ENT: Ear Problems 2 Flashcards
(82 cards)
What is tinnitus?
Tinnitus is the perception of sound in the absence of sound from the external environment. It may be described as a ringing, hissing, buzzing, sizzling, whistling, or humming, and can be constant or intermittent, and unilateral or bilateral
NOTE: thought to be due to cochlea producing a background sensory signal and the signal not being filtered out by central auditory system
What is primary tinnitus?
- Tinnitus with no identifiable cause
- Often occurs with sensorineural hearing loss
Secondary tinnitus refers to tinnitus with an identifiable cause; state some example causes
Example causes:
- Impacted ear wax
- Ear infection
- Ménière’s disease
- Presbyacusis
- Noise exposure
- Medications (e.g., loop diuretics, gentamicin and chemotherapy drugs such as cisplatin)
- Acoustic neuroma
- Multiple sclerosis
- Trauma
- Depression
- TMJ disorders
Tinnitus may also be associated with systemic conditions:
- Anaemia
- Diabetes
- Hypothyroidism or hyperthyroidism
- Hyperlipidaemia
What is subjective tinnitus and what is objective tinnitus?
State some example causes of objective tinnitus
- Subjective tinnitus (more common) if the perceived sound can only be heard by the affected individual. This is caused by abnormal activity in the inner ear or central nervous system.
-
Objective tinnitus (affecting 1% of people with tinnitus) if the sound can be heard by the affected individual and the examiner (by auscultating with a stethoscope around ear); This often originates from an identifiable and correctable source that produces sound near to, or within, the ear. Example causes:
- Carotid artery stenosis (pulsatile carotid bruit)
- Aortic stenosis (radiating pulsatile murmur sounds)
- Arteriovenous malformations (pulsatile)
- Eustachian tube dysfunction (popping or clicking noises)
When asking a pt about tinnitus, state some questions you should ask (focused around characteristics/pattern of symptoms)
- Uni or bilateral
- Frequency
- Duration
- Severity
- Pulsatile or non-pulsatile
- Additional symptoms such as hearing loss, dizziness, vertigo, balance problems, jaw pain or clicking, facial weakness, or sensitivity to loud noises
Then also ask:
- Impact on life
- PMH & past surgical history
- Medications
What investigations may be done for someone with tinnitus?
NICE suggest:
- Blood tests:
- FBC (anaemia)
- Glucose (diabetes)
- TSH (thyroid disorders)
- Lipids (hyperlipidaemia)
- Audiology (assess hearing)
- CT or MRI imaging (not often required but may be required to investigate e.g. acoustic neuroma)
State some tinnitus red flags that could indicate serious underlying pathology that needs specialist assessment
- Unilateral tinnitus
- Pulsatile tinnitus
- Hyperacusis (hypersensitivity, pain or distress with environmental sounds)
- Associated unilateral hearing loss
- Associated sudden onset hearing loss
- Associated vertigo or dizziness
- Headaches or visual symptoms
- Associated neurological symptoms or signs (e.g., facial nerve palsy or signs of stroke)
- Suicidal ideation related to the tinnitus
Discuss the management of tinnitus
- Reassure that it to tends to improve & resolve over time without intervention
- Treat identifiable underlying causes e.g. wax
- Measures to help improve & manage symptoms:
- Hearing aids
- Sound therapy (add background noise to mask tinnitus)
- CBT
- Support groups
What is vertigo?
A false sensation of movement (spinning or rotation) of the person or their surroundings in the absence of any actual physical movement.
