasal obstruction
• Snoring
• Mouth breathing

Management
• Surgery if symptomatic = Simple endoscopic septoplasty
• Combine with rhinoplasty if external deformity

56
Q

Thyroid lump investigations

A
Check TFTs
Refer if (routine)
•	New lump
•	Increase in size
•	Abnormal TFTs
•	Sudden pain
Refer (2ww)
•	Hoarseness
•	Cervical adenopathy
•	Rapidly enlarging painless mass
•	Thyroid nodule in child

Same day referral if stridor

57
Q

Thyroidectomy risks

A

Bleeding –> Airway obstruction
Recurrent laryngeal n injury
HYPOCALCAEMIA

58
Q

Neck lumps main caused

Investigations

A

85% are LNs especially if <3 weeks - Infection/Cancer
8% are goitres
7% others e.g. sebaceous systemic or lipoma

Investigation
TRIPLE ASSESSMENT: Clinical, US, Cyto/Histo (apiration or biopsy)

59
Q

Laryngocele

Define

Characteristic feature

A

Cystic dilatation of the laryngeal saccula
Anterior non-pulsatile neck lump

Exacerbated by blowing

60
Q

Dermoid cysts

A

Causes
Developmental inclusion of the epidermis along the lines of skin fusion

Presentation

  • common <20yo
  • Found at junction of embryological fusion
    • neck midline
    • lateral angles of eyebrow
    • under tongue

Contains ectodermal elements

  • Hair follicles
  • Sebaceous glands
61
Q

Cystic hygroma

Presentation

Rx

A

Congenital multiloculated lymphagioma arising from the jugular lymph sac

Presentation

  • Infant
  • Lower posterior triangle lump - may extend into axilla
  • increases in size when kid cries
  • Transilluminates bilaterally

Rx
- Excision of hypertonic saline sclerosant

62
Q

Cervical rib neck lump

Presentation

A

Overdevelopment of transverse process of C7

Presentation

  • Mostly asymptomatic
  • Hard swelling
  • Reduced radial pulse on abduction and external rotation of arm

CAN –> vascular syndrome

  • compresses subclavian a.
  • Raynauds
  • Subclavian steal
  • Reduced venous outflow = oedema

Can –> Neuro symptoms

  • Compresses lower trunk of brachial plexus, T1 nerve root. Wasting of intrinsic hand muscles
  • Paraesthesisa along medial border of hand
63
Q

Causes of salivary gland enlargement

A

1) Whole gland
- Parotitis
- Parotitis
- Sjogrens
- Sarcoid
- ALL
- Chronic liver disease
- Anorexia/bulimia

2) localised
- Tumours
- Stones

64
Q

Acute parotitis causes

A

Viral: Mumps, coxsackie A, HIV
Bacterial: S.aureus (associated with calculi and poor oral hygiene

65
Q

Salivary gland neoplasms

A

80% parotid
80% pleomorphic adenoma
Deflection of ear outwards is classic sign
CN VII palsy = malignant

Classification

1) Benign
a) Pleomorphic adenomas
b) Adenolymphoma (Warthin’s)
2) Malignant (CN7 palsy and fast growing)
a) Mucoepidermoid
b) Adenoid cystic

Investigations

  • ENT exam
  • US +/- CT
  • FNAC
66
Q

Benign salivary gland lymphomas

A

1) PLEROMORPHIC ADENOMAS
- Commonest salivary gland neoplasm
- Presentation: 90% in parotid, middle age, F>M, Benign and slow growing
- Histo: Different types
- Rx: Superficial parotidectomy

2) Adenolymphomas (Warthin’s tumour)
- Benign cystic tumour
- Older men
- Enucleation

67
Q

Adenoid-cystic carcinoma

Define

Features

Management

A
Highly malignant salivary tumour and one of most common
= Rapid growth
- Hard fixed mass
- Pain
- Facial n palsy

Surgery

  • Superficial or radical parotidectomy
  • Fascia lata face lift for facial palsy

Complications

  • Facial palsy
  • Salivary fistula
  • Frey’s syndrome (gustatory sweating - red and sweaty when thinking about food lol)