Remind yourself of the 3 sensory inputs for maintaining balance and posture
- Vision
- Proprioception
- Signals from vestibular system
Remind yourself of the structure of the vestibular system and how it works to maintain balance and posture
Structure
- Vestibular apparatus in inner ear
- Made up of the semi-circular canals, utricle and saccule
- Stereocilia in utricle & saccule respond to linear acceleration and static pull of gravity
- Stereocilia in semicicrucular canals respond to rotational acceleration in 3 different planes
- Semi-circular canals filled with endolymph and are orientated in different directions to detect various head movements
- When head moves, fluid in the canals moves
- Fluid movement is detected by stereocilia
- Stereocilia generate action potential
- Signal carried, by vestibular nerve, to the vestibular nucleus in brainstem and to the cerebellum
- Vestibular nucleus sends signals to oculomotor, trochlear and abducens nuclei that control eye movements and also to the thalamus, spinal cord and cerebellum
- Cerebellum helps coordinate movement throughout the body
Vertigo can be caused by a peripheral or a central problem; explain what we mean by each
- Peripheral: due to problem with vestibular system
- Central: due to problem with brainstem or cerebellum
State some peripheral causes of vertigo (highlighting the 4 most common)
Four most common
- Benign paroxysmal positional vertigo
- Ménière’s disease
- Vestibular neuronitis
- Labyrinthitis
Others
- Trauma to the vestibular nerve
- Vestibular nerve tumours (acoustic neuromas)
- Otosclerosis
- Hyperviscosity syndromes
- Herpes zoster infection (often with facial nerve weakness and vesicles around the ear – Ramsay Hunt syndrome)
State some central causes of vertigo
Central problems disrupt signals from vestibular system and cause sustained, non-positional vertigo.
Most common causes
- Posterior circulation infarction (stroke)
- Tumour
- Multiple sclerosis
- Vestibular migraine
What is the first thing you must establish in a patient presenting with ‘dizziness’?
Whether it is vertigo (rotatory or spinning symptoms) or a non-rotatory dizziness e.g. light-headedness, off-balance
**NICE states to consider asking:
- ‘When you have dizzy spells, do you feel light-headed or do you see the world spin around you as if you had just got off a playground roundabout?’
- If the person has nystagmus it is likely that their dizziness is vertigo.
Discuss how you can distinguish between peripheral and central vertigo based on:
- Onset
- Duration
- Hearing loss
- Tinnitus
- Coordination
- Nausea
Alongside features of vertigo, there are other things you can enquire about in history to try and determine cause; state these
- Recent viral illness (labyrinthitis or vestibular neuronitis)
- Headache (vestibular migraine, cerebrovascular accident or brain tumour)
- Typical triggers (vestibular migraine)
- Ear symptoms, such as pain or discharge (infection)
- Acute onset neurological symptoms (stroke)
If a pt presents with vertigo, what examinations must you do? (5)
- Ear examination including otoscopy & hearing tests (infection or other pathology)
- Neurological examination (assess for central causes)
- Cardiovascular examination (assess for cardiovascular causes of dizziness)
- Cerebellar examination
- Special tests:
- Romberg’s test (looks for sensory ataxia)
- Dix-Hallpike manoeuvre (diagnose BPPV)
- HINTS examination
What is the HINTS examination used for?
What does it involve?
Used to distinguish between central and peripheral vertigo. It stands for:
- HI – Head Impulse
- N – Nystagmus
- TS – Test of Skew
Explain how to perform the head impulse test and explain what the results mean
How to perform
- Sit pt upright and ask to look at examiners nose (and continue looking at it throughout)
- Examiner holds pt’s head and rapidly jerks it 10-20 degrees in one direction
- Slowly return head back to centre and repeat in opposite direction
Results
- Normal vestibular system: eyes remain fixed on examiners nose
- Abnormal vestibular system/peripheral cause of vertigo: eyes will make a corrective saccade before eventually fixating back on examiners nose
Explain how to check for nystagmus and what the results mean
How to perform
- Get pt to look to the left or right and hold the gaze
- Observe eye movements
Results
- Unilateral/unidirectional horizontal nystagmus: more likely peripheral
- Bilateral horizontal or vertical nystagmus: more likely central
Explain how to perform the test of skew (also know as alternate cover test) and explain what the results mean
How to perform
- Sit pt upright and ask pt to fixate/look at examiners nose
- Examiner covers one eye at a time, alternating between the two eyes
Results
- Eyes should remain fixed on examiners nose
- If there is vertical correction when eye is uncovered (meaning eye has drifted up or down when covered so needs to move vertically to re-fixate on nose) = indicates central cause of vertigo
Summarise HITT examination findings in peripheral and central causes of vertigo
Remind yourself of features of cerebellar disease (and hence what to assess in cerebellar examination)
- D – Dysdiadochokinesia
- A – Ataxic gait (ask the patient to walk heel-to-toe)
- N – Nystagmus (see below for more detail)
- I – Intention tremor
- S – Speech (slurred)
- H – Heel-shin